Acute abdominal pain Flashcards

1
Q

How does ruptured AAA present?

What are some signs on examination of ruptured AAA?

A

Presentation of ruptured AAA

  • sudden onset severe pain radiating to back, abdomen, groin
  • collapse

Signs on examination
-rigid abdomen and PULSATILE, EXPANSILE MASS
-shock-low BP, high HR, poor perfusion (absent femoral pulses)
-signs of ecchymosis (bleeding under skin)
• Cullen’s sign
• Grey-Turners sign

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2
Q

What size dilatation is a AAA categorised as?

When is unruptured AAA big enough to be operated on?

A

AAA is a permanent dilatation >3cm

<5.5cm = watch and wait 
-regular USS monitoring
      (3-4.5 do yearly USS
      (4.5-5.5 do 3 monthly USS)
-modify risk factors (stop smoking, control HTN (ACEi), DM, chol (statin), Aspiring potentially)

> 5.5cm OR >1cm/yr OR symptoms = surgery

  • endovascular stent repair
  • or laparotomy with prosthetic graft
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3
Q

What is most common site for AAA?

A

Infrarenal (just below kidneys)

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4
Q

Risk factors for AAA?

A
  • Over 50 years old (age related changes in elastin + collagen + smooth muscle)
  • Male(screened)
  • Risk factors for developing atheroma in the aorta:
  • Hypertension
  • Smoking
  • Male
  • Hyperlipidaemia
  • Obesity
  • Genetics
  • Marfan’s
  • Collagen disorders(Elher’s Danos syndrome)
  • Syphilis
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5
Q

Investigations for AAA?

A

Investigations for AAA
- Urgent USS is diagnostic for AAA - but if it has already ruptured should not waste time doing this

- USS, ECG AND BLOODS: 
• FBC 
• group and save and a cross match for 10U or more
• amylase 
• LFTs 
• Us and Es
• Clotting
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6
Q

How should you manage AAA rupture?

A

AAA management
-CALL THEATRE ASAP (vascular surgeon with experienced anaesthetist)
ABCDE
- High flow oxygen 15L/min via non rebreathe mask
- 2 wide bore cannulas in antecubital fossa
- Bloods - emergency cross matching 10U, FBC, clotting, amylase, U+Es, LFTs
- Give fluids in major hypovolaemia but avoid excess(aim for systolic <100)
-IV morphine(prevent tachycardia and hypertension)- IV antiemetics - 50mg cyclizine
-IV prophylactic antibiotics -METRONIDAZOLE AND CEFUROXIME

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7
Q

Symptoms of appendicitis?

A

symptoms appendicitis

  • INITIAL GENERALISED abdo pain which localised to the RIF just a few hours later
  • Nausea and vomiting
  • Anorexia
  • Constipation or diarrhoea
  • Frequent urination (irritation-can mimic UTI)
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8
Q
Signs of acute appendicitis?
What are: 
Rovsigns sign
Copes sig/obturator 
Psoas sign
A

Signs of acute appendicitis

  • Tachycardia, tachypnoea, pyrexia
  • Tenderness at McBurney’s point (2/3rds umbilicus to ASIS)
  • Guarding (localised peritonitis)
  • Rovsigns sign -pain in RIF when pressing on LIF
  • Cope/obturator sign - pain on flexion and internal rotation of R thigh
  • Psoas sign- get patient to lie on LHS and extend their right leg> PAIN
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9
Q

What causes acute appendicitis?

What should some differentials for acute appendicitis be?

A

appendicitis CAUSES

  • infected faecolith
  • gut microorganisms arrive
  • causes immune cells to kick off
  • inflammation

appendicitis DIFFERENTIALS

  • diverticulitis
  • gastroenteritis
  • acute flare of IBD
  • ectopic pregnancy
  • ovarian cyst
  • mesenteric ischemia (always think about this)
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10
Q

What investigations should be done in acute appendicitis?

A

Appendicitis investigations

  • mainly clinical
  • CRP!!!!!!

