Acute chronic periodontitis Flashcards

1
Q

What is acute periodontitis?

A

clinical conditions of rapid onset that involve the periodontium, or associated structures, and may be characterized by pain or discomfort, tissue destruction and infection

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2
Q

Examples of acute Periodontology disease?

A

Gingival abscesses

Periodontal abscesses

Nectrotizong periodontal diseases

Hermetic gingivostomatitis

Pericoronoitis

Endo-periodontal lesions

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3
Q

what is a periodntal abscess?

A

Is a localised accumulation of pus located within the gingival wall of the periodontal pocket/sulcus

Resulting in a significant tissue breakdown

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4
Q

primary signs and symptoms of a periodontal abscess?

A

Ovoid elevation in the gingiva

Along the lateral part of the lot

Bleeding on probing

Pain

Suppuration on probing

Deep periodontal pockets

Increased tooth mobility

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5
Q

is tissue break down associated with a periodontal abscess fast or slow?

A

Rapid

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6
Q

primary signs and symptoms to aid diagnosis of a periodntal abscess?

A

Ovoid elevation in the gingiva along the lateral part of the tooth

Bleeding on probing

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7
Q

Secondary signs and symptoms to aid diagnosis of a periodontal abscess?

A

pain

Suppuration on probing signs

Deep periodontal pockets Increased

Increased tooth mobility

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8
Q

are teeth with associated periodontal abscess vital?

A

Yes likely to be vital

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9
Q

tx for periodontal abscess?

A

Incise/drain

Debridement

Extraction

+/- antimicrobial therapy f warranted

+/- referra; t specialist of bone deformity present or deep pockets may require surgical periondianl care

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10
Q

why do periapical abscesses occur?

A

Pulp necrosis

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11
Q

acute vs chronic abscess?

A

Acute non-draining and painful

Chronic drains

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12
Q

2 types of people with periodntal disease?

A

Pre-existing periodontal pocket

Non- pre- existing periodontal pocket

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13
Q

what happens f you don’t clean to the ful depth of the poet?

A

The coronal section heals and traps in the apical area with the infection still present

Causing a periodontal abscess

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14
Q

if the periodontal abscess doesn’t heal, what are differential diagnosis?

A

Tumour lesions

Squamous cell carcinoma

Pyogenic granuloma

Osteomyelitis

Cysts

Self-inflicted gingival injuries

Sickle cell anemia

Abscesses after surgical procedures

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15
Q

what is acute necrotising conditions?

A

Are a groups of periodontal diseases witha characteristic clinical phenotype (papilla necrosis, bleeding and pain)

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16
Q

Periapical abscess vs periodntal abscess?

A

Unlike periapical lesion, teeth will likely be vital, no pain to retinal compression/percussion but may have pain on horizontal pressure

17
Q

NPDs infectious or non-infectious?

A

Infectious

Based on host response to oral flora

18
Q

risk factors for NPD?

A

Host response. Immune dysfunction

  • pre-existing periodontal condition
  • HIV/AIDs
  • malnutrition
  • psychological stress and sleep deprivation
  • smoking and alcohol consumption
  • poor OHI
  • young age (15-34)
  • previous NPDs
  • local factors such as restorations
19
Q

what are the 4 zones in NP/NG?

A

Superficial bacterial layer
Neutrophil rich zone - bodies response
Necrotic zone
Spirochete zone - studies have shown this to be a key marker

20
Q

bacteria related to spirochete?

A

T. denticola

21
Q

How to diagnose NP?

A

Clinical observation:
- necrosis/ulcer o the interdental papillae, gingival bleeding, halitosis, pain, and rapid bone loss

  • secondary signs/symptoms are pseudomembrane formation, lymphadenopathy, and fever
22
Q

how to diagnose NG?

A

Primarily include nercrosis/ulcer of the interdental papillae, gingival bleeding and pain

Secondary signs/symptoms include halitosis, pseudomembrane formation, regional lymphadenopathy, fever

23
Q

necrotisin stomatitis?

A

Soft tissue necrosis that extends beyond the gingiva, with bone denudation that may occur through alveolar mucosa

Large areas of osteitis and formation of bone dequestrum

Soft tissue necrosis that extend beyond the gingiva

24
Q

who is likely to have necrotising stomatitis?

A

systemically compromised patients (hiv/aids, severe malnutrition)

Must consider other systemic conditions, such as vesicular-bulbous diseases

Consider referral to OM

25
tx for NPDs?
Occur eth rapi breakdown of tissues 4 phases 1. Acute 2. Re-evaluation 3. Management of pre-existing condition 4. Management of residual lesions 1) Acute - perform daily (2-4 days), careful ultrasonic debridement. - use of CHX MW 2x (or 3% hydrogen peroxide 1:1) daily. Avoid brushing direct n wounds - in severe cases with systemic involvement consider systemic antibiotics 2) Re-evaluation - strict adherence to oh and continue debridement - close monitoring (daily of possible) until complete debridement of all deposits - a cause from their mental health, refer to gp 3) Management of pre-existing condition - essentially complete stage 1 of the BSPerio tx algorithm - initial cause related therapy 4) Management of residual lesions/sequale - correct tissue malformations cased by dense - may need specialist referral (major black triangles)
26
What is the ab to take for NPD?
400 mg for 3 times a day 3 days