633 - Sheep Flashcards

1
Q

What is the biggest expenses on a farm?

A

Feed

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2
Q

What are some challenges associated with excellerated lambing systems?

A

Feed supply variations - you may have to supplement as you won’t always have the advantage of natural pasture

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3
Q

What are the main drivers of profitability? What are some things that reduce profitability on a sheep farm?

A

Stocking rate (production/Ha) - influenced by soil and therefore pasture quality
Weaning % - influenced by total lambs, disease, weather, genetics, nutrition
Other things that reduce profitability: drench resistance, lice, flies, Johne’s disease

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4
Q

What economic effect does drench resistance have?

A

> $2/weaner loss on returns from wool
$4/sheep if using an ineffective drenching product

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5
Q

At what points should vet visits happen ideally and what are we doing at these points?

A

Pre-joining - breeding soundness exams, nutrition, BCS
Post-joining - pregnancy diagnosis, scanning for empty/singles/twins to allow for cohorting and more appropriate feed management
Pre-lambing - BCS, nutrition management to prevent metabolic diseases, ensuring good quality milk in udders
Marking - vaccination, provision of pain relief
Weaning - data collection (especially weaning weight) to allow us to improve the next season

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6
Q

Why is is so important to increase feeding rate close to lambing?

A

2/3rds of foetal growth happens in the last trimester of pregnancy, we need to increase our feeding rates so that the ewes don’t lose condition while the foetus still gets fatter as this increases chance of dystocia

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7
Q

What is the target benchmark for abortive disease?

A

<2%

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8
Q

What is the target benchmark for perinatal mortality? Which complex contributes highly to this rate?

A

20-25% of lambs
SME complex = starvation, mismothering, exposure

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9
Q

What is the target benchmark for weaner death rate?

A

<4%

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10
Q

What is an acceptable lambing rate for merinos?

A

150-180%

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11
Q

What factors determine lambing rate?

A

Good quality rams
Joining ratio 1:50, but can get it higher
Good nutrition to ensure multiple ovulations - can also use drugs but you need to have good condition ewes

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12
Q

What are the nutritional requirements of ewes in their last pregnancy and how does it change when they are carrying singles vs twins vs triplets?

A

3.5-4% BW for ewes, 3-3.5% BW for goats
Singletons - requires nearly twice as much feed to prevent loss of body condition compared to non-pregnant ewes
Twins - requires 2.5 times as much than the non-pregnant ewe
Triplets - requires 3 times as much

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13
Q

What 4 major principles result in high productivity and efficient production?

A
  1. Maintaining ewe BW throughout year
  2. Growing young sheep at steady rate to reach adult BW at 19 months old
  3. Utilisation of superior genetics
  4. Remove non-productive sheep from flock
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14
Q

What is the target LW for most breeds of sheep?

A

55kg

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15
Q

What are the target weight gains expected from birth until weaning and then after weaning for slaughter lambs?

A

Birth to weaning - 280g/day
After weaning - 100g/day

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16
Q

What are the benefits of maintaining ewe bodyweight?

A

Steady foetal growth
Higher lamb survival
Higher milk production
High wool growth

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17
Q

Name 3 strategies that can be put into place when there is insufficient nutrients available from pasture.

A
  1. Alter stocking rate - sell or adjust stock or change priorities for different classes of stock
  2. Supplementary feeding - cereal grains, conserved pasture or forage crops during drought and periods of low pasture availability
  3. Accept the loss from underfeeding - not recommended
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18
Q

Discuss the 3 primary components of sheep production that have to be addressed in order to increase productivity.

A
  1. Lambing % - improve nutrition, use superior genetics, identify excessive losses
  2. Nutrition - assess pasture quantity and quality, supplementary feed as needed, leads to efficient weight gains in growing sheep and maintains ewes in a productive state throughout the year
  3. Animal Health Management - put programmes into place to ensure minimal morbidity and mortality
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19
Q

At what point do we cull/remove ewes from the flock?

A

If not pregnant at PD
If not rearing a lamb
If aged and no longer have sound mouths + udders

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20
Q

How do we measure performance?

