Alterations to the Normal Swallow Flashcards

1
Q

What are some warning signs of dysphagia?

A
  • Reports problem
  • Drooling/poor oral hygiene
  • Dysarthria +/- dysphonia
  • Gurgle voice
  • Increased time with meals OR refusal to eat or drink
  • Coughing on food/fluids
  • Recurrent chest infections/unexplained
    temperature spikes
  • Fluctuating level of consciousness
  • Primitive oral reflexes
  • Weight loss
  • Confused mental state
  • Diminished/absent cough
  • Certain medications
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2
Q

How does aspiration impact swallowing function, before, during and after the swallow?

A

Before:
i.e. food/fluid content from a swallow will enter airway before the swallow is triggered (eg. Poor oral control allows spillage of bolus over base of tongue and down into pharynx and airway before a patient even triggers a swallow)

During:
i.e. food/fluid content from a swallow will enter airway while the swallow is happening (e.g. as the bolus is passing through the pharynx and part of it enters the airway because of failure to protect the airway e.g., failure of the vocal folds to close)

After:
i.e. food/fluid content from a prior swallow will enter the airway at a later time-point (e.g. residue left post swallow sits in oral cavity/pharynx and can later enter airway)

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3
Q

How does labial contraction/closure impact the person’s swallow/risk of aspiration?

A

Impact to oral prep and oral stage

  • Impaired ability to remove food from spoon
  • Food or liquid may leak/spill from the mouth
  • In moderate to severe weakness – lack
    of saliva control, drooling
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4
Q

How does impaired tongue function impact the person’s swallow/risk of aspiration?

A

Impact to oral prep, oral stage & consequences for pharyngeal stage

BEFORE:
↓ability to form and hold bolus (at tip & base) – lack of bolus control in oral stage
particles may fall over the base of the tongue during oral stage and be aspirated before swallow is initiated

AFTER:
↓ ability to control/manipulate food in the mouth during mastication
food inadequately masticated, food/fluid spreads throughout oral cavity + bolus separated into and swallowed in “bits” rather than a cohesive whole – piecemeal deglutition
Residue may remain in mouth that can later fall over the base of the tongue and be aspirated after that swallow has happened - the residue will fall down into an unprotected airway later

Poor tongue base retraction impacts hyoid movement, laryngeal elevation and epiglottic deflection…. impairs airway protection

Impaired lingual propulsion & tongue retraction – impacts hyoid elevation & there is lack of pressure behind bolus tail to assist bolus flow through pharynx → residue in pharynx

Residue may remain in pharynx, valleculae and pyriform sinus, and be aspirated at a time point later after that swallow has happened

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5
Q

How does reduced jaw strength and function impact the person’s swallow/ risk of aspiration?

A

Impacts oral prep stage

  • Food inadequately masticated into size for safe swallowing
  • increased risk of choking
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6
Q

How might reduced buccal tension impact the person’s swallow/ risk of aspiration?

A

Impacts oral prep stage

  • Food may fall into the lateral sulcus during chewing and remain there as residue ……….need assistance to be removed from active tongue movement / other (eg., finger)
  • particles may later fall over the base of the tongue and be aspirated at a time point later after that swallow has happened
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7
Q

How might reduced oral sensitivity impact the person’s swallow/ risk of aspiration?

A

Impacts oral prep, oral stage, swallow initiation

BEFORE
* Inability to appropriately prepare bolus for swallowing (size, texture, wetness)

Lack of sensory awareness leads to premature spillage of bolus over base of tongue → before swallow is initiated
aspirated before the swallow

AFTER
Material falls/lodges in areas of reduced sensitivity & left as residue in mouth
food particles may later fall over the base of the tongue and be aspirated after the swallow.

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8
Q

How might impaired velopharyngeal function/SOFT PALATE impact the person’s swallow/ risk of aspiration?

A

Impacts oral and pharyngeal stage

BEFORE
* VP fails to drape against base of tongue during oral stage. If there is also poor tongue function, lack of additional protections from VP can also contribute to aspiration before the swallow is initiated

AFTER
* Failure to contract and seal nasal cavity - Material may enter the nasal cavity – leading to discomfort + reside that can later fall into pharynx → aspiration after the swallow

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9
Q

How might delayed/absent swallow impact the person’s swallow/ risk of aspiration?

A

Impacts pharyngeal stage

BEFORE

  • If absent - Bolus is transferred into pharynx and the airway is not protected, causing aspiration of bolus
  • If delayed, bolus will reach pyriform sinuses and potentially overspill into laryngeal inlet before airway protection is in place – smaller amounts of aspiration

→ aspiration before the swallow is initiated

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10
Q

How might reduced laryngeal closure impact the person’s swallow/ risk of aspiration?

A

Impacts pharyngeal stage
DURING
Airway protection is compromised as larynx/airway is not closed allowing aspiration to occur during the swallow

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11
Q

How might reduced laryngeal elevation impact the person’s swallow/ risk of aspiration?

