Age and Sex Considerations in Sport and Exercise Flashcards

1
Q

What are some physiological responses to acute exercise when a person is growing?

A
• Strength
• Cardiovascular, respiratory function
• Metabolic function
– Aerobic capacity
– Running economy
– Anaerobic capacity
– Substrate utilization
• Strength as muscle mass with age
– Peaks at ~20 years for women
– Peaks at 20 to 30 years for men
• Strength, power, skill require myelination
– Peak performance requires neural maturity
– Boys experience marked change at ~12 years
– Girls more gradual, linear changes
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2
Q

What are some physiological responses in terms of the CV system when a person is growing?

A

• Resting and submaximal blood pressure
– Lower than in adults (related to body size)
– Smaller hearts, lower peripheral resistance during
exercise
• Resting and submaximal stroke volume, HR
– Lower SV: smaller heart, lower blood volume
– Higher HR: almost compensates for low SV
– Slightly lower cardiac output than an adult
– (a-v-)O2 difference will increase to further compensate
• Maximal HR higher than in adults
• Maximal SV lower than in adults
• Maximal cardiac output lower
– Limits performance: less O2 delivery
– Not a serious limitation for relative workloads

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3
Q

What are some physiological responses to acute exercise in terms of lung function when a person is growing?

A
• Lung function
– Lung volume increases with age
– Peak flow rates increase with age
– Postpuberty: girls’ absolute values lower than boys’
due to smaller body size
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4
Q

What are some physiological responses to acute exercise in terms of metabolic function when a person is growing?

A

– Increases with age
– Related to muscle mass, strength, cardiorespiratory
function

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5
Q

What are some CV changes in response to acute exercise in a growing person?

A

• Cardiorespiratory changes during exercise
accommodate muscles’ need for O2
• Cardiorespiratory changes with age permit
greater delivery of O2
– V•O2max in L / min increases with age (boys, girls)
– V•O2max in ml / kg / min steady with age in boys
– V•O2max in ml / kg / min decreases with age in girls
– L / min more appropriate during growth year

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6
Q

How would one approach scaling data for size?

A

• V•O2max relative to body weight is
considerably different from absolute values,
as shown in figure 17.7
• Using body surface area or weight to the
0.75 power is the best way to reduce the
effect of body size on data

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7
Q

What are some physiological responses to acute exercise in children?

A

• Children’s economy of effort worse than
adults’
– Child’s O2 consumption per kilogram > adult’s
– With age, skills improve, stride lengthens
• Endurance running pace increases with age
– Purely result of economy of effort
– Occurs regardless of V•O2max changes, training
status

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8
Q

Explain the differences between a child’s vs. an adult’s anaerobic capacity for exercise?

A
• Children limited anaerobic performance
compared to adults
• Lower glycolytic capacity in muscle
– Less muscle glycogen
– Less glycolytic enzyme activity
– Blood lactate lower
– Mean and peak power increase with age
• Resting stores of ATP-PCr similar to adults’
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9
Q

What are the endocrine responses to acute exercise in a child?

A

– Exercising growth hormone and insulin-like growth
factor surge higher than in adults
– increased Stress response to exercise compared to adults
– Hypoglycemic at exercise onset
– Immature liver glycogenolytic system

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10
Q

What are substrate utilisation responses to acute exercise in a child?

A

– Relies more on fat oxidation compared to adults

– Exogenous glucose utilization high

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11
Q

How do body weight & composition respond to physical training in a child?

A

– Respond to physical training similarly to adults
– Training - decreased body weight/fat mass, increased FFM
– Significant bone growth

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12
Q

Is weight lifting safe for children to engage in?

A
• Weight lifting safe and beneficial
– Should be prescribed, supervised
– Low risk of injury
– Protects against injury
– Child: strength gains only via neural mechanisms,
no hypertrophy
– Adolescent: neural + hypertrophy
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13
Q

Describe some changes in regards to aerobic training in children?

A

– Little or no change in V•O2max

– Performance increased due to improved running economy

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14
Q

Describe some changes in regards to aerobic training in adolescents?

A

– More marked change in V•O2max

– Likely due to increase in heart size, SV

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15
Q

What are some physiological responses to anaerobic training in a child?

A

– increased Resting PCr, ATP, glycogen
– increased Phosphofructokinase activity
– increased Maximal blood lactate

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16
Q

Can adult anaerobic training programs be utilised to train a child?