A

Monthly pasture covers
Faecal egg counts + antiparasitic treatments
Wool production + hogget average daily wool growth
Meat production as kg/Ha and kg/kg ewe mated
Lambing %, proportion of pregnant ewes, proportion of ewes rearing lambs, proportion of twin lambs
Weaning %
Fertility index
Death rates at various stages
Financial data - cost of production, income, profitability (all per head and per hectare)

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21
Q

Explain to a farmer the best time for lambing.

A

We want lambing to happen ideally in July, August when the high rainfall coincides with strong pasture growth, so that by the time they are weaned in November there is a good supply of pasture and the farmer won’t have to spend too much (hopefully) on supplementary feeding ($$$$$).

A longer joining season pushes lambing back to happen at the time of maximal pasture growth, which means there will be more costs for supplementary feeding weaners for market as the farmer will have to buy in feed.

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22
Q

Explain the importance of correct feeding for ewes, especially for pregnant and lactating ewes.

A

Ewes need a massive amount of nutrients especially in their 3rd trimester to not lose condition as 2/3 of foetal growth is happening in this stage. If the plane of nutrition is only enough to support the foetus and the ewe is losing condition then there is not enough energy left over for production of good quality milk nutrition for the lamb which leads to high lamb mortality. If the lambs do survive they often have poor quality wool.

Correct feeding for ewes is also important to prevent them falling into negative energy balance and getting severely metabolically compromised - pregnancy toxaemia, hypocalcaemia, hypomagnesaemia, etc.

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23
Q

How can you differentiate between pregnancy toxaemia, hypocalcaemia, and hypomagnesaemia?

A

Pregnancy toxaemia - inc. BOHB levels; may also see dec. Ca and Mg; last month of pregnancy; slow onset of signs, only in pregnant ewes and does

Hypocalcaemia - total serum Ca lower than 2mmol/L; late pregnancy/early lactation; rapid onset of signs; pregnant + non-pregnant ewes/does

Hypomagnesaemia - total serum Mg lower than 0.5mg/dl (0.21mmol/L); early lactation/1-4 weeks post lambing; rapid onset of signs; pregnant + non-pregnant ewes/does

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24
Q

What are the clinical signs of pregnancy toxaemia?

A

Early Signs: separate from mob, low BCS, enlarged abdomen, appear blind, stop eating, wool break

Later Signs: drowsiness, nervous signs, recumbency, bruxism, coma + death in 3-4d, foetal death then apparent recovery then relapse

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25
Q

What are the 5Cs of mastitis management?

A

CONFORMATION - symmetrical udder with teats at 4 and 8 o’clock
CONDITION - BCS 3 or more reduces mastitis risk
CULL - ewes with chronic mastitis or old, thin ewes
CONSIDER SUPPLEMENTATION - to ensure they lactate well
COVER - cover exposed udders from wind and water

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26
Q

How can we induce parturition in a ewe? Why does it change depending on her stage of pregnancy and how is it different to inducing parturition in a doe?

A

Ewes: Before d55 you can use PG as the sole source of progesterone is the CL, during the second trimester the foetal-placental unit provides some progesterone so need to do PG + corticosteroids (dexamethasone), in the last trimester the foetal-placental unit is the sole source of progesterone so only need corticosteroids to terminate/induce.

Does: PG at any stage as they rely on the functional CL for progesterone the entire gestation length.

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27
Q

Describe the importance of pregnancy diagnosis in a sheep enterprise.

A

Allows for cohorting of ewes (even if just pregnant vs empty) so that they can be appropriately fed. Non-pregnant ewes can then be sent to market, which can add money back into the farm. Pregnant ewes can be fed appropriately which lowers the chance of metabolic disease and other complications.

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28
Q

How long is the ideal joining period for sheep? Explain.

A

5 weeks
65% of sheep will fall pregnant after a single mating cycle, reaching 85% pregnancy rate with 2 cycles, of about 34d or just under 5 weeks.
The benefit of having the ram in for another cycle and extending the joining period is minimal as the ewes that haven’t gotten pregnant in the first 2 cycles have poorer fertility and is not enough to outweigh the added costs of having a later and extended lambing period.

