abgs Flashcards

1
Q

abg use

A

gives best representation of pH and PaCO2
used to assess acid=base status and determine adequacy of oxygenation and ventillation

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2
Q

vbg

A

PaCO2 higher
so pH change and PO2 not accurate

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3
Q

PaCO2 normal

A

35 - 45
resp parameter

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4
Q

HCO3- normal

A

24 - 29
metabolic parameter, reported as CO2 on chm panel, calculated in ABG

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5
Q

primary event

A

problem that initiates acid-base imbalance -> hyper/hypovent, v/d, etc

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6
Q

primary disorder

A

results from primary event -> resp acidosis, met alkalosis, etc

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7
Q

compensation mechanisms

A

phys process that adjusts pH back to normal, lungs v kidneys
lungs = H2CO3, respiratory, not prolonged compensation
kidneys = HCO3, metabolic

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8
Q

metabolic alkalosis

A

too much bicarb or not enough carbonic acid
primary = high HCO3, compensation = high CO2 to decrease pH
give H2 blockers, PPI to retain H

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9
Q

metabolic aklalosis: causes

A

causes: too much baking soda, prolonged v (stomach acid loss), NG suction, diuretics (esp loop), aklaseltzer -> hypoK bc K goes into cell and H comes out

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10
Q

metabolic aklalosis: cm

A

CNS over excitability, confusion, tremor, muscle cramp, paresthesias (tingling of fingers and toes), coma, n/v/d, resp dep -> hold in to CO2, cardiac cm
restlessness followed by lethargy, dysR (tachy), compensatory hypovent, confusion (decreased LOC, dizzy, irritable), hypoK

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11
Q

respiratory alkalosis

A

H2CO3 (carbonic acid) deficit in ecf
hypervent = primary event, blow off CO2, can also happen with ventilation
give rebreather

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12
Q

respiratory alkalosis: causes

A

hypervent, ventilation, increased metabolic demands (fever, sepsis), meds, acute anx, hypoxia, PE or lung disease, CNS lesions, vent settings

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13
Q

respiratory alkalosis: cm

A

CNS overexcitability
tachypnea (deep and rapid, hypervent), light headed, confusion, blurred vision, paresthesia, hyperactive reflexes, seizure, coma
tachy, decreased or normal BP, hypoK, lethargy and confusion, light headed, n/v

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14
Q

resp acidosis

A

acute or chronic
hypovent = primary event -> CO2 retained (H+)
acute = resp arrest, chronic = COPD or oversedation
H2CO3 excess in ecf (too much acid/H+)
ABG low pH, CO2 high, HCO3- normal in acute (no time for kidneys to compensate)
HCO3- high and pH normal in chronic bc kidneys have time to compensate
put on vent, dont really give sodium bicarb

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15
Q

resp acidosis: causes

A

cardiopulm arrest, head injury, narcotics/sedatives, anesthesia, pain and abd distention (cant take deep breath)
pulm distress = acute asthma, CPOD exacerbation, pna, resp failure, drug OD, atelectasis
airway obstruction, chest wall deformities, NM problems bc muscles not strong enough (ALS, guillain-barre)

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16
Q

resp acidosis: cm

A

CNS dep causing hypovent and dyspnea, resp distress, shallow resp, anesthesia, HA, restless, confusion, tachy, arrhythmias (hypoerK), decreased LOC, stupor, coma
decreased BP with vasodilation, drowsy, dizzy, disorientation, muscle weak, hyperreflexia

17
Q

metabolic acidosis

A

HCO3 (bicarb) deficit in ecf, excess acids added or bicarb loss
rapid acting insulin, sodium bicarb

18
Q

metabolic acidosis: causes

A

renal fail
fistulas - F+E loss
DM - type 1 - DKA
lactic acidosis - not enough O2
prolonged d - relative increase in acid d/t decreased HCO3
starvation - usually fat for E, ketosis
shock
cardiac arrest

19
Q

metabolic acidosis: cm

A

CNS: lethargy, drowsy, confusion, tremor, muscle cramp, paresthesia
hypoT, hyperK, deep breathing (kussmal resp - DKA)

20
Q

resp acidosis: w/o compensation

A

low pH
high CO2
normal HCO3

21
Q

resp acidosis: full compensation

A

pH normal
high CO2
high HCO3

22
Q

resp alkalosis: w/o compensation

A

high pH
low CO2
normal HCO3

23
Q

resp alkalosis: full compensation

A

pH normal
low CO2
low HCO3

24
Q

met acidosis: uncompensated

A

low pH
normal CO2
low HCO3

25
Q

met acidosis: full compensation

A

pH normal
low CO2
low HCO3

26
Q

met alkalosis: uncompensated

A

high pH
CO2 normal
high HCO3

27
Q

met alkalosis: full compensation

A

pH normal
CO2 high
HCO3 high

28
Q

uncompensated

A

pH abn
acid or base abn

29
Q

partially compensated

A

pH abn
acid and base abn (bc compensation kicking in)

30
Q

compensated

A

pH wnl -> neutralized but not corrected acid/base balance, acid or base components are abn but balanced, arrows in same direction

31
Q

corrected

A

pH wnl, all acid/base parameters wnl
COPD never reach