5. Orthognathic and Obstructive Sleep Apnea Flashcards

1
Q

Normal incisor angulation

A

Maxillary incisors 102 degrees to SN

Mandibular incisors 90-95 degrees to mandibular plane

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2
Q

What is Bolton Analysis

A

Determines disproportion of size of permanent maxillary and mandibular teeth (tooth size discrepancy between upper and lower teeth). Two ratios can be calculated (overall ratio and anterior ratio).

Overall ratio: sum of MD width first molar to first molar of mandibular teeth divided by sum of MD width first molar to first molar of maxillary teeth (91.3%)

Anterior ratio: canine-canine 77.2%

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3
Q

How much orthodontic expansion can be achieved

A

~5mm (greater than 5mm = severe maxillary transverse discrepancy –> SARPE or segmental osteotomy).

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4
Q

How are vertical facial proportions analyzed (facial thirds)

A

Upper third: trichion (Tr) to glabella. Assess eyebrow shape, position, symmetry.

Middle third: glabella to subnasale. Includes eyes, nose, and cheeks. Scleral show, flattening of cheek bones may indicate midface deficiency.

Lower third: subnasale to menton. Ratio of middle third to lower third vertical height should be 5:6.

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5
Q

How are transverse facial proportions assessed? (rule of fifths)

A

Divide sagitally into 5 equal parts. Each segment is the width of one eye.

Outer canthi coincide with gonial angles. Medial canthi coincide with alar bases of nose. Interpupillary distance coincides with corners of mouth.

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6
Q

Describe the Steiner analysis.

A

Assess maxillary AP position: Maxillary AP positions in relation to anterior cranial base (S-N). SNA 82* is considered normal.

Assess mandibular AP position: mandibular AP position in relation to anterior cranial base (S-N). SNB 80* is considered normal

Assess AP maxillomandibular relationship. Normal relationshiop is indicated by ANB of 2*. Class III <2; Class II >2.

Maxillary incisor axillary position should be 22* to NA and most anterior point should be 4mm ahead of NA. Facial surface of maxillary incisor should be 4-6mm ahead of vertical line through A point.

Mandibular incisor angulation to NB line should be 25* and most labial point of incisor should be 4mm anterior to the line.

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7
Q

Describe Ricketts analysis

A

Ricketts analysis uses maxillary depth. Measures angle at the intersection of FH line and NA line. Angle of 90 +/- 4* is ideal. Angle less than 86* indicates retrognathia, while angle greater than 94* indicates prognathism.

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8
Q

Describe McNamara analysis

A

Assess maxillary AP position: measures distance from A point to nasion perpendicular (a line that crosses N and is perpendicular to FH), normal range 0-1mm. A negative number indicates retrognathia, while a positive number greater than 1 indicates prognathism.

Assess mandibular AP position: measures the distance from Pog to N perpendicular. Ideal number for mixed dentition is -8 to -6mm, adult female -4 to 0mm, adult male -2 to 2mm.

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9
Q

Describe Downs analysis

A

Assesses AP position of mandible with facial angle.

Indicates relative AP position of mandible to cranium.

An angle formed by intersection of the facial line, N-Pog’ line and FH line. Mean is 82-95*.

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10
Q

What is Wits Appraisal?

A

Linear relationshiop between maxilla and mandible not influenced by cranium.

Points BO and AO established by dropping perpendicular lines from A point and B point onto occlusal plane.

BO 1mm ahead of AO in males

BO and AO coincide in females.

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11
Q

Describe mandibular plane angle. What is normal? What is associated with an increase or decrease in plane angle?

A

Mandibular plane angle (Steiner) formed between mandibular plane (Go-Gn) and anterior cranial base (S-N), normal value 32*

> 39* is high; <28* is low.

Increased plane angle = dolichocephaly, class II malocclusion, vertical maxillary excess, apertognathia.

Decreased plane angle: bradycephaly, skeletal deep bite, notched gonial angles.

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12
Q

What assessment is used for chin position?

A

Holdaway ratio:

Extend NB line to inferior border of the mandible and compare the distance between L1 (incisal edge of mandibular incisor) and Pog from this line.