Bedside

  • urinalysis -can be normal (rule out UTI). It is abnormal 50% of the time because bladder is close to appendix- white cells (inflammation)
  • pregnancy test (rule out ectopic)

Bloods

  • CRP!!
  • FBC (increased neutrophils)
  • Us Es/LFTs/amylase/crossmatch

Imaging
-not required clinical is enough
can do CT or USS if unsure

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11
Q

How should we manage acute appendicitis in ED?

A

Appendicitis management
• Obtain IV access and resuscitate if necessary
• Commence fluids if dehydrated
• Antibiotics (metronidazole and cefuroxime) (guidelines)
• IV analgesia and anti-emetics

• Refer to general surgeons for prompt laparoscopic appendectomy

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12
Q

What causes cholecystitis?

A
  • Stone or sludge obstruction of the neck of the gall bladder
  • Causing INFLAMMATION of the gall bladder
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13
Q

What are some signs and symptoms of cholecystitis?

A

Symptoms of cholecystitis

  • Continuous epigastric or RUQ pain
  • May radiate to right shoulder
  • Worse when eating (especially fatty foods)
  • Vomiting

Signs
- Fever- the presence of fever distinguishes from just biliary colic
- local peritonism- tender o/e with possible GB mass with guarding and some rigidity
○ MURPHY’S SIGN 2 fingers over RUQ and ask patient to breath in . (only +ve if the same test in the LUQ does not cause pain)

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14
Q

What is the most common cause of cholecystitis?

A

Gall stones

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15
Q

What investigations should be done in a patient with potential cholecystitis?

A

Cholecystitis investigations

  • FBC (high WCC)
  • LFTs (marginal elevation of ALP, bili and ALT possible - but highly elevated levels might be more suggestive of bile tract obstruction)
  • USS might show thick walls and shrunken GB, might see stones and might see a dilated CBD
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16
Q

How should we initially manage cholecystitis in ED?

A

Cholecystitis management

  • NBM
  • analgesia
  • IV fluids
  • IV antibiotics (guidelines) (cefuroxime)

Refer for laparoscopic cholecystectomywithin 7 DAYS

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17
Q

What is ascending cholangitis and how does it differ from cholecystitis?

Treatment for cholangitis?

A
Cholangitis
-This is similar to cholecystitis PLUS JAUNDICE
-It's bad! likely septicaemia!
-Infection of the gall bladder 
-CHARCOT'S TRIAD:
 •RUQ pain 
 •Fever and rigors 
 •Jaundice 

Treatment
-antibiotics (cefuroxime and metronidazole) (guidelines)

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18
Q

What are some symptoms and signs in a patient with a bowel obstruction?

A
  • Vomiting, nausea, anorexia.
    If the obstruction is long-term then the faecal contents behind the obstruction might start to ferment and this can cause FAECAL VOMITING
  • Constipation
  • Colic
  • Abdominal distension
    o/e distension, rigidity, absent or tinkling bowel sounds
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19
Q

What difference will there be between the presentation in patients with small and large bowel obstructions?

A

Small bowel

  • central distention (less severe than large bowel)
  • bilous vomiting will be predominant early symptom
  • colic pain +++

Large bowel

  • flank distention
  • constipation is early sign
  • more constant pain
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20
Q

What are some common causes of bowel obstruction? (dynamic and adynamic) give examples of both

A
Dynamic -> mechanical obstruction
 •hernias 
 •fecal impaction
 •volvulus 
•adhesions
 •TB 

Adynamic -> paralytic ileus (inactivity of the bowel)
•recent abdo surgery
• electrolyte imbalance e.g. loss of K+ after surgery or hypercalceamia
•spinal injury
•pseudo obstruction

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21
Q

How would you approach a patient presenting with ?Bowel obstruction in ED

A

Bowel obstruction managment (emergency)

  • ABCDE approach
  • ABDO EXAM and end pieces
    • stool
    • hernia-femoral more likely to cause obstruction
    • rectal exam (1st line)
    • urine-cathertise
  • BLOODS
    • FBC
    • UsEs
    • LFT
    • clotting
    • group and save (surgery)
    • glucose
    • amylase
    • calcium,
  • Imaging:
    • AXR -bigger than 3 (small),6, (caecum) 9 (large)
    • CXR erect (pneumoperitomium))
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22
Q

What specific treatment can be done for bowel obstruction?