A

• Adult anaerobic training programs can be
used with children and adolescents
– Be conservative to reduce risk of overtraining, injury,
loss of interest
– Explore variety of activities and sports

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17
Q

What are the typical physical activity patterns among youth?

A

• Physical activity patterns established in
childhood carry into adulthood
• Intervention strategies aimed at getting
children more active have been mostly
ineffective

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18
Q

Explain sports performance and specialisation in children/young adults?

A

• Sport performance in children and
adolescents improves with growth and
maturation
• Early specialization in one sport reduces
“fun” physical activities - reduced lifelong
physical activity

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19
Q

Explain thermal stress in children?

A

– Children have a larger surface area:mass ratio
– decreased Evaporative heat loss (less sweat)
– Slower heat acclimation
– Greater conductive heat loss, gain
• More research needed; be conservative

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20
Q

Explain a child’s growth when training?

A

– Little or no negative effect on height
– Affects weight, body composition with intensity
– Peak height velocity age unaffected
– Rate of skeletal maturation unaffected
• Maturation with training: effects on markers
of sexual maturation less clear

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21
Q

Explain the chantes in height, weight and body composition in an aging person.

A

• Height decreases with age
– Starts at 35 to 40 years
– Compression of intervertebral discs
– Poor posture
– Later, osteopenia, osteoporosis
• Weight increases, then decreases
– increases 25 to 45 years: decreased physical activity, increased caloric intake
– decreases 65+ years: loss of body mass, less appetite
• Body fat content tends to increase
– Active versus sedentary older adults vary
– Older athletes have lower body fat content
– Older athletes have lower central adiposity
• Fat-free mass decreases starting around age 40
– decreased Muscle, bone mass
– Sarcopenia (protein synthesis decreases)
– Due (in part) to lack of activity
– decreased Growth hormone, insulin-like growth factor 1
• Bone mineral content decreases
– Bone resorption > bone synthesis
– Due to lack of weight-bearing exercise
• Body composition variables
– Body weight
– Percent body fat
– Fat mass
– Fat-free mass (FFM)

22
Q

How does training alter age-related body composition changes?

A

– decreased Weight, percent body fat, fat mass
– increased FFM (more likely with resistance training than with aerobic training)
– Men > women
• Biggest results with diet + exercise

23
Q

Describe the physiological responses to acute exercise in an aging person.

A

• Strength and neuromuscular function decrease
with age
– Interferes with activities of daily living
– Manifests ~50 to 60 years of age
– Results from decreased muscle mass
• Strength decrease offset by resistance exercise
• Type II fiber loss with aging
– Decrease in type II motor neurons
– Type I neurons innervate old type II fibers?
– Higher percent type I fibers
• Training slows or stops fiber-type change
• Size and number of muscle fibers decrease with
age
– Size of both type I and type II
– Lose 10% per decade after age 50
• Endurance training - no impact on decline
in muscle mass with age
• Resistance training - reduces muscle
atrophy, increased muscle cross-sectional area
• Reflexes slow with age
– Exercise preserves reflex response time
– Active older people ≈ young active people
• Motor unit activation decreases with age
– Exercise retains maximal recruitment of muscle
– Some studies show decreased strength due to local muscle (not neural) factors
• Exercise maintains muscle physiology
– Number of capillaries unchanged
– Oxidative enzyme activity only mildly reduced

24
Q

What are some CV responses to acute exercise in an aging person?

A

• Central and peripheral cardiovascular
decrements with age
• Reduced maximal HR
– Reduction varies considerably
– Electrical and receptor changes with age
– Same for active and sedentary people
• HRmax = [208 – (0.7 x age)]
• Maximal stroke volume (SV) decreases with age
– decreased Contractility, response to catecholamines
– Partial loss of Frank-Starling mechanism
– LV, arterial stiffening
– Exercise attenuates decline in SVmax
• V•O2max decreased with age due to decreased Q•max
– Due more to decreased HRmax, less to decreased SVmax
– Exercise attenuates decline in V•O2max
• Sedentary habits increase risk for vascular aging
– decreased Cardiac and arterial compliance
– Endothelial dysfunction
– Reduced vasodilation
• Exercise -> decreased risk
– Less arterial stiffening, endothelial dysfunction
– Preserved vasodilator signaling
– Research ongoing on proper exercise dose for
cardiovascular benefit
• Peripheral blood flow decreases with age
– ~10 to 15% reduction even with exercise
– Due to increased vasoconstriction, decreased vasodilation
– increased (a-v-)O2 difference compensates for decreased flow
• Effects of primary aging versus
cardiovascular deconditioning
– Which changes result from aging alone?
– Which changes result from reduced activity?