A

Impacts pharyngeal stage

BEFORE

  • If larynx doesn’t elevate then its not “tucked” under tongue
    aspiration may occur before the swallow as the airway is not fully protected when the bolus transitions from oral cavity to pharynx (i.e. before swallow trigger)

AFTER
* if larynx is not elevated, you get poor epiglottic deflection → can cause residue to trap in valleculae
Aspiration can occur after the swallow when any residue in valleculae can later slide into airway

  • if larynx is not elevated, then the CP sphincter is also probably not being pulled open very well – causing problems with bolus flow & causing residue in pyriform sinus
    Aspiration can occur after the swallow when that residue can enter into airway
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12
Q

How might reduced pharyngeal contraction impact the person’s swallow/ risk of aspiration?

A

Impacts pharyngeal stage

  • Impacts laryngeal elevation (long muscles of pharynx assist elevation)

AFTER
* Impaired bolus transition through pharynx → residue may remain in the valleculae & pyriform sinuses and particles may later fall into the airway, aspiration may occur after the swallow

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13
Q

How might weak cricopharyngeus muscle impact the person’s swallow/ risk of aspiration?

A

Impacts pharyngeal and oesophageal stage

AFTER

  • Failure to close behind the tail of the bolus
  • Peristaltic activity in may force some of the bolus that had passed through UES to return into the pharynx through the semi-open sphincter - and spill into the airway, causing aspiration after the swallow.
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14
Q

How might poor esophageal peristalsis/ physical obstruction impact the person’s swallow/ risk of aspiration?

A

Impacts oesophageal stage and can impact pharyngeal stage

AFTER
* Boluses may not transit efficiently (slowing/stasis)…causing subsequent boluses to build up behind prior slow moving boluses (or any physical obstruction) and ultimately stop any further contents from passing through UES

  • Subsequent boluses backflow/remain in pharynx with nowhere to go…but into the opened airway post swallow, causing aspiration after the swallow.
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15
Q

What 6 other factors influence a swallow?

A

Age
Volume
Viscosity
Delivery of bolus
Impaired respiratory support
Saliva, taste, flavour perception

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16
Q

How would age influence swallowing?

A

Infant to adult swallow
Developmental differences – learn more in peads lectures to some!

Young vs Old Adults

Oral stage
Older:- longer oral stage of the swallow . Also produce extra hyoid gestures “tongue pumping”

Residue
Older:- normal to have more small amounts of oral and pharyngeal residue

Trigger of pharyngeal stage
Younger:- triggered sooner - when bolus passes anterior faucial arches

Older:- triggered later - by the time at which the base of tongue crosses lower rim of mandible

Differences in laryngeal elevation:
Older men elevate larynx just enough to open the UES but no more, whereas young men continue to elevate larynx beyond when upper esophageal sphincter (UES – also called cricopharyngeal sphincter) has opened

Reduction in sense of smell and taste
with advancing age

Changes to oesophageal transit and clearance
slower and less efficient with increasing age

IMPORTANT TO RECOGNISE WHAT IS NORMAL AGE RELATED CHANGES (Presbyphagia) ….VERSUS WHAT ARE SIGNS OF ACTUAL DYSPHAGIA OR SARCOPENIC DYSPHAGIA

17
Q

How does volume influence swallowing?

A

Small volume (1-3ml)
Oral phase followed by pharyngeal phase then oesophageal phase

Saliva swallows
1ml = saliva size

Larger volume (10-20ml)
Simultaneous oral and pharyngeal activity - necessary to clear large bolus from oral cavity and pharynx
Can see delay in contact between tongue base and pharyngeal wall

18
Q

How does viscosity influence swallowing?

A

Thin, Thick, Solid

  • ↑ tongue force
    ↑submental and infrahyoid activity
  • ↑pharyngeal contraction
  • ↑transit time from oral to pharyngeal phases
  • ↑total swallow duration
  • NO CHANGE to laryngeal closure time
  • Normal for SOLID material to
    enter and dwell in the valleculae prior to the swallow
    have some premature spillage:- During active chewing, soft palate is NOT down and forward, and premature spillage is common and is entirely normal.
19
Q

How does respiratory support influence swallowing?

A

Poor / reduced respiratory capacity

Unable to manage with apnea periods associated with swallow – may fatigue, de-saturate

Respiratory need overrides laryngeal protection - may inhale too soon

May need addition oxygen supplementation at mealtimes, or pattern of intake

20
Q

How does the delivery of bolus influence swallowing?

A

Fed vs self feeding
Self feeding more natural
Impulsivity

Manner of delivery
Cup drinking, sequential sipping vs sipping
Issues: controlling size and amount of bolus taken in, unsafe swallow position

Straw drinking
Can help, but also make things worse when have uncoordinated inhalation

21
Q

How does taste, saliva and flavour influence swallowing?