A

Bowel obstruction treatment
ABCDE management
-Drip and suck
-IV fluids
-NG ryles tube (reduce contamination of peritonium)
-IV antibiotics
-Correct electrolytes
-Analgesia and antiemetic (if necessary)
-Catheter (monitor fluid input AND output)
-PPI
-Refer to surgery (exploratory laparotomy for patients that are haemodynamically unstable or signs of ischaemia/necrosis)

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23
Q

What is diverticulosis/diviticular disease/diverticulitis?

In which diverticula does diverticulitis most often occur?

What are some complications of diverticulitis?

A

Increase in lumen pressure>out pouching
Diverticulosis= asymptomatic
Diverticular disease=symptomatic
Diverticulitis (infected)

In the diverticula of the descending/sigmoid colon (LIF)

Complications of diverticulitis

  • abscess (swinging fever, WCC)
  • perforation -surgery
  • haemorrhage
  • fistulae
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24
Q

What are some common symptoms of acute diverticulitis?

What are some common signs on examination would you see?

A

Acute diverticulitis

  • SEVERE PAIN IN THE LEFT ILIAC FOSSA (normally relieved by defecation)
  • Fever
  • Nausea
  • Altered bowel habit
  • Flatulance
Examination
• Febrile 
• Tachycardia (pain) 
• Tenderness and guarding in LIF
• Localised or generalised peritonism
• Diarrhoea ± Bleeding (melaena)
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25
Q

What investigations should be done in patients with diverticulitis?

What score should you use to classify severity?

A

BLOODS:
•FBC (wcc raised)
•ESR and CRP raised

IMAGING

  • CT abdo with IV contrast (can also look for abcess)
  • Erect CXR to look for perforation
  • Can also do USS: thickened bowel walls and pericolic collections

Hinchey score – severity classification

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26
Q

How should diverticulitis be managed in the ED?

What investigation must you AVOID in acute diverticulitis?

A

Acute diverticulitis

  • Mild attacks an be treated as an outpatient with oral co-amoxiclav
  • If they are in considerable abdo pain or cant tolerate oral fluids consider admission for IV fluids, IV co-amoxiclav and analgesics (NBM)

DO NOT DO A COLONOSCOPY IN AN ATTACK OF DIVERTICULITIS(risk of perf)

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27
Q

What are risk factors for ectopic pregnancy?

What are some clinical features of ectopic pregnancy?

A

Ectopic pregnancy

RISK FACTORS

  • endometriosis
  • intrauterine contraceptive devise
  • pelvic inflammatory disease
  • pelvic surgery/edhesions
  • PMH ectopic
  • IVF
  • progesterone only pill

FEATURES

  • Collapse/syncope
  • Recurrent lower abdominal pain (haemorrhage may cause pain to radiate to shoulder)
  • Vaginal bleeding
  • Missed period
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28
Q

What investigations should be done with suspected ectopic pregnancy?

What investigation should be AVOIDED?

A

Ectopic pregnancy
-Trans-vaginal USS
- Urine pregnancy test and serum bHCG in blood
-Bloods: FBC, crossmatch to determine rhesus status
think about other causes (UsEs, LFT, amylase, Ca, glucose)

Do not do bimanual examinationin ectopic pregnancy

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29
Q

How should we manage ectopic pregnancy?

A

Ectopic pregnancy

  • Fluid resuscitation if shocked
  • Depends on size and certain factors (B-hCG and FHR)
  • Refer to gynaecology
  • Watch and wait while measuring Hcg OR Methotrexate (if diagnosed later) OR surgery (heamodynamically unstable)
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30
Q

How might a miscarriage present?

A

Miscarriage presentation

  • Acute vaginal bleeding with or without pain/cramping.
  • Sometimes bleeding can be profuse and cause hypotension/collapse/faintness
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31
Q

What might cause a miscarriage?

