A&P for Cardiac Flashcards

1
Q

describe the pericardium

A

fibrous outer sac, serous parietal layer that minimizes friction during contraction, and visceral layer aka epicardium

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2
Q

what are the three layers of the heart

A

epi, myo, and endocardium

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3
Q

where is the tricuspid valve

A

between RA and RV

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4
Q

where is the pulmonary valve

A

between RV and Pulm Artery

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5
Q

where is the mitral valve

A

between the LA and LV

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6
Q

Without neural influence, the impulse from the SA node beats at _______ bpm (inherent rhythmicity)

A

100

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7
Q

what is the purpose of the AV node

A

delay A-V impulse until atria have had time to eject their contents into the ventricles

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8
Q

what is the inherent rhythmicity of the AVN

A

40-60 bpm

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9
Q

what is the p wave

A

atrial contraction

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10
Q

what is the QRS wave

A

ventricular contraction

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11
Q

what is the T wave

A

ventricular relaxation

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12
Q

how does the vagus nerve exert its influence over the heart?

A

parasympathetically - decreases HR

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13
Q

how do the upper thoracic nerves and rami of the sympathetic trunk exert their influence over the heart?

A

sympathetically - inc HR

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14
Q

the sympathetic receptors of the heart are primarily _________, and stimulation by ________ increases overall activity of the heart

A

beta-adrenergic; norepinephrine

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15
Q

the sympathetic receptors of the peripheral vasculature are primarily __________, and when activated they can cause systemic _________

A

alpha-adrenergic; vasoconstriction

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16
Q

describe the pressure-resistance relationship of the R side of the heart

A

low pressure system with little resistance from the pulm arteries

17
Q

describe the pressure-resistance relationship of the L side of the heart

A

high pressure system with high resistance from systemic circulation

18
Q

what is the primary determinant of oxygen delivery

A

cardiac output!

CO = SV x HR

19
Q

what are the three factors that influence stroke volume

A

preload (starling effect), afterload, and myocardial contractility

20
Q

how does preload influence stroke volume

A

increased preload = increased SV

21
Q

how does afterload influence stroke volume

A

increased afterload = decreased SV

22
Q

what is the frank-starling effect

A

increase in ventricular volume increases systolic force but excessive stretching in fluid overload impairs actin/myosin interaction thus impairing contractility

23
Q

blood pressure is a product of __________ and ____________

A

CO and TPR

24
Q

what is pulse pressure and what can it tell you about heart disease

A

pulse pressure is the difference between SBP and DBP and increased pulse pressure is a risk factor for heart disease

25
Q

where in the body do we regulate BP?

A

vasomotor center in the medulla which mediates sympathetic and vagal inputs

26
Q

what is the short term regulator of BP

A

arterial baroreceptors - if BP increase, CO and TPR decrease

27
Q

what is the long term regulator of BP

A

renin-angiotensin-aldosterone system - kidneys compensate for BP decrease by activating angiotensin 2 which leads to vasoconstriction

28
Q

water retention in the kidneys is another long term regulatory mechanism in response to low blood volume, but how does it work?

A

low blood volume stimulates posterior pituitary ADH which leads to vasopressin upregulation