- ABDOMINAL PRESENTATIONS - Flashcards

1
Q

Outline the clinical manifestations of GIT bleeding

A
  • Abdominal pain
  • Nausea & vomiting
  • Jaundice
  • Ascites
  • Explosive diarrhoea
  • Haematemesis
  • Malena
  • Haematochezia
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2
Q

Outline the clinical manifestations of a bowel obstruction

A
  • vary according to the location of the obstruction
  • pain
  • colicky cramps (more sever the higher the obstruction)
  • vomiting (upper obsruction)
  • distention (the lower the more distended)
  • altered bowel sounds
  • intitially high pitched, hyperactive then hypoactive and absent
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3
Q

Outline the clinical manifestations of Ulcerative colitis

A
  • bloody diarrhoea (up tp 20 times a day)
  • abdo pain (lower abdo cramping, generalised - perferetion)
  • mucous in stool
  • fever
  • weight loss
  • tachycardia
  • dehydration
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4
Q

Outline the clinical manifestations of Chron’s disease

A
  • abdo pain
  • diarrhoea
  • nausea and vomiting
  • weightloss
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5
Q

Outline the clinical manifestations of cholecystitis

A
  • Pain
  • Epigastric to RUQ, severe and constant
  • May radiate to (R) scapula area / back
  • Abdo rebound tenderness and guarding
  • Nausea/vomiting
  • Fever
  • Jaundice if CBD obstructed
  • +Murphy’s sign
  • Usually no peritoneal signs unless complications
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6
Q

Outline the clinical manifestations of pancreatitis

A
Range from mild to severe
Airway/breathing:
– tachypnoea
– hypoxaemia
– pleural effusion
– atelectasis 
Circulation:
– hypotension
– tachycardia
– oliguria, renal failure
– coagulation abnormalities
Disability:
 - Low grade fever
- Hyperglycaemia
- Nausea and vomiting
- Hypoactive or absent bowel sounds
- Jaundice
- Raised WCC
- Discoloration of abdominal
wall (Cullen’s sign, Grey Turner sign)
- Abdominal pain / distention:
– LUQ / epigastric
– radiates to back
– sudden onset
– severe, deep, piercing,
continuous
– worse with eating
– guarding
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7
Q

Outline the clinical manifestations of hepatitis

A
  • jaundice
  • fatigue
  • abdo pain
  • arthralgia
  • anorexia
  • nausea and vomiting
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8
Q

Outline the clinical manifestations of liver failure

A
  • jaundice
  • ascites
  • coagulopathy
  • hhepatic encephalopathy
  • haemodynamic changes
  • electrolyte disturbance
  • renal failure (hepatorenal failure)
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9
Q

Outline the clinical manifestations of peritonitis

A
  • decreased bowel sounds
  • abdo pain
  • anorexia
  • N,V,D
  • tachycardia
  • hypotension
  • fever
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10
Q

Outline the management of GIT bleeding

A

Airway/breathing:
– high flow oxygen
– pulse oximetry
– monitor work of breathing

Circulation:
– IV fluids
      \+/- packed cells
– cardiac monitoring
– IDC
– nil by mouth
– ECG
Disability
– GCS
– BGL
– temperature
– focused abdominal assessment
– PR exam / endoscopy
Interventions:
• NGT
• Medications
– Octreotide
– Antacids
– Histamine-2 receptor antagonists
• Cimetidine, Ranitidine, Famotodine etc.
– Betablockers
• Balloon tamponade ( see slides as for variceal
bleeding)
• Endoscopic control of bleeding
– Adrenaline injection
– cautery
• Surgical interventions
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11
Q

Outline the management of a bowel obstruction

A
  • history
  • physical exam
  • x-rays
  • NGT
  • ?enema if in large bowel and no suspected perferation
  • colonoscopy
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12
Q

Outline the management of ulcerative colitis

A
  • FBE, EUC, serum protein and electrolytes
  • stool sample (examine for blood and pus then culture
  • drug therapy
  • scopes
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13
Q

Outline the management of chron’s disease

A
  • in acute phase rest GI tract; fasting or fluids only
  • path FBE,EUC,Electrolytes
  • dietary management\
  • specialist follow-up
  • drug therapy
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14
Q

Outline the management of cholecystitis

A
  • path (esp amylase, EUC, billiruben, urine)
  • ECG (to exclude cardiac involvement)
  • IV crystalioid
  • antiemetics
  • ?NGT for gastric decompression
  • ?IVABS if infection is suspected
  • narcotic analgesics
  • intake/output
  • vitals
  • Sx laparotomy or laproscopic cholecystectomy
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15
Q