The other benefit of having a shorter joining period is that the lambing period will be condensed and have reduced costs of extra labour during that time.

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29
Q

What are some consequences of under or over feeding ewes during pregnancy?

A

Dystocia, poorer quality wool from offspring, poorer average daily gain of offspring, lowered perinatal survival, metabolic diseases (pregnancy toxaemia for example).

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30
Q

Discuss the appropriate nutritional management of ewes carrying twins or triplets (multiples) in the last week of pregnancy.

What feeds should be avoided in late pregnant ewes and why?

A

High energy supplementation (barley) during the last week to boost colostrum production, feed whole barley grain @1% LW in addition to what they normally get, making sure to gradually introduce it to avoid acidosis.

Avoid clover, lupins + lucerne as they often contain oestrogenic compounds so can lead to a higher chance of abortion in late pregnant ewes.
Maize with mould also has oestrogenic compounds, and maize can be calcium binding which predisposes to hypocalcaemia.

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31
Q

When would abortions warrant further investigation? How would you go about investigating abortion in small ruminants?

A

When to investigate - abortion levels greater than 5%, abortions occurring over a short period of time, abortions occuring in the same locations.

Get a history and physical exam
Take samples - put 1/2 on ice for virus isolation, PCR + bacterial culture, and the other half in 10% buffered formalin for histology.
Samples of - foetal stomach contents, liver, lungs, brain, placenta (including placentoma), blood for serology (paired at time of abortion and then another in 2 weeks or so to allow for titre comparison).

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32
Q

Explain the pathogenesis of pregnancy toxaemia/ketosis/twin lamb disease.

A

Ineffective gluconeogenesis leads to increased glucose demand from foetuses –> hypoglycaemia –> lipid metabolism –> inc. ketone bodies/cortisol –> hyperketonaemia + hyperketonuria

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33
Q

Describe the signalments associated with hypocalcaemia.

A

Ewes in last month of pregnancy + early lactation
Young sheep grazing green oats/young grasses low in Ca
Sheep fed cereal grain only diets
Stress/fasting, shearing, crutching, transport
Older animals - have dec. mobilisation of Ca
Sheep fed on oxalate containing plants or given long courses of tetracyclines - both bind Ca

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34
Q

Describe the signalments associated with hypomagnesaemia.

A

Animals on lush green young rapid growing pasture
Pastures fertilised with nitrogen and potassium
Young cereal pastures
Late gestation/early lactation ewes + does
Stress + temporary food deprivation

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35
Q

When should you intervene in a lambing?

A

If the ewe has been having active stage 2 contractions (abdominal tenesmus) for more than 30 mins
Total period of lambing exceeding 90 mins
If the lamb/s have any of the following presentations: head but no legs, legs but no head, tail no legs, 1 front leg and 1 hind leg

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36
Q

How can we prepare ewes for weaning to prevent mastitis?

A

Cut grain feeding 3-5d before weaning to reduce milk production
Replace grain with grass hay
Withhold water and food for 12h before weaning

The more flaccid and empty the udder is the less likely it is for microbes to enter and cause problems

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37
Q

What is the normal temperature, HR, RR, and weight for newborn lambs/goat kids?

A

Temp - 38.6-39.9 degrees C
HR - 80-100bpm
RR - 36-48bpm
Weight - 3-4kg for kids, >4kg for lambs depending on the number born/ewe

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38
Q

What are some management strategies we can implement to decrease the risk of congenital disease outbreaks?

A

Not using live vaccinations in the first trimester
Avoid teratogens including joining ewes on alkaloid/bitter lupins
Not breeding defective sheep
Cull lambs with defects

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39
Q

List 4 congenital diseases of lambs/kids.

A
  1. Entropion
  2. Cerebellar atrophy/Daft Lamb Disease
  3. Congenital goitre (iodine deficiency)
  4. Congenital selenium deficiency
  5. Congenital copper deficiency
  6. Border Disease
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40
Q

What is entropion? What are the clinical signs?