1:1 is ideal in males and 0.5-1 in females.

Only of value if lower incisors are in the proper position!

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13
Q

Describe your technique for a LeFort Osteotomy

A
  • K wire nasofrontal suture
  • Maxillary vestibular incision from one zygomaticomaxillary buttress to the other (5mm non-keratinized mucosa for closure).
  • Bony exposure via FTMPF
  • Dissection to nasal aperture and protect nasal mucosa by lifting it up with freer elevator. Tunnel to pterygoid plates bilaterally.
  • Dissect ANS free, dissect nasal floor off palatal shelf using freer.
  • Horizontal cut from posterior maxilla to piriform rim bilaterally.
  • Nasal septum separated with nasal-guarded osteotome
  • Pterygoid plate osteotomies with pterygoid osteotome
  • Lateral nasal wall osteotomies with guarded osteotome.
  • Vertical interdental osteotomies at this time if multiple-piece.
  • Induced hypotension MAP 50-65 mm Hg
  • Downfracture
  • Trim bony interferences of septum and lateral walls
  • Ensure mobility of maxilla
  • If multi-piece, cut palatal paramedian osteotomy just lateral to nasal septum.
  • Place splint, MMF
  • Rotate maxillomandibular complex up. Grind as needed to achieve correct vertical position. Place maxilla into new position with plates at piriform and zygomaticomaxillary buttresses.
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14
Q

Bleeding sources LF1 osteotomy

A

Pterygoid plexus
Posterior superior alveolar artery
Greater palatine artery
Terminal branches of maxillary artery.

Note that internal maxillary artery is 25mm superior to the base of the junction of the pterygoid plates in a normal maxilla. Pterygoid osteotome is 15mm in height, leaving 10mm margin for safety.

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15
Q

How do you treat bleeding from the maxillary artery after LF1

A

Attempt pressure packing with gauze or hemostatic agent. If no resolution, try to identify vessel for cautery. If continues, consider IR intraoperative consult for embolization.

Prevention: in extremely small maxillas i.e. cleft or syndromic patient, pre-op CT angiogram may be useful to ID these vessels.

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16
Q

Anterior open bite after MMF release in LF1

A

Condyles were not seated in fossa or area of premature bone contact. Remove fixation, check for bony interferences, ensure passive condylar positioning, replace fixation.

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17
Q

Epiphora following LF1

A

More common in high LeFort osteotomies due to damage of nasolacrimal system, nasoseptal deviation, or swelling. If no resolution after 6 weeks, CT scan to r/o source. May require dacryocystorhinostomy or nasoseptoplasty depending on etiology.

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18
Q

How much transverse maxillary expansion can you achieve by dental tipping?

A

5mm with healthy periodontium and upright teeth.

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19
Q

Tipping vs. expansion children vs. adolescents

A

Orthodontic rapid palatal expansion takes advantage of growth potential in growing children and adolescents.

Transpalatal dental-borne and/or micro implant borne (MARPE) orthopedic expander opens midpalatal suture, tips teeth, and bends and remodels the alveolus.

Older patients have more sutural resistance that results in less expansion and more dental tipping, lateral tooth displacement, and periodontal defects.

Children 50% tipping and 50% expansion

Adolescents 65% tipping and 35% expansion

High relapse (40-60% depending on age) with up to 50% overcorrection recommended.

More widening at canines than molars (3:2)

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20
Q

Indications of SARPE

A

> 7mm expansion
Desire to avoid segmental maxillary surgery
Thin, delicate soft tissue with gingival recession in bicuspid-canine region
Significant nasal stenosis
Level occlusal plane
Constricted V-shaped arch form

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21
Q

Benefits of SARPE

A

Greater arch expansion
May avoid extractions
Better orthodontic alignment before definitive orthognathic surgery
Improved periodontal health, esthetics, and buccal corridor

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22
Q

Segmental LF vs. SARPE + single piece LF

A

For expansion >7mm, SARPE stability (30% relapse at canine and molars) far exceeds segmental LF and RPE (50% relapse).

