6.1.5 Recognises common ocular abnormalities and refers when appropriate Flashcards

1
Q

What is cataract and what are the symptoms?

A

Cataract – Caused by denaturation of protein fibrils within the lens due to oxidative
stress, increasing age and metabolic disturbance
Symptoms of a Cataract;
Reduced VA
Reduced CS
Increased Glare

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2
Q

What is nuclear sclerotic cataract?

A

light scattering associated with Brunescence.
* Poorer Diet
* Low socio-economic status
* Age
* Smoking
* Larger Lens
* Higher Ambient Temperature
Signs:
* Yellowish hue
* Myopic Shift

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3
Q

What is cortical cataract?

A

Appearance of spokes
* Sunlight (UVB)
* Lens size
* Age
* Diabetes
* Smoking
* Female
* Non-Caucasian
Signs:
* Increased astigmatism
* Monocular Diplopia

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4
Q

What is posterior sub capsular cataract?

A

abnormal epithelial cells and granular material at posterior pole
due to swelling and breakdown of lens fibres
* Diabetes
* High Myopia
* Steroids
* Age
* Male
* Thyroid Hormone Use
Signs:
Rapidly progressing loss of visual acuity

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5
Q

What is the referral criteria for cataract?

A

no clear cut off, when sufficient cataract present to limit:
* The quality of life (mobility, glare)
* ability to work
* ability to drive
* willingness to have surgery
College Management Guidelines – ROUTINE Referral

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6
Q

What is the prevalence of cataract?

A

o 40 – 49 – 2.5%
o 50 – 59 – 6.8%
o 60 – 69 – 20%
o 70 – 79 – 42.8%
o 80 – 89 – 68.3%
o Nuclear Sclerotic – 18.5%
o Cortical – 12.9%
o Posterior Sub-capsular – 10.8%

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7
Q

What is normal tension glaucoma?

A
  • VHG4
  • IOPs below 24mmHg
  • Visual fields show glaucomatous changes
  • Optic Nerve head shows glaucomatous changes
  • C:D ratio asymmetry of >0.2 bilaterally
  • Loss of ISNT rule
  • Notching of the RNFL
  • Baring, Bayonetting and Overpassing of the vessels around the optic nerve head
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8
Q

What is primary open angle glaucoma?

A
  • VHG4
  • IOPs above 24mmHg
  • Visual fields show glaucomatous changes
  • Optic Nerve head shows glaucomatous changes
  • C:D ratio asymmetry of >0.2 bilaterally
  • Loss of ISNT rule
  • Notching of the RNFL
  • Baring, Bayonetting and Overpassing of the vessels around the optic nerve head
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9
Q

What is ocular hypertension?

A
  • VHG4
  • IOPs above 24mmHg
  • Visual fields show NO glaucomatous changes
  • Optic Nerve head shows NO glaucomatous changes
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10
Q

What is the prevalence of glaucoma?

A

o COAG - 2% over 40
o OHT - 3-5% over 40
o FHG 4 to 9 times more at risk

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11
Q

What is blepharitis?

A

Consider DailyDisposable to reduce chances of infection
Grade 3 or 4: Cease lens wear until resolved as risk factor for corneal infection due to
pathological microbes close to ocular surface.
Lid Hygiene:
* Warm compress
* Tea tree oil wipes
* Ocular lubricants

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12
Q

What is MGD?

A
  • Lens wear can be continued if this is tolerated by the px, as more likely to
    experience discomfort due to dryer eyes
  • Artificial tears
  • Low water content lens: prevents CL from acting like a sponge and absorbing the
    tear film
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13
Q

Describe evaporative dry eye?

A

Pre-disposing
Factors
* Wind
* Air Con
* Visually attentive tasks where
blink rate is reduced: reading,
VDU
Causes
* Lid Disease: Blepharitis, MGD
* Thyroid eye disease
* Allergic eye disease
Treatment
* Modify local environment
* Lid hygiene
* Omega 3
* Lubricants
Appearance
* Streaky Pattern on Tearfilm

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14
Q

Describe aqueous deficient dry eye?

A

Pree-disposing factors:
* Medications (Oral,
antihistamines, Betablockers, antipsychotics,
antidepressants)
Causes:
* Inflammation of lacrimal glands
* Sjogren’s Syndrome
* Age
Treatment:
* Lubricants
* Omega 3
* Punctal Plugs
Appearance:
* Circular pattern on tear film

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15
Q

What is dry AMD?

A

A natural dysregulation of biological retinal processes with time causes a build-up of
waste product generated by the photoreceptors. This product sits between the RPE and
Bruch’s membrane and is called Drusen.

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16
Q

What is the signs of dry AMD?

A

Dry AMD * Multiple small drusen
* Few intermediate drusen
* RPE pigmentary changes
Intermediate AMD * Extensive intermediate drusen (63-124
ym)
* At least 1 large drusen (>125ym)
* Geographic atrophy not involving the
fovea
At this stage, the AMD may progress into
either Late DRY AMD, or WET AMD
Late AMD * Geographic Atrophy involving fovea

17
Q

What is wet AMD and what are the signs?

A

VEGF release causes angiogenesis, triggering neovascular vessels to grow within the
choriocapillaris and through into bruch’s membrane.
These vessels are extremely fragile, causing breakage and subsequent fluid leak of fluid &
blood beneath the macula.
Signs at the macula:
* Drusen
* Hard exudates
* Haemorrhages
* Sub-retinal fluid
* Sub-RPE fluid
* Hyper & Hypopigmentation