A

Rolling in of the eyelids (usually the bottom/lower eyelid) leading to blepharospasm, epiphora, matting of wool around the eyes, corneal opacity, blindness, lowered growth rates

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41
Q

How can we treat and prevent entropion?

A

Manual eversion for less severe cases - often recurrs
Surgical staples for more severe cases
Inject 2ml sterile saline or air or benzathine penicillin or oxytetracycline SC into affected lid
Surgery

Prevent - cull rams whose progeny have entropion, do not breed affected sheep

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42
Q

How do we manage cereballr atrophy in lambs in a commercial setting?

A

Euthanasia

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43
Q

A farmer comes to you because he has had several Angora goat kids born with swellings posterior to the larynx which died within their first week of life. The does have been kept on pastures used to grow sorghum and brassica. What is your likely diagnosis and how are you treating it?

A

DDx - Congenital goitre (iodine deficiency)

Cx - enlargement of thyroid glands, death of neonatal kids/lambs

Tx - potassium iodide PO

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44
Q

Border disease is similar to which disease of cows?

A

BVDV

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45
Q

In congenital selenium deficiency which organ is lesions usually found?

A

Cardiac muscle (heart)

46
Q

How do we prevent congenital selenium deficiency?

A

Supplementing selenium to ewes 6 weeks prior to lambing

47
Q

Congenital copper deficiency can cause still birth. If the lambs/kids are born alive what clinical signs do they show?

A

Blindness
Paralysis
Moribund
Head tremours

48
Q

Describe the treatment of congenital copper deficiency.

A

It’s unfortunately not treatable
Euthanasia if born alive

49
Q

Describe the clinical signs of border disease.

A

Abortion
Weak, poor, small lambs
Increased lamb mortality or reduced growth
Hairy shakers = PI lambs

50
Q

What are the outcomes for a lamb whose dam was infected with border disease <60d gestation?

A
  1. Abortion, early foetal death, mummification, barren ewe
  2. Foetus survives - either a hairy shaker (PI) or be clinically healthy; Ag positive, Ab negative
51
Q

What are the outcomes for a lamb whose dam was infected with border disease between day 60-85 of gestation?

A
  1. Abortion, early foetal death, mummification, barren ewe
  2. Foetus survives - either a hairy shaker (PI) or be clinically healthy; Ag positive, Ab negative
  3. Foetus survives to be born normal - eliminated virus; Ag negative, Ab positive
52
Q

What are the outcomes for a lamb whose dam was infected with border disease >85d gestation?

A
  1. Foetus survives to be born normal - eliminated virus; Ag negative, Ab positive
53
Q

Discuss the diagnosis of border disease.

A

Serology - heart blood, serum, thoracic fluid
Histopath - brain + spinal cord (hypomyelination + hypercellularity of white matter), fleece, skin (primary follicle enlargement, inc. fibre size, inc. number of medullated fibres)
Virology - thyroid, kidney, brain, spleen, gut, LNs, placenta

PM findings:
Brain + spinal cord - hydrancephaly, microcephaly, cerebellar hypoplasia, doming of head

54
Q

What is the current rate of perinatal mortality in Australia as a whole compared to just in WA?

A

20-25% as a whole of Australia average
15-20% as a WA average

55
Q

What are the common causes of perinatal mortality?

A

Birth stress (60%) - thermogenesis effects, sucking, locomotory behaviour, meconium staining

Starvation-mismothering-exposure complex (SME) (25%) - aberrant maternal behaviour, hypothermia, inadequate milk supply

Disease (15%) - malformations, infections, trace element deficiency, predation

56
Q

When do a majority of perinatal mortality (lamb deaths) occur?

A

Between day 1 and 3 of life (53%)

57
Q

Name the predisposing factors of starvation and mismothering.

A

The first 48 hours of life
Poor morthering ability - i.e. maiden ewes
Ewe cannot feed lamb/lamb is weak and/or uninterested in sucking or unable to suck
Birthing injury

58
Q

Name the predisposing factors for exposure/hypothermia.

