Adoption Study Heston 1966 Flashcards

1
Q

aim of heston 1966

A

how many adopted children of biological mothers with SZ would go on to develop it

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2
Q

sample of heston 1966

A
  • 50 adoptees with diagnosed SZ mothers, matched to 47 adoptees with non-SZ mothers
  • all from Oregon state
  • all separated within days of birth and contacted by letter for study
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3
Q

procedure of heston 1966

A
  • adults were interviewed in their homes via a general medical and environmental questionnaire to assess whether they had developed SZ
  • they were then given the Minnesota Multiphasic Personality Inventory
  • they were evaluated blindly and independently by 2 psychiatrists and a final evaluation was made by a 3rd researcher
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4
Q

results of heston 1966

A
  • out of those interviewed, 5 had been hospitalised with SZ
  • 3 of these were chronically ill
    -thus 10% of the adopted children of SZ mothers developed SZ
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5
Q

conclusions of heston 1966

A

genetics do play some role in the onset and development of SZ

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6
Q

generalisability of heston 1966

A

Low
- narrow sample of 47 adopted ppts from Oregon state
- ethnocentric so can’t be generalised to other cultures
- only studied SZ so can’t be generalised to genetic/environmental roles in other mental disorders/behaviours

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7
Q

reliability of heston 1966

A

High
- used a standardised procedure of structured interviews
- this allows for the study to be replicated to test for consistency of findings

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8
Q

validity of heston 1966

A

Low
- self-report data was used from interviewing ppts which is subjective and can lead to biased results

High
- high ecological validity as ppts were interviewed at their homes
- meant that ppts were less likely to present demand characteristics and felt slightly more at ease

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9
Q

ethics of heston 1966

A

Fair
- informed consent was obtained as ppts were contacted by letter asking if they wanted to take part

Low
- some questions may have caused stress as it was very personal, highly socially sensitive

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10
Q

applications of heston 1966

A

can assist with treatments for SZ
- if we know there is a genetic component we can develop drugs or therapies that prioritise the idea of rehabilitation or epigenetics

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