A's Exam Review MCQ 2011 Flashcards

1
Q
Where is:
- umbilicus level
- aortal bifurcation level
- depth of aorta average
How many cm below aorta bifurcation is Lt common illiac vein located
A
  • L3-4
  • usually L4 - 5, caudal if obese
  • 6 cm
  • 3-6cm caudal, umbilicus always cephalad to that point
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2
Q
  1. Where is Palmer’s Point?
  2. Lateral Trochar placement
    - 2 vessels and 2 nerves to avoid?
    - steps to avoid injury
A
  1. 3 cm below Lt costal margin, midclavicular
    2.
    - inf and superior epigastrics, illioinguinal, illiohypogastric
    - place lat ports superior to asis, 6+ cm lateral to midline
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3
Q

At what level does an epidural work for 1st and 2nd stage labour.

A

1st stage contractions: T10-L1

2nd stage: must extend S2,3,4 late stage 1 and stage 2

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4
Q

DDx Vaginal Bleeding in Peds (Life threatening and common)

A

Life threatening:
- Malignancy (endodermal sinus tumor or rhabdomyosarcomas) usually present < 3yrs
- Trauma (abuse, water jets, recreational injury, stradle inj)
Common:
- Neo withdrawal PVB
- Foreign Body
- Infection (GAS and Shigella a/w PVB specifically but also have discharge)
- lichen sclerosus (purpura and hematomas)
- urethral prolapse
Uncommon:
- exogenous estrogen
- precocuscious puberty
- hypothyroidism

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5
Q
  1. CI to regional anesthesia
  2. ThromboProphylaxis
    - time between last does and initiation
    - time between last dose and removal of catheter
    - time between neuraxial and restarting anticoag
A

1.

  • coagulopathy
  • increased ICP
  • infection to lower back
  1. Thromboprophylaxis:
    *insertion:
    UH no delay (up to 10 000 then 4h)
    LMWH: 10-12h
    * removal:
    UH 4h
    LMWH 10-12h
    *restart:
    UH 1-8h
    LMWH 6-8h
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6
Q

Where i estrogen produced?

Testosterone secretion?

A
  • E2 from granulosa cells of developing follicles and conversion of E1 (estrone to peripherally)
  • Testosterone secretion: 25 % ovary, 25% adrenals, 50% peripheral conversion of androstenedione
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7
Q

VARICELLA:

  • Incubation period
  • Does Shingles have fetal sequelae
  • Infectious period?
  • Pneumonitis: risk of getting, most risk for death in what trimester, what day of infection would it usually occur?
A
  • Incubation period 10-21 days
  • not associated with viremia or fetal sequela
  • infectious period: 48 hours before the rash until the vesicles crust over
  • 5-10% pregnant women with varicella, T3, Day 4 usually
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8
Q

Classification of Operative Vaginal delivery

A
OUTLET: 
- scalp visible at introitus without separating labia
- fetal skull has reached pelvic florr
- fetal head at or on perineum
- Direct OA, OP or  LOA/ROA 
- Rotation does not exceed 45 degrees
LOW:
- Spines + 2 or more then either - rotation < 45 or > 45 degrees
MID: Station spines 0-2cm
HIGH: NO
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9
Q

Requirements and CI to OVD

A

Prereqs:

  • vertex, engaged
  • Fully, ROM
  • exact position known
  • adequate pelvis
  • analgesia
  • bladder empty
  • informed consent
  • appropriate personel/backup
  • skilled operator
CI:
ABSOLUTE:
- no vertex or brow
- unengaged head
- not fully
- evidence of CPD
- fetal coagulopathy
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10
Q

Complications of OVD

A

Both Vacuum and forceps:
- Mat trauma
- Fetal scalp bruising/lacs, bleeding
- RARE: ICH, retinal hemorrhage, facial nerve injury, skull fracture
FORCEPS: also rarely minor ext occular trauma

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11
Q

Risks of cocaine use in pregnancy?

