ABG's Flashcards

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1
Q

Assessment and Care of Patients with Acid-Base Imbalances

A
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2
Q

What is pH

A

˜balance of acids and bases in body fluids

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3
Q

What is the normal Arterial pH balance

A

7.35 to 7.45

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4
Q

What is the normal Venous pH balance

A

7.31 to 7.41

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5
Q

What is the normal pCO2

A

35-45 mmHg

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6
Q

What is the normal HCO3

A

22-26 mEq/L

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7
Q

What pH levels can be fatal

A

˜pH <6.9 or >7.8 usually fatal

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8
Q

Respiratory system more sensitive to which imbalance

A

˜acid-base changes; can begin compensating in seconds to minutes

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9
Q

Kidneys are more powerful in what?

A

result in rapid changes in ECF composition; fully triggered for imbalance of several hours to days

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10
Q

What are the types of Acid-Base Imbalances

A

˜Metabolic acidosis
˜Respiratory acidosis
˜Metabolic alkalosis
˜Respiratory alkalosis

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11
Q

Acid-Base Mnemonic—ROME

A

˜Respiratory

˜Opposite

Alkalosis ↑ pH ↓ PaCO2

Acidosis ↓ pH ↑ PaCO2

˜Metabolic

˜Equal

Acidosis ↓ pH ↓ HCO3

Alkalosis ↑ pH ↑ HCO3

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12
Q

Laboratory Assessment:
Metabolic Acidosis

A

˜pH <7.35

˜Bicarbonate <21 mEq/L

˜Pao2 normal

˜Paco2 normal or slightly decreased

˜Serum potassium high

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13
Q

Respiratory Acidosis

A

˜Respiratory function is impaired, causing problems with O2 and CO2

˜Retention of CO:

Respiratory depression

Inadequate chest expansion

Airway obstruction

Reduced alveolar-capillary diffusion

˜Hallmark of respiratory acidosis:

Decreased Pao2 with rising Paco2

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14
Q

Laboratory Assessment:
Respiratory Acidosis

A

˜pH <7.35

˜Pao2 low

˜Paco2 high

˜Serum bicarbonate variable

˜Serum potassium levels elevated (if acute acidosis)

Serum potassium levels normal or low (if renal compensation present

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15
Q

Acidosis: Patient-Centered Collaborative Care

A

˜History

˜CNS changes

˜Neuromuscular changes

↓ Muscle tone, deep tendon reflexes

˜Cardiovascular changes

Early: ↑ Heart rate, cardiac output changes

Worsening: Hyperkalemia; ↓ heart rate; T wave peaked and QRS widened; weak peripheral pulses; hypotension

˜Respiratory changes
Kussmaul respiration
˜Skin changes (metabolic and respiratory acidosis)
Warm, dry, and pink (vasodilation)
˜Psychosocial assessment

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16
Q

Interventions: Metabolic Acidosis

A

˜Hydration

˜Drug therapy

Insulin to treat DKA

Antidiarrheals

Bicarbonate (only with low serum level)

17
Q

Interventions: Respiratory Acidosis

A

˜Focus is on improving ventilation and oxygenation, maintaining patent airway

˜Drug therapy

Bronchodilators

Anti-inflammatories

Mucolytics

˜Oxygen therapy

˜Pulmonary hygiene

˜Ventilation support

Prevention of complications

18
Q

Metabolic Alkalosis

A

˜Base excess – Excessive intake bicarbonates, carbonates, acetates, citrates

˜Acid deficit – Prolonged vomiting, excess cortisol, hyperaldosteronism, thiazide diuretics, prolonged NG suction

˜Hallmark of base excess acidosis:

ABG result with ↑ pH and ↑ bicarbonate level with normal O2 and CO2 levels

19
Q

Respiratory Alkalosis

A

˜Hyperventilation – Anxiety, fear, improper vent settings, stimulation of central respiratory center due to fever, CNS lesion, salicylates

˜Hallmark of respiratory alkalosis:

ABG result with ↑ pH coupled with low CO2 level

O2 and bicarbonate usually normal

20
Q

Alkalosis: Patient-Centered Collaborative Care

A

˜Assessment (same for metabolic and respiratory alkalosis)

˜Hypocalcemia

˜Hypokalemia

˜CNS changes – Positive Chvostek’s and Trousseau’s signs

˜Neuromuscular changes – Tetany-twitching of facial muscles

˜Cardiovascular changes

Respiratory changes

21
Q

Interventions: Alkalosis

A

˜Prevent further losses of hydrogen, potassium, calcium, chloride ions
˜Restore fluid balance
˜Monitor changes, provide safety
˜Modify or stop gastric suctioning, IV solutions with base, drugs that promote hydrogen ion excretion

22
Q
A