5. Paediatrics (MSK) Flashcards

1
Q

Paeds fractures - general (3)

A

Generally small kids’ bones bend and buckle, rather than breaking.
Repeat in 7-10 days if unsure, periosteal reaction is a sign of healing fracture.
Kids heal completely, often with no sign of fracture

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2
Q

Paeds fracture - involvement of physis (3)

A

Major concern is growth arrest, can be asked by showing a physeal bar (early bony bridge crossing the growth plate).
Can get bars from prior infection, but Hx of trauma suggests fracture.

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3
Q

Salter harris classification (4)

A

Type 1: Slipped
- Complete physeal fracture, +/- displacement
Type 2: Above (or Away from joint)
- Involves metaphysis. Most common (75%)
Type 3: Lower
- Involves epiphysis.
- Chance of growth arrest, often need surgery to maintain alignment
Type 4: Through
- Involves metaphysis and epiphysis.
- Often end up with growth arrest or focal fusion.
- Require anatomic reduction and often surgery
Type 5: Ruined
- Compression of growth plate, due to axial loading injuries.
- Poor prognosis, easy to miss and often found when looking at comparisons.
- “Bony bridge across physis”

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4
Q

Toddler’s fracture (3)

A

Oblique fracture of the midshaft of tibia, seen in child just starting to walk (new stress on bone)
If spiral fracture, sus for NAI.
Typically 9 months to 3 years.

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5
Q

Stress fracture in children (3)

A

Occurs after repetitive trauma, usually after new activity like walking.
Common site is tibia, proximal posterior cortex (Toddler fracture).
Other classic ones include Calcaneal fracture, seen after child has had cast removed and returns to normal activity.

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6
Q

Paeds Elbow (3)

A

Elevation of fat pad suggests effusion and occult fracture.
Can see thin anterior pad, but should never see posterior pad.
Commonest fracture is supracondylar (in kids, >60%), then lateral condyle (20%), then medial epicondyle (10%).

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7
Q

Radiocapitellar line (2)

A

Line through centre of radius, should intersect the middle of the capitellum on every view.
If radius is dislocated it will NOT pass through centre of capitellum

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8
Q

Anterior humeral line (3)

A

Need true lateral.
Line along anterior surface of humerus should pass through middle third of the capitellum.
Supracondylar fracture causes this line to pass through the anterior third.

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9
Q

Ossification centres of elbow (6)

A

Capitellum (age 1)
Radial head (age 3)
Internal (medial) epicondule (age 5)
Trochlea (age 7)
Olecranon (age 9)
External (lateral) epicondyle (age 11)

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10
Q

Lateral condyle fractures (3)

A

Second most common distal humerus fracture in kids.
A fracture that passes through the capitello-trochlear groove is unstable (Milch II).
Difficult to tell this, so treatment is based on displacement of the fracture fragment (>2mm or <2mm).

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11
Q

Trochlea (2)

A

Can have multiple ossification centres and therefore a fragmented appearance, NOT a fracture

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12
Q

Medial epicondyle avulsion (5)

A

Because it’s an extra-articular structure, its avulsions will not necessarily result in a joint effusion.
Can get interposed between the articular surfaces of humerus and olecranon.
Avulsed fragments can get stuck in the joint, even when there’s no dislocation
If there’s a dislocation, ask 1) is the patient 5 and 2) where is the medial epicondyle.
Should never see the trochlea without the medial epicondyle. If you can, it’s probably a displaced fragment.

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13
Q

Common vs Uncommon elbow fractures (4)

A

Common:
- Lateral condylar
- Medial epicondylar
Uncommon
- Lateral epicondyle
- Medial condyle

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14
Q

Nursemaids elbow (3)

A

When a child’s arm is pulled on, the radial head may sublux into the annular ligament.
X-rays don’t help, ulness you supinate the arm during lateral position, which often relocates the arm

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15
Q

Avulsion injuries (pelvis) (7)

A

Kids tendons tend to be stronger than their bones, unlike adults.
Iliac crest # due to Abdominal muscles
ASIS # due to Sartorius
AIIS # due to Rectus femorus
Greater trochanter # due to gluteal muscles
Lesser trochanter # due to iliopsoas
Ischial tuberosity # due to hamstrings
Symphysis # due to adductor group

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16
Q

Sindig-Larsen-Johansson (3)

A

Chronic traction injury at the insertion of the patellar tendon on the patella.
Seen in active adolescents between 10-14.
Kids with cerebral palsy are prone to it.

