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ANS Flashcards

1
Q

Anesthesia cart drugs 19

A

Atropine, glyco, scopolamine, ipratropium, ephedrine, epi, norepi, dopamine, dobutamine, isoproterenol, neosynephrine, albuterol, prazosin, phentolamine, propranolol, metoprolol, atenolol, esmolol, labetolol

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2
Q

Neuron myelination: preganglionic, postganglionic

A

Pre myelinated, post unmyelinated

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3
Q

SNS

Where preganglionic neuron cell bodies are located

A

T1-L2/3 spinal cord, intermediolateral horn grey matter

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4
Q

SNS where postganglionic neuron cell bodies are

A 
Paravertebral chains on either side of spinal column 
Paravertebral ganglia (celiac, superior, inferior, mesenteric in abd)
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5
Q

SNS pre/postganglionic neuron length

A

Short pre

Long post

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6
Q

PSNS

Pre ganglionic neuron location

A

Cranial- medullary- CN 3, 7, 9, 10

Sacral- spinal cord- S2-4

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7
Q

PSNS post ganglionic neuron cell body location

A

Target organs. Ganglia in head and neck (ciliary ganglia)

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8
Q

PSNS neuron cell length pre/post ganglionic

A

Long pre, short post

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9
Q

SNS vs PSNS action to eye

A

SNS- dilation

PSNS- constriction

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10
Q

SNS vs PSNS salivary glands

A

SNS- decreased bf and secretions, dries

PSNS- secretions made

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11
Q

Lacrimal gland pns

A

Able to cry

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12
Q

SNS

Vessels in head and neck

A

Constrict in skin, no constriction to brain

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13
Q

SNS

Heart

A

Stronger contraction, faster rate, higher 02 demand. Blood return to heart increased via constriction in legs/skin/sphlancnic beds

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14
Q

PNS

Heart

A

Some innervation, SA node rate decreased from vagus

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15
Q

SNS vs PNS

Lungs

A

Bronchodilation B2 receptors.

Bronchoconstriction

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16
Q

SNS adrenal medulla

A

B2 receptors, epi released from here

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17
Q

SNS

Liver

A

Increase glucose release, glycogenolysis, lipolusis, insulin increases (to uptake glucose by muscle)

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18
Q

SNS
GI tract
Vessels everywhere

A

Sphincters contracted

Depends where. B2- dilation. A1- constriction.

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19
Q

PNS

GI

A

Motile, inc gastric acid secretion and pancreatic enzymes

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20
Q

PNS

Bladder

A

Detrusor gains tone, sphincter loses tone, able to void

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21
Q

Organs innervated by both systems: 3

A

Gi, cardiac muscle, bladder

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22
Q

Only innervated by sns: 2

Only innervated by PSNS: 2

A

SNS: sweat glands, blood vessels (muscarinic receptors tho)
PSNS: ciliary eye muscle, bronchial smooth muscle (B2 receptors tho)

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23
Q

Receptors can be present in a tissue and not be ____. If this is case, receptor can only respond when what

A

Innervated. Drug or hormone is circulating in the blood.

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24
Q

SNS

__ response with ___ innervation. 3 stim

A

Amplification, diffuse. Postural changes, exercise, emergency response

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25
PSNS | __ and ___ __ responses
Discrete, narrowly targeted
26
Both systems exhibit __ __ at rest. Heart rate: __ predominance, blood vessels: ___ tone
Baseline tone Vagal SNS
27
Somatic efferent muscle Receptor What works here
ACh nicotinic on skel muscle | Muscle relaxers
28
SNS Receptors in blood vessels Never releases what, always what
ACh nicotinic to norepi in vessel | Never epi, always norepi
29
SNS | Sweat gland receptors
ACh nicotinic on a ACh muscarinic on gland.
30
SNS | Adrenal medulla receptor
ACh nicotinic
31
PNS receptors
ACh (nicotinic) on an ACh (muscarinic) in location
32
PSNS cholinergic receptors:
Nicotinic ACh: Nm, Nn | Muscarinic ACh: M1-5
33
PSNS Adrenergic receptors
A1, A2 | B1, B2, B3
34
3 Overall anomalies in pns
Adrenal medulla, sweat glands, blood vessels
35
How adrenal medulla is an anomaly
Acts like ganglia but releases ne and epi as hormones. 20% NE, 80% epi
36
How sweat glands are an anomaly
Innervated anatomically by SNS. Post ganglionic nerve releases Ach on a muscarinic Ach receptor. Sympathetic cholinergic fiber
37
How blood vessels are an anomaly
No PSNS innervation but there are muscarinic ach receptors present. They activate w/NO and dilate if Ach circulating
38
M1 (Ach) Receptor Signal
Gaq | Excitatory CNS, modulatory ganglia
39
M1 (Ach) | 2nd messengers
PLC activated IP3 and DAG PKC and inc Ca Decreased K conductance
40
M1 What NT Physiologic response
Acetylcholine | Inc CNS activity, modulation at ganglia
41
M2 (Ach) Receptor Signal
Gai | Inhibitory cardiac
42
M2 (Ach) | 2nd messenger
Inhibit Adenylate cyclase Decrease cAMP Increase K conductance
43
M2 NT Physio response
Ach | Decreased cAMP slows HR and decreases contractility
44
M3 (Ach) Receptor Signal
Gaq | Excitatory smooth muscle and glands
45
M3 Ach | 2nd messengers
PLC activated IP3 and DAG PKC and inc Ca
46
M3 NT Physio response
Ach | Smooth muscle contraction
47
Nn (Ach) Receptor Signal
Ligand gated ion channel | Excitatory ganglia CNS
48
Nn (Ach) | 2nd messengers
Increased K and Na permeability
49
Nn NT Physio response
Ach | Depolarization
50
Nm (ACH) Receptor Signal
Ligand gated ion channel | Excitatory NMJ
51
Nm (Ach) | 2nd messengers
Increased Na and k permeability
52
Nm NT Physio response
ACH | Depolarization
53
A1 (epi/NE) Receptor Signal
Gaq | Excitatory blood vessels
54
A1 (epi/NE) | 2nd messengers
PLC activated IP3 and DAG PKC and inc Ca
55
A1 NT Physio response
NE, epi | Smooth muscle vasoconstriction
56
A2 (NE, epi) Receptor Signal
Gai | Inhibitory blood vessels pre-synaptic CNS, post synaptic
57
A2 (NE, epi) | 2nd messengers
Inhibit Adenylate cyclase Decrease cAMP Increase K conductance
58
A2 NTs Physio response
NE, epi | Decreased CAMP leads to Increased smooth muscle contraction, hyperpolarization inc K
59
B1, 2, 3 (epi, norepi) Receptor Signal
Gas | Excitatory or inhibitory depending on cAMP
60
B1, 2, 3 | 2nd messengers
Activate Adenylate cyclase | Increase cAMP
61
B1, 2, 3 NTs Physio response
NE, Epi | Increased cAMP relaxes smooth muscle, stim cardiac contractility, increases rate

Pharmacology (19 decks)

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