ABG Flashcards
(32 cards)
purpose of ABG’s
to assess acid-base status and determine adequacy of oxygenation and ventilation
ABG: pH
balance of H+
7.35-7.45
< = acidic
> = basic
PaCO2
35-45 mmHg
respiratory parameter
carbonic acid dissolves into CO2 and H2O
partial pressure of CO2 in ARTERY
HCO3
22-26 mEq/L
metabolic parameter
*measured HCO3 is reported as CO2 on a chemistry panel (serum CO2)
venous blood gas:
pH
PvCO2
pH: 7.31-7.41
PvCO2: 41-51 (partial pressure of CO2 in VEIN)
basic metabolic panel (BMP):
serum CO2 =
HCO3 level
22-26 mmol/L
primary EVENT
PROBLEM that initiates imbalance
*hypoventilation
*hyperventilation
*V/D
primary DISORDER
what RESULTS from the primary event
*respiratory acidosis (hypovent)
*metabolic acidosis (hypervent)
+ “” alkalosis
compensation mechanisms
physiologic processes that adjust the pH back to normal range
*if lungs are problem - kidneys compensate
*if kidneys are problem - lungs compensate
metabolic cause
HCO3 (bicarbonate) level changes d/t METABOLIC alterations (KIDNEY)
respiratory cause
H2CO3 (carbonic acid) level changes d/t RESPIRATORY alterations (LUNGS)
4 types of imbalances are a result of
changes in ventilation
increase/decrease in CO2
changes in H+ or bicarbonate ions
ROME
respiratory - opposite
metabolic - equal
metabolic acidosis: primary/compensation/pH
decrease HCO3
decrease CO2
decrease pH
metabolic alkalosis: primary/compensation/pH
increase HCO3
increase CO2
increase pH
respiratory acidosis: primary/compensation/pH
increase CO2
increase HCO3
decrease pH
respiratory alkalosis: primary/compensation/pH
decrease CO2
decrease HCO3
increase pH
metabolic alkalosis
too much HCO3 OR not enough carbonic acid
pH > 7.45
CO2 35-45
HCO3 > 26
causes of metabolic alkalosis
-excess baking soda/sodium bicarb
-prolonged vomiting
-NG tube (GI suctioning)
-diuretics (esp. loop)
clinical manifestation of metabolic alkalosis
CNS over excitability –> leads to fatigue
confusion
tremors
muscle cramps
paresthesias
coma
N/V/D
respiratory depression - compensatory hypoventilation
dysrhythmias (tachycardia)
hypokalemia
respiratory alkalosis
H2CO3 (carbonic acid) DEFICIT in ECF
pH > 7.45
CO2 < 35
HCO3 22-26
cause of respiratory alkalosis
hyperventilation (CO2 blown off)
increased metabolic demands (d/t fever, sepsis)
meds
acute anxiety
hypoxia
PE or lung disease
CNS lesions
ventilator settings
clinical manifestations of respiratory alkalosis
CNS over excitability
tachypnea + hyperventilation (deep and rapid)
light headedness
confusion, blurred vision
paresthesia
hyperactive reflexes
seizures
coma
tachycardia
hypokalemia
respiratory acidosis
excess carbonic acid/ H2CO3
*hypoventilation – retained CO2
pH < 7.35
CO2 > 45
HCO3 22-26