25
Q

What are some respiratory responses to acute exercise in an aging person?

A

• Respiratory function with sedentary aging
– decreased Vital capacity and FEV1.0, increased residual volume, total lung capacity unchanged
– Less air exchanged
– decreased Lung and chest wall elasticity with age
– But does not limit exercise capacity
• Exercise maintains ventilatory capacity
– Pulmonary ventilation does not limit aerobic capacity
– Oxygen saturation remains high
• V•O2max changes with aging
– Measured in L/min or ml/kg/min?
– Absolute versus relative decrement
• V•O2max in normally active older people
– Declines steadily from 25 years to 75 years
– ~1% per year (~10% per decade)
• V•O2max in older male athletes
– 5 to 6% decline per decade in active adults
– 3.6% decline over 25 years in elite athletes
– 15% decline per decade in previously active adults
• V•O2max in older female athletes
– Fewer studies, but similar to men
– ~1% decline per decade
– Longitudinal changes > cross-sectional changes
• Percent decline in V•O2max related to
intensity of training before and during aging
• Factors that affect rate of decline
– Genetics
– General activity level
– Intensity and volume of training
– Age-related body composition changes
– Age range

26
Q

What are some changes in lactate threshold in response to acute exercise in an aging person?

A

• Lactate threshold (as % V•O2max) increases
– Not predictive of running performance with aging
– Percent V•O2max may not be best measure
– Remember: absolute V•O2 decreases with age
• Lactate threshold (as absolute V•O2) decreases

27
Q

What are the effects of resistance training on an aging person?

A

• Effects of resistance training on strength
– increased Strength (men, women: 30%; some studies of men: 50-200+%)
– Fiber hypertrophy
– increased Cross-sectional area of types I, II
– Neural adaptations
• increased Muscle mass, muscle size, bone mineral
density
• Improved activities of daily living, decreased risk of
falls

28
Q

What are VO2max improvements with training in an elderly population?

A

– Independent of sex, age, initial fitness
– Young: increased maximal cardiac output (central)
– Older: increased oxidative enzymes (peripheral)

29
Q

What are some anaerobic capacity changes with training in an elderly population?

A

– Less known than aerobic training results

– Lactate threshold bad predictor of performance

30
Q

Explain the changes of sports performance in an aging population.

A

• Running performance decreases with age.
– Rate of decline independent of distance
– Both 100 m, 10 km records slower with age
– Decline accelerates past age 60.
• Swimming performance decreases with age.
– Decline accelerates past age 70.
– Decline in women is greater than decline in men
• Cycling performance
– Peaks between 25 and 35 years
– Speed then decreases by 0.7% per decade
• Weight-lifting performance
– Peaks between 25 and 35 years
– Sum of power lifts then declines 1.8% per year

31
Q

Explain any special issues e.g. hyperthermia & the risks associated in an elderly population.

A

• Higher risk of death from hyperthermia
– Higher core temperature than young subjects
– Metabolic heat gain related to absolute V•O2
– Heat loss related to relative percent V•O2max
• Physical training affects thermoregulation
– Improves skin vasodilation (convection)
– Improves sweat rate (evaporation)
– Improves redistribution of cardiac output
• Exercise in cold = increased risk of hypothermia
– Risk not as great as hyperthermia
– Reduced ability to generate metabolic heat
– Excessive convective heat loss
– Core temperature can drop even with mild cold
stress
• Must add behavioral thermoregulation
• Exercise and longevity
– Mild caloric restriction increases longevity
– Exercise may contribute to caloric balance
– Exercise - compression of mortality
• Exercise can lead to injury
– Tendon injury (rotator cuff, Achilles)
– Cartilage injury (meniscus, focal injuries)
– Stress fractures
• Exercise can reduce risk of falls

32
Q

What is the difference between sex vs. gender?

A
• Sex is biologically determined.
• Gender is culturally determined.
• Most basic physiological comparisons to
date have established sex differences.
• IAAF policy on hyperandrogenism: Women
must undergo testing if too “masculine.”
33
Q

In relation to body size and composition, what is the effect of testosterone?

A

• Testosterone leads to
– increased Bone formation, larger bones
– increased Protein synthesis, larger muscles
– increased EPO secretion, increased red blood cell production

34
Q

In relation to body size and composition, what is the effect of estrogen?

A
• Estrogen leads to
– increased Fat deposition (lipoprotein lipase)
– Faster, more brief bone growth
– Shorter stature, lower total body mass
– increased Fat mass, percent body fat
35
Q

Explain body size and composition adaptations in females.