A

Birthing injury - hypoxia leads to decreased thermoregulation
First 36 hours of life - cannot regulate their own core temperature, + they are born wet so lose heat through evaporation
Cold wet weather
Low birth weight - multiples
Lambs which received no colostrum or inadequate milk

59
Q

At what temperature is a lamb/kid considered moderately hypothermic? How about severely hypothermic?

A

Moderate hypothermia - <38 degrees C
Severe hypothermia - <37 degrees C

60
Q

How long after birth does a hypothermic lamb become likely to have hypoglycaemia?

A

> 5 hours old (especially after 10 hours old)

61
Q

Describe the treatment of hypothermia.

A

Dry them off and warm them up
Feed colostrum 50ml/kg
If older than 5 hours then give 10ml/kg 20% dextrose intraperitoneal

62
Q

What measures would you recommend to decrease predation on neonatal lambs?

A

Electrical fences
Alpacas
Anatolian Shepherd dogs - livestock guardians

63
Q

Explain the pathogenesis of watery mouth disease.

A

Usually causes death of lambs/kids younger than 3d old
Lambs ingest a high number of non-enteropathogenic or non-enteropathotoxic strains of E. coli from the contaminated environment soon after birth
These lambs/kids often have insufficient colostrum and gut hypomotility which leads to rapid multiplication of bacteria in the gut and production of bacterial endotoxins which leads to clinical signs of disease

64
Q

List the clinical signs of watery mouth disease.

A

Dullness, cold extremities, hypothermia
Excessive mucoid salivation
Abdominal distension
Not suckling
Affected lambs die within 6-24 hours

65
Q

What preventative measures can be taken against watery mouth disease?

A

Provide adequate colostrum
Ensure ewes lamb in a clean, dry shed/pen/paddock

66
Q

What level of TSP is considered failure of passive transfer in lambs/kids?

A

<4.2g/dL

67
Q

What are the 3 most common post-natal conditions affecting lambs/kids between day 2 and 7?

A
  1. Orphaning/desertion by ewe, mismothering
  2. Starvation
  3. Enteritis/diarrhoea - usually result of FPT
68
Q

What are the 3 most common post-natal conditions affecting lambs/kids between week 1 and 4? How do they manifest?

A
  1. Delayed selenium deficiency
  2. Delayed copper deficiency
  3. Enterotoxaemia/pulpy kidney
69
Q

What breed of sheep are susceptible to copper toxicity?

A

Texel

70
Q

Discuss the aetiology of delayed swayback/copper deficiency.

A

Primary - dietary deficiency, less fresh green pasture vs hay, deficient soils

Secondary - diet adequate but conditioning factors interfere with copper utilisation, high molybdenum and sulphur, zinc suppression, iron may inhibit Cu metbaolism, high in pasture/feed

71
Q

What 3 effects does copper deficiency have on the developing lamb/kid?

A

Spinal cord demyelination –> hindquarter paralysis
Anaemia (microcytic, hypochromic)
Straight wool fibres/steely wool

72
Q

What procedures are encompassed in the term “marking”?

A

Eartagging and notching
Tail docking
Castration
Vaccinations and drenching
Mulesing

73
Q

Where on the ear should an ear tag be placed?

A

Proximal 3rd of the ear between the bottom and middle cartilages

74
Q

Where should vaccinations be given on a sheep and why?

A

At the base of the ear or high up on the neck to preserve the carcass value

75
Q

How long should we be leaving the tail when tail docking?

A

Long enough to cover the vulva to protect from solar radiation, SCC, and melanoma formation, and to prevent rectal prolapse (if done too proximally you can damage the muscles of the perineal diaphragm)

76
Q

Name 3 complications of castration.

A

Tetanus
Flystrike
Uroliths

77
Q

What are some alternatives to mulesing?

A

Control of internal parasites to prevent diarrhoea
Breeding sheep with less skin folds
Wool clips - not practical

78
Q

What are some adverse effects/problems with activities that happen at marking?

