ACGIH BEIs / TLVs Flashcards

1
Q

TLV definition

A

Airborne concentrations of chemical substances that represent conditions under which it is believed that nearly all workers may be repeatedly exposed, day after day, over a working lifetime, without adverse health effects

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2
Q

What does a TWA measure?

A

Concentration for a conventional 8-hour workday and 40-hour workweek, to which it is believed that nearly all workers may be repeatedly exposed, day after day, for a working lifetime without adverse effects

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3
Q

What does a STEL measure?

A

A 15-minute TWA exposure that should not be exceeded at any time during a workday, even if the 8-hour TWA is within the TLV-TWA

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4
Q

What does a STEL protect workers from?

A

Workers can be exposed to the STEL without suffering from 1) irritation 2) chronic or irreversible tissue damage 3) dose-rate dependent toxic effects, or 4) narcosis

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5
Q

How often can you be exposed to the STEL?

A
  • Exposure should always be less than 15 minutes
  • Should occur no more than 4x per day
  • There should be at least 60 minute between successive exposure in this range
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6
Q

TWA SL

A

The concentration on workplace equipment and facility surfaces that is not likely to result in adverse effects following direct or indirect contact

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7
Q

Ceiling value

A

The concentration that should not be exceeded during any part of the working exposure

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8
Q

Peak exposure - when are they applicable

A

When a STEL does not exist for a substance (even if there’s a TLV-TWA)

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9
Q

Peak exposure rules

A

May exceed 3x the TLV-TWA for no more than 15 minutes at a time, for no more than 4 occasions spaced out one hour apart during a work day

May never exceed 5x the TLV-TWA when measured as a 15 minute TWA

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10
Q

When are ceiling values more appropriate to use rather than TLV-TWAs?

A

When substances are fast acting and whose TLV is more appropriately based on the concentration associated with this particular response

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11
Q

What are BEIs?

A

They are guidance levels for evaluating biological monitoring results. BEIs generally represent the levels of determinants that are most likely to be observed in specimens collected from healthy workers who have been exposed to chemicals to the same extent as workers with inhalation exposure

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12
Q

Biological monitoring can assist with:

A
  • determine absorption via the skin or gastrointestinal route
  • assess body burden
  • reconstruct past exposure
  • detect nonoccupational exposures among workers
  • test the efficacy of PPE and engineering controls
  • Monitor work practices
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13
Q

What are the specimens used in BEIs?

A

Urine, blood, or exhaled air

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14
Q

Prior to shift sampling time (BEI)

A

16 hours after exposure ceases, but before any exposure on sampling day

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15
Q

Prior to last shift sampling time (BEI)

A

Prior to last shift of a workweek

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16
Q

Increase during shift sampling time (BEI)

A

Requires pre and post shift sample collection

17
Q

End of shift sampling time (BEI)

A

As soon as possible after exposure ceases

Carbon monoxide
Organophosphates

18
Q

End of workweek sampling time (BEI)

A

After 4 or 5 consecutive working days with exposure

19
Q

Discretionary/non critical sampling time (BEI)

A

At any time

Lead
Cadmium

20
Q

For urine sample, creatinine levels must be within what range to be valid?

A

0.3 g/L < X < 3 g/L

21
Q

Cadmium determinant / sampling time (BEI)

A

Cadmium in urine | not critical (sampling time)
Cadmium in blood | not critical (sampling time)

22
Q

Carbon monoxide determinant / sampling time (BEI)

A

Carboxyhemoglobin in blood | end of shift (sampling time)
Carboxyhemoglobin in end-exhaled air | end of shift (sampling time)

23
Q

Mercury determinant / sampling time (BEI)

A

Mercury in urine | prior to shift (sampling time)

24
Q

Parathion determinant / sampling time (BEI)

A

Total p-Nitrophenol in urine| end of shift
Acetylcholinesterase in red blood cells | end of shift

25
Q

Western red cedar TWA

26
Q

All other species of wood TWA

27
Q

Oak and beech carcinogenicy

28
Q

Birch, mahogany, teak, walnut carcinogenicity

29
Q

Western red cedar carcinogenicity