A

• Distinct female fat deposition pattern
• Rapid storage on hips and thighs due to increased
lipoprotein lipase activity
• decreased Lipolytic activity makes regional fat loss
more difficult
• Lipoprotein lipase decreases, lipolysis increases during third trimester of pregnancy, lactation

36
Q

How does muscle strength differ between sexes?

A

– Upper body: women 40 to 60% weaker
– Lower body: women 25 to 30% weaker
– Due to total muscle mass difference, not difference
in innate muscle mechanisms
• No sex strength disparity when expressed
per unit of muscle cross-sectional area

37
Q

What are the causes of upper body strength disparity?

A
– Women have more muscle mass in lower body
– Women utilize lower body strength more
– Altered neuromuscular mechanisms?
• Women: smaller cross-sectional areas
• Similar fiber-type distribution
• Research indicates women more fatigue
resistant
38
Q

How does cardiovascular function differ between the genders?

A

• For same absolute submaximal workload
– Same cardiac output
– Women: lower stroke volume, higher HR
(compensatory)
– Smaller hearts, lower blood volume
• For same relative submaximal workload
– Women: HR slightly higher , SV lower, cardiac output lower
– Leads to decreased O2 consumption
• Women compensate for decreased hemoglobin via increased (a-v-)O2 difference (at submaximal
intensity)
– (a-v-)O2 difference ultimately limited, too
– Lower hemoglobin, lower oxidative potential

39
Q

What are some sex differences in terms of respiratory function?

A

– Due to difference in lung volume, body size
– Similar breathing frequency at same relative
workload
– Women increase frequency at same absolute workload
• Women’s V•O2max < men’s V•O2max
• Untrained sex comparison unfair
– Relatively sedentary nonathlete women
– Relatively active nonathlete men
• Trained sex comparison better
– Similar level of condition between sexes
– May reveal more true sex-specific differences
• Can scale V•O2max to other body variables
– Height, weight, FFM, limb volume
– Sex difference minimized or gone with scaling
• Simulated women’s fat mass on men
– Reduced sex differences in treadmill time,
submaximal V•O2(ml / kg), V•O2max
– Women’s additional body fat major determinant of
sex-specific difference in metabolic responses
• Women’s lower hemoglobin limits V•O2max
• Women’s lower cardiac output limits V•O2max
– SVmax limited by heart size, plasma volume
– Plasma volume loading in women helps
– Submaximal absolute V•O2: no sex difference in SV
• Sex differences in lactate, threshold
– Peak lactate concentrations lower in women
– Lactate threshold occurs at same percent V•O2max

40
Q

What are some changes in body composition between the sexes?

A

• Body composition changes
– Same in men and women
– decreased Total body mass, fat mass, percent body fat
– increased FFM (more with strength vs. endurance training)
• Weight-bearing exercise maintains bone
mineral density
• Connective tissue injury not related to sex

41
Q

Explain strength gains in women vs. men?

A

– Less hypertrophy in women versus men, though
some studies show similar gains with training
– Neural mechanisms more important for women
• Variations in weight lifted for equivalent
body weight
– For given body weight, trained men have more FFM
than trained women
– Fewer trained women
– Factors other than FFM?

42
Q

Explain cardiorespiratory changes in the sexes? Are these changes sex-specific?

A

• Cardiorespiratory changes not sex specific
• Aerobic, maximal intensity
– increased Q•max due to increased SVmax (increased preload, contractility)
– increased Muscle blood flow, capillary density
– increased Maximal ventilation
• Aerobic, submaximal intensity
– Q• unchanged
– increased SV, decreased HR

43
Q

Explain VO2max changes between the sexes? Are these changes sex-specific?

A

• V•O2max changes not sex specific
– ~15 to 20% increase
– increased Q•max, increased muscle blood flow
– Depends on training intensity, duration, frequency
• Lactate threshold increased
• Blood lactate for given work rate decreases
• Women respond to training like men do

44
Q

Explain sports performance differences between the sexes.

A
• Men outperform women by all objective
standards of competition
– Most noticeable in upper-body events
– Gap narrowing
• Women’s performance drastically improved
over last 30 to 40 years
– Leveling off now
– Due to harder training
45
Q

Explain menstruation and athletic performance in females?