More expansion at canines than molars due to lateral nasal wall and palatine bone resistance

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23
Q

Describe the SARPE surgical procedure

A

Maxillary vestibular incision from one zygomaticomaxillary buttress to the other. FTMPF.
Bilateral maxillary osteotomies from piriform rim to pterygomaxillary junction
Release of nasal septum
Dissect mucosal tunnel between #8-9 from alveolar crest to the nasal floor.
Use thin spatula osteotome between 8 and 9 and extend osteotomy to PNS.
Osteotomy of anterior portion of lateral nasal walls for 1.5cm.
Bilateral pterygoid plate osteotomies
Activate distractor to allow passive expansion of 3-4mm. Then decrease expansion to a bony gap of 0.5-1.5 mm at the end of the procedure.

Close any perforations of palatal tissue
Close with alar base cinch and V-Y closure

5-7 days latency
Rate and rhythm 0.25mm twice/day (0.5mm/day)

Palatal expansion within 4 weeks of surgery. Allow 4 months retention before removing expander.

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24
Q

Timing of SARPE. Latency, rate/rhythm, retention.

A

5-7 days latency
Rate and rhythm 0.25mm twice/day (0.5mm/day)
Palatal expansion must occur within 4 weeks of surgery. Allow 4 months of retention before removing expander.

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25
Q

Expansion is asymmeteric following SARPE. Explain.

A

Most common expansion complication. Results from incomplete release of the pterygomaxillary junction on one side. Half the time, the asymmetry self-corrects. Others may require segmental osteotomy to correct asymmetry at least 4 weeks after SARPE.

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26
Q

Expansion stops prematurely after SARPE

A

Can result in pain, dental tipping, periodontal breakdown, palatal tissue impingement by expansion device, relapse. Treat with adequate mobilization and removal of bony interferences.

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27
Q

How much can a BSSO be used to advance or set back the mandible?

A

Mandibular setback less than 8mm (greater potential for bony interference and posterior border step defect if proximal end of distal segment is passed beyond the posterior border of the proximal segment. Be cognizant of posterior airway space during planned setbacks.

Mandibular advancement of 12mm or more are unstable, more prone to relapse.

28
Q

What are the names of the modifications of the BSSO?

A

Hunsuck modification
Dal Pont modification
Epker-Schendel modification

29
Q

What is the Hunsuck modification?

A

Medial osteotomy of BSSO does not extend to posterior ramus as opposed to original Obwegeser medial cut. Allows for shorter split, less soft tissue stripping, and improved mandibular contour.

30
Q

What is the Dal Pont modification?

A

Vertical osteotomy on buccal cortex of the BSSO was advanced between 1st and 2nd molars allowing for greater advancement by allowing more bony contact surface area.

31
Q

What is the Epker Schendel modification?

A

Reduced stripping of the masseter and soft tissue of the medial ramus. Cut at inferior border to extend to lingual side, including entire inferior border in prosimal segment. Decreased postop swelling, hemorrhage, and reduced manipulation of NV bundle.

32
Q

Describe your technique for BSSO

A

Incision over anterior border of ramus into mandibular vestibule to second molar region, leave 5mm cuff of non-keratinized tissue to aid in closure.
-Dissection laterally with FTMPF over body of mandible to inferior border
-Dissection proceeds up anterior ramus to coronoid process, fibers of temporalis are freed.
-Medial dissection subperiosteally above the lingula and mandibular foramen to identify entrance of the nerve. Lingula usually at level of occlusal plane.
-Make medial corticotomy just above lingula. Extend anteriorly through ramus paralleling buccal plate to the medial aspect of the second molar region. Drop a vertical corticotomy in second molar region to inferior border.
-Chisels carefully used to slowly expand the corticotomies to split the mandible. IAN in distal segment.
- Release medial pterygoid attachment on distal segment for advancement with J stripper.
- Interim or final splint is placed and mandible is moved to new position.
- Pickle fork to push proximal segment posterior and superior. Eval alignment of inferior borders.
- Fixate with plates or screws.

