Altered LOC and Coma Flashcards

1
Q

Concussion:

  • what is this?
  • caused by?
  • results in what?
A

WHat: trauma induced alteration in mental status may or may not involve loss of conciousness.
Concussion = Mild TBI

Caused by:
-direct blow to the head, face, neck, or body

Results in neuropathological changes- function not structural.

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2
Q

What is the glasgow coma scale?

A

Eye opening:

  • spontaneous = 4
  • response to verbal command = 3
  • response to pain = 2
  • no eye opening = 1

Best verbal response:

  • oriented = 5
  • confused = 4
  • inappropraite words = 3
  • incomprehensible sounds =2
  • no verbal response = 1

Best motor response:

  • obeys commands = 6
  • localizing response to pain =5
  • withdrawl response to pain = 4
  • flexion to pain =3
  • extension to pain =2
  • no motor response =1
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3
Q

What is a TBI?

-what would their GCS score be for mild moderate and severe?

A

-head injury d/t contac and/or acceleration/deceleration forces

Mild TBI: 13-15 measured 30minutes after injury

Moderate TBI: 9-12

Severe TBI: less than 8

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4
Q

What are the two most common causes accounting for TBI?

What is the leading cause of death persons aged 1-45?

A
  • MVAs
  • Falls

-TBI is leading cause of death, males affected greater than females

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5
Q

what are the two phase of brain injury?

A

Primary: cortical contusion; may be coup or contracoup injury

  • Coup= direct blow to brain
  • Contracoup = injury to brain on opposite side of brain from where it was struck.

Phase 2: molecular injury mechanisms

  • continues for hours to days
  • free radical injury, apoptosis leading to ischemia
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6
Q

Describe mild and severe primary injury in TBI.

A

Severe: axonal rupture, can get generalized cerebral edema

Mild: diffuse axonal injury; leads to axonal swelling

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7
Q

TBI

  • clinical features
  • Sx
  • signs
A

Clinical features: +/- LOC, confusion, stupor, amnesia
important to know how long theyve had them.

Sx: HA, dizziness, disorientation, N/V

Signs: vacant stare, inability to focus, gross in-coordination, memory difficulties, delayed verbal expression, slurred or incoherent speech, emotion out of proportion to events.

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8
Q

Complicated TBI

-what sx indicate a more serious injury or rising intracranial pressure?

A
  • seizures
  • focal neurologic signs
  • worsening HA, confusion, lethargy
  • protracted N/V
  • other injury to head and neck
  • decreasing GsC (repeat every 5-10mins)
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9
Q

Pt comes in with suspected TBI/concussion what would you like to examine?

A

pupils, are they moving their extremities, fingers, cranial nerves, moving facial muscles, strength, reflexes, orientation.
…the longer the frame of amnesia the more serious the injury!

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10
Q

Guidlines for CT scan in the ER?

A
  • GSC less than 15
  • open or depressed skull fx
  • any sign of basilar skull fx: CSF leak
  • two or more episodes of vomiting
  • 65yrs or older
  • amnesia impact of 3 or more minutes
  • dangerous mechanism (ejected from vehicle)
  • seizure
  • intoxication
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11
Q

What are signs of basilar skull fx?

can you detect basilar skull fx on CT?

A

-raccoon eyes, battle signs, blood behind eardrum.

Basilar skull fx is hard to detect on CT, you will need MRI

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12
Q

CT scan abnormalities found with TBI?

A
  • subdural hematoma
  • intracranial bleeding
  • cerebral edema
  • skull fx
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13
Q

Who do we hospitalize with TBI?

A

GSC less than 15 or deteriorating

Abnormal CT

Seizures

abnormal bleeding parameters

those who do not have someone to care for them.

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14
Q

Outpatient Observation of TBI:

-what are signs you need to be aware of indicating that you should return to the ER?

A
  • somnolence or confusion
  • difficulties w/ vision
  • severe or worsening HA
  • urinary or bowel incontinence
  • weakness or numbness
  • unsteadiness or seizures
  • N/V greater than 2x
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15
Q

Sequelae from TBI

A
  • post-concussion syndrome
  • post-traumatic HA
  • post traumatic seizures
  • post traumatic vertigo
  • other cranial nerve injuries
  • second impact syndrome (person develops second concussion before the first one resolves leading to brain swelling)
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16
Q

Post concussion syndrome:

  • sx
  • dx
  • tx
A

Sx: HA, dizziness, neuropsychiatric, cognitive impairment including noise sensitivity

Dx: clinically with Hx, if severe sx get MRI

Tx:

  • education
  • resolves in 3mo
17
Q

Coma:

  • what is this?
  • common causes
A

What: unarousable and unresponsive

Common Causes:

  • TBI
  • hypoxic ischemic encephalopathy (post CPR hypoxic brain)
  • drug overdose
  • intracranial hemorrhage
  • CNS infection
  • brain tumor
  • bilateral and unilateral cerebral disorders
  • brain stem disorders (IICP)
  • systemic (toxins, metabolic=sepsis, renal/hepatic failure)
  • endocrine (panhypopituitarism)
18
Q

Coma pathophysiology

A

focal lesion in the upper brainstem can alter alertness by damaging the ARAS (ascending reticular activating system)

ARAS: neurons project from the pons/midbrain up to the cortex.

Coma related to toxic, metabolic, & infectious etiologies is not well understood.

19
Q

What is decorticate posturing? decerebrate?

A

Decorticate posturing: UE addiction (flexion oat the elbows and fingers) w/ LE extension. Dysfunction of cerebral cortex or thalamic damage; better outcome.

Decerebrate: UE extension, adduction and pronation with LE extension. Injury to diencephalon, midbrain, or pons. Worse outcome,

20
Q

Describe each of the following breathing patterns.

  • cheyne stokes
  • hyperventilation
  • apneustic
  • ataxic
A

Cheyne stokes: cyclic pattern, hypernea & apnea

Hyperventilation: increased RR.

Apneustic: prolonged pausee at the end of inspiration

Ataxic: irregular in rate and tidal volume.

21
Q

Dx coma

A

figure out underlying cause via:

  • neuroimaging (CT)
  • LP
  • EEG
  • any lab you can think of
  • Physical exam
22
Q

Management of Coma

A
  • protect airway
  • hydrate
  • monitor blood glucose and electrolytes