AH II Test 3 Flashcards
Right Brain Damage
- paralyzed left side or hemiplasia
- left sided neglect
- spatial-perceptual defecits
- denies/minimizes problems
- rapid perfromance, short attention span
- impulsive, safety problems
Left-Brain Damage
- paralyzed right side, hemiplegia
- impaired speech/language aphasia
- impaired right/left discrimination
- slow performance, cautious
- aware of defecits, depression, anxiety
- impaired comprehension
Risk Factors for Stroke
- heredity
- age
- gender (males)
- race (African American)
- HTN
- Obesity
- DM
- smoking
- hyperlipidemia
- heavy ETOH, cocaine
- inactivity
- carotid stenosis
- oral contraceptives
- atrial fib
Status Epilepticus
- 2+ sz’s between which there is incomplete recovery of consciousness
- neurological emergency-high mortality rate due to hypoxia, hypoglycemia, systemic acidosis, hyperthermia, exhaustion
- precipitated by-abrupt stopping of AEDs, fever, acute ETOH withdrawal, head trauma, metabolic disturbances, infection
- Tx=AEDs (IV Dilantin), IV valium, ativan, O2, cool, protect, treat underlying cause
TIA
- cerebral ischemia without infarction
- symptoms resolve after an hour (usually)
- temporary loss of vision, hemiparesis, numbness, aphasia (disturbance of the comprehension and formulation of language caused by dysfunction in specific brain regions), tinitus, vertigo, ataxia
Symptoms of Stroke
- sudden weakness in face, arm, leg
- diff understanding of speech, loss of speech
- dizziness
- visual distrubances
- unexplained, severe HA
- change in personality or mental status–confusion
- sudden loss of consciousness or syncope
(older adults can have atypical presentation–falls, failing to eat/drink, functional decline, memory loss, urinary incontinence)
Time is Brain!
- penumbra=compromised area–has an ischemic core
- penumbra will necrose in 3-6 minutes
- inflammatory process outside of necrosis
- cellular death in 4-6 minutes
- average time bt onset of stroke and beginning of medical care = 24 hours
Ischemic Stroke
- 85% of strokes
- Thromolytic or Embolytic
- thrombolytic=usually where athreosclerotic plaques have narrowed blood vessel
- embolytic–most common=A Fib, can also be fat (long bone fx), air, tumor, bacteria)
Hemorrhagic Strokes
- bleeding into the brain (intracerebral) or into the subarachnoid space or ventricular space
- due to ruptured vessel
- 30-50 mls is a hemorrhage
- causes: HTN (esp during activity), aneurysms, AV malformation, coag disorders, anticoag drugs, trauma, bleeding, brain tumor
- sudden onset, severe HA
- complications=hydrocephalus, Sz, vasospasm, rebleeding (1,2,14d post stroke)
Dx Stroke
- CT scan to determine hemorrhagic/ischemic
- cerebral angiogram
- carotid ultrasound or doppler (use if carotid stenosis is believed to be cause of ischemia)
- LP for RBCs in CSF
- ECG (dysrhyth) or Echocardiogram (clots)
- coag studies-PT/INR, PTT, plts
Tx Stroke
- O2 for sats >92
- VS’s-MAP <130 to maintain CPP
- assess neuro status (GCS, pupils, motor/sensory function)
- IV with NS
- Labs (hyponatremia, hyper/hypoglycemia can cause more brain damage)
- sz control/precautions
- assess gag reflex
Tx for
Ischemic Strokes
- after CT scan—
- tPA-activates plasminogen which > fibrolytic process
- within 3.5-4 hr of stoke
- adverse effect=bleeding
- contraindications=taking anticoags, abn PTT, plts, BP > 185/110, recent GI bleed, stroke or head trauma within 14d, PG
- post tPA (or those ineligible)-anticoags, ASA, Plavix, Pradaxa
- take BP down slowly over time
- mechanical embolius removal in cerebral ischemia (MERCI)–done in interventional radiology-arteriorgram
- Sx-carotid endarterectomy (90% occlusion or more)
Tx for Hemorrhagic Strokes