A

Fetal:

  • SA
  • IUFD
  • Abruption
  • Prematurity
  • IUGR

Maternal:

  • CV cocaine toxicity: HTN (dont use beta blockers! cause end organ ischemia, use hydralazine)
  • stroke
  • MI
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12
Q

Complications of Midtrimester PPROM

A
PTB
Chorio, endometritis
abruption
cord prolapse
IUFD
Retained placenta
Neo: morbidity, pulm hypoplasia, msk deformities
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13
Q

Prevalence of Pulmonary Hypoplasia in midtrimester PPROM

Mortality and predictors

A

Prevalence 9 % in this pop mortality 70-90%
Predictors:
- GA a time of PPROM (rare after 26 wks)
- Degree of oligo

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14
Q

Most common cause of hypothyroidism in pregnancy (1st world)

A

Chronic Autoimmune Thyroiditis

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15
Q

Risk of untreated overt hypothyroidism

A

PTB
LBW
SA
?Neurodevelopmental

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16
Q

Risk of untreated overt hyperthyroidism

A
PTD
LBW
IUFD
preeclampsia
cardiomyopathy --> pulm hypertension --> heart failure (8-90%)
17
Q

Treatment Hyperthyroidism

A

T1 PTU (propylthiouracil) as lower risks of birth defects, then methimazole T2/3 (risk of hepatotoxicity with PTU)

18
Q

Complications of long term beta blocker use

A

IUGR, fetal bradycardia, neo hypoglycemia

19
Q

List US findings suspicious for fetal hyperthyroidism (Trab positive complicaitons)

A
Fetal tachycardia
IUGR
goiter
accelerated bone maturation
heart failure
hydrops
20
Q

Thyroid Storm and or hyperthyroid related cardiomyopathy Treatment

A

ICU

  • Thionamides (PTU) (blocks periph conversion T4->T3)
  • HR control: BBlockers
  • Iodine
  • K Iodide
  • Lugols (blocks release of thyroid hormone)
  • Corticosteroids (dec peripheral conversion T4->T3
21
Q

DDX precocious puberty

A
Most common: Idiopathic Central Precocious Puberty (GnRH dependent)
- Intracranial lesions
Peripheral: 
- hormone secreting tumors (granulosa, sertoli/Leydig, gonadoblastoma)
- hypothyroid
- exogenous hormones
- adrenal tumor
- McCune Albrigth Syndrome
22
Q

What is McCune Albright Syndrome

A

Triad of precocious puberty, irregular cafe au lait spots and fibrous bone dysplasia.

23
Q

List 3 factors that affect MSAFP values

A
GA
Maternal Weight
DM
Fetal anomalies
Multiples
Race
Fetal viability
24
Q

Components of Apgar

A
Heart rate
Respiratory Effort
Muscle Tone
Reflex irritability
Color
25
Q

Do we use apgars as predictors of neo death/outcomes

A

NO. Not precise enough to use to Dx asphyxia (cord gases are used for this)

26
Q
  1. What is the only type of CP a/w acute interruption of blood supply
  2. Incidence of term CP
  3. Factors a/w CP
A
  1. spastic quadriplegia
  2. 2-3/1000
    • Maternal Co-morbidities
    • Multiples
    • PTB
    • IUGR
    • autoimmune, infection
    • Asphyxia
    • CNS, developmental, metabolic abnormalities
    • Substance abuse
27
Q
  1. What percentage of children with CP had a demonstrated IP asphyxia?
  2. Four essential criteria to associate CP with IP asphyxia
  3. Criteria which suggest an IP insult
A
  1. 10-20%
    • Evidence of metabolic acidosis (ph < 7, BE > 12)
    • Early onset Neo Encephalopathy (> 34wks)
    • CP type spastic quadriplegia or dyskinetic type
    • Exclusion of other etiologies (trauma, coagulopathy, genetic anomaly etc)
      3.
    • sentinal hypoxic event
    • sudden/sustained fetal bradycardia or no variability in presence of lates/variables, when pattern previously normal
    • apgars 0-3 beyond 5 minutes
    • onset of multi system involvement within 72 h of birth
    • early imaging of acute, non focal, cerebral anomalies
28
Q

Risk factors for fetal NTD

A
  • Personal/Family Hx both 1st and 2nd degree
  • GI malabsorption d/o
  • Advanced liver/renal disease
  • Pre-gestational DM, Obesity
  • Anti-epilepsy or anti-folate meds
  • Smoking, ETOH
    low SES, food restrictions
29
Q

Classification or Risk for fetal NTD and related supplementation

A

Low: 0.4mg - 1mg
Mod: (Personal or family history of NTD or folate sensitive anomalies, Mat DM, meds, GI malabsorption) 1mg
High: (Parent with personal Hx NTD, previous child with NTD) 4mg

30
Q

What anomalies are thought to be folate sensitive

A
NTD
Cardiac
 limb
cleft palate
Urinary tract
 congenital hydrocephaly