Anxiety and Depression Flashcards

1
Q

What is Psychopathology Psychopathology is a term which refers to either the study of mental ill_____ or mental dis______ or the manifestation of be________ and exp_________ which may be indicative of mental illness or psychological imp________.

A

What is Psychopathology Psychopathology is a term which refers to either the study of mental illness or mental distress or the manifestation of behaviours and experiences which may be indicative of mental illness or psychological impairment.

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2
Q

What is psychological-good health? ‘A state of complete phy____, me____ and soc__ well-being and not merely the absence of di___ or in______.’ (WHO, 1946)

A

What is psychological-good health? ‘A state of complete physical, mental and social well-being and not merely the absence of disease or infirmity.’ (WHO, 1946)

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3
Q

What are the ‘biological’ nomothetic explanations?

A

Genetic and Neurochemical

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4
Q

What are the Cognitive nomothetic explanations?

A

Thought patterns

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5
Q

What are the learnt nomothetic explanations?

A

Reinforcements and experiences

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6
Q

Which qualities also have ideographic qualities

A

Approaches to research from cognitive and learnt theories

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7
Q

What is the ideographic approach?

A

The approach tends to include qualitative data, investigating individuals in a personal detailed way.

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8
Q

What is the nomothetic approach?

A

Psychologists who adopt this approach are mainly concerned with studying what we share with others (i.e. similarities between people.

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9
Q

Who assesses mental illness

A

Technically only a medical doctor (usually a psychiatrist can make an actual diagnosis).

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10
Q

Who is the DSM-5 intended for

A

It can be used by mental health and other health professionals, including psychiatrists, social workers, nurses, occupational and rehabilitation therapists and counsellors.

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11
Q

What are the genetic risk factors for depression and anxiety?

A

While there is strong evidence for a family history of depression and anxiety. There is little evidence of a significant genetic risk.

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12
Q

Is there a causal relationship for disorders and genetic risk?

A

No, even disorder with high genetic risk is still not fully causal. e.g. Schizophrenia

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13
Q

Has a specific gene been found that is known to cause mental conditions?

A

No, there is no evidence for single or a small numbers of genes. Many genes influence each other.

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14
Q

What is the prevalence of depression during a lifetime?

A

Lifetime prevalence is 2.6%

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15
Q

By what percentage does having a parent or sibling with depression increase the risk of also getting depression?

A

Sibling or parent with depression, increases risk by 3, therefore 7.8

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16
Q

What is seen as various (debatable) causes of depression?

A
  • Allergy
  • Changes in digestive system
  • Obesity
  • Sedentary lifestyle
  • Dental disease
  • Poor sleep
  • Poor diet
  • Smoking
  • Stress
  • Low vitamin D
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17
Q

How can a person with a biological risk to depression offset the chances of getting the condition?

A

A hypothetical person with a bioloical risk but with good coping and support is likely to offset the risk.

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18
Q

Is depression purely a biological/genetic condition?

A

No, there are environmental causes of depression (e.g. separation poverty, stress) lead to lower levels of brain serotonin.

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19
Q

How does serotonin affect the chances of getting depression?

A

Depression may involve decreases in levels of brain serotonin, and can be helped by drugs increasing brain serotonin. Does this mean that depression is a biological/genetic condition?

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20
Q

What treatments are available to help treat depression?

A

Biological (drug) treatments and psychological therapies are equally effective at treating mild to moderate depression i.e. we can influence brain serotonin either directly (drugs) or indirectly (psychological therapies)

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21
Q

What is the link between serotonin and depression?

A

In theory, all anti-depressant increase 5HT availability in the brain, by increasing release or blocking reuptake. Getting direct evidence for drug-related changes in 5HT activity difficult to collect. However, tryptophan is an amino acid essential for 5HT production; lowering dietary tryptophan leads to depression relapse in SSRI-treated patients; may lead to depression in control.

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22
Q

What is the evidence of drug treatment in depression?

A
  • Found to work very effectively
    • Another issue is that many people do not respond to treatment.
    • However, double-blind controlled studies sometimes show little or no advantage over placebo; mainly because of large placebo response in anxiety and depression.
  • -Psychological factors underestimated in anxiety and depression
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23
Q

What are the criticism of drug treatments

A
  • As much as 90% of effects may be placebo effects -Do not “cure” the disorder, they manage the symptoms but not all the symptoms. - Placebo effect -While they may manage/reduce the biological risk factors they cannot address all of them. - Do not increase/ improve protective factors - High relapse rate and aversive “discontinuation syndrome” when clients stop taking drugs.
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24
Q

What other problems are connected with anti-depressants

A
  • There is much evidence that antidepressant medications are not benign treatments - Many antidepressants are cardiotoxic, have dangerous side effects, and are often used in suicide attempts. - They also result in: relatively poorer compliance than psychotherapy, have a higher drop-out rate, result in as much as a 60% non response rate with some patient populations.
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25
Q

Why are there variations in the prescription of anti-depressants?