Outline the management of pancreatitis

A
• Airway / breathing
– monitor respiratory rate
– monitor oxygen saturation
– treat hypoxaemia with oxygen
– maximise lung expansion
– chest x-ray
• Circulation:
– haemodynamic monitoring
– IV access
– early aggressive intravenous hydration (
recommendation in ACG Guidelines, 2013)
– ? Lactated ringers the preferred choice of crystalloid
– manage losses – anti-emetics
– correct electrolytes
– +/- CVC, IDC
– +/- inotropes 

• Disability:
– analgesia – opiates, PCA, epidural anaesthesia

• Nutritional support
– in mild AP – oral feeding start immediately ( if no N & V )
– low fat solid diet
– in severe AP – enteral nutrition is recommended
– NG and NJ enteral feeding are both comparable in efficacy
and safety

  • Monitor BGL
  • Monitor for signs of infection
    – antibiotics – no longer recommended as routine
    – proton pump inhibitors
    – Histamine H2-receptor antagonists
    – insulin
Investigations:
– pathology (lipase)
– chest & abdominal x-ray
– abdominal U/S
– abdominal CT

Surgery:
– ERCP +/- cholecystectomy
– laparotomy

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16
Q

Outline the management of hepatitis

A
  • treat dehydration
  • antipyretics (mindful of paracetamol)
  • analgesia
  • antiemetics
  • cease ETOH
  • decrease fatty foods
  • antiviral therapy
17
Q

Outline the management of liver failure

A
  • Path
  • ultrasound
  • airway protection (vomiting)
    • pharmacology (vasoactive drugs)
  • sepisis common
  • monitor renal function
  • lifestyle factors
  • ?AWS
18
Q

Outline the management of peritonitis

A
  • antibiotics
  • peritoneal lavage
  • laparotomy
  • analgesia
  • nasogastric decompression
  • antiemetics
  • volume support
  • management of sepsis
19
Q

Discuss the pathophysiology of pancreatitis

A

Pancreas:

  • inflammation of the pacreas
  • back up of pancreatic secretions
  • activation and release of enzymes (trypsin, chymeotrypsin, lipase, elastase)
  • autodigestion
  • Vascular damage, coagulative necrosis, fat necrosis and formation pseudocysts
  • oedema
  • ischaemia and necrosis

Systemically:

  • Pro-inflammatory cytokines & vasoactive peptides released
  • Activation leucocytes
  • Injury to vessel walls
  • Coagulation abnormalities
  • third spacing of fluid in the peritoneum
  • Vasodilation
  • Hypotension
  • Shock
  • Multisystem failure
  • SIRS
20
Q

Discuss the pathophysiology of Liver failure

A
  • injury to liver (hepatitis, ETOH, drugs, poisoning, obesity, DM, metabolic and genetic disorders)
  • inflammation
  • immune system stimulation
  • increase in oxygen free radicals
  • decrease in hepatic perfusion
  • hepatic atrophy
  • hepatic fibrosis (cirrhosis)
  • portal hypertension
  • fuild shift
  • ascites, peripheral oedema, oesophageal varices
  • hepatic decompensation
  • renal failure
21
Q

Discuss the pathophysiology of GIT bleeding

A
Aetiology
Upper GI bleed:
– peptic ulcers
– Mallory- Weiss tear
– drug induced erosions
– gastric cancer
Lower GI bleed:
– haemorrhoids
– diverticulitis
– colonic polyps
– colon cancer
– colitis
– ulcers
– gastritis

Patho:

  • injury to the epithelial layer of the GI tract
  • GIB
  • hypovolaemia
  • compensate with peripheral vasoconstriction and increased CO
  • Renal Na and water retention
  • blood flows to vital organs
  • further bleeding or inadequate compensation
  • hypovolaemic shock
  • insufficient blood to organs
  • MODS
  • death
22
Q

Discuss the pathophysiology of a Bowel Obstruction

A

. - obstruction occurs

  • lack of rapid movement of HCI and chyme
  • bacteria flourish as a result
  • fluiid, gas and intestinal products accumulate proximal to the obstruction
  • distal bowel collapses
  • distention reduces absorption
  • increased pressure leads to increassed capillary permeability and leak of fluid into the peritoneal cavity
  • this results in hypotesion and hypovolaemic shock
23
Q

Define cholecyctitis

A
  • Inflammation of the gallbladder due to obstruction of the CBD by gallstones
  • Causes distension of the gallbladder
  • pressure decreases blood flow
  • bacterial invasion / infection : (E.coli, streptococcus or salmonella)