8 - Post-op complications 4 Flashcards

1
Q

What is an OAC?

A
  • oral antral communication
  • acute problem
  • self heal or surgical, if present for more than 6 weeks then becomes OAF
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2
Q

What is an OAF?

A
  • oral antral fistula
  • chronic problem
  • epithelial lined sinus tract
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3
Q

How do you diagnose an OAC?

A
  • size of tooth
  • radiographic position of roots in relation to antrum
  • bone at trifurcation
  • bubbling of blood
  • nose holding test
  • direct vision
  • good light and suction
  • blunt probe
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4
Q

How can you diagnose an OAC with suction?

A

Will echo if OAC present

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5
Q

How do you manage an OAC?

A
  • inform patient
  • if small, encourage clot and suture margins
  • if large or lining torn, close with buccal advancement flap
  • POI
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6
Q

What are POI for an OAC?

A
  • use a straw
  • don’t blow nose
  • sneeze with mouth open
  • may required decongestants
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7
Q

How do you manage an OAF?

A
  • excise sinus tract
  • close with a flap (buccal advancement ± buccal fat pad, palatal rotation, collagen membrane)
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8
Q

How do you confirm a root/foreign body has entered the antrum?

A

Radiographically by OPT, occlusal or PA

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9
Q

What are the different methods of retrieving a root/foreign body from the antrum?

A
  • via OAF/through socket
  • Caldwell-Luc approach
  • ENT via endoscope
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10
Q

How do you retrieve a FB via an OAF?

A
  • open fenestration with care
  • use suction
  • small curettes
  • irrigation or ribbon
  • close as OAC
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11
Q

How do you retrieve a FB with the Caldwell-Luc approach?

A
  • lift a buccal flap in the sulcus
  • drill away bone to open a window into the antrum
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12
Q

What is osteomyelitis?

A
  • infection of the bone
  • rare
  • patient is usually systemically unwell
  • most common in mandible
  • site of extraction tender
  • deep infection near IAN may alter sensation
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13
Q

Describe the development of osteomyelitis.

A
  • begins in medullary cavity in the cancellous bone
  • spreads to cortical bone
  • then spreads to periosteum and causes overlying mucosa to become red and tender
  • oedema in the enclosed space causes ischaemia of supplying BVs and soft tissue necrosis
  • blood borne defences cannot access site to combat bacteria and bacterial proliferation continues until antibiotic ± surgical therapy occurs
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14
Q

Why is osteomyelitis more common in the mandible?

A
  • maxilla has a rich arterial blood supply
  • mandible has dense cortical bone and is supplied primarily by the inferior alveolar artery
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15
Q

What are predisposing factors to osteomyelitis?

A
  • odontogenic infections
  • fracture of mandible
  • compromised host defence (diabetes, alcoholism, IV drug use, malnutrition, cancer patients)
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16
Q

How long does it take for acute osteomyelitis to be detectable on a radiograph?

A

10-12 days

17
Q

Describe the radiographic appearance of osteomyelitis.

A
  • increased radiolucency
  • either uniform or patch (moth-eaten appearance)
  • may be patches of radiopacity within the radiolucent region due to bony sequestra
  • surrounding sound bone may be more radiopaque due to inflammatory response (involucrum)
18
Q

What bacteria are involved in osteomyelitis of the jaw?

A
  • streptococci
  • anaerobic cocci
  • anaerobic gram negative rods eg fusobacterium and prevotella
19
Q

What is the management of osteomyelitis?

A
  • medical and surgical
  • check culture for antibiotics
  • investigate host defences (FBC, glucose levels)
20
Q

What is the antibiotic treatment of osteomyelitis?

A
  • penicillin is first line drug (good bone penetration)
  • 6-12 week course or via IV (can be up to 6 months for chronic cases)
21
Q

What is the surgical treatment of osteomyelitis?

A
  • drain pus
  • remove any non-vital teeth in area
  • remove sequestra
  • remove any wires/screws etc if fractured mandible
  • corticotomy (removal of bony cortex)
  • perforation of bony cortex
  • excision of necrotic bone until actively bleeding bone is reached
22
Q

What is ORN?

A
  • osteoradionecrosis
  • seen in patients who receive radiation to the head and neck for cancer
  • bone within beam becomes non-vital
  • endarteritis (reduced blood supply)
  • turnover of remaining viable bone is slow and self repair is ineffective
  • effects increase with time and dose
23
Q

What are treatments for ORN?

A
  • careful routine extraction
  • surgical extraction, alveoplasty primary closure of soft tissues
24
Q

How do you prevent ORN?

A
  • PMPR and chlorohexidine mouthwash prior to extraction
  • careful technique
  • antibiotics and chlorohexidine mouthwash
  • hyperbaric oxygen before and after extraction
25
Q

What is MRONJ?

A
  • medication related osteonecrosis of the jaw
  • caused by medications that inhibit osteoclast activity and bone renewal
  • occurs post XLA, denture trauma or spontaneous
  • ranges from asymptomatic to bone exposure
26
Q

Give examples of bisphosphonates.

A
  • alendronate
  • ibandronate
  • zoledronate
27
Q

What other drugs can cause MRONJ?

A
  • RANKL inhibitors (denosumab)
  • monoclonal antibody drugs
  • antiangiogenic drugs (bevacizumab)
28
Q

What are the risk factors for developing MRONJ?

A
  • impact on bone eg XLA
  • trauma from dentures
  • infection
  • periodontitis
  • duration of drug therapy
  • implants
29
Q

What is the management of MRONJ?

A
  • prevent invasive treatment
  • XLA can be conducted in primary care
30
Q

What is actinomycosis?

A
  • rare bacterial infection
  • bacteria has low virulence and must be treated in the area
  • erodes through tissue rather than follow fascial planes
  • presents with multiple skin sinuses and swellings
  • pus is thick and lumpy (like cottage cheese)
31
Q

How do you treat actinomycosis?

A
  • incision and drainage of pus
  • excision of chronic sinus tracts
  • excision of necrotic bone and FBs
  • high dose antibiotics (often IV)
  • long term antibiotics to prevent recurrence
32
Q

What antibiotics can be used to treat actinomycosis?

A
  • penicillins
  • doxycycline
  • clindamycin
  • based on culture sensitivity
33
Q

What is the guidance surrounding IE?

A
  • infective endocarditis
  • antibiotic prophylaxis used to be standard
  • now choice is discussed with patient and cardiologist regarding risk and procedure
34
Q

What prophylaxis antibiotics can be prescribed for someone at risk of IE?

A
  • amoxicillin
  • clindamycin
  • azithromycin (second line if allergy)