A

• Normal menstrual function
– Menstrual (flow) phase
– Proliferative phase (estrogen)
– Ovulation—follicle stimulating hormone (FSH),
luteinizing hormone (LH)
– Secretory phase (estrogen, progesterone)
• Cycle length ~28 days, can vary
• No reliable data indicate altered athletic
performance across menstrual phases
• No physiological differences in exercise
responses across menstrual phases
• World records set by women during every
menstrual phase

46
Q

Explain menstrual dysfunction and athletic performance in females?

A
• Menarche: first menstrual period
– May be delayed in certain sports (e.g., gymnastics)
– Delayed menarche: after age 14
• Delayed-menarche athletes self-select?
– Sport may not - delayed menarche
– Small, lean athletic girls (delayed menarche
candidates) may gravitate to sport
• Menstrual dysfunction
– Seen more in lean-physique sports
– Eumenorrhea: normal
– Oligomenorrhea: irregular
– Amenorrhea (primary, secondary): absent
– Can affect 5 to 66% of athletes
• Menstrual dysfunction ≠ infertility
• Secondary amenorrhea—caused by energy
deficit (inadequate caloric intake)
– decreased LH pulse frequency
– decreased T3 secretion
– decreased Estrogen, progesterone
– May also involve GnRH, leptin, cortisol
• As long as caloric intake adequate, exercise
does not lead to secondary amenorrhea
47
Q

Explain any pregnancy concerns?

A
  1. Acute reduction in uterine blood flow
    (shunt to active muscle) -> fetal hypoxia
  2. Fetal hyperthermia from increase in
    maternal core temperature
  3. Maternal CHO usage increased, thereby decreasing CHO availability to fetus
  4. Miscarriage, final outcome of pregnancy
    • decreased Uterine blood flow may not lead to hypoxia
    – Uterine (a-v-)O2 difference increase may compensate
    – Fetal HR increases due to maternal catecholamines
    • Fetal hyperthermia: unresolved
    • CHO availability: unresolved
    • Miscarriage, final pregnancy outcome
    – Data scarce, conflicting
    – Many studies show favorable (or no) effects
48
Q

Explain physical activity recommendations for pregnant women?

A

• Mild-to-moderate exercise 3 times / week
• No supine exercise after first trimester
• Stop when fatigued
• Non–weight-bearing exercise preferable
• No risk of falling, loss of balance, etc.
• Ensure adequate caloric intake
• Dress and hydrate to avoid heat stress
• Prepregnancy exercise routine should be
gradually resumed postpartum
• No scuba diving
• Benefits > risks if cautiously undertaken

49
Q

Explain osteopenia & osteoporosis and how hormone deficiencies contribute to these diseases (risk factors between the sexes?)

A
• Osteopenia versus osteoporosis
– Risk greater in women especially after menopause
– Slowed and retarded by weight-bearing exercise
• Major contributing factors
– Estrogen deficiency
– Inadequate calcium intake
– Inadequate physical activity
– Amenorrhea, anorexia nervosa
• Physical activity affects bone health
– Maximize bone density early in life
– Diet, weight-bearing physical activity
• Menstrual status affects bone health
– Particular concern for postmenopausal women;
exercise can protect against bone loss
– Women with amenorrhea or anorexia nervosa often
suffer low bone mass
50
Q

Explain eating disorders and risk factors between the sexes.

A

• Anorexia nervosa
– Refusal to maintain minimal normal weight
– Distorted body image, fear of fatness
– Amenorrhea
• Bulimia nervosa
– Recurrent binge eating
– Lack of control during binges
– Purging behaviors (vomiting, laxatives, diuretics)
• Young women at highest risk
• Eating disorder versus disordered eating
• Worse in certain sports
– Appearance sports: diving, figure skating, ballet
– Endurance sports: distance running, swimming
– Weight-class sports: jockeys, boxing, wrestling
– Perfectionists, competitive, under tight control
• Self-reporting underestimates prevalence
• Eating disorders considered addictions
– Behavior reinforced by media, parents, coaches
– Very difficult to treat
– Often accompanied by denial
– Life threatening, expensive to treat
• Must seek out trained clinical specialist

51
Q

What is the Female Athlete Triad?

A

• Syndrome of interrelated conditions
– Energy deficit  secondary amenorrhea  low
bone mass
– Disordered eating may (not) be involved
• Three disorders can occur alone or in
combination, must be addressed early
• Treatment: increased caloric intake, decreased activity (in
some cases)

52
Q

What is menopause & how can exercise be utilised for this condition?

A

• Usually occurs between ages 45 and 55
• Symptoms can lower quality of life in some
• Exercise is recommended to improve mood,
decrease depression, and improve sleep