33
Q

What is the best operation for V-shaped mandible (divergent ramus pattern) for setback

A

BIVRO

34
Q

What are indications for BIVRO vs. BSSO

A

-V-shaped mandible setback
-Concern about paresthesia (incidence is less)
-Very thin mandibular ramus with little marrow space (increased risk of unfavorable split with BSSO)
-Large mandibular asymmetrical prognathism. VRO may reduce incidence of segmental inteference on setback side.
-Patients who have symptomatic TMD pre-operatively that have not been addressed either non-surgically or with TMJ surgery (less potential pressure on intracapsular tissues).

35
Q

What are some downsides of BIVRO vs. BSSO

A

Need for period of IMF

36
Q

Describe IVRO technique

A

Incision over anterior border of ramus into mandibular vestibule.
FTMPF to expose lateral ramus to sigmoid notch.
-J stripper to release attachments at inferior border
-Bauer retractors into sigmoid notch and along inferior border. Some use Merrill-Levasseur retractor at posterior border.
-Identify antilingula as this marks the anterior limit of the osteotomy.
-Make vertical cut utilizing oscillating saw blade beginning in midramus region posterior to antilingula to the inferior border (if you cannot identify antilingula, cut 7-10mm anterior to posterior border).
- Superior osteotomy completed last as there is risk to the masseteric artery.
- On medial aspect, judiciously dissect subperiosteal pocket to accept overlapping segment.
- Establish occlusion with MMF (6 weeks).
- Rigid fixation with 2-3 screws can be done through trocar.

37
Q

Genioplasty surgical technique

A

Incision half the distance between vestibule and wet-dry line of the lower lip, canine-to-canine.
- Carry incision through mentalis to bony mandible.
- Subperiosteal dissection, identify mental nerves.
- Mark midline.
- Recip saw to make cut beneath mental foramen (5mm below foramina and 5mm below apices of teeth.
- Bony chin mobilized and repositioned.
- Close in layers (reapproximate mentalis to prevent ptosis of the chin).

38
Q

Hierarchy of Stability of Orthognathic Movements

A

STABLE
- Maxilla up
- Mandible forward
- Maxilla up/mandible forward
- Maxilla forward/mandible back
- Mandible back
- Maxilla down
- Maxilla wider
UNSTABLE

39
Q

Specific considerations for cleft orthognathics

A

-Intubation
-Incision design (vascularity)
-Osteotomy and downfracture
- Management of residual oronasal fistulas and altered nasal anatomy.
- Rigid fixation and bone grafting.

40
Q

Incision design in maxilla and vascularity in cleft orthognathics

A

-Shortened circumvestibular incision leaving large buccal soft tissue pedicle
-An anterior midline pedicle can be left with a vertical incision at midline and two lateral vestibular incisions
-Preserve descending palatine vessels if possible
- Note that small alveolar cleft graft may be weak and prone to fracture during mobilization and downfracture of the segment (consider palatal splint)

41
Q

When is cleft distraction osteogenesis considered?

A

Patients with large AP discrepancies that would not be possible to correct in a single orthognathic procedure or which would require unnecessary mandibular setback procedures.

42
Q

What is obstructive sleep apnea?

A

A sleep disorder characterized by obstructive apneas and hypopneas caused by collapse of the upper airway during sleep

43
Q

What is central sleep apnea?

A

The absence of respiration associated with an absence of respiratory effort

44
Q

What is polysomnography?

A

Polysomnography (PSG) is a diagnostic test used for evaluation of sleep disorders.

Components include EEG, EOG, EMG, ECG, and pulse oximetry

Gold standard in diagnosis of sleep apnea

45
Q

What is apnea?
What is hypopnea?

A

Apnea: cessation of airflow at the nostrils and mouth for at least 10 seconds while sleeping.

Hypopnea: reduction of airflow resulting in a drop in oxygen saturation followed by an arousal.
- 50% reduction in airflow for 10 seconds with a 3% drop in oxygen saturation
- 30% reduction in airflow for 10 seconds with a 4% drop in saturation

46
Q

What is the apnea hypopnea index?

A

AHI: the average number of apnea and hypopnea events per hour

47
Q

What is the respiratory disturbance index?

