Anomalies pt 2 Flashcards
MIH - what teeth must it affect for diagnosis? what appear on teeth? where to lesion start/progress? how does colour of lesion change from deeper to more superficial?
must affect at least 1 first permanent molar
well demarcated lesions - white, cream to brown
lesion start in inner aspect of enamel and work outwards
more superficial lesions are whiter, browner lesions further into enamel
what does hypomineralised enamel mean? what is it prone to? how do bond strengths differ?
right quantity of enamel but defect in quality - softer enamel prone to post eruptive breakdown
poorer bond strengths
when should first permanent molars be XLA’d in MIH? what will this allow?
8-10yrs
space will spontaneously close w/ 7s erupting in (7s erupt around 11-13yrs)
what will happen if 6s XLA’d too early or too late?
too early = 7s will drift distally
too late = 7s will not close space as already erupted/partially erupted
explain balancing and compensating XLA approach - what does each approach ensure?
balancing = take out contralateral tooth at same time - maintains midline
compensating = take out opposing tooth at same time as XLA - prevents overeruption
how is LA affected in MIH teeth? what LA techniques are often required? possible resolution?
LA harder to achieve as inflamed pulp
buccal/palatal infiltration w/ articaine, IDB and intraligamentary often required
could do comp dressing of pulp to help settle down inflammation so easier to achieve LA at next appt
what could be done to FPMs prior to XLA time to stabilise?
hall crown
how do margins and caries resistance of white patches on fluorosis differ to MIH?
fluorosis show diffused patches - whereas MIH patches are well demarcated
fluorosis teeth caries resistant whereas MIH caries prone
list 6 ways to manage MIH & fluorosis?
microabraison - removes white patches to match rest of teeth
bleaching - matches normal tooth to whiter patches
resin infiltration
localised comps on lesions
veneers
is amelogeneis imperfecta caused by genetic or environmental factors? how does this differ from MIH/fluorisis?
completely genetic - no environemntal factors (unlike MIH & fluorosis)
name 3 types of AI - explain appearance
hypomaturation - enamel thinner and less dense/hypomineralised/pitted
hypoplastic - normal strength but not right shape (thin or absent enamel)
hypocalcified - enamel contains less calcium so thinner and weaker
what type of AI is associated w/ taurodontism?
hypomature enamel
give differing physical differences in the types of AI
hypomature - snow capped (white/brown striations), mottled/spotted/pitted
hypoplastic - not right size/shape but normal hardness, may be sensitive due to thin/absent enamel
hypocalcified - discoloured, opaque, chalky, extremely sensitive
chronological enamel hypoplasia - cause? presentation of enamel?
caused by environmental factor
thin or absent enamel - teeth look yellow
what is a turner tooth? what are they prone to? clinically present as?
permanent tooth that has been affected by infection or insult to primary tooth
prone to post eruptive breakdown and caries
may be sensitive