acute neuro pt 1 Flashcards
acronym for remembering cranial nerves sensory vs motor
some say marry money but my brother says big brains matter more
normal ICP
5-15 mm Hg
> 20 emergency
CPP =
MAP-ICP
60-80
s/s of IICP
cushing’s triad - bradycardia, resp depression, hypertension
decreased LOC, HA, pappiledema, pupil changes/coma - herniation, hypo/hyperventilation
strategies to decrease ICP
ventriculostomy
elevate HOB
mannitol/hypertonic saline
glucocorticoids
sedation
hyperventilation (CO2 30-35)
pressers to ensure CP >60
gold standard for ICP monitoring
ventriculostomy
type of hydrocephalus most common in adults
normal pressure hydrocephalus
d/t trauma, aneurysm, meningitis, Paget disease
3 cardinal features of hydrocephalus
gait difficulty (not responsive to levodopa)
cognitive disturbance
urinary incontinence
hydrocephalus diagnostics & mgmt
B12, thiamine, electrolytes
MRI - ventriculomegaly in absence of sulcal enlargement
CSF removal
mgmt via shunting (VP or VA)
first line for seizures
loraz 2mg (up to 10)
diazepam 5-10 mg q 5-15 min
lacunar infarct
small (<5 mm), non cortical ischemic stroke
associated w poorly controlled DM/HTN
cerebral infarct
thrombotic stroke - atherosclerosis causes embolus
may develop over hours, pt likely has history of TIAs
embolic stroke - particles of debris (not atherosclerosis) block arterial access to brain
CVA diagostics/mgmt
ABCs / glucose
non contrast head CT stat
H&P - last known normal?
NIHHS
reverse coagulopathy (Coumadin)
permissive HTN - hold home antiHTN, lytics <185/110, no lytics <220/110
contraindications to thrombolytics
> 4.5 hours
unknown LNK
recent bleeding
MI
surgery
thrombocytopenia
SBP>185
CVA interventions
thrombolysis if candidate
endovascular intervention within 6 h
ASA 325 ASAP or 3 hr post thrombolysis
ASA & plavix 21 days, then just aspirin
ICH treatment
d/c anticoagulation/antiplatelets, reverse if possible
fever mgmt
manage glucose
isotonic IVF, osmotic diuretics,
ICP drains
BP - treat <160/90 labetalol/nicardipine
SAH presentation, diagnostics
“worst HA of my life” progress to obtundation
HCT with angiography to ID source
LP with xanthrochromic fluid
SAH treatment
phenytoin to prevent seizures
tx with surgical clipping
BP mgmt - DBP <100 nimodipine
AVM
congenital vascular malformations that result from localized poorly developed part of the primitive vascular plexus and abnormal arteriovenous communications that are without intervening capillaries
sudden onset SAH and ICH
recurrent seizures, HA, misc complaints
CT for symptoms, then MRI arteriogram
treatment - neurosurgery excision of AVM, embolization