A

Xerostomia – impaired/reduced saliva
Common post radiotherapy (XRT) + a common side effect of many medications
Impact to bolus movement – dryness impedes flow
Impact to taste reception – taste molecules carried to taste receptors by saliva, thus minimal saliva impacts taste detection at receptors on tongue

Dysgusea (impaired), phantogusea/parageusia (altered)
Impaired and/altered taste perception common post XRT
Normal aging creates reduced taste and smell perceptions – impacting flavour perceptions
impact s diet and consumption

22
Q

What are the 6 key clinical populations for dysphagia?

A

Acute neurological
Degenerative neurological
Critical care and trauma
Mechanical/ structural
Pulmonary
Iatrogenic

23
Q

What are acute neurological populations?

A

Stroke
- Impact determined by size and location of cortical damage
- Brain stem damage impacts processing sensory and motor aspects on swallow
- Significant pharyngeal stage impairment
- Often present with absent swallow initially
- May take weeks to recover

CORTICAL: Left CVA
- Oral and pharyngeal stage deficits
- Unilateral weakness
- Impact of associated aphasia on dysphagia management

CORTICAL: Right CVA
- Oral and pharyngeal stage deficits - - Unilateral weakness
- Behavioural issues impacting as well as motor deficits eg., verbose, ↓attention; poor new learning, Impulsive ++, Left neglect

Multiple strokes

Head injury:
- Diverse damage
- Direct head injury, brainstem twist, puncture wound, laryngeal fracture
- Extent of injury and swallowing severity correlation
- Impact of management; medications, intubation, surgery

Swallow patterns:
- Oral disorders: lip closure, tongue function, oral reflexes
- Pharyngeal: triggering swallowing, reduced laryngeal elevation and CP opening, reduced airway closure
- Cognitive overlay: impulsiveness, reduction in new learning
- Pulmonary: ventilation
- Recovery: varies

24
Q

What are degenerative neurological populations?

A

Parkinsons, MND, Dementia ++

Parkinsons:
- Rigidity, tremor, bradykinesia
- Dysphagia not early feature
- Effect of medications
- Complication of associated dementia

Oral stage
- Tongue pumping ++
- Lingual tremor
- Inefficient mastication
- Piece meal deglutition, premature spill
- Buccal retention

Pharyngeal stage
- Delayed swallow initiation
- Pharyngeal residue secondary to poor BOT to PPW
- Later on reduced laryngeal excursion

Motor neuron Diseases

  • Chronic disease characterized by progressive degeneration of motor neurons in spinal cord, brain stem and cerebral cortex
  • Aetiology unknown (suggest metabolic); relatively rare
  • No medical treatment, palliative, steady progression until death
  • Oral stage & Pharyngeal stage significantly impacted (aspiration often final cause of death)
    - Muscle weakness, poor respiratory support, delays+++

Dementia
- Reduced interest/ desire to eat
- Easily distracted
- Food agnosia: confusing what is/what isn’t food, utensil use, inappropriate bolus size
- Long time of food in oral cavity
- Delayed trigger of pharyngeal swallow

25
Q

What are critical care and trauma populations?

A

Spinal injury, burns etc

Multiple factors influence swallow

Neurological damage

Structural damage

Management
- Brace/collars (spinal), positioning (burns)
- Iatrogenic damage eg., surgery, intubation

Medical fragility & instability
- Long periods ventilation & intubation

Assessment issues!!
- Team work critical

26
Q

What are mechanical/structural populations?

A

Cervical osteophytes:
- Bony outgrowths from C vertebrae
- Impacts swallowing
- Narrow pharynx
- Sensation of food getting stuck

Facial fractures:
- Internal fixation can allow oral intake
- Modified consistencies should be given for at least 6 weeks to minimise pressures or stress on bones

Pharyngeal Pouch:
- Zenkers Diverticulum
- Side pocket formed when pharyngeal muscle herniates
- Occurs in CP/UES region
- Hypertonic CP muscle requires increased pressure to drive bolus through
- Can cause aspiration - airway unprotected

Oesophageal disorders:
- Structural or functional issues with normal oesophageal function which causes problems with
- Swallowing large amounts of food
discomfort/ tightness in chest
- Backflow of materials back through CP sphincter
- Can aspirate

GERD
- Backflow may cause aspiration
- Burning sensation in pharynx
- Frequent coughing or gagging
- If aspirated acid - lungs burn
- Gastroenterologist intervention needed

27
Q

What are pulmonary populations?

A

COPD
- Aerophagia: swallowing air, anxiety, hypoxia, dyspnea, reduced apetite, fatigue

  • Delayed pharyngeal swallow, delayed laryngeal closure, premature laryngeal opening , reduced airway protection
  • Slow and effortful bolus preparation, pharyngeal residue, slow clearance
  • Swallow-respiratory incoordination
28
Q

What are Iatrogenic populations?

A

Medications:
- Sedatives
- Antipsychotics
- Reduced saliva production
- Reduce awareness and alertness
- Delay swallow

Surgery
- Structure, function, innervation of structures or impact

Intubation
- Trachy tube

Certain treatments
- Chemotherapy
- Radiotherapy