Arrhythmias- 1 Flashcards

1
Q

What are the two Rough anatomical types of arrhythmias?

A
  • Supraventricular (anything above ventricles)

- Ventricular

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What are Ectopic beats?

A

Beats originate from places other than the SA node.

Ectopic beat- may cause single beats, or take over the heart dictating overall beat.

Dangerous- if they affect the CO -vely.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What are three categories of arrhythmias?

A
  1. Supraventricular- AF, atrial flutter, SVT, Sinus bradycardia
  2. AV Node- AV block, AVN re-entry, accessory pathway
  3. Ventricular- VF, VT, Premature ventricular complex.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What are the clinical causes of arrhythmias?

A
  1. Anatomical- LVH, accessory pathways. congenital HD.
  2. Autonomic:
    - Sympathetic stimulation: nervousness, exercise, CHF
    - Increased vagal tone- bradycardia, heart block

3, Metabolic- hypoxic or ischaemic myocardium

  1. Inflammation- myocarditis
  2. Drugs
  3. Genetic mutations
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What are the three physiological mechanisms of arrhythmias?

A
  1. Altered automaticity
  2. Triggered activity
  3. Re-entry
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What type of changes occur in the AP graph in altered automaticity?

A
  • Change in slope
  • Change in threshold
  • Change in RMP
  • Decrease Phase 4 slope and hyper-polarise
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What physiological factors can change the slope of nodal conductance? (Phase 4)

A
  1. Increases slope
    - hyperthermia
    - hypoxia, hypercapnia
    - hypokalemia (more ectopic beats, prolonged repolarisation)
    - Cardiac dilatation
  2. Decreases slope
    - Hypothermia
    - Hyperkalaemia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What happens in triggered activity?

A

Terminal phase of AP (phase 3), a small depolrisation may occur.
If this reaches threshold, it may trigger sustained depolarisations= triggered activity

Causes= digoxin, long QT syndrome, hypokaleamia.

If after-depolarisation in phase 3= pause-dependent triggered activity
if in phase 4= catechol dependent.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What are the requirements for Re-entry?

A
  1. Available circuit
  2. Unidirectional block
  3. different conducting speeds of the limb circuits.

Conditions that depress conduction velocity or increase refractory period favour re-entry.
Eg- MI, AV nodal reentry, reciprocating tachycardia.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What happens in re-entry?

A

A transient block on one side of the conduction system
- Impulse travles down the other side
- Then travels through the block in a retrograde manner
- if the tissue is excitable, the impulse travels down non occluded branch.
- Result is a circus movement.
If in the AV node can cause echo beat, or nodal tachycardia.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What are the symptoms of arrhythmia?

A
  • Palpitations
  • Syncope
  • SOB
  • Dizzy
  • sudden cardiac death
  • worsen existing condition- angina, hf
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What investigations would you do?

A
  1. ECG:
    - Exercise ecg- asses for ischaemia
    - 24hr ecg- detect paroxysmal arrhythmia
  2. ECO gram- assess structural heart disease like LVH
  3. Electrophyisiological study
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is an atrial ectopic beat? Symptoms + treatment?

A

Abnormal (Not sinus) beat produced in the atria, causing an extra beat to be produced during sinus rhythm.

Symptoms- Asymptomatic. Palpitations

Treatment- B blocker may help. Avoid stimulants like coffee, cig

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What is sinus bradycardia? What can cause it? Treatment?

A

Sinus (normal) rhythm is slower than 60bpm.

Seen in- anterior MI, Heart block, (b blocker) , athletes

Treatment- Atropine (if acute)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is sinus tachycardia? Causes? Treatment?

A

Sinus rhythm faster than 100bpm.

Seen in- Drugs, physiological (tension, fever, hypotension, anaemia).

Treatment- B blocker.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What are some causes of SVT?

A
  1. AV nodal reentrant tachycardia
  2. Accessory pathway “ (eg Wolff parkinson white syndrome)
  3. Ectopic atrial tachycardia
17
Q

What is the management (acute/ chronic) for SVT’s?

A
  1. Acute
    - Vagal manoeuvres, carotid massage
    - IV adenosine
    - IV Verapamil
  2. Chronic
    - Avoid stimulants
    - Radiofrequency ablation
    - Anti arrhythmic (II, IV)
18
Q

What is ablation?

A

Selective cautery (burn) of cardiac tissue= prevent tachycardia

19
Q

Which cardiac conditions can cause AVN conduction disease?

A
  • acute MI
  • Drugs
  • infiltrative disease
  • myocarditis
  • aortic valve disease
20
Q

What are three types of AV block?

A

1st Degree- not proper block. Prolonged PR interval.
Treatment- not needed. Long term follow up needed tho.

2nd Degree- intermittent AVN block. (dropped beats)
Mobitz I- progressive PR interval increase. Eventual dropped beat.
Mobitz II- Pathological. Dropped beat every 2nd/3rd cycle. May progress to complete block.

3rd Degree- no AP gets from the SA node/atria to the AV node.
Treatment- ventricular pacing

21
Q

What are the two delivery options for pacing?

A
  1. Transcutaneous (painful)

2. Transvenous (via IJV, subclavian or femoral veins)

22
Q

What are the two types of pacemakers?

A
  • Single chamber (used in defective SA, or as ventricular pacemakers)
  • Dual chamber (used in AVN disease)
23
Q

What are premature ventricular ectopics? what factors cause it?

A

Heartbeat initiated by purkinjee fibres in the ventricles instead of SA node. Felt as missed beat- as ventricles contract before SA node can initiate and fill the ventricles fully with blood.

Causes- ischaemic changes, hypertension, inherited cardiomyopathy.
Usually asymptomatic. Treat with B blockers

24
Q

What happens in VT?

A

Ventricles beating abnormally fast. Cannot properly contract hence CO poor. Life threatening.

Most patients- significant disease like CAD, MI.

25
Q

What Signs to check in VT?

A

Stable or unstable

Chest pain, hypotension, CHF, altered conscious level

26
Q

What is VF?

A

Spasmic ventricular electric activity. Needs defibrillator

27
Q

What is the acute/ chronic treatment for VT

A

Unstable- DC cardioversion
Stable- Anti arrhythmic drugs (B blockers, Ca, Na, K drugs)

Management- look for causes ie ischaemia, electrolytes, hypoxia

Long term- correct ischaemia if possible. AAD can be worse. Implantable defibs in worst cases.