Applied Psychology Flashcards

1
Q

What is a physiological explanation for criminal behaviour?

A

Brain Dysfunction- Raine- Focused on the pre frontal cortex, low activity in this area of the brain can be indicated by a low resting heart rate. It is involved in planning and regulating of behaviour, it also enables us to focus. If there is low arousal in the prefrontal cortex then these abilities are impaired. Raine investigated the relationship between low activity in the prefrontal cortex and impulsive violent criminal behaviour. PET scans were used to investigate the differences in brain activity in murderers who pleaded NGRI and non-murderers.
Genetics- Brunner- suggested an “association” rather than a “cause” between genes and aggressive behaviour, “Warrior gene” was related to aggressive and violet behaviour. MAOA low activity variant may increase susceptibility to antisocial traits and aggression.

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2
Q

What are the non-physiological explanations for criminal behaviour?

A

Upbringing-Farrington- Cambridge study in delinquent development. Large study of 411 boys aged 8 when the study began and were interviewed as adolescents and adults too, their criminal record was reported as part of the study. The study was comprehensive and identified a range of risk factors for criminal behaviour; family criminality, daring or risk-taking, low school attainment, poverty and poor parenting

Learning-Sutherland- The differential association hypothesis states the more contact someone has with attitudes favourable to criminal behaviour, and more exposure they have to criminal behaviour in their family and friends, the more criminal behaviour they will commit themselves

Cognitive- Palmer & Hollin- those who commit crime do so because they operate at an immature level of moral reasoning. The Kohlbergian preconventional stage of moral reasoning suggests that behaviour is seen as right and wrong only in terms of the outcome for the individual

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3
Q

What was the aim of Raine et al.’s study into brain abnormalities of murders?

A

To find out if there is a difference in the structure of brain activity between people who have committed murder (NGRIs) and non-murderers.

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4
Q

What did Raine et al. hypothesise?

A

1). Serious violent individuals pleading NGRI have relatively localised brain dysfunction in the prefrontal cortex, angular gyrus, amygdala, hippocampus, corpus callosum and thalamus
2). Seriously violent individuals pleading NGRI show no dysfunction in other brain areas that are not associated with violence

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5
Q

What was the method used by Raine in his study into brain abnormalities of murders?

A

Quasi experiment as the IV is naturally occurring (murderers pleading NGRI and non murderers). The DV was whether the participant showed evidence of brain dysfunction in their prefrontal cortex.

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6
Q

What experimental design did Raine et al. use in his study into brain abnormalities of murders?

A

Used matched pairs design, matching on age and gender (matched with controls)

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7
Q

Who were the sample for Raine in his study into brain abnormalities of murders?

A

Murders- 41, 39M 2F who were tried in California for murder or manslaughter who pleaded not guilty by reason of insanity
Control- 41 39M 2F non-murderers who take no medication and have no history of mental illness

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8
Q

What was the procedure for Raine in his study into brain abnormalities of murders?

A

-Participated under protocols and concent forms
-all murderers were kept in custody and were medication free for two weeks prior to the brain scanning. non-murderers did not take medication.
-before receiving FDG injection participants were given practice trials on the continuous performance task (CPT) and 30s before scan they stared the CPT
-participants were then taken to an adjacent PET scanner where 10 slices of their brain were taken
-Was very precise so it could be replicated

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9
Q

What did Raine et al. find in his study into brain abnormalities of murders?

A

in murderers who pleaded NGFRI had reduced activity in areas linked to violence, pre-frontal cortex, left angular gyrus and corpus collosum
-abnormal asymmetries; reduced activity on the left half of the brain compared to right, which are areas linked to violence and aggressive behaviours like hippocampus

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10
Q

What conclusions can be made about Raine’s research in his study into brain abnormalities of murders?

A

Murderers who plead not guilty by reason of insanity have significant differences in metabolism of glucose in a number of brain areas than non-murderers
-results do not show that violent behaviour is determined by biology alone

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11
Q

Evaluate the strengths of Raine’s study into brain abnormalities of murders

A

method-high control over extraneous variables so results on brain abnormalities in murderers who pleaded not guilty by reason of insanity are more likely to be reliable as the experiment can be replicated due to standardised procedures.

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12
Q

Evaluate the weaknesses of Raine’s study into brain abnormalities of murders

A

-Sample- small so is unlikely to be representative of all murderers from California so brain abnormalities in murderers cannot be generalised to other murderers from other places// androcentric sample as there are significantly more male than females
-Method- using matched pairs is inaccurate as you cannot exactly match people, so is low in ecological validity

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13
Q

What did Raine argue is the cause for criminal behaviour?

A

Criminal behavior comes from a combination of biological, environmental and neurological factors, and brain dysfunction, genetic predisposition and birth complications

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14
Q

What are Raine’s biological risk factors for antisocial and criminal behaviour?

A

-Children of mothers who smoke during pregnancy have a 3-fold risk of becoming violent offenders compared to children who’s mothers didn’t smoke during pregnancy
-Birth complications risk factor for antisocial behaviour and crime
-Poor nutrition of a mother during pregnancy doubles rates of anti-social behaviour

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15
Q

Explain how biological interventions work.

A

-100 children received a 3 part intervention: nutrition, physical exercise, cognitive simulation. Raine found that when older they could focus their attention better than controls, and scored significantly lower on conduct disorder rating than control, and were less cruel to others.
-Regularly taking omega 3 supplements can help to reduce aggressive and violent behaviours making them less likely to partake in crime
-not smoking during pregnancy so testosterone levels will be kept at a suitable level therefore the child is less likely to grow up to be violent

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16
Q

What is the background for Hall & Player?

