anesthesia for orthopedic surgery Flashcards

1
Q

what are common characteristics seen with orthopedic patients?

A
  • many have arthritic joints/deformities
  • many have limited ROM in neck
  • require careful positioning
  • may need help moving to OR table
  • fracture patients may have full stomachs
  • may have increased bleeding
  • increased risk of fat emboli, DVT, and PE
  • exposure to large amounts of radiation
  • 80% young and healthy; 20% old w/ fall or bad arthritis (inactive)
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2
Q

describe rheumatoid arthritis

A
  • immune related, progressive inflammation of synovial joints (not just joint wear and tear)
  • systemic manifestations
  • multiple joint involvement, deformity
  • chronic NSAID use (GI bleeding, renal toxicity, platelet dysfunction)
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3
Q

what are issues seen r/t joint deformity?

A
  • invasive lines and monitoring challenges
  • immobility and decreased endurance of patients
  • cervical and TMJ issues: check neck ROM, mouth opening
  • atlantoaxial subluxation: C-spine films to evaluate and determine if fiberoptic intubation is indicated if greater than 5 mm instability exists
  • cricoarytenoid arthritis: hoarseness, airway obstruction
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4
Q

what are CV effects of rheumatoid arthritis?

A
  • pericardial thickening and effusions
  • myocarditis
  • coronary arteritis
  • conduction defects
  • cardiac valve fibrosis (AR)
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5
Q

what are pulmonary effects of rheumatoid arthritis?

A
  • pleural effusions
  • pulmonary nodules
  • interstitial pulmonary fibrosis
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6
Q

what are hematological effects of rheumatoid arthritis?

A
  • anemia
  • platelet dysfunction
  • thrombocytopenia
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7
Q

what are endocrine effects of rheumatoid arthritis?

A
  • adrenal insufficiency (on steroid treatment)

- impaired immune system

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8
Q

what are dermatologic effect of rheumatoid arthritis?

A

thin atrophic skin

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9
Q

describe positioning for orthopedic cases

A
  • usu. done after pt. is anesthetized
  • usu. supine, sitting lateral decubitus or prone
  • protect face from injury: eyes/nose, pad face
  • sitting increases risk for air embolus: head and neck neutral position; avoid N2O
  • arms over chest may impede ventilation
  • sitting can reduce CPP
  • stretch injuries
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10
Q

describe used of regional anesthesia for orthopedic cases

A
  • excellent advantages but can be unreliable
  • supplement w/ IV opioids
  • may use more for upper extremity surgery
  • may be used in combo w/ GA for post op pain control
  • reduced blood loss in some surgeries
  • bier block commonly w/ wrist and hand surgeries
  • neuraxial or peripheral nerve catheters allow continuous infusions in the post op period
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11
Q

describe shoulder surgery

A
  • lateral or “beach chair” position (lateral is best)
  • open or arthroscopic (90%) technique
  • positioning challenge: ears, eyes, boney areas
  • may use LMA
  • interscalene block combine w/ GA
  • controlled hypotension (low 90s systolic allows surgeon to see shoulder joint)
  • irrigation pressure during arthroscopic surgery must be higher than systemic pressure (too high can lead to injury)
  • associated w/ nerve injury
  • VAE possible in “beach chair” position
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12
Q

how can CPP be estimated?

A
  • level of the external auditory meatus and tragus correlates w/ the Circle of Willis, where CPP can be measuered
  • lower pressure than level of heart d/t the vertical column and hydrostatic pressure difference
  • *each 10 cm distance from heart equals 7.5 mmHg lower
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13
Q

describe arthroscopic surgery

A
  • knee, shoulder, elbow, wrist, hip , ankle
  • less invasive w/ reduced blood loss
  • bloodless surgical field
  • minor incision w/ less post op pain
  • reduced blood pressure on non tourniquet joints optimizes surgical field
  • irrigation fluid 60-80 mmHg distends joint
  • high pressure w/ long duration associated w/ sub q emphysema and tension pneumothorax
  • keep systemic BP as low as possible to still maintain CPP so surgeons won’t have to increase their pressure
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14
Q

describe spinal surgery

A
  • most often to decompress cord or nerve root
  • neuro and ortho surgeons perform
  • multilevel instrumentation w/ plates, rods, and screws for spinal instability
  • GA w/ or w/o paralysis
  • if transthoracic, may need to deflate the lung
  • airway challenges: prone position, cervical immobility
  • can be done posterior or anterior approach
  • may develop CSF leak requiring a vital capacity maneuver (VCM or valsalva)
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15
Q