A

Infection - tetanus, arthritis, spinal cord and vertebral body abscesses
Cutaneous myiasis/flystrike
Tail lesions
SCC
Rectal prolapse
Vaccination site reactions

79
Q

Discuss the artificial rearing of lambs and kids, especially in terms of nutritional management.

A

Make sure you have addressed any hypothermia or hypoglycaemia
Ensure they are sheltered and warm - take their rectal temperature

If they did not get colostrum than you need to give some to prevent failure of passive transfer - minimum 200ml/kg BW given in the first 24 hours is usually adequate

Nutritional management:
50ml/kg/feed milk replacer (full cream powdered milk, 200g powder with warm water to make up 1L then split it)
Cool milk is less likely to cause abomasal bloat

80
Q

How does sheeps milk differ to cows and goats milk in composition?

A

Higher fats, proteins, and total solids

81
Q

Describe how to collect and manage colostrum.

A
  1. Catch a ewe that has given birth in the last 2 hours
  2. Give 5IU oxytoxin IV or 10IU IM
  3. Wait 1 minute
  4. Milk off 500mL
  5. Freeze the colostrum in a labelled container - can last up to 1 year
  6. When you need it, thaw in a 37 degree C water bath (no microwave)
  7. Administer by bottle or stomach tube within the first 12-18 hours of life
82
Q

How can you tell if a goat kid is going to develop horns?

A

Pointed raised bumps on top of the head between the ears
Double whorls of hair on the top of the head between the ears

83
Q

When and how is goat dehorning ideally done?

A

As a kid between 2-7 days old
Electric disbudding hot iron is ideal

84
Q

Describe the process of goat dehorning (as a kid).

A
  1. Sedate with diazepam
  2. Clip hair from the horn base
  3. Ring block around the horn bulbs with 2% lignocaine/dilute to 0.5% lignocaine with the total dose not exceeding 10mg/kg to avoid toxicity
  4. Apply the hot iron with a rotating motion for 5 seconds at a time or remove the horn bud with a scalpel blade and then cauterise with the hot iron
  5. Should see perfect circles around where the horn bud was
85
Q

What are some signs of lignocaine toxicity that we need to look out for when doing a dehorning procedure?

A

Drowsiness
Respiratory depression

86
Q

What other structure can be removed at the same time as an adult surgical dehorning?

A

Scent (horn) glands in bucks

87
Q

Describe the process of adult dehorning.

A
  1. GA or sedate with diazepam and a ring block or block the cornual branches of the infratrochlear nerve and lacrimal nerve with 2-3mL 2% lignocaine
  2. Incise skin 1cm from the base of the horn
  3. Remove the horns with embryotomy wire or a saw
  4. Seal the sinuses with epoxy or bandage the head and remove in 7-10 days

Monitor for flystrike, vaccinate against tetanus, and watch for sinusitis

88
Q

What time of year do most weaner problems happen?

A

Summer months when there is little to no green feed available

89
Q

How much lower is the risk of mortality between weaning at 14 kg vs 12kg?

A

The additional 2 kg LW decreases the risk of weaner mortlity by 38%

90
Q

Describe the diagnostic approach to investigating weaner ill thrift.

A

Inspect the sheep in the paddock at rest and following exercise
Bring the mob into the yards (watching for a long weaner mob tail) and BCS, get actual weights on 10% of the mob or 50 sheep (whatever is less)
Calculate the % of weaners in the tail of the mob when bringing them in - benchmark is 4%
Thorough physical exam of 5-10 animals
Collect samples from live animals (3-10 animals per mob) - serum, whole blood in EDTA and lithium heparin; 10 faecal samples (10g/sheep)
Necropsy affected weaners - more than 3 ideally, take samples

91
Q

What are the 5 points to check for any evidence of parasite infection in sheep/goats?

A

Eyes - anaemia (FAMACHA score)
Back - low BCS
Tail - diarrhoea
Coat - wool break
Jaw - submandibular oedema

92
Q

What samples do you need to diagnose selenium deficiency?

A

Blood in lithium hepatin tube for glutathione peroxidase - 5 sheep
Serum for CK and AST - 10 sheep

93
Q

What samples do you need to diagnose cobalt deficiency?