A

In response to Dror’s research that fingerprint experts are affected by cognitive bias . They thought it was important to ascertain if the normal working practices employed by the metropolitan police fingerprint bureau introduce an emotional bias.

17
Q

What is the aim to Hall & Player?

A

to test if context had an affect on fingerprint identification by fingerprint experts, and attempted to answer; does the written report of a crime, as routinely supplied with the fingerprint evidence, affect a fingerprint experts interpretation of a poor quality mark?/ Are the fingerprint experts emotionally affected by the circumstances of the case?

18
Q

What is the method to Hall & Player?

A

-laboratory experiment
-task was artificially generated and participants depressingly allocated a condition out of two
-IV was weather the participant was allocated to the low-context or the high-context group
-DVs: read crime scene examination prior examining finger print \ weather participant considered the finger print to be identification, not an identification, insufficient, or insufficient detail to establish identity

19
Q

What is the sample to Hall & Player?

A

-volunteer sampling
-group of fingerprint practitioners chosen to represent a wide variation of experience
-70 fingerprint experts working for metropolitan police fingerprint bureau
-low context group - 35 ppts
-high context groups- 35 ppts

20
Q

What materials were used in Hall & Player?

A

-finger print impression from a known source
-volunteers right finger was inked and introduced to a price of paper
-then scanned into a computer and superimposed on a scanned image of a £50 note
-14 copies made and were compared for consistency
-allowed a fingerprint magnifying glass and a tussle comparator

21
Q

What was the procedure to Hall & Player?

A

-asked to participate during work time for it to be naturalistic
-randomly asigned in groups of 8
-no time limit and were told to treat the experiment as an ordinary case
-low - context groups: 35 ppts were given an examination report referring to forgery “suspect entered premises and tried to pay for goods with a forged £50 note”
-high - context groups: 35 ppts were given a report referring to murder ‘suspect then fired two shots at victim before decamping’
-researcher stayed with each group at all times to answer any questions
-they were then asked to consider whether the mark was an identification, not an identification, insufficient, or insufficient detail to establish identity
-were then given a feedback sheet which asked weather or not they had referred to the crime scene examination and if they had they were asked if the report had effected their analysis and if so how?

22
Q

What were the results to Hall & Player?

A

-57 of 70 ppts read crime scene examination prior to examining prints 30 were high context condition
-52% of 30 high context condition felt they were effected by report indicating that there is a relationship between type of context and the perceived effect on the experts
Identification; H=6 L=7
No identification; H=1 L=0
Insufficient; H=15 L=12
Insufficient to establish identity; H=13 L=16

23
Q

What were the conclusions to Hall & Player?

A

-emotional context affects a fingerprint experts analysis but has no actual affect on their final decision
-severity of case affects the fingerprint experts analysis but does not have any affect on their final decision

24
Q

What is the medical model?

A

Suggests that mental health has a physical cause and that symptoms are grouped together and classified as a disorder. Mental disorders are due to biological factors within the brain

25
Q

How does serotonin lead to a disorder?

A

Serotonin carries messages between nerve cells and regulates mood, an imbalance leads to anxiety

26
Q

How does noradrenaline lead to a disorder?

A

if too high can cause anxiety disorders and panic attacks

27
Q

How does dopamine lead to a disorder?

A

Dopamine causes feelings of pleasure so increased levels leads to addictive behaviours causing anxiety or schizaphernia

28
Q

How does Glutamate lead to a disorder?

A

Allows for learning and memory, can cause hallutionations and delusions- symptoms of schizoprenia

29
Q

What are the biochemical explinations of mental illness?

A

Suggest that imbalances in neurotransmitters can affect how the brain passes signals between neurons and lead to things like anxiety, low mood, and hallucinations

30
Q

What happens biochemically for someone with depression?

A

People with depression have low levels of serotonin which can be due to experiencing a stressful event or it can be caused by genetics. When serotonin is released into the synapse it doesn’t bind effectively to the receptors on the post synaptic neuron before being taken back up through reuptake channels.

31
Q

What happens biochemically for someone with schizophrenia?

A

Increased levels of dopamine are released into the synapse and binds to D2 receptors on the post synaptic neuron and too much dopamine binds and less is taken back up which causes dellusions and hallutionations

32
Q

How can we treat depression with drug therapy?

A

SSRI(selective serotonin reuptake inhibitors) this blocks the reuptake channel on the pre synaptic neuron so serotonin stays in the synapse so it has more time to bind

33
Q

How can we treat schizophrenia with drug therapy?

A

Antipsychotic drugs balance the levels of dopamine by blocking D2 receptors on post synaptic neuron allowing normal amount of dopamine to bind resulting in no hallutinations.

34
Q

Describe Gottesman’s research.

A

He investigated couples who have psychiatric illnesses and their offspring to calculate the risk of their offspring also being diagnosed with a psychiatric illness. The aim of the study was to investigate the importance of genetic influence, where both parents have been admitted with a severe psychiatric disorder. Patients were diagnosed with ICD-8 and ICD-10, the study measures the cumulative incidences of schizophrenia and bipolar disorder in offspring up to 52 years old

35
Q

Explain the findings to Gottesman’s research.

A

Schizophrenia- both parents= 27% one parent= 7% no parent= 0.9%
Bipolar- both parent= 25% one parent= 4% no parent= 0.5%
One parent with Schiz/ One parent with Bipoar= 16% of schiz 12% of Bipolar