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17
Q

Osgood Schlatter (3)

A

Due to repeated microtrauma to the paterllar tendon on its insertion into the tibial tuberosity.
25% bilateral.
More common in boys

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18
Q

Distal femoral metaphyseal irregularity (6)

A

Cortical desmoid.
Lucency seen along the back of the posteriomedial aspect of the distal femoral epiphysis.
Lateral knee X ray with irregularity or lucency at the back of the femur is probably this.
Often bilateral.
Buzzwords include “scoop like defect” with an “irregular but intact cortex”.
Incidental finding, no clinical importance.

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19
Q

Blounts (5)

A

Tibia Vara.
Varus angulation at the medial aspect of the proximal tibia (varus bowing occurs at the metaphysis, not the knee).
Often bilateral, not seen before age of 2.
Later in disease progression, the medial metaphysis will be depressed and an osseous outgrowth classically develops.
Can see in 2 age groups, early (around age 2) and late (12).

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20
Q

Congenital rubella (2)

A

Bony changes seen in 50%, classic buzzword being “celery stalk” appearance, from generalised lucency of the metaphysis.
Usually seen in first few weeks of life

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21
Q

Syphillis (4)

A

Bony changes seen in 95% of cases.
Bony changes do NOT occur intil 6-8 weeks of life (Rubella are earlier).
Metaphyseal lucent bands and periosteal reaction along long bones.
“Wimberger sign” or destrucion of the medial portion of the proximal metaphysis of the tibia.

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22
Q

Caffey disease (3)

A

Self limiting disorder of soft tissue swelling, periosteal reaction and irritability seen in first 6 months of life.
Really hot mandible on bone scan.
Mandible is commonest location, then clavicle and ulna.

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23
Q

Prostaglandin therapy (3)

A

Prostaglandin E1 and E2, often used to keep PDA open, can cause periosteal reaction.
X-ray will show sternotomy wires or other hints of congenital heart, then periosteal reaction in arm bones

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24
Q

Neuroblastoma mets (1)

A

Only childhood malignancy that occurs in newborns and mets to bones

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25
Q

Physiologic growth (8)

A

aka physiologic periostitis of the newborn.
Name is false, doesn’t occur in newborns, seen around 3 months old, should resolve by 6.
Proximal involvement (femur) comes before distal (tibia).
Always involves the diaphysis.
NOT physiologic periosititis if
- Seen before 1 month
- Seen in tibia before femur
- does not involve the diaphysis

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26
Q

Langerhand cell histiocytosis (6)

A

Also known as eosinophilic granuloma.
Twice as common in boys.
Skeletal manifestations are highly varied, classic ones are:
Skull: most common site. Bevelled edge fron uneven distribution of the inner and outer tables. Round lucent lesion in the skill of a child - think this or neuroblastoma mets
Ribs: Multiple lucent lesions, with expanded appearance
Spine: Vertebra plana

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27
Q

Osteomyelitis (3)

A

Usually occurs in babies (30% of cases less than 2 years old).
Usually haematogenous spread (adults tends to spread directly from a diabetic ulcer)
Some changes that occur over time, potentially testable.

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28
Q

Osteomyelitis in newborns (2)

A

Open growthplates, and perforating vessels which travel from the metaphysis to the epiphysis.
Infection usually starts in metaphysis (most blood supply because it’s fastest growing) and spread via these perforators to the epiphysis

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29
Q

Osteomyelitis in kids (3)

A

Later in childhood, these perforators regress and the avascular physis stops infection from crossing over.
This creates a septic tank scenatio, where infection stays in the metaphysis.

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30
Q

Osteomyelitis in adults (2)

A

When the growth plate fuses, barrier of an avascular plate is no longer present and infection can again cross over into the epiphysis

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31
Q

Osteomyelitis trivia (4)

A

Haematogenous spread more common in kids, direct spread in adults.
Metaphysis is most common location.
Bony changes don’t occur on X-ray for around 10 days.
Can rapidly destroy cartilage if it spreads into joint.