- depending on cause–may require craniotomy to clip aneurysm or remove blood from brain
- serial CTs to monitor re-bleeding
- clipping, wrapping, coagulation, resecting, coiling
Stroke Deficits
- apraxia-inability to carry out learned movements (dressing, combing hair)
- agnosia-inability to use an item correctly
- aphasia-difficulty with language (expressive and/or receptive) *left brain damage
- dysphagia-difficulty swallowing
(wernicke=receptive, broca=expressive)
Stroke Dysphagia Screening
- GCS (cut off at <13)
- facial symmetry
- tongue symmetry
- palatal symmetry
- water test
Levels of SCI
- Quadriplegia-C1 through C8 (bowel and bladder)
- Paraplegia-T1 through L4 (bowel and bladder)
Muscles of Respiration
C2-C8: Accessory rib muscles
C2-C5: diaphragm-phrenic nerve
T1-T7: Intercostal muscles
T6-T12: Abdominal muscles
(for deep breathing and coughing)
Incomplete Cord Injuries
- Anterior Cord Syndrome–often flexion injury-motor function, pain, temp sensation lost below injury, no loss of reflexes (touch, position, vibration and motion remain in tact)
- Posterior Cord Syndrome-motor function, pain and temperature sensation in tact (loss of vibr, crude touch, and position sensation)
-Central Cord Syndrome-lesion in central cord; motor weakness and sensory loss in upper and lower extremities, more pronounced in upper
-Brown-Sequard Syndrome-penetrating injury that causes hemisection of cord-affects half of cord; characterized by ipsilateral loss of motor function and contralateral loss of sensory function.
Neurogenic Shock
- during acute phase
- complication of SCI
- hypotension
- brady
- warm, dry extremities
- inability to reg temp
- areflexia below injury
- no sensation, movement (flaccid)
Tx=dopamine for hypotension
Complications of SCI
Acute (30 minutes-6 weeks)
Neurogenic Shock=hypotension, brady, warm, dry extremities, Tx=dopamine
Hyperkalemia (due to muscle loss)–Kayexylate, diuretics, insulin
Chronic
autonomic dysreflexia (above T5-6, disconnect bt parasympathetic/sympathetic)=HA, HTN, brady, diaphoresis
hazards of immobility
bowel/bladder mgt
Autonomic Dysreflexia/Hyperreflexia
- complication of SCI
- more likely above T5-6
- disconnect bt parasympathetic and sympathetic
- intact lower motor neurons sense painful stimuli below level of injury (full bladder, bowel), trasmit message up cord, at SCI, pain signal stops
- ascending info reaches splenic sympathetic outflow (T5-6) and sitmulates sympathetic response
- S/S=sudden, severe HTN, pounding HA, brady, arterial dilation, flushed skin, sweating above T6, nasal congestion, cool skin and goose bumps below injury
- Tx-raise HOB, monitor BP, determine cause, vasodilators (apresoline, procardia, NTG)
- Complications=stroke, MI, Sz, renal hemorrhage, retinal hemorrhage
HNP
(herniated nucleus pulposus)
- Herniated discs
- L4-S1 most common (sciatica)
- C5-C7=second most common
Risk Factors: age >40, prolonged sitting, obesity, smoking, trauma, genetic predisposition
S/S=change in posture/gait, position change, muscle spasm, DTR loss, effect on ADLs, bowel/bladder status, lower extremity radiculopathy-sciatica (if orig L4-S1)
Tx-2 d bed rest, PT, ice/heat/massage, traction, NSAIDs, opioids, muscle relaxants, steroids. Sx-discectomy (removal of soft, gel stuff), laminectomy (removal of entire lamina)–log roll, do not ambulate w/o an order, do not raise HOB, draw sheet, neuro checks (motor loss, urinary retention, sensory loss), stool softener, wound check, TCDB q2, ATB, TLSO (vest), CSF leak, retroperitoneal hemorrhage, meningitis
Spinal fusion- for multiple laminectomies, chiropractic, acupuncture, spinal injections (steroids), chemonucleolysis therapy (enzyme injected to east herniated disk)-SE=anaphylaxis and paralysis