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oral path 1 final > Allergies and immunologic Diseases > Flashcards

Allergies and immunologic Diseases Flashcards

1
Q

canker sores
familial relationship,
unknown pathogenesis; immune mediated process
most common in children and young adults

A

recurrent aphtous ulcerations

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2
Q

what are the 3 clinical variations of recurrent aphthous ulcerations?

A

minor
major
herpetiform

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3
Q

most common recurrent aphthous ulcerations

A

minor aphthous ulcerations

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4
Q

most common form
only on NONKERATINIZED MUCOSA
most common sites are buccal and labial mucosa ventral tongue, floor of mouth and soft palate
1-5 PAINFUL ulcers preceded by erythematous macule and prodromal symptoms
ERYTHEMATOUS HALO

A

minor aphthous

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5
Q

which one of the aphthous ulcerations heal without scaring?

A

minor

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6
Q

3-10 mm diameter -
1-3 cm diameter-
1-3 mm diameter -

A

minor
major
herpetic

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7
Q

1-5 PAINFUL ULCER -
1-10 lesions per episode
many present per episode

A

minor
major
herpetic

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8
Q

heal in 7-14 days
heal in 2-6 weeks
heal in 7-10 days

A

minor
major
herpetic

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9
Q

buccal and labial mucosa, ventral tongue, floor of the mouth, soft palate

labial mucosa, soft palate, tonsillar fauces

any oral mucosal surface

A

minor

major

herpetic

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10
Q

true or false

minor aphthous does not cross the MGJ

A

true

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11
Q

true or false

major aphthous has lesser erythmatous halo compared to minor

A

true

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12
Q

which of the the following has resemblance to primary HSV?
minor
major
herpetiform

A

Herpetiform

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13
Q

which of the following has most frequent recurrences?
minor
major
herpetiform

A

herpetiform

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14
Q

what is the medication for Recurrent Aphthous Ulcerations?

A

topical HIGH-potency corticosteroids

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15
Q

multisystem disorder with oral aphthous like ulcerations
affects genetically predisposed individual
Highest prevalence in Turkey, Japan and Eastern mediterranean countries

A

Behcet’s syndrome

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16
Q

what is the triad for behcet’s syndrome

A

oral ulcerations
genital ulcerations
ocular disease

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17
Q

which of the following has cutaneous leisions, arthritis, CNS involvement, cardiovascular, GI, pulmonary, renal?

oral
genital
ocular

A

ocular disease of the behcet’s syndrome

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18
Q

posterior uveitis

A

ocular

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19
Q

you have a positive pathergy test. what is the diagnosis?

A

behcet’s syndrome

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20
Q

what is the treatment for behcet’s syndrome?

A

topical or systemic immunosuprressive therapy

early aggressive therapy for severe cases

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21
Q

non-necrotizing granulomatous inflammation,
persistent non-tender swelling
LIPS most common site, may involve any oral mucosal or perioral location

A

orofacial ganulomatosis

22
Q

what are the two forms of orofacial granulomatosis?

A

cheilitis granulomatosa

melkersson-rosenthal syndrome

23
Q

which of the two orofacial granulomatosis have only lips involvement

A

chelitits granulomatosa

24
Q

A person has bell’s palsy, fissured tongue, non-tender lip swelling.
non-necrotizing granulomatous inflammation

A

melkerssson-rosenthal syndrome (one of the two orofacial granulomatosis

25
Q

what are the microscopic features of orofacial granulomatosis?

A

superfical vascular ectasia, vasculitis, edema

non-necrotizing granulomas

no evidence of foreign material and specific stains to rule out specific infection

26
Q

medication for orofacial granulomatosis

A

intralesional corticosteroids work best

multiple injections may be necessary

27
Q

necrotizing granulomatous process with vasculitis
can affect upper airway, lungs, kidneys, skin and mucosa
wide range of age group

A

wegener’s granulomatosis

28
Q

what are the 3 categories of wegener’s granulomatosis

A
  1. Generalized wegener’s
  2. limited
  3. superficial
29
Q

generalized wegeners

A

upper and lower respiratory tract, kidneys

30
Q

limited wegener’s

A

upper and lower respiratory tract

31
Q

superficial wegener’s

A

lesions primarily of skin and mucosa

32
Q

STRAWBERRY GINGIVITIS - panthognomonic

this is very specific to this condition

A

wegener’s granulomatosis

33
Q

destruction of nasal septum (saddle-nose deformity)
dry cough, hemoptysis, dyspea, chest pain
proteinuria,
palatal ulcer with oral-antral fistula

A

wegnener’s granulomatosis

34
Q

what is the microscopic appearance of wegener’s granulomatosis?

A

subepithelial hemorrhage,
vasculitis
poorly formed granulomas, mixed inflammation

35
Q

autoantibodies directed against neutrophils have been identified in high percentage of these patients (ANCA)

A

wegener’s

36
Q

what is the prognosis of wegener’s granulomatosis

A

complete response in 75%
relapse in 30%
prednisone and cyclophosphamide vs methotrexate

37
Q

what are the two types of allergic reaction to systemic drugs?

A

fixed drug eruption

lichenoid drug reaction

38
Q

fixed drug eruption?

A

inflmaatory alteration that recur at the same site after the administration of a medication

39
Q

lichenoid drug reaction

A

medicaiton induced mucosal alterations which mimic appearche of lichen planus which is white line or stria

40
Q

what are there 3 allergic contact stomatitis?

A
  1. dentrifice-related sloughing
  2. orla mucosal cinnamon reaction
  3. lichenoid amalgam reaction
41
Q

what is associated with sodium lauryl sulfate and sodium pyrophophate?
found in toothpastes

A

dentrifice-related sloughing

42
Q

clinical varation of oral mucosal cinnamon reaction

toothpaste:

chewing gum/candy:

A

diffuse, gingiva

localized

43
Q

patient has erythema with overlying SHAGGY HYPERKERATOSIS,

pain and buring

A

oral mucosal cinnamon reaciton

44
Q

how long does it take to resolve oral mucosal cinnamon reaction

A

1 week following discontinuation of the cinnamon product

45
Q

lichenoid amalgam reaction

A

associated with old amalgam restorations undergoin corrosion and release of metallic ions

mucosal alterations mimic lichen planus HOWEVER are LOCALIZED.

may be white or erythematous

46
Q

what is the differential diagnosis of lichenoid amalgam reaction?

A

true leukoplakia would the differential diagnosis.

key is to look if the amalgam is contacting the mucosa

47
Q

will smith reaction in mr. hitch

A

angioedema

48
Q

IgE mediated hypersensitivity reaction

A

angioedema

49
Q

NON-PAINFUL pruritus and erythema may be seen
solitary or multiple sites may be involved
relatively rapid onset of sof ttissue swelking

A

Angioedema

50
Q

what inhibitor is involved in angioedema?

A

C1 esterase inhibitor and

ACE inhibitor

51
Q

excess bradykin

A

angioedema

oral path 1 final (4 decks)

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