32
Q

Skeletal dysplasia vocab: fingers (6)

A

Short fingers = brachydactyly
Too many fingers = polydactyly
Two or more fused fingers = Syndactyly
Contractures of fingers = Camptodactyly
Inclined fingers (usually 5th) = Clinodactyly
Long, spider like fingers = Arachnodactyly

33
Q

Skeletal dysplasia - limb vocab (6)

A

Absent limb = Amelia
Mostly absent limb = Meromelia
Hands/feet (distal limbs) short = Acromelic
Forearm or lower leg (middle limbs) short = Mesomelic
Femur or Humerus (proximal limbs) short = Rhizomelic
Short all over = micromelic

34
Q

Dwarfs - types (3)

A

Achondroplasia
Thanatophoric
Asphyxiating thoracic dystrophy (Jeune)

35
Q

Achondroplasia (5)

A

Commonest skeletal dysplasia.
Due to fibroblast growth factor receptor problem.
Rhizomelic (short femur, short humerus) dwarf.
Often large heads, trident hands (3rd and 4th fingers are long) and narrowing of the interpedicular distance, and tombstone pelvis.
Advanced paternal age is a risk factor.

36
Q

Thanatophoric (5)

A

Most common lethal dwarfism.
Rhizomelic shortening (humerus, femur).
Femurs are sometimes called telephone receivers.
Short ribs and long thorax, and small iliac bones.
Vertebral bones are flat (Platyspondyly) and skull can be cloverleaf shaped.

37
Q

Asphyxiating thoracic dystrophy (Jeune). (4)

A

Usually fatal.
“bell shaped thorax” with short ribs.
15% will have polydactyly.
Associated with chronic nephritis.
Normal vertebral bodies

38
Q

Additional dwarfism trivia (3)

A

Ellis-van crevald is the dwarf with multiple fingers.
Pseudoachondroplasia - not present at birth and spares the skull
Pyknodystosis = osteoporosis in a dward, with wide angled jaw and osteoacrolysis.

39
Q

Osteogenesis imperfecta (5)

A

Collagen defect and make brittle bones.
Can be lethal or mild.
Totally lucent skull or multiple fractures with hyperplastic callus.
Can also show legs with fibula longer than tibia.
Wormian bones, and often flat or beaked vertebral bodies.
Also get blue sclera, hearing impairment (otosclerosis).

40
Q

Osteopetrosis (5)

A

Defective osteoclasts lead to weak, disorganised bone (prone to fracture).
Several types with varied severity.
Infantile type is lethal, destroys bone marrow.
Less severe forms cause abnormal deminished osteoclastic activity, that varies during skeletal growth, resulting in sclerosis parallel to growth plate.
Can be shown as bone in bone appearance in the verteral body or carpals.
Picture frame vertebrae is another buzzword.
Can also show diffusely sclerotic lesion with diffuse loss of corticomedullary junction in the long tubular bones.

41
Q

Pseudodysostosis (2)

A

Osteoporosid + wormian bones + acro-osteolysis.
Can also have wide (obtuse) angled mandible.

42
Q

Klippel Feil (3)

A

Congenital fusion of the cervical spine.
Cervical vertebral bodies will be tall and skinny.
Often sprengel deformity (high riding scapula).

43
Q

Hunters/Hurlers/Morquio (6)

A

All are mucopolysaccharidoses.
Findings include oval shaped vertebral bodies with anterior beak, which is mid in Morquio and inferior in Hurlers.
Clavicles and ribs are often thick (narrow more medially).
Pelvis shape is opposite of achondroplasis, the iliac wings are tall and flaired.
Hand X-ray is most commonly shown, with wide metacarpal bones with proximal tapering.

44
Q

Morquio trivia (3)

A

They’re dwarfs.
Most common cause of death is cervical myelopathy at C2.
Bony changes progress during first few years of life

45
Q

Gauchers (6)

A

Most common lysosomal storage disease
Big spleen and liver.
AVN of femoral heads
H shaped vertebra
Bone infarcts
Erlenmeyer flask shaped femurs.

46
Q

Caudal regression syndrome (2)

A

Spectrum including sacral and or coccyx agenesis.
Seen in VACTERL and Currarino Triad.