describe cervical spine surgery considerations

A
  • prone, sitting, or supine position
  • arthritic conditions may cause ROM issues (may require FO)
  • major arteries and veins are near by (check distal pulses; can use ear probe oximeter)
  • potential recurrent laryngeal nerve injury (some surgeons don’t allow muscle relaxant use)
  • airway swelling could be post op issue
  • identify and document preexisting neuro deficits
  • prevent too much neck and head traction (pad)
  • coughing or bucking must be prevented (LTA kit)
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16
Q

what arthritic conditions may cause ROM issues?

A
  • TMJ dysfunction: limited jaw opening
  • atlantoaxial instability: limited neck ROM
  • potential airway management challenge, may require F/O intubation
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17
Q

describe lumbar spinal surgery

A
  • can be minimally invasive or more extensive
  • prone position using prone chest rolls
  • brachial plexus at risk
  • keep arms and shoulders less than 90 degrees
  • abdominal compression
  • impedes venous return
  • diaphragm is cephalad, decreasing FRC and Vt
  • biggest fear: eye injury or vision loss
  • long procedures lead to large blood loss
18
Q

describe spinal fusion

A
  • fusion of two or more vertebra are indicated
  • bone grafting autologous or cadaveric
  • artificial disk is inserted into space
  • plates, rods, screws stabilize the two vertebra
  • restores the correct distance b/w vertebra as a “cage” is constructed to hold the vertebra in place and prevent collapse
  • risk hitting vertebrae, tear dura, or hit spinal cord
19
Q

what are anesthesia implications for spinal fusion?

A
  • identify and document preexisting neuro deficits
  • pressure off eyes, ears, nose, breast, genitalia
  • blindness can occur
  • ischemic nose or chin
  • limited access to airway, monitoring leads
  • longer procedures: warm patient, temp monitors
  • greater blood loss: cell saver, typed and cross matched
  • scoliosis pt. may have restrictive lung disease
  • major vessels are in close proximity to spine
  • monitor blood loss and temp*
20
Q

describe hip fracture

A
  • typically elderly, frail, debilitated, dehydrated pts. w/ existing comorbid conditions
  • supine position
  • fracture table
  • traction applied to fracture
  • perineal post
  • ipsilateral arm is placed on chest
  • hypobaric and hyperbaric SAB can be used
  • fat embolism common
21
Q

describe hip replacement

A
  • typical pt. is elderly, likely arthritic history
  • blood loss can be large (watch blood loss!)
  • lateral decubitus position allowing greater ROM
  • large incision
  • prosthetic can be cemented or uncemented
  • bilateral hip surgery contraindicated if declining pulmonary function occurs after first hip surgery (wait 6 mth.)
  • axillary/chest roll
  • up arm supported and stabilized
  • down ear should be flat or no pressure
22
Q

what causes declines in pulmonary function during hip replacements?

A

when banging prosthetic in, micro emboli will scatter from hip bone and will see a drop in the O2 saturation as micro emboli reach lungs

23
Q

describe knee replacements

A
  • usu. elderly, arthritic
  • supine position
  • tourniquet used
  • more painful than hip
  • high incidence of DVT
  • many regional anesthetic options
  • mostly older, at risk, out of shape population vs. young, active, in shape arthroscopic knee surgery
  • prosthetics cemented to femur and tibia
24
Q

what regional anesthetic options are available for knee replacements?

A
  • femoral 3 in 1 block (LFC, Obt, FN) combined w/ spinal

- femoral catheter used post op for pain control

25
Q

describe closed reduction

A
  • usu. very short procedures
  • hips, shoulders, wrist, elbow, fingers (many times popped out of place)
  • short acting paralysis usu. required (muscle tone may be working against surgeon popping back in )
  • may be done w/ mask ventilation
  • done many times w/ propofol bolus since short time (usu. minutes) are required for reduction success
26
Q

describe methylmethacrylate cement

A
  • used to bind prosthetic to bone
  • exothermic reaction occurs which hardens cement and expands (causes lysis of blood cells and marrow)
  • intermedullary hypertension: embolization of air, fat, marrow, and cement
  • systemic absorption: decreased SVR d/t vasodilatory effects; release of tissue thromboplastin, platelet aggregation, microemboli formation
  • *drop in O2 Sat
  • *cant smell if pregnant
27
Q

