Alterations in Special Sensory Function Flashcards

1
Q

what is conductive hearing impairment? mechanisms? etiology?

A

-Decreased sensitivity to sound in external or middle ear
-Occurs when sound cannot reach cochlea
-Generally correctable with medical or surgical therapy

4 mechanisms
1. Stiffness effect (otosclerosis)
2. Obstruction (cerumen impaction)
3. Mass loading (middle ear effusion)
4. Discontinuity (ossicular disruption)

Etiologies: Wax in outer ear, ossification of bones, middle ear infections & edema

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2
Q

what is otosclerosis? patho? CM?

A

-abnormal growth in ear that cause progressive conductive sensorineural or mixed hearing impairment.
- >50% family hx, women>men, onset 20-30 yrs.
-May occur following pregnancy.

Patho:
-Bone resorption followed by overgrowth of new, hard sclerotic bone.
-The stapes is immobilized against oval window.
-Decrease in transmission of sound to the inner ear.

Clinical manifestations: Tinnitus, hearing loss, poor speech discrimination

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3
Q

what is otitis media? CM?

A

-Inflammation or infxn of middle ear with presence of effusion
-Children at risk d/t shorter, horizontally positioned eustachian tubes
-Risk factors: pacifiers, second-hand smoke, GI reflux, daycare, propped bottles

Acute: Usually short duration.
-Often follows URI
-Pathogen enters from pharynx via Eustachian tube.
-Puss forms & possible TM rupture.

Clinical Manifestations:
Sudden onset of ear pain, fever, poor hearing, dizziness, vertigo, mastoid pain.

Chronic
-Chronic E. tube obstruction impairs equalization of air pressure in middle ear.
-Damage d/t atrophy or perforated TM.
Clinical manifestations: Poor
hearing, “snapping & popping”, retracted TM, pain, vertigo, purulent DC

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4
Q

what is otitis externa? etiology? CM?

A

Inflammation of outer ear, known as swimmer’s ear

Etiology: Excess moisture in ear canal, fungi, bacteria, trauma.
-Common organism Pseudomonas aeruginosa.
-Removal of cerumen

Manifestations: Pain; touching tragus, fullness in ear, decreased hearing, white particles on sides of canal.

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5
Q

what is sensorineural hearing impairment? etiology?

A

Hearing mechanism disturbed in inner ear

Etiologies: long-term exposure to loud sounds, trauma, ototoxic rxs, aging, metabolic causes, certain disease states.
-Often irreversible: d/t drugs or trauma (loud sounds, CHI).

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6
Q

what is presbycusis? CM? categories?

A

-Gradual onset, bilateral, degenerative hearing loss
-May be progressive

Clinical manifestations: Hearing loss, dizziness, difficulty hearing high-pitched sounds & conversational speech

Four categories:
1. Sensory: atrophy & degeneration of sensory & supporting cells
2. Neural: loss of neurons in cochlea & CNS
3. Metabolic: atrophy of wall of cochlea
4. Mechanical: middle ear undergoes changes in properties

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7
Q

what is meniere disease? CM?

A

Chronic disorder of inner ear.
-Accumulation of endolymph in labyrinth; cochlea degenerates

Clinical manifestations: Tinnitus, vertigo, feeling of fullness, fluctuating hearing loss
-Onset of vertigo usually sudden, reaches maximal intensity within few mins, usually lasts for an hour or more
-Tinnitus: low buzzing/blowing sound; louder before vertigo attack

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8
Q

what is vertigo?

A

-Exaggerated sensation of motion with no actual movement
-Associated with nystagmus & nausea, vomiting, diaphoresis
-Tx aimed at correcting cause.
-Cardinal symptom of disorders of inner ear or cochlear nerve.

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9
Q

what are the types of errors of refraction?

A

Myopia: Nearsightedness
Elongated eye, image focused in front of retina rather than on it; fuzzy image

Hyperopia: Farsightedness
Shortened eye, image focused behind retina; fuzzy image.

Presbyopia: Unable to see near objects clearly
Loss of accommodative capacity, corrected with reading glasses

Astigmatism: Irregular curvature of cornea or lens
Distorted image, corrected with lens of opposite curvature

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10
Q

what is strabismus disorder (age-related)? CM? etiology? tx?