47
Q

Scoliosis (3)

A

Lateral curvature of the spine, usually idiopathic in girls.
Can be due to vertebral segmentation problems.
NF can cause it as well.

48
Q

Radial dysplasia (6)

A

Absence of hypoplasia of the radius, usually with missing thumb.
Differential case, caused by:
- VACTERL
- Holt oram
- Fanconi anaemia
- Thrombocysopenia absent radius
TAR kids will have a thumb

49
Q

Neurofibromatosis (2)

A

Type 1 can cause anterior tibial bowing and pseudoarthrosis at the distal fibula.
Can also get scoliosis

50
Q

Hand foot syndrome (4)

A

Classic Hx of hand or foot pain/swelling in infant with sickle cell.
Dactylitis, due to ischaemia.
Will resolve on it’s own after a few weeks.
Radiographs can show a periosititis 2 weeks after pain subsides.

51
Q

Congenital feet: vocab (10)

A

Talipes = congenital
Pes = foot or acquired
Equines = “plantar flexed ankle”, heel cord is often tight and heel wont touch the floor
Calcaneus = opposite of Equinus. Calcaneus is angled up
Varus = forefoot in
Valgus = forefoot out
Cavus = high arch
Planus = Counter part of cavus - flat foot
Supination - inward rotation - sole of foot in
Pronation - outward rotation - sole of foot out

52
Q

Hindfoot valgus vs varus (6)

A

Normal
- Consider acute angle the talus and calcaneus make on lateral view.
Hindfoot valgus
- Talus sliding node down off the calcaneus, making the angle wider.
- If the talus slides off, you lose longitudinal arch, which caracterizes hindfoot valgus.
- Nose down, nearly vertical, appearance of talus.
Hindfoot varus
- Opposite situation, narrowing of the angle between talus and calcaneus.
- 2 bones lay nearly parallel, like 2 clubs laying on top of each other

53
Q

Flat foot (valgus) (6)

A

Pes planus.
Can be congenital or acquired.
Congenital can be flexible or rigid (flexible is more commoin in kids).
Distinction can be made with plantar flexion views (flexible improves with stress).
Rigid subtyoes can be further divided into tarsal coalition and vertical talus.
All have hindfood valgus

54
Q

Tarsal coalition (5)

A

2 main types, talus to the calcaneus and talus to the navicular.
50% are bilateral.
Can have bony or fibrous/cartilaginous types.
Fibrous/cartilaginous types are more common than bony.
Talocalcaneal: occurs at middle facet, has continuous C-sign produced from an absent middle facet, on lateral view. Talar beaking (spur on anterior talus) is seen in 25%
Calcaneonavicular: Occurs at the anterior facet. Has anteater sign.

55
Q

Vertical talus (3)

A

Equinus hindfoot valgus
Sometimes called rocker bottom foot, because talus is in extreme plantar flexion with the dorsal dislocation of the navicular resulting in locked talus in plantar flexion.
Often associated with myelomeningocele.

56
Q

Club foot (Talipes Equino Varus) (6)

A

Congenital plantar flexed ankle forefoot.
More common in boys, and 50% bilateral.
Toes are pointed down (Equines) and the talocalcaneal angle is acute (varus) (hindfoot varus).
Medial deviation and inversion of the forefoot.
Elevated plantar arch.
Most common surgical complication is overcorrection, resulting in “rocker bottom” deformity.

57
Q

Developmental dysplasia of the hip (5)

A

More commonly in females, children born breech, oligohydramnios.
Physical exam: asymmetric skin or gluteal folds, leg length discrepancy, palpable clunk or delayed ambulation.
1/3 are bilateral.
US to evaluate, useful until bones ossify, then needs X ray.
Laxity immediately after birth (related to maternal oestrogen) can affect measurements

58
Q

Hip dysplasia angles (5)

A

Alpha angle should be >60 degrees. (acute angle between femoral shaft and acetabulum)
Anything less means the acetabulum isn’t deep enough to hold the femoral head.
Plain film equivalent is the acetabular angle, which is the complimentary angle and should be <30.
Acetabular angle should decrease from 30 at birth to 22 at age 1.
DDH causes increased angle, but neuromuscular disorder can also increase it.