describe bone cement implantation syndrome

A
  • emboli migrate to pulmonary system
  • hypotension
  • hypoxia
  • reduced cardiac output
  • dysrhythmias
  • shunt
  • pulmonary hypertension
  • increase FiO2
  • adequate hydration
28
Q

describe the pneumatic tourniquet

A
  • utilized to minimize blood loss and optimize surgical field
  • exsanguination: esmarch bandage; distal to proximal
  • cuff overlap should be 180 degrees from nerve bundle
  • inflation pressure: determined by BP
  • typically 100 torr lower ext. and 50 torr upper ext. over SBP
  • venous tourniquet can occur
  • neurological damage may occur if over 2 hours or if overlap is over nerve bundle
29
Q

what are physiologic effects of tourniquet inflation?

A
  • initial rise in SVR, CVP, PVR
  • displaced blood volume (300-500 ml) w/ exsanguination
  • prolonged inflation increased HR and BP (cant control)
30
Q

what are physiologic effect of tourniquet deflation?

A
  • metabolic acidosis: increased potassium and CO2 levels
  • HR increase
  • temp decrease (warm blood goes back to cool limb and cools rest of body
  • hypotension (most common)
  • sudden reduction in SVR and PVR
  • wash out of ischemic metabolites (thromboxane)
  • *during inflation, distal to TQ, anaerobic cellular respiration occurring creating lactic acid and CO2 which is washed back into system w/ deflation (acidosis, hypotension)
31
Q

what are neurological effects of TQ?

A
  • abolition of SSEP and nerve conduction within 30 min
  • more than 60 min causes TQ pain and HTN
  • more than 2 hrs can result in post op neuropraxia
  • evidence of nerve injury may occur at the skin level underlying the TQ
32
Q

what are muscle effects of TQ?

A
  • cellular hypoxia within 2 min
  • cellular creatinine value declines
  • progressive cellular acidosis occurs
  • endothelial capillary leak after 2 hrs.
33
Q

describe TQ pain

A
  • usu. an hour after inflation
  • increase in HR and BP
  • ischemia, nerve fiber compression
  • burning, dull aching, throbbing pain (c fibers)
  • IV analgesics minimally effective
  • opioids added to local anesthetic have been shown to help
  • deflation for 10-15 min can help
  • quality/intensity of block vs. level of block
34
Q

describe C fibers

A
  • unmyelinated
  • slow conducting
  • burning, dull aching, throbbing pain w/ TQ inflation
35
Q

describe A delta fibers

A

-pinprick, tingling pain after deflation

36
Q

describe long bone fractures

A
  • fracture of femur and tibia
  • fat released into the circulation
  • fat embolism syndrome (triad)
  • DVT and PE can occur
  • VAE can also occur
37
Q

describe fat embolus

A
  • common in fractures of femur and tibia (long bones)
  • fat globules are released into the blood
  • fat embolism syndrome triad occurs 12-24 hrs. later
  • tachycardia and/or ST segment changes may occur
  • fat in the urine and sputum, conjunctiva may be present
  • coexisting lung disease at greater risk
38
Q

what are the consequences of fat globules released in the blood?

A
  • impaired pulmonary perfusion
  • endothelial damage to pulmonary capillaries
  • alveolar wall damage
  • pulmonary congestion
39
Q

what makes up the fat embolism syndrome triad?

A
  • petechiae (axillary, subconjunctival)
  • dyspnea
  • confusion, mental changes
40
Q

what is treatment for fat embolism syndrome?

A
  • O2
  • fluids
  • steroids if indicated (fat emboli can cause inflammatory reaction in lungs)
  • aggressive ventilation required
41
Q

describe DVT and PE

A
  • more common after total hip and total knee arthroscopy
  • mechanisms: venous stasis, hypercoagulable state, platelet aggregation
  • TQ greatly contributes to problem: venous stasis
  • epidural or spinal reduces DVT (higher levels of plasminogen and plasminogen activators; hyperkinetic blood flow)
  • may be attributed to allowing early ambulation
  • utilization of anticoagulants
42
Q

describe post op pain management

A
  • NSAIDS (if not contraindicated) and IV opioids
  • systemic opioids via PCA device (improved anesthesia, decreased total opioid consumption, increased pt. and nurse satisfaction)
  • intra-articular injection: combo of opioids and LA
  • continuous post op neuraxial and peripheral analgesia: better pain relief, faster ambulation and overall rehabilitation, better post op joint mobility