7 bones and joints Flashcards

1
Q

causes of genu varum (bowleg)

A

metabolic bone disease,
asymmetric growth arrest,
bone dysplasia and congenital and neuromuscular disorder

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2
Q

causes of genu valgum (knock knees)

A
metabolic bone disease, 
skeletal dysplasia, 
posttraumatic physeal arrest, 
tumors, 
infection
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3
Q

pain over the tibial tubercle in a growing child

A

Osgood-Schlatter Disease

  • traction apophysitis of the tibial tubercle growth plate and the adjacent patellar tendon
  • more common in males, athletes, 10-15 years old
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4
Q

abnormal morphology and development of the acetabulum

A

Acetabular dysplasia

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5
Q

partial contact between the femoral head and acetabulum

A

Hip sublaxation

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6
Q

hip with no contact between the articulating surfaces of the hip

A

Hip dislocation

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7
Q

-assess potential for dislocation of the non-displaced hip
-adducts Flexed hip and gently pushes the thigh
posteriorly in an effort to dislocate the femoral head
-(+) test: hip will slide out of the acetabulum

A

Barlow provocative maneuver

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8
Q

-attempt to reduce a dislocated hip
-grasp thigh between the thumb and Index finger and
with the 4th and 5th fingers, lift the greater trochanter
while simultaneously abducting the hip
-(+) test: femoral head will slip Into the socket with

A

Ortolani test (Reverse of Barlow)

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9
Q
  • femoral head disorder of unkown etiology
  • temporary interruption of the blood supply to the
    bony nucleus of the proximal femoral epiphysis,
    leading to impairment of the epiphyseal growth and femoral head deformity
A

Legg Calve Perthes Disease

-delayed skeletal maturation, shorter than normal
-most common symptom: limp of varying duration
-pain is activity related, localized
-antalgic galt after strenuous activity
-limited hip motion, atrophy of thigh muscles, calf,
buttocks secondary to pain
-classic portrait: small, thin, extremely active child
-plain radiographs: primary imaging tool

Goal of treatment:
create a spherical, well covered femoral head with hip range of motion close to normal

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10
Q

most common organism in all age groups of osteomyelitis

A

S.aureus

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11
Q

most common organism in 6y/o osteomyelitis

A

most cases are caused by S. aureus, Streptococcus, or Pseudomonas aeruginosa

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12
Q

second most common cause of osteomyelitis in children

A

Kingella kingae

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13
Q

Diagnostics used for Osteomyelitis

A
  • Blood culture
  • history and physical findings
  • Aspiration for Gram stain
  • Culture: confirms the diagnosis
  • Aspiration of bone pus: best specimen
  • elevated ESR and CRP: assess response to therapy or identify complications
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14
Q

treatment for osteomyelitis in neonates

A
  • Antistaphylococcal Penicillin
    :Nafcillin or Oxacillin (150-200 mkd q6) and
    Cefotaxime (150-200 mkd q8)
    MRSA: Vancomycin and aminoglycoside
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15
Q

treatment of osteomyelitis in older children

A
  • Cefazolin (100-150mkd q6) or nafcillin (150-200mkd q6)

- Cefotaxime or Ceftriaxone if no H. Influenza vaccine

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16
Q
  • have the potential to cause permanent disability
  • frequency is increased in infants and toddlers
  • Staphylococcus aureus Infection is most common
  • Gonococcus in Sexually active adolescents
  • 50% occur by 2 y/o, % of cases in 5 y/o
  • majority of infections in otherwise healthy children are of hematogenous origin
  • can follow penetrating injuries
A

Suppurative Arthritis/Septic Arthritis

17
Q

diagnostics for suppurative arthritis

A
  • Blood culture
  • Gram stain and culture definitive diagnostic technique
  • Elevated ESR and CRP
  • Plain films: widening of the Joint capsule, soft tissue edema, and obliteration of normal fat lines
  • Ultrasonography: highly sensitive in detection of joint effusion, particularly for the hip joint (aid in aspiration)
  • CT and MRI; useful in confirming presence of joint fluid
18
Q

treatment for suppurative arthritis

A
  • Neonates: nafcillin or oxacillin and cefotaxime
  • Children: cefazolin or nafcillin
  • MRSA: Clindamycin and vancomycin
  • Immunocompromised: vancomycin and ceftazidime or
  • extended-spectrum penicillins and B-lactamase inhibitors
  • with an aminoglycoside
  • Dexamethasone for 4 days
  • Daily aspiration of synovial fluid: Joints
  • Surgical emergency: hip
19
Q

achondroplasia

A
  • prototype of chondrodysplasia
  • at birth: short limbs, long narrow trunk, large head with midfacial hypoplasia and prominent forehead
  • limb shortening: greatest in the proximal segments, and the fingers often display a trident configuration
  • Most joints are hyperextensible, but extension is restricted at the elbow
  • an autosomal dominant trait
  • delayed motor milestones, normal intelligence,
    exaggerated lumbar lordosis
  • Infants and children: progressively fall below normal standards for length and height, large heads
20
Q
  • large calvarial bones
  • small cranial base & facial bones
  • short vertebral pedicles, decrease interpedicular
    distance, 1st to the 5 lumbar vertebra
  • fibula is disproportionately long compared with tibia
  • Surgical correction for severe spinal canal stenosis
A

Achondroplasia

21
Q
  • most common genetic cause of osteoporosis
A

Osteogenesis Imperfecta (brittle bone disease)

  • generalized disorder of connective tissue
    • structural or quantitative defects in type I collagen
    (primary component of the extracellular matrix of bone and skin)
  • autosomal dominant
22
Q

TRIAD of Osteogenesis imperfecta

A

fragile bones
blue sclerae
early deafness

23
Q
  • autosomal dominantly inherited disorder
  • abnormal biosynthesis of fibrillin-1 (major constituent of microfibrils provide scaffolding network of elastin and have an anchoring function in nonelastic tissue- aortic adventitae & eye’s suspensory ligament)
A

Marfan Syndrome