A

Miscarriage causes

  • Might be spontaneous or might be repeated (infertility)
  • Chromosomal abnormalities of fetus
  • Uterine malformations (fibroids or cervical changes) - this is more likely to be the cause if the miscarriage is in the second or third trimester
  • Drugs (isotretinoin)
  • Trauma
32
Q

What investigations should be done in a patient with potential miscarriage?

A
Investigations post miscarriage 
1st line: pregnancy test 
2nd line: USS (determine if unteruterine) 
3rd line: 
Intrauterine: look at cervix (see if open) 
Not seen in uterus: beta hcg levels 
         -suboptimal rise=ectopic
         -declining=miscarriage 

Others

  • FBC, Rhesus status
  • Pelvic examination
33
Q

How should miscarriage be managed in the ED?

A
  • Resuscitate if needed
  • Treat pain
  • Refer to gynaecology and obstetrics for examination
  • Counselling
34
Q

How might an ovarian cyst present in an acute setting?

A
  • Lower abdominal pain, can be a dull ache or a sharp pain
  • May radiate lower back
  • Exacerbated by intercourse
  • Bloating or swelling in the abdomen
  • Urinary symptoms if pressing on bladder
  • Very heavy/irregular periods
    -Difficulty getting pregnant

Ruptured cysts
• Severe sudden sharp pain
• Vomiting
• Vaginal bleeding
• Shock

35
Q

What are some important causes of ovarian cyst?

A

Cysts are either:
FUNCTIONAL
- very common and form as part of the menstrual cycle
- usually these are harmless, short-lived and asymptomatic

PATHOLOGICAL

  • much less common
  • they are an abnormal growth and while the majority are benign some can be cancerous
  • can be associated with endometriosis/PCOS
36
Q

What investigations should be done in people with suspected ovarian cyst?

A

Investigations for ovarian cyst

  • Pregnancy test (blood and urine) to exclude ectopic
  • USS
  • CA125 for ovarian cancer (high levels doesn’t necessarily mean cancer)
  • Urinalysis to exclude UTI
37
Q

How should we initially manage ovarian cyst in the ED?

A
Ovarian cyst management 
-Resuscitation if shocked 
-No treatment required in most cases  
- If the woman is post-menopausal then she might have a slightly higher risk of cancer and so might suggest monitoring over a year
- Consider surgeon referral (lap cystectomy) if ovarian cysts are:
• Large >5cm
• Symptomatic
• Cancerous
• Signs of torsion
38
Q

What are some clinical features of acute pancreatitis?

Signs on examination?

A

Initially: -severe epigastric pain (relived by sitting forward) with nausea and vomiting
Then: pain radiates to back (involvement of peritoneum)

Signs O/E:

  • Tachycardia + hypotension(fluid shift)
  • Mild fever
  • Jaundice if gallstones is cause (may have RUQ pain)
  • Epigastric tenderness/local peritonism (rigidity/guarding )
  • GREY-TURNER’S (flank) or CULLEN’S (central)
  • Oliguria
  • Hypocalceamia (lipase breaks down fat that bind to free calcium)
39
Q

What actually is acute pancreatitis?

What are some causes of acute pancreatitis?

A

Acute pancreatitis

  • Self-perpetuating pancreatic inflammation (enzyme-mediated auto-digestion)
  • Odema and fluid shift (to gut, peritoneum, retroperitoneum) → HYPOVOLAEMIA (worsened by vomiting)
GET SMASHED 
G - Gall stones (38%)
E - Ethanol (35%)
T - Trauma 
S - Steroids 
M - Mumps 
A - Autoimmune 
S - Scorpion venom 
H - Hyperlipidaemia, hypercalcaemia, hypothermia 
E - ERCP and emboli 
D - Drugs
40
Q

What investigations should we do in someone in whom we’re suspecting acute pancreatitis?