A
  • Drugs only recommneded for severe depression - but given to people with mild/moderate depression - Mild/Moderate group would be better treated by psychological treatments.
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26
Q

What are other bio-social factors?

A

Links to depression - low levels of vitamin B and high levels of homocysteine - low levels of omega 3 fatty acid - high in-take of refined sugar - Vitamin D and mood - a range of diet and lifestyle factors has a dramatic impact on depressive disorders.

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27
Q

Environmental factors can lead to biological changes

A

A child raised in a caring environment - Better emotional regulation - more stable Has an impact on neurochemical release - but also more likely to cope with stress as an adult.

28
Q

What is the importance of serotonin and where is it found?

A
  • Not only found in the brain but around the body - According to Reigstad et al (2015), around 80% of the body’s serotonin is made by microbes in the gut. -Clark et al. Low levels of good bacteria in the gut leads to low serotonin, which can lead to low mood and anxiety. However, although replenishing guy bacteria can alleviate symptoms of anxiety it doesn’t correct the abnormal neurochemical markers.
29
Q

Explain what a chemical imbalance is?

A

Chemical imbalance is psychological terms: refers to variations of the neurotransmitters (serotonin, dopamine, norepinephrine, and epinephrine) of the brain. Neurotransmitters are responsible for transmitting messages. When these levels are at less than “optimum” levels it can cause physical and emotional difficulties such as depression, bipolar disorder, anxiety, and other psychological disorders.

30
Q

Can depression be considered an allergic reaction?

A
  • Illness, inflammation and infections can trigger depression - Similarly, certain food and diets can trigger cytokines (a family of proteins that witch the brain into sickness mode) e.g. forces the body to recuperate. - However, stress associated with social rejection and loneliness can also trigger these. - Therefore as people are generally overeating, less active and more isolated depression its is likely we will see depression increase.
31
Q

Can you learn to be depressed?

A

Seligman 1974: dogs given unavoidable shocks fail to escape when given the chance. Hiroto & Seligman 1975: same results with humans and unavoidable white noise.

32
Q

Why is depression linked to stressful events?

A

So depression is linked to stressful events over which you believe you have no control The problem is clearly related to individual differences.

33
Q

What is the negative triad?

A

The negative view of the self, the world and the future?

34
Q

What are cognitive biases?

A

A cognitive bias is a type of error in thinking that occurs when people are processing and interpreting information in the world around them. The human brain is powerful but subject to limitations. Cognitive biases are often a result of your brain’s attempt to simplify information processing. They are rules of thumb that help you make sense of the world and reach decisions with relative speed.

35
Q

What are examples of cognitive biases?

A

Arbitrary inference, (a cognitive error whereby a person draws a conclusion that is either unrelated to or contradicted by the evidence at hand), selective abstraction (a type of cognitive bias or cognitive distortion in which a detail is taken out of context and believed whilst everything else in the context is ignored), magnification (When you magnify the negative), minimisation (minimize the positive in your life) etc

36
Q

What could learned helplessness also be considered as for someone on the negative triad?

A

Learned hopelessness.

37
Q

What causes Generalised Anxiety Disorder?

A

The exact cause of GAD isn’t fully understood, although it’s likely that a combination of several factors plays a role.

38
Q

What are the factors considered to play a role in Generalised Anxiety Disorder? (1)

A

Research has suggested that these may include:

  • overactivity in areas of the brain involved in emotions and behaviour
  • an imbalance of the brain chemicals serotonin and noradrenaline, which are involved in the control and regulation of mood.
39
Q

What genetic factors play a role in Generalised Anxiety Disorder?

A
  • the genes you inherit from your parents – you’re estimated to be 5 times more likely to develop GAD if you have a close relative with the condition.
40
Q

Which health issues are considered to play a role in Generalised Anxiety Disorder?

A

having a painful long-term health condition

  • such as arthritis
  • having a history of drug or alcohol misuse
  • But many people develop GAD for no apparent reason.
41
Q

What is the prevalence of anxiety in the UK?

A

In 2013, there were 8.2 million cases of anxiety in the UK. In England women are almost twice as likely to be diagnosed with anxiety disorders as men.

42
Q

What is the instrument used for screening, diagnosing, monitoring and measuring severity anxiety?

A

The GAD-7 form

43
Q

What are anti-anxiety drugs designed to do?

A
  • Designed to reduce anxiety without affecting alertness or concentration - Slow down excitatory synaptic activity in the nervous system
44
Q

What are the different types of anti-depressants used for depression and anxiety disorders.