A

RDI: average number of apnea events, hypopnea events, and respiratory event related arousals (RERAs) per hour

RERA - an event that causes an arousal or decrease in oxygen saturation without qualifying as apnea or hypopnea

48
Q

What is Cheyne Stokes breathing?

A

A breathing pattern marked by crescendo-decrescendo changes in airflow and respiratory effort that often ends with apnea (typical of central sleep apnea syndrome).

49
Q

What is Mueller’s maneuver?

A

Inhalation with the nasal passages occluded and the mouth closed with an endoscope inserted through one nostril to observe the location of airway collapse.

50
Q

What is the Fujita classification?

A

A classification system developed to indicate the level of obstruction identified by nasopharyngoscopy in conjunction with a Mueller’s maneuver or during sleep-induced nasopharyngoscopy.

Type I: upper pharynx to include palate, uvula, and tonsils
Type II: upper and lower pharynx
Type III: lower pharynx to include tongue base, lingual tonsils, and supraglottic region.

Can guide what surgical interventions may be useful.

51
Q

AHI values for normal, mild, moderate, and severe OSA

A

Normal 0-4
Mild 5-15
Moderate 15-30
Severe >30

52
Q

Describe the cycles of sleep

A

Non-REM and REM sleep
N1, N2, N3 and finally to REM.

N1 lightest stage. Slow rolling eye movements and low-amplitude, mixed EEG frquencies.
N2 sleep spindles and K-complexes
N3 deep sleep with low-frequency, high amplitude EEG waves.

53
Q

REM sleep characteristics

A

Low voltage, mixed EEG pattern (saw-toothed waves), rapid eye movements, muscle atonia.

Phasic and tonic phases.

Delayed or suppressed by alcohol, sedative-hypnotics, barbiturates, antiepileptic drugs, beta antagonists, MAOIs, SSRIs, stimulants.

54
Q

Pathophysiology of OSA

A

Increased sympathetic tone, autonomic arousals.

Hypoxia – sleep fragmentation and restriction.

Hypoxia followed by oxygenation can lead to production of free radicals and endothelial damage via hypoxia-reperfusion injury. Activation of PMNs and release of inflammatory mediators.

Chronic inflammatory state.

55
Q

Medical conditions associated with OSA

A

Hypertension
Arrhythmias
CHF
MI
Stroke
Parkinson disease
Seizures
Diabetes
Cognitive function
Depression
Pulmonary hypertension
Acid reflux
Decreased wound healing
Headaches
Immune system impairment
Secondary polycythemia
Erectile dysfunction

56
Q

STOP BANG

A

Snore
Tired
Observed apnea
Pressure (treated for HTN)
BMI >35
Age >50
Neck circumference >16 inches
Gender (male?)

57
Q

Diagnostic aids for OSA

A

Nasopharyngoscopy
Mallampati classification
Drug-induced sleep endoscopy (DISE)
Polysomnography
Home Sleep Apnea Testing (HST)

58
Q

Stanford Protocol for OSA

A

Created to standardize surgical approach to OSA, prevent excessive operations.
- Surgery addresses site of obstruction
- Divided into two phases

Phase I (directed to site of obstruction) 61% success
- Nasal obstruction: septoplasty, turbinectomy, alar collapse, valve deformities, etc
- Retropalatal obstruction: UPPP with tonsillectomy
- Retrolingual obstruction: genioglossus advancement

Phase II
- MMA
- Tracheostomy

59
Q

What is a uvulopharyngopalatoplasty (UPPP)?

A

Reduction, tightening, and/or repositioning of the soft palate and related oropharyngeal structures as well as removal, reduction, or reconfiguration of the uvula.

50% success rate for OSA
Complications: pain, post-op bleeding, dysphagia, nasal regurgitation, velopharyngeal incompetence, subjective globus.

Can have effect on ability to move maxilla forward during MMA.

60
Q

What is a hyoid suspension?

A

Used for patients with low-lying hyoid bone.

Advance and stabilize hyoid to thyroid cartilage or inferior border of mandible. This