Acute Visual Loss Flashcards

1
Q

Important info to get from pt history

A

Is the visual loss transient or persistent; monocular or binocular?
Over what time frame did it occur (rapidly, hours, days, weeks, etc.)?
Pt’s age, race, medical conditions?
Pain associated w/ the loss?

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2
Q

Media opacities

A

irregularities of the clear refractive media; cornea, anterior chamber, lens, vitreous; won’t cause an APD but can physically alter the pupil

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3
Q

corneal edema: appearance, cause, implications

A
  • dulling of the reflection of incident light off the cornea
  • ground glass appearance
  • caused by increased IOP or damage to corneal tissue by dystrophies, trauma or surgery
  • can cause acute infections or corneal inflammation
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4
Q

acute angle closure glaucoma: symptoms, tx

A
  • a true emergency!
  • S&S = intense pain, fixed pupil, very red eye, corneal edema, very high IOP
  • tx by laser iridotomy
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5
Q

hyphema: definition, causes

A
  • blood in the anterior chamber
  • usually traumatic but occurs spontaneously in rubeosis (neovascularization of the iris)
  • neovascularization can be caused by diabetes, tumors, status post surgery, chronic inflammation
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6
Q

hypopion

A
  • WBCs forming pus in the anterior chamber
  • usually from infection
  • can be sterile (accumulation of inflammatory debris)
  • could cause cornea to look like it’s “melting”
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7
Q

lenticular changes

A
  • cataracts usually develop slowly
  • advanced cataracts may acutely cause inflammation or glaucoma (rare in the US)
  • sudden changes in blood sugar or electrolytes may cause lens edema and shift in refractive error – yay diabetes!
  • everyone gets cataracts to a certain degree; the leading cause of preventable blindness worldwide
  • appears as a yellowing/browning of the lens; could look like cinder in the middle of the red reflex
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8
Q

vitreous hemorrhage

A
  • reduction in vision from opaque blood blocking light; will have poor red reflex
  • could be caused by trauma or conditions causing neovascularization (diabetes mellitus or branch retinal vein occlusions)
  • could be associated w/ a subarachnoid hemorrhage
  • if you see the lens is clear but you can’t see the fundus suspect a vitreous hemorrhage
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9
Q

retinal detachment

A
  • pt c/o flashing lights, floaters, curtain/cloud in vision W/O pain
  • could only present w/ flashes of light and new onset floaters
  • could cause an APD, might see elevated retina w/ folds
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10
Q

Macular disease

A
  • painless reduction in VA
  • most commonly caused by subretinal neovascularization from age related macular degeneration
  • # 1 cause of blindness in US (and other developed countries)
  • may or may not have APD
  • central metamorphosia (wavy vision)
  • wet and dry versions; can slow progression from dry to wet w/ nutritional supplements
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11
Q

retinal vascular occlusion

A
  • transient monocular loss is amaurosis fugax (something blocked blood flow temporarily and then it cleared - vision loss occurs then goes back to normal/near normal)
  • if pt is 50+ suspect carotid circulation as cause i.e. an embolus from the carotid
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12
Q

central retinal artery occlusion

A
  • sudden painless usually complete visual loss (NLP)
  • permanent damage to ganglion cells and inner retina
  • early findings include narrowed arterioles; cell death occurs causing white retinal edema after several hours
  • will see a “cherry red spot” in the fovea (like you would see in Tay-Sachs)
  • true emergency!
  • tx by trying to change the intravascular pressure/resistance: attempts to dislodge the embolus by compression or paracentesis of anterior chamber
  • 50% 5-year mortality rate; the CRAO doesn’t cause death but the underlying cardiovascular condition does
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13
Q

retinal vein occlusion

A
  • painless subacute loss of vision
  • caused by HTN, DM, vasculopathies, AV nicking
  • optic disc swells, venous engorgement, cotton wool spots, diffuse retinal hemorrhages; “blood and thunder” fundus
  • can tx w/ ASA QID; some forms may need laser tx; can lead to neovascular glacoma
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14
Q

optic neuritis

A
  • inflammation of optic n.
  • can be associated w/ MS, intracranial mass, aneurysm
  • reduced VA, often +APD, contrast sensitivity and visual field diminished
  • usually pain w/ eye movements
  • get MRI to r/o tumor, aneurysm, etc.
  • if MS you can tx w/ high dose methylprednisolone 1g IV over 45 minutes QID x 5d –> DO NOT tx w/ PO steroids always use IV first
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15
Q

papillitis v. papilledema

A

papillitis = optic n. inflammation
papilledema = swelling of optic disc from increase ICP; has to be BILATERAL finding; caused by tumor, hemorrhage, malignant HTN, etc.
- test question setup = 20-30yof overweight w/ dull headache w/ unremarkable PE except for papilledema –> answer = pseudotumor cerebri (idiopathic increased ICP)

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16
Q

ischemic optic neuropathy

A
  • optic n. edema causing painless vision loss
  • 2 types = arteritic and nonarteritic
  • arteritic = medical emergency; giant cell (temporal) arteritis; can wipe out the optic n. very quickly
  • nonarteritic = not caused by inflamed a./vessel; not an emergency; anterior ischemic optic neuropathy
17
Q

giant cell (temporal) arteritis

A
  • systemic disease rarely in pts when pt moves mouth/talks the jaw tightens
  • if suspected get a sed rate and CRP
  • if sed rate over 60 tx w/ 100mg corticosteroids and an H2 blocker or PPI to protect the stomach and get a temporal a. biopsy for a definitive dx - don’t wait to tx
18
Q

traumatic optic neuropathy

A
  • hx of trauma, vision loss, +APD, +/- other ocular damage
  • shearing of vascular supply to optic n.
  • compression - hematoma in optic canal, optic n. avulsion, bone fragment impinging n.
  • poor prognosis: tx w/ high dose steroids, could require orbital or neurosurgery
19
Q

homonymous and bitemporal hemianopia; what the losses are and the most common location of injury causing them

A

homonymous hemianopia = same side loss in both visual fields; usually lesion behind the optic chiasm
bitemporal hemianopia = loss of the right half of the OD field and left half of the OS field (tunnel vision); usually lesion at optic chiasm i.e. pituitary tumor

20
Q

cortical blindness

A
  • aka central or cerebral blindness
  • extensive b/l damage to cerebral visual pathways
  • complete loss of vision, normal pupillary light response and normal fundus exam
21
Q

functional (nonphysiologic) visual loss

A
  • preferred term over hysterical or malingerer
  • hysteria = extreme emotional stress; malingering = seeking secondary gain, often obnoxious and uncooperative
  • PE = none other than psychophysical
  • vision change may sound bizarre when described
  • can do your own placebo effect experiment = “magic drop” test