acyanotic CHD Flashcards

1
Q

LIST acyanotic

A

ASD

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2
Q

Atrial septal defect
ASD Definition:

A

Defect in interatrial septum

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3
Q
A
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4
Q
A
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5
Q
A
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6
Q

Types of ASDs

A

secundum
primum
sinus venosus

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6
Q

Ostium secundum defect
occurence
where
association

A

-The commonest type.
-Lies in the middle part ofthe septum at the site of fossa ovalis.
- Association: may be with Holt Oram syndrome (Absent radii , l 51 degree
heart block , ASD)

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7
Q

Ostium primum defect

A
  • Lies in the lower part of the septum
  • Association: usually with cleft of mitral valve leaflet.
  • mitral regurge.
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8
Q

Hemodynamics of ASD

A

Blood is shunted from left atrium to right atrium
-right ventricle -pulmonary blood flow
(more with primum defects).

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9
Q

General manifestations of ASD

A

1- Asymptomatic in most cases.
2- Large ASD (especially primum defect) may present with features of increased pulmonary blood flow .

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10
Q

Auscultation in ASD
2 things

A
  1. pulmonary area
    a- wide fixed splitting of S2:
    -wide splitting due to large filling of right ventricle & fixed (not vary with
    respiration) due to constant filling of right ventricle in all phases of respiration

b- murmur of a relative pulmonary stenosis:
- ejection systolic.
-soft.
- no thrill.
- no propagation
- with accentuated P2*

2- Apex: pansystolic murmer of mitral regurge in ostium primum defect.

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10
Q

Investigations
cxr
ecg

A

1- Chest X-ray:
-Cardiomegaly with RVH & RAD.
- Plethoric lungs.

2- ECG:
- RVH & RAD.
- Right bundle branch block is common.

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11
Q

Treatment of ASD

A

1- Medical:
-Control heart failure (diuretrics, digoxin, vasodilaters).
- Prophylaxis against infective endocarditis usually not needed.
- Antibiotics for chest infections.
-Follow up with ECG & Echo to confirm to spontaneous closure.

2- Surgical: Transcatheter or open heart surgical closure at 3-5 years.

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12
Q

Prognosis of ASD

A

40% of ostium secundum defects close in I st four years spontaneously

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13
Q

ventricular septal defect (VSD) definition

A

Defect in interventricular septum

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14
Q

Types ofVSDs:

A

membranous
muscular
supracristal

15
Q

hemodynamics of VSD

A

Blood is shunted from left ventricle to right ventricle
___.t pulmonary blood flow ___.t input to left atrium
and left ventricle.

16
Q

General manifestations

A

1- Small VSD: Usually asymptomatic, discovered accidentally.
2- Large VSD: - Features of increased pulmonary blood flow
- Congestive heart failure with tachypnea, tachycardia
& enlarged tender liver.

17
Q

Precordial Examination vsd

A
  • Evidence of biventricular enlargement (L VH & RVH)
  • Systolic thrill on lower left sternal border.
18
Q

Auscultation VSD

A

1- Murmer ofVSD:
- pansystolic.
- on lower left sternal border.
- propagate all over the heart.
- Harsh (louder if small).

2- Pulmonary area: Accentuated P2 & soft systolic murmer indicate pulmonary hypertension.

3- Apical: Soft mid diastolic murmer may be heard due to relative mitral stenosis.

19
Q

Investigations VSD
cxr
ecg

A

1- Chest X-ray:
-Large VSD ~Cardiomegaly with biventricular enlargement (LVH & RVH).
& increased pulmonary vascular markings (Plethora).

2- ECG:
-Large VSD ~ Biventricular enlargement (LVH & RVH) & LAD.

20
Q

Treatment VSD
medical
surgical

A

1- Medical:
-Control heart failure (diuretrics, digoxin, vasodilaters).
- Prophylaxis against infective endocarditis.
- Antibiotics for chest infections.
-Follow up with ECG & Echo to confirm spontaneous closure.

2- Surgical:
-Types:
a- Palliative: Pulmonary artery banding (less favoured).
b- Direct closure of the defect.

21
Q

indication for surgery vsd

A

a- Symptomatic large defects.
b- Growth failure uncontrolled medically.
c- Pulmonary hypertension.
d- Supracristal VSD (aortic cusp may herniate inside resulting in aortic regurge).

22
Q

Prognosis VSD

A

30-50% of small defects (especially muscular) close spontaneously within I 51 2-years.

23
Q
A