A
  • Serotonin Reuptake Inhibitors (SSRIs):
    • Fluoxetine (Prozac), Sertraline, Citalopram.
  • Serotonin-norepinephrine Reuptake Inhibitors (SNRIs)
    • Duloxetine, Venlafaxine Tricyclic antidepressants (TCAs)
  • Tricyclic antidepressants (TCSs):
    • Amitriptyline, Clomipramine, Nortriptyline
  • Monoamine oxidase inhibitors (MAOIs)
    • Selegiline, Phenelizine, Tranylcypromine
45
Q

How does stress behave as a trigger to issues with the body?

A
  • Activates hormones that affect neurotransmitter systems
  • Turns on certain genes
  • Affects circadian rhythms
  • Activates dormant psychological vulnerabilities (i.e., negative thinking)
  • Contributes to sense of uncontrollability
  • Fosters a sense of helplessness and hopelessness
46
Q

Environmental factors can lead to biological changes

A
  • A child raised in a caring environment
    • Better emotional regulation
    • More stable
  • Impact on neurochemical release
    • but also more likely to cope with stress as an adult
47
Q

What is the psychoanalysis etiology of anxiety disorder?

A

Psychoanalytic Theories: it results from psychic conflicts between sexual or aggressive wishes and threats from external reality

48
Q

What is the behavioural etiology of anxiety disorder?

A

Classical conditioning or/and Social learning Theory: people learn to respond excessively to stress (e.g. modelling)

49
Q

What is the cognitive etiology of anxiety disorder?

A

People misperceive situations as dangerous when they are not. They overestimate danger or harm and underestimate own ability

50
Q

What is a main strategy of the nomothetic approach?

A

Focus on similarities and differences between groups of individuals.

51
Q

What is the aim of the nomothetic approach?

A

To identify a basic structure or category to be able to describe personality or a disorder universally.

52
Q

What is the research method of the nomothetic approach?

A

Quantitative methods, to ensure the structures are explored, produced valid measurements/assessments, and to explore the relationship between variables across groups.

53
Q

What types of data is used in the nomothetic approach?

A

Self-report Questionnaires Physical Assessments

54
Q

What is the strategy for psychological well-being?

A

What differentiates the psychological healthy from those who are ‘unwell’.

55
Q

What is the aim of psychological well-being?

A

What criteria make up one disorder and is distinct from another.

56
Q

What research is used in understanding psychological well-being?

A

Are the categories and definitions of each disorder valid and reliable 1. What are these disorders correlated with in terms of lifestyle and inter-group factors. 2. Use of valid measurements/assessments, and to explore the relationship between variables across groups.

57
Q

What type of data is used in understanding psychological well-being?

A

Behaviour, self-report (what is said to doctor/psychiatrist), questionnaires, and occasionally physical assessments.

58
Q

What is used to assess mental illness?

A

Diagnostic and Statistical Manual of mental Disorders, Fifth Edition (DSM-5)

59
Q

What are the side effects of SSRIs?

A
  • Agitation
  • Anxiety
  • stomach aches
  • constipation
  • insomnia
  • headaches
  • low sex drive
  • difficulties with orgasm and erection
  • increased risk of suicidal thought
  • thought of self-harm
  • long-term use increase of developing type 2 diabetes
  • change of appetite
60
Q

What are the side effects of Serotonin-norepinephrine Reuptake Inhibitors (SNRIs)

A
  • Dry mouth
  • Sleepiness
  • Fatigue
  • Constipatin
  • Loss of appetite
  • Sweating
61
Q

What are the side-effects of Tricyclic antidepressants (TCAs)?

A
  • Dry mouth
  • blurred vision
  • Constipation
  • Problems urinating
  • Drowsiness
  • Dizziness
  • Weight gain
  • Excessive sweating
  • Heart Rhythm problems
  • long-term use increase of developing type 2 diabetes
62
Q

What are the side-effects of Monoamine oxidase inhibitors (MAOIs)?

A
  • Dizziness
  • drowsiness
  • tiredness
  • insomnia
  • constipation
  • digestive and urination problems
  • fainting
  • mental changes
  • muscles stiffness
  • termor
  • stolen limbs
  • change of vision
63
Q

How does stress weaken proper function in the immune system?

A
  • Possibly responsible for increased risk of illness
64
Q

What effect does the nature of a stressor have on specific immune functions of the body?

A
  • Natural Immunity
    • Occurs quickly and is relatively non-specific in nature.
  • Specific Immunity
    • more targeted process; takes longer to occur
65
Q

What does “diathesis” mean?

A

A hereditary predisposition of the body to a disease, a group of diseases, an allergy, or another disorder.

66
Q

What prior experiences can have an effect on developing Generalised Anxiety Disorder?

A

having a history of stressful or traumatic experiences

  • such as domestic violence
  • child abuse
  • bullying.