9/11 - Movement Considerations II Flashcards

1
Q

Abnormal Muscle Tone

A

hypertonia, hypotonia, spasticity, rigidity

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2
Q

Paresis

A

Motor weakness/abnormal motor control

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3
Q

Dysmetria

A

lack of coordination of movement typified by the undershoot or overshoot of intended position

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4
Q

Hypometria

A

a condition of cerebellar dysfunction in which voluntary muscular movements tend to result in the movement of bodily parts (as the arm and hand) short of the intended goal

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5
Q

Hypermetria

A

a condition of cerebellar dysfunction in which voluntary muscular movements tend to result in the movement of bodily parts (as the arm and hand) beyond the intended goal

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6
Q

Involuntary movements

A

tremor, ballismus

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7
Q

Abnormal cerebellum

A

hypotonia, incoordination, intention tremor, impaired error correction affecting motor learning

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8
Q

co-activation

A

primitive/unrefined; normal in early learning

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9
Q

coordination

A

sequencing, timing, and grading of the activation of multiple muscle groups

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10
Q

incoordination

A

movements are awkward, uneven, inaccurate

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11
Q

Timing problems

A

problems initiating movement; slowed execution time; problems terminating movement

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12
Q

problems initiating movement

A

Time between the persons decision to move and the movement itself; Requires ability to overcome inertia, gravity, and antagonistic restraint; Deficits could be in ROM, force production, motivation, postural control

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13
Q

dysdiadochokinesia

A

problems terminating a movement

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14
Q

dexterity

A

ability to fractionate fine movement; depends on ability to move and respond to environment; most important in manipulative functions

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15
Q

coordination exam: non-equilibrium

A

finger to nose, heel to shin; quality of movement; subjectively graded 1-5

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16
Q

primary impairments of NM system

A

abnormal muscular tone; motor weakness/abnormal motor control; abnormal synergies; co-activation and coordination problems; involuntary movements

17
Q

Abnormal basal ganglia

A

hypokinetic disorders (bradykinesia, akinesia); rigidity; resting tremor; hyperkinetic disorders (choreiform, athetoid movements, dystonia)

18
Q

tremor

A

rhythmic and oscillatory

19
Q

dystonia

A

sustained muscle contractions, often twisting, repetitive, and abnormal

20
Q

associated movements

A

unintentional movements of one limb during the voluntary movement of another limb

21
Q

General interventions: Biomechanical

A

Sensory modalities: icing, vibration, approximation (weak evidence)

22
Q

General interventions: Neurophysiologic

A

Stretching, serial casting, casts, orthoses

23
Q

General interventions: Muscle Reeducation

A

Recruitment and type (fast or slow); Alignment issues; Fast and slow speeds
Accuracy; Intensity

24
Q

Hedman temporal sequence

A

initial conditions preparation initiation execution termination outcome

25
Q

Abnormal muscle tone

A

hypertonia; hypotonia; spasticity; rigidity

26
Q

Abnormal muscle tone treatment

A

pharm, surgical, physical

27
Q

Pharma tone treatments - botox

A

Botulinum toxin Type A
Injected directly into the muscle at the motor point
Blocks the neuromuscular junction by reducing the release of Ach
Initial effects in 3-7 days
Wearing off in 4-6 months

28
Q

Surgical tone treatments

A
Selective posterior rhizotomy
Selected roots of L2-S2
Variable results
Risk of reducing function
Tendon lengthening, release or transfer
Variable results
Generally successful for improving ROM
Risk of reducing muscle strength
Selective posterior rhizotomy
29
Q

PT tone treatments

A
Saladin Videos of 
Rhythmic rotation in sidelying
Rotation, Counter Rotation in sidelying
Rhythmic Rotation hooklying
Rhythmic Rotation LEs on ball
30
Q

Weakness and motor control interventions

A

Patient, family and entire team education
Encourage functional use
Consider stationary bicycle with vital sign monitoring
Guarded ambulation if appropriate
PREs of individual muscle groups Task specific training and strength training CVA ? Winstein, 2004
Bimanual Training
FES ? Sullivan and Hedman, 2004

31
Q

CVA secondary effects

A

Immobilization and shortened position causes atrophy
Loss of sarcomeres
Accumulation of intramuscular connective tissue
Increased skeletal muscle fat content
Degenerative changes at myotendinous junction
Increase in mechanical spindle stimulation by stretch

32
Q

Dodd systemic review

A

Systematic review that strength training programs improve strength in those with CP without increased spasticity

33
Q

Ada systemic review

A

Systematic review in those with stroke
Acute, very weak; acute weak; chronic very weak; chronic weak
Strength training is effective early after stroke,
It is not harmful (no increased spasticity)
It may be worthwhile, but adequate strengthening interventions applied to several muscles of a limb and tests need to address this in future research e.g. 10 meter walk or 9 hole peg test

34
Q

Sullivan and Hedman 2004

A

Sensory Stimulation 2 hours per day
FES to wrist extensors during a lifting task
15 minutes, twice a day
18 weeks of home exercise and 6 home PT visits

35
Q

Coordination Interventions

A

Repetitions of functional movements with increasing demands on accuracy
Assist with knowledge of results and/or knowledge of performance
Weight-bearing activities for LEs
Repetitive non-functional movements

36
Q

Timing problems - interventions

A

Give a time constraint to a task
Metronome
Time an activity
Verbal, visual, manual feedback

37
Q

Scaling problems - interventions

A

Practice grading force
Faster movements tend to be easier
Repetition of functional activity that will carry over

38
Q

Treatment (intervention) of involuntary movements

A

Reduced effort/increase efficiency
Weight bearing and approximation
Distal fixation
Limb is weighting-controversial