A
ACUTE PANCREATITIS 
Bloods
-ABG (critically ill patient)  
-FBC (↑WCC)
-UsEs (↑Urea)
-LFT and clotting 
-↑CRP (marker of severe)
-↑Amylase (3x upper limit-can also be normal)_
-↑Lipase (more specific and sensitive) 
-↑Glucose 
-↑lactate (
-Ca2+ ( may be ↑)

Imaging
-CT (standard- rule out pnuemoperitoneum)
-AXR
(no psoas shadow-high retroperitoneal fluid)
(sentinel loop sign-solitary air-filled dilatation)
-CXR (rule out other causes e.g. bowel perf)
-USS (if gallstones)

Sentinel loop sign (solitary air-filled dilatation) indicates gall stone ileus
3. MRCP – determine if gall stone obstructing CBD

41
Q

How should we initially manage someone with acute pancreatitis in the ED?

A

Acute pancreatitis is managed by surgeons

  1. Stabalise
    - Admit to ward and monitor vital signs
    - IV access (FLUIDS FLUIDS FLUIDS)
    - If vomiting, NBM (NJ tube)
    - Analgesia (IM pethidine-avoid morphine-sphincter of oddi contraction)
    - Antibiotics if necrotic
  2. Treat cause
    - hypercalceamia
    - gallstones (ERCP to relive obstruction within 72 hours. call general surgery for cholecystectomy)
  3. Now what?
    - Glasgow imrie score within 48 hours (assess severity)
    - 3+ then transfer to ICU
42
Q

What are some clinical features of someone presenting with peptic ulcer disease ?

A
PUD: *Sharp, easily located epigastric pain*
(50% assymptomatic)
S-Epigastric, can easily point to where it is 
O-
C-sharp
R- 
A
-heart burn
-taste changes (more metallic) 
-bloating/early satiety
ALARMS
-Anemia (tiredness, palpitations) 
-Loss of weight 
-Anorexia 
-Recent onset 
-Melena/heamatemisis(not a lot)-may relieve. 
-Swalling problems 
T-
E
-food RELIEVES DUODENAL 
-food EXACERBATES GASTRIC 
S-
43
Q

What are some important causes of peptic ulcer disease?

A

Duodenal ulcer is the most common

  1. Helicobacter pylori - bacteria which produces ammonia to neutralise stomach acid which is toxic to epithelial cells
  2. DRUGS: NSAIDs, steroids SSRIs

Others

  • Smoking
  • Alcohol
  • Stress
  • Aggravating food
  • Elderly (gastric)
44
Q

What investigations should we do in patients with peptic ulcer disease?

(what is diagnostic -wat do you do for it)

A

BEDSIDE
-ECG to rule out cardiac pain

BLOODS

  • FBC (↓Hb – indicates anaemia, refer)
  • Us and Es

SPECIAL TESTS -

  1. Non-invasive H.Pylori test
    - 1st line is carbon urea breath test
    - or stool antigen
  2. Endoscopy (if DYSPHAGIA or 55+ with ALARMS signs)
    - DIAGNOSTIC: Upper GI endoscopy (∆) – exclude malignancy and determine extent of oesophagitis
    - Stop PPI 2 wks before
    - Multiple Biopsies and histology (H.Pylori CLO)
    - recheck after treatment to see if malignant
45
Q

How should we manage peptic ulcer disease initially?

A
  1. Stop drugs (NSAIDS)/lifestyle and review in 4 weeks
  2. If no improvement then check for H pylori
    -If H pylori -ve then give PPI (4 weeks)
    -If H pylori +ve with NSAID use> PPI first then irradication
    - If H pylori +ve and no NSAID use> TRIPPLE THERAPY
    •PPI + amoxicillin, + either clarithromycin/metronidazole
    for 1 week

(if pen allergic, give PPI, metronidazole and clarithromycin

  • *in ED just test for H pylori (skip step 1)
  • *only irradiate H pylori twice-if still +ve seek expert advice
46
Q

What are some clinical features of PID?

What is a sign on examination?

A

This is an inflammation of the upper part of the female reproductive tract (ovaries, fallopian tubes, uterus and surrounding pelvis)

  • Pelvic and lower abdominal pain
  • New or different discharge
  • Pain during sex
  • Painfull/irregular periods
  • Dysuria

On examination
-cervical motion tenderness

47
Q

What are some important causes of PID?

