AMD 1 Flashcards
What’s AMD- this question is to make u feel good that u aren’t actually dumb
age related macular disease
Wet amd
TREATABLE
10% of people
severe type of visual loss
whys it called macula lutea
u see the yellow pigment there
where do problems for AMD arise from
bruchs membrane- thickens- drusen- comprimese RPE- and then photoreceptors- va affected function detoriates with age
wet AMD aka
neovascular AMD- exudative AMD- choroidal neovascular membranes growing from under bruschs membrane causing heommarahges and scars
late AMD
discoform scarring- round circular scarring
end stage of wet AMD
poor va
dry AMD
aka geographic atrophy, looks like a country on the map
AMD- eyes behave symmetrically- likely to be same in other eye
drusen
under retina- in basement membrane under RPE- samll bv over drusen- SUBRETINAL
soft drusen RF fro CNV
CNV
networks of bv- that grow from choroid through basement membrane- BRUCHS
classed by location and pattern of angiographic leakage
most occur subfovealy
Angiographically, 29.8% of CNV lesions are predominantly classic, 20.4% minimally classic, 19.5% occult no classic, and 2.1% Pigment Epithelium Detachemnts.
how would u manage amd px
Counselling in clinic ECLO (Eye Clinic Liaison Officer) Low vision Clinic and advice Registration as partially sighted or severely sight impaired Macular Disease Society/ RNIB
wet amd management
-Laser photocoagulation-Few cases suitable shad too be outside fovea Tendency to undertreat Destructive treatment High rate of recurrence -PDT laser- veterphorin Subfoveal /juxtafoveal lesions Vision 6/60 or better Lesion predominantly classic cnv Avoid minimal classics and occult Damage limitation Destructive treatment VPDT cohort study -AntiVEGF’s -Single v combination therapy -Detection and early treatment -Prevention