59
Q

Position of femoral epiphysis (3)

A

Should be below Hilgenreiner’s line (horizontal line between tops of acetabula) and medial to Perkin’s line (vertical line through femoral heads).
Shenton’s line should be continuous.

60
Q

Proximal focal femoral deficiency (3)

A

Congenital condition, ranges from absent proximal femur to hypoplastic proximal femur.
Varus deformity.
Mimic of DDH but DDH will have normal femoral length

61
Q

Septic arthritis (3)

A

Most urgent cause of painful hip in child.
Wide joint space (lateral displacement of femoral head) should proceed to US promptly, which should prompt a joint aspiration.

62
Q

Slipped capital femoral epiphysis (5)

A

Type 1 salter harris fracture through the proximal physis of the femur.
Bad prognosis if not fixed, unlike other type 1s.
Classic Hx: overweight adolescent black teenager with hip pain.
1/3 are bilaeral.
Frog leg view is best to diagnose.
Klein’s line: drawn along edge of the femur, should normally intersect with the lateral superior femoral epiphysis. If it doesn’t, this suggests SCFE.

63
Q

Legg-Calve-Perthes (6)

A

AVN of proximal femoral epiphysis.
Boys > girls 4:1, favours white children aged 5-8.
10% are bilateral.
Subchondral lucency (crescent sign), best seen on frog leg X ray.
Other signs include asymmetric small ossified femoral epiphysis.
MRI is more sensitive.
Flat, collapsed femoral head.

64
Q

Rickets (4)

A

Vit D deficiency, affects most rapidly growing bones (knees and wrists).
Buzzwords include “Fraying, cupping and irregularity along the physeal margin”.
Increased risk of SCFE.
“Rachitic rosary” appearance from expansion of the anterior rib ends at the costochondral junctions.
Never seen in newborn due to maternal vitamin D.

65
Q

Hypophosphatasia (4)

A

Looks like Rickets in a newborn.
Frayed metaphyses and bowed long bones.
Underlying pathology is deficient serum ALP.
Variability in severity, with lethal perinatal/natal forms and milder adult forms.

66
Q

Scurvy (7)

A

Vit C deficiency.
Rare nowadays.
Doesn’t occur before 6 months (aternal vit C stores)
Bleeding disorders are common.
Subperiosteal haemorrhage, lifts up periosteum.
Haemarthrosis.
Scorbutic rosary appearance from expansion of costochondral junctions (similar to rickets)

67
Q

Lead poisoning (3)

A

Kids who eat paint chips.
Wide, sclerotic metaphyseal line (lead line) in an area of rapid growth (knee).
Will not spare the fibula, as a normal variant line might.

68
Q

Lucent metaphyseal bands (4)

A

Classic paeds DDx
- Leukaemia
- Infection (TORCH)
- Neuroblastoma mets
- Endocrine (Rickets, Scurvy)

69
Q

NAI (2)

A

Any suspicious fracture should prompt skeletal survey.
Suspicious fractures include highly specific fractures (metaphyseal corner fracture, posterior rib fractures), or fractures that don’t make sense (toddler fracture in non ambuatory child).

70
Q

Posterior medial rib fracture (2)

A

In a child under 3, pretty reliable sign of NAI.
Due to squeezing a child

71
Q

Metaphyseal corner fractures (3)

A

In non ambulatory patient (infant), is highly specific of NAI.
Only exception is obstetric trauma.
After age 1, this becomes less specific.

72
Q

Skull fracture (1)

A

Anything other than a parietal bone fracture (seen more with actual accidents) is concerning.

73
Q

Solid organ and lumen injury (2)

A

Duodenal haematoma and traumatic pancreatitis in an infant suggestive of NAI.
Just think “abdo pain in kid that’s too young to fall on the handle bars of their bike”

74
Q

Dating fractures (3)

A

Periosteal reaction: Suggests fracture is less than a week old.
Complete healing: occurs in around 12 weeks.
Exceptions: Metaphyseal, skull and costochondral junction fractures will often heal without any periosteeal reaction

75
Q

Child abuse mimics (3)

A

Rickets and OI, can have multiple fractures at different sites, and are the 2 most commonly described mimics.
Wormian bones and bone mineral density issues are clues that you’re dealing with a mimic.
Will have to show you one or the other in questions.