A

Blood in lithium hepatin for methylamonic acid or vitamin B12 - 10 sheep
Blood in EDTA for CBC if sheep appear anaemic - 3-10 sheep
Necropsy at least 3 affected sheep - sample liver, kidney, cardiac + skeletal muscle, brain and spinal cord

94
Q

Discuss the treatment of selenium and cobalt deficiency.

A

Supplementation for Se
Injections, drenches, rumen pellets, lick blocks
Fertilisers
Pasture sprays for cobalt
Vitamin E drenches and feeding vitamin E treated grain for selenium deficiency

95
Q

Name 4 causes of weaner illthrift.

A

Parasites
Mineral deficiencies - cobalt, selenium
Inadequate nutrition - feeding low DE/protein/Ca deficiency/insufficient food amount
Energy deficiency - dry pasture in WA during summer and autumn is a limiting factor
Chronic infections - pneumonia, facial eczema, caseous lymphadenitis

96
Q

What are the energy and protein requirements of weaner sheep?

A

7-9 MJ ME/kg DM
15% protein

97
Q

At what age does the rumen become functional? How does this affect the recommended age of weaning?

A

Rumen becomes functional at 8 weeks old
Weaning should therefore be done at least 8 weeks from the end of the lambing period
Since a normal joining period is 5 weeks the recommended weaning time is 13 weeks from the start of lambing so that the youngest lambs to be weaned will still have a functional rumen

98
Q

When should lamb marking be done?

A

6-7 weeks after the start of lambing

99
Q

When should you start teaching creep feeding to lambs?

A

3-4 weeks before weaning

100
Q

What 2 management strategies can we implement to reduce weaner deaths?

A
  1. Nutritional management - 2.5kg/head/months of oats + lupins if no green feed, aim for 0.25-0.5kg weight gain per month
  2. Do not shear weaners in autumn - increases the risk of death x4 if shearing is done between March and June
101
Q

At what age and weight can british breed ewe lambs be bred?

A

7-9 months old
>45kg LW

102
Q

At what age and weight can merino ewe lambs be bred?

A

8-9 months old
>37kg LW

103
Q

Why is the lambing rate lower for merino ewe lambs compared to british breed ewe lambs?

A

Merinos have less resources to stimulate their HPA axis to produce GnRH and quality oocytes in turn
There is a high incidence of silent oestrus, incomplete ovulations and incompetent oocytes resulting in a lower lambing rate

104
Q

Why is diarrhoea a problem?

A

Contamination of fleece
Blowfly strike
Loss of body condition score + impaired growth
Dehydration, acidosis, death
Decreased wool production
Extra crutching required - increased labour costs
Need to replace the dead sheep
Need to spend money on drenches

105
Q

Name the most common causes of diarrhoea in lambs/kids between 0-4 weeks old.

A

Rotavirus
Colibacillosis
Lamb dysentery
Dietary diarrhoea

106
Q

Name the most common cause of diarrhoea in lambs/kids from 4-12 weeks old.

A

Coccidiosis

107
Q

Name the most common causes of diarrhoea in lambs/kids from 12 weeks to 1 year old.

A

Salmonellosis
Nematodiasis
Plant poisons
Nutritional/managerial

108
Q

Name the most common causes of diarrhoea in adult sheep/goats.

A

Salmonellosis
Nematodiasis
Plant poisons
Nutritional/managerial

109
Q

List the clinical signs and presentation of lambs/kids with salmonellosis.

A

Acute tenesmus
Depressed
Temperature above 40 degrees C
Tachypnoea + tachycardia
Blood tinged, mucoid, fibrinous faeces
Abortion
Death in 2-5 days

110
Q

What are the rules of thumb for treatment of diarrhoea?

A

Fluids - at least 15% BW over 24 hours
NSAIDs - flunixin
Antibiotics are not always necesary
Correct metabolic acidosis
Potassium as part of therapy might be helpful - 10-20mEq/L
Keep feeding milk/milk replacer
Don’t give oral electrolytes and milk in the same feeding - separate by 2 hours

111
Q
A