A

-Ocular misalignment

Manifestations: Squinting, close 1 eye to see, difficulty picking up objects, dizziness, HA.

Etiology: Abnormal fusion mechanism of visual system.

TX: Occlusion therapy, corrective lenses, surgery or eye exercises.

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11
Q

what is amblyopia disorder (age-related)? etiology? tx?

A

-Poor vision of 1 or both eyes despite optical correction.
-Normal-appearing retinal & optic nerve pathways.
-Decreased visual acuity with no organic causes.

Etiology: Untreated strabismus

TX: atropine or patching of stronger eye.

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12
Q

what are cataracts? etiology? CM? tx?

A

-Clouding or opacity of lens; causes eventual loss of sight.
-Usually bilateral and most common > 65 yrs.

Etiologies: Aging, trauma, congenital factors, metabolic dse, rx´s

Clinical Manifestations
-White opacity of the lens
-Gradual blurred vision
-Altered color perception
-Visual distortion
-Decreased night vision & increased glare at night
-Abnormal presence of light in the visual field
-Decline in near and distant vision
-Loss of red reflex

Tx: Surgical removal of cataract with lens replacement.

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13
Q

what is retinal detachment? etiology? patho? cm?

A

Separation of retina with sudden appearance of floating spots.
-Usually spontaneous but can be d/t trauma.

Etiologies: Tumors, inflammation, trauma, cataract extraction.

Patho: Retina folds back on itself. Breaks in retina allow fluid from vitreous cavity to seep into the defect.
-Separation causes ischemia & death of retinal neurons & total blindness in eye.

Manifestations: floaters, flashes of light in visual fields, loss of visual acuity, sense of veil coming down in visual field.

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14
Q

what is diabetic retinopathy?

A

-Disorder of the retinal vessels that cause micro aneurysms, hemorrhage (non proliferative) or neovascularization and subsequent leakage and retinal detachment (proliferative)
-Blurred, darkened, distorted vision; visual changes fluctuate.
-Preventive: tight glycemic control & careful monitoring.
-Surgery and laser procedures may be used.

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15
Q

what is macular degeneration? etiology? patho? CM?

A

Degeneration of macular area of retina with progressive loss of central vision.
-Leading cause of blindness >55yrs.

Etiologies: Idiopathic, injury, inflammation, nutritional, hereditary.

Patho: Failure of outer layer of retina; waste products/toxins accumulate & cell death occurs.

Manifestations: Loss of central vision, yellow spots on macula, visual distortion

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16
Q

what is glaucoma? chronic open-angle? acute angle closure?

A

-Increased intra-ocular pressure (IOP) & progressive vision loss.
-Accounts for 12%-15% of blindness in U.S.
-Aging and family history

Chronic Open-Angle
-IOP increased by decrease in outflow of aqueous humor
-Gradual loss of vision in periphery results in tunnel vision
-Tx: Increase drainage of aqueous humor & decrease IOP

Acute Angle Closure
-Angle between pupil & cornea is narrow; blocks aqueous humor outflow when pupil dilated
-Severe eye pain, n/v, blurred vision & halos, red eye, dilated pupil nonreactive to light
-Medical emergency!!
-Tx: decrease IOP

17
Q

what is conjunctivitis?

A

-“Pink Eye”
-Inflammation of the conjunctiva.
-Conjunctiva becomes irritated & blood vessels enlarge making eye appear red in color.
-Most common causes are viral, allergic & bacterial (staphylococcus) infections

18
Q

what is epistaxis? etiology?

A

-Nosebleed
-Men affected more than women.
-Anterior & posterior bleeds.

Etiologies:
Anterior: rx’s, drying, infection, trauma
Posterior: blood dyscrasias, HTN, DM, trauma (more severe).

19
Q

what is sinusitis? CM?

A

-Inflammation of mucous membranes of paranasal sinuses.
-Classified as acute, subacute, or chronic.
-Inflammation causes obstruction; ideal for bacterial growth which causes swelling and congestion.
-Common organism is streptococcus.
-Manifestations: pain, sinus tenderness, HA, fever, malaise, purulent discharge