Acute and Critical care Flashcards

1
Q

Side effects following critical care

Post Intesive Care Syndrome = pICS

A
Physical deconditioning - Exhausted for 3 months 
HA infections
Functional and cognitive impairment
DElerium
PTSD and anxiety
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2
Q

When do you do an emergency Circithyrotomy

A

When you cant intubate, cant ventillate (or oxygenate)

- Cannula first and if that fails, Surgical

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3
Q

C Spine check?

A
Canadian C spine
> 65?
Dangerous mechanism?
Paraesthesia in extremities 
Unable to actively roatate neck 45 degrees?

If yes –> radiography

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4
Q

GCS

A

E4, V5 M6

Eyes
4 = open
3 = opens to command
2 = open to pain

Voice
5 = orientated
4 = confused 
3 = talking confused / inappropriate words
2 = grunts
1 = no verbal response 
Motor
6=  follows command
5 = localises
4 = withdraws from pain 
3= flex
2= extends
1 = no motor response
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5
Q

Monroe KEllie Doctrine

A

Volume inside cranium is fixed
Blood, CSF and brain and in equilibrium
Increase volume in one must be compensated by decrease volume in another

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6
Q

Treatment of brain swelling

A
Correct hypotension 
MAP >90 
Treat sezirues 
Elevate head 30 degrees
Mannitol to reduce brain swelling 
Corticosteroids if oedema
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7
Q

Cerebral perfusion pressure =

A

Mean arterial pressure - ICP
As ICP increases, CCP decreases
The response is to increase BP
causes Cerebral ischaemia, midline shift, hydrocephalius and herniation

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8
Q

Invasive cardiac monitoring?

A

Central venous pressure = measured through a central venous line aka PICC line if in arm (can also take bloods and deliver fluids and meds)

Invasive Arterial Pressure = art line/ a line
- can take ABGs from and monitor real BP

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9
Q

Hypovolaemic shock

A

Bleeding and dehydration = cool and pale

Give more fluid

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10
Q

Distributive

A

Warm, vasodialted –> Give Vasopressin eg Noradrenaine

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11
Q

Cardiogeneic

A

Pump failure, vascular defects, rhtym abnormalities

Give IONOTROPES

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12
Q

Obstrucive

A

Eg massive PE- treat obstructive cause

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13
Q

Adrenaline

A

B1 = HEART RATE
increased Heart rate and blood pressure
central only

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14
Q

Noradrenaline

A

A1 agonist= increase blood pressure - peripheral vessels
central only
vasopressor –> give in distributive shock

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15
Q

Dobuatamine and Dopexamine

A

B1 agonist = increase heart rate - ionotrope

and decrease BP

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16
Q

Salubtamol

A

b2 receptrors = lung

17
Q

Phenylepherine and Metaraminol

A

A1 = increases blood pressure

decreases HR

18
Q

Goal directed therapy in ICU

A
Normalise Lactate 
Urine Output >0.5ml/kg/hr
MAP >65mmHg 
Central venous sats >70%
Central venous pressure 8-12
19
Q

Atropines affect on heart

Adenonsines affect on heart

Amiodarone

A

increase HR

decrease HR

Amiodarone - rhythm + for tachys

20
Q

Dead space

Shunt

A

dead space = ventilated lung without perfusion

Shunt = blood but no ventillation

21
Q

type 1 respiratory falure treatment ICU

A

CPAP
- 02 delivered with positie pressure
decreases work of breathing

22
Q

Type 2 resp failure ICU - hypercapnea

e.g copd retainers, neuromuscular

A

bipap = NIV which is basically CPAP + additional pressure to support inspiration

23
Q

If resp failure is refractory to other treatment / low GCS …

A

Mechanical ventillation and circulatory support
Requires endotracheal tube + sedation
Try get off ventillator day 5/6 and by day 8 consider Tracheoatomy to help with weaning

24
Q

Renal replacement therapy indicaitons

A

1) Acute- potassium >6.5 despite medical mx or acidosis <7.2
2) Within 24 hrs urea >40 and creatinine >400
3) CKD stage 5

25
Q

Indications for plasma exchange

A

used as therapy for some acute immune mediated conditions

- guillian barre, myasethenia gravis, SLE, vasculitis

26
Q

Signs of liver failure

A

Encephalopathy
Bleeding (INR >1.5 as liver not making clotting factors)
Jaundice, N+V, ascites

27
Q

Diagnosing death - cardiac death

A
  1. Establish CPR failed or DNACPR
  2. Observe for 5 minutes for an absence of a central pulse on palpation and absence of heart and breath sounds on auscultation or ECG
  3. Confirm brainstem has been damaged by examining for lack of pupillary light reflexes, corneal and motor response to suborbital pressure
    - Time of death is stated when tests are completed

diagnosis needs 2 clinicians, one must be consultant

Must be no doubt its reversible or brainstem death