7 Cold injuries Flashcards
Causes of Frostnip & Frostbite
Low air temperature • Windchill • Exposed skin • Moisture on skin & in clothing • Poor insulation • Direct skin contact with supercooled metal or liquid • Reduced blood circulation • Tight clothing or shoes • Dehydration • Localised pressure
Frostnip:
- Only outer layer of skin
- like a 1st degree burn
- Rewarm instantly, no long term damage
Superficial Frostbite:
- Some underlying skin tissue frozen
- White or grey, hard on surface, soft below, blisters.
- Rapid rewarming required
Severe Frostbite:
- Crystalisation of fluids in skin, tissue death
- Pale, hard, numb
- Extreme pain, swelling and blisters
- Take to A&E without thawing
Non-Freezing cold injuries:
- Trench/Immersion Foot
- Chilblain
- Pernio
Trench/Immersion Foot:
- Prelonged exposure to 0-20(C)
- Reduced CIVD response
- Damage caused by prolonged intense vasoconstriction
- Should be rewarmed slowly
Phases of Non-Freezing Cold Injury
Phase 1: During cold exposure:
• Cold, pale, numb
Phase 2: Following cold exposure / rewarming:
• Swollen, mottled pale blue, numb
Phase 3: Hyperaemia
• Red, hot, flushed, intense pain, swelling, blistering &
ulceration
Phase 4: Following hyperaemia
• Cold sensitivity, pain, loss of sensation, hyperhydrosis
Treatment of Non-Freezing Cold Injury
-Rewarm slowly
-Rewarming of injured tissues increases metabolic demand above the supply capability
of injured blood vessels
-Alleviate pain in early stage
Mechanisms of frostbite:
Local vasoconstriction -> Reduced blood flow -> Clot
Freezing of tissue (ice crystals) -> cell damage and dehydration
Mechanisms of Non-Freezing Cold Injury
Local vasoconstriction -> Reduced blood flow -> Clot
Local nerve cooling -> Local nerve damage