A
90% associated with sexually transmitted infections:
- Gonorrhoea 
- Chlamydia 
- Bacterial vaginosis increases the risk of developing PID

Organisms
• Chlamydia trachomatis
• Neisseria gonorrhoea
• Mycoplasma hominis
• Ureaplasma urealyticum
48
Q

What investigations should we do in patients with PID?

A
  • FBC (high WCC)
  • Pelvic exam
  • Pregnancy test
  • Swabs of cervix and vagina> look for STIs
  • Urinalysis-exclude UTI
  • USS (abbess)
49
Q

How should we consider managing PID?

A
  • Gynaecological review
  • Pain killers
  • Empirical antibiotics
50
Q

What is pathophysiology of renal colic?

What are some clinical features of renal colic?

A

REAL COLIC

  • intermittent severe pain in flank/loin/groin (testes-not not tender)
  • ureteric peristalsis

Symptoms

  • suprapubic pain
  • anorexia
  • nausea and vomiting
  • sweating

urine problems
-change in smell/ colour (heamaturia)/amount (anuria/dysuria)

51
Q

What is the most likely cause of renal colic?

A

Kidney stones (haematuria and raised inflammatory markers)

52
Q

What investigations should we do in someone with renal colic?

A

Bloods

  • FBC (wcc)
  • UsEs
  • CRP
  • Calcium

Urine

  • urine dip (blood and leucocytes due to inflammation)
  • pregnancy test

Urgent Imaging within 24 hours

  • CT non contrast
  • if young or pregnant do KUBUSS
53
Q

How should we consider managing a patient with renal colic in ED?

A

< 5mm stones

  • NSAIDS (PO diclofenac or IV ketorolac)
  • Fluids- important to keep pt hydrated-help to pass stones
  • AB if infection

> 5mm

  • alpha blocker to relax (tamsulosin)
  • or surgical treatment
54
Q

What are some clinical features of urinary tract infections?

A

-suprapubic pain (loin/flank pain>pylonephritis)
- URINARY SIGNS:
○ Urgency
○ Frequency
○ Feeling of incomplete emptying
○ Haematuria
○ Dysuria
○ Smelly urine
- Fever, rigors and tenderness are also suggestive of an upper UTI
- Confusion in the elderly-easily missed

55
Q

What are some important causes of UTIs?

A
  • Very common in women, especially those who are sexually active
  • Common in the elderly
  • Gram negative organisms: E.col
    ○ Consider immunocompromisation in pt not in high risk group
56
Q

What investigations should we consider in someone with a UTI?

A
  • Urine dip and culture
  • Pregnancy test
  • Bloods: FBC, U+;E
57
Q

How can we manage a UTI in the ED?

A
  • Antibiotics (follow guidelines, trimethoprim, nitrofurantoin, pivmecillinam)
    ○ Cefuroxime if UUTI
  • Encourage high intake oral fluids, IV if pt is unwell
58
Q

What is binary colic?
How does it present?
Investigations?
Treatment?

A

Bilary colic
-Gallstones temporarily obstruct cystic duct, then are passed into common bile duct

-Will cause RUQ and might have some jaundice. However no fever as the GB is NOT inflamed

-Investigations (rule out other conditions) 
○Urinalysis 
○ Bloods 
○CXR 
○ECG 
-Treatment 
○NBM 
○analgesia 
○IV fluids 
○elective laparoscopic cholecystectomy
59
Q

How would liver enzymes look in obstructive jaundice (post hepatic)

A

Obstructive jaundice/ post hepatic jaundice

  • ALP +++
  • GGT+++
  • Unconjugated bilirubin+ Conjugated bilirubin+++
  • ALT and AST normal or +
60
Q

Complications of peptic ulcer disease?

A

Complications of peptic ulcer disease

  • UPPER GI BLEED if erosion of vessels(Haematemesis/Melaena)
  • PERFORATION-can cause acute abdomen (epigastric pain → generalised rigidity (CXR – pnuemoperitoneum)

PYLORIC STENOSIS– due to scarring of the duodenum
Weight loss + Projectile Vomiting

61
Q

What is the severity score for acute pancreatitis?

How does this effect your managment?

A
Acute pancreatitis 
-Do the Glasgow imrie score within 48 HOURS (assess severity)
P- PaO2 <8
A- >55
N- Neutrophilia >15x109
C- Calcium <2mmol 
R- Renal function urea >16 
Enzymes -LDH (>600) and AST (>200)
A-Albumin <32
Sugar (blood glucose >10mmol) 

-If 3+ then transfer to ICU

62
Q

Complications of acute pancreatitis? (early and late-3)

A
Complications of acute pancreatitis
EARLY 
-shock (FLUIDS) 
-renal failure (FLUIDS) 
-sepsis 
-DIC 
-ARDS (acute lung injury due to systemic illness) 

LATE

  1. Pseudocyst (mass and persistent high amylase)-cystogastrostomy if symptoms dont resolve
  2. Pancreatic abscess-infected pseudocyst (drain)
  3. Necrotising pancreatitis: Imipenem if > 30% necrosis
63
Q

Complications of diverticulitis? (how do you treat)

A

Complications of diverticulitis
1. Haemorrhage (sudden and painless)
→ Transfusion

  1. Abscess- swinging fever, ↑↑WCC/Neut, localising signs e.g. boggy rectal mass
    → antibiotics – Co-Amoxiclav
    → image guidedpercutaneous drainage (rectally)
  2. Perforation - Ileus, peritonitis ± shock
    → Laparotomy w/ Hartmann’s procedure (temporary colostomy + partial colectomy)
64
Q

Differential for aneurysm?

A

Pseudoaneurysm/false aneurysm

-blood vessel wall is injured and the leaking blood collects in the surrounding tissue

65
Q

What would you see on x-ray for bowel obstruction?

How do you know if small/large bowel?

A

Dilated bowel

  • Small bowel dilatation if >3cm
  • Large bowel dilatation if >6cm
  • Cecal dilatation if > 9cm
  • no gas distal
  • *multiple fluid levels is abnormal
  • **riglers sign(both sides of bowel wall can be seen)= perf and pneumoperitoneum

Small bowel obstruction - valvulae conniventes are visible (lines completely cross the bowel)

Large bowel obstruction - haustral lines visible (just on edges)

66
Q

What are some causes of small bowel obstruction?

A
  • ADHESIONS most common (prior abdo surgery, TB)
  • Incarcerated hernias
  • Crohn’s disease
67
Q

What are some causes of large bowel obstruction?

A
  • TUMOURS most common e.g. colon carcinoma
  • Constipation
  • Volvulus
  • Diverticular stricture
68
Q

How can you tell the difference between mechanical and functional bowel obstruction?

A

‘functional’ or dynamic:

  • absent bowel sounds
  • pain tends to be less

mechanical/dynamic:
-bowel sounds tinkling

69
Q

Best imaging for determining cause of bowel obstruction?

A

CT scan (with IV contrast)

70
Q

What is a simple/closed loop/strangulated bowel obstruction and how can you tell difference?

A

Simple: just one obstruction point, no vascular compromise

Closed loop: obstruction at 2 points (sigmoid volvulus-risk of perf)

Strangulated: compromised blood supply

  • PERITONISM!!! (sharper more localised pain)
  • ILLER THAN YOU’D EXPECT
  • ↑WCC
71
Q

How can you tell the difference between functional and mechanical bowel obstruction?

A

‘functional’ or dynamic:

  • absent bowel sounds
  • painless
  • no flatus (large amount of gas in rectum)

mechanical/dynamic: bowel sounds tinkling

72
Q

What can be done for palliative treatment for obstructive large bowel malignancys?

A

Endoscopic stenting – useful in palliative + elderly pts.

73
Q

What would you see on imaging for sigmoid volvulus (2 different images)

A

CT abdo-pelvis with IV contrast - dilated sigmoid colon with ‘whirl sign’

AXR - coffee-bean sign arising from LIF

74
Q

What is sigmoid volvulus?

Who does it normally occur in?

A

Large bowel twists on mesentery- can cause strangulated obstruction

(tends to occur in elderly, comorbid, constipated patient)

75
Q

Treatment of sigmoid volvulus?

A
  • Flatus tube/sigmoidoscopy to decompress

- If that doesnt work: sigmoid colectomy