Adult Consequences of Childhood Trauma Flashcards
when is root development complete for deciduous and permanent teeth
deciduous - 1.5 years after eruption
permanent - 3 years post eruption
what timings are acute , subacute and delayed treatment in regards to dental trauma
acute - within 3 hours
subacute - 3 to 24 hours
delayed - more than 24 hours before treatment received
why might a tooth see yellow discolouration post trauma
canal obliteration
should monitor for loss of vitality
why might a tooth see pink discolouration post trauma
haemmhorage in pulp chamber seeing blood leak into dentinal tubules
if no necrosis of pulp discolouration may reverse as pulp revascularises
if necrosis , discolouration may worsen
why might a tooth see brown/grey/black discolouration after trauma
in non vital teeth pulp death sees bleeding into dentine.
Hydrogen sulphates from bacteria can convert iron within blood to dark coloured iron sulphates making it darken over time.
Why might pulp necrosis and apical periodontitis occur after trauma
if revascularisation has failed
pulp will undergo sterile necrosis but subsequent bacterial infection may occur
diagnostic indicators of pulp necrosis and apical periodontitis
no response to vitality tests
TTP
possible sinus tract
periapical radiolucency on radiographs
grey/brown discolouration of crown
Requires endo or extraction
what is infra occlusion and why might it occur
occluding surface/ incisal edge of affected tooth is at insufficient height in comparison to adjacent teeth (think intrusion)
Most likely in teeth that have experienced severe PDL trauma e.g intrusion
occurs if ankylosis or replacement root resorption as a result of injury
not easily fixed with ortho
Extrinsic causes of tooth discolouration
smoking
tannins (red wine, tea, coffee, guiness)
chlorhexidine - in excess
iron supplements
chromogenic bacteria (more common in children , leaves black/green stains)
intrinsic causes of tooth discolouration
fluorosis
tetracycline (old antibiotic that can cause banded discolouration if taken during tooth development)
non vitality (blood products leach into dentine)
dental materials staining (amalgam)
physiological age changes
medical conditions (porphyria sees red deciduous teeth, cystic fibrosis pts may have grey teeth)
What should the first stage of treating extrinsic vital staining always be
hygiene phase therapy
What is the science behind external vital tooth bleaching
discolouration is caused by formation of chromogenic products which are long chain organic molecules, the aim of bleaching is to oxidise these compounds which leads to smaller molecules which are lighter in colour
What is the active agent in vital external bleaching and how does it work
active agent = hydrogen peroxide
Forms an acidic solution in water that breaks down to form hydrogen and oxygen. The free radical hydroperoxyl (HO2) is formed which is the active oxidising agent
What is the hydrogen peroxide containing chemical normally used in teeth whitening products
carbamide peroxide
consists of hydrogen peroxide and urea
what is in carbamide peroxide and what are each ingredients purpose during teeth whitening (6)
hydrogen peroxide - active ingredient
urea - increases pH to counteract H2O2
thickening agent - Carbopol
surfactant - allows gel to wet tooth surface
desensitising agent e.g Ca phosphate
Fluoride - prevents erosion by acidic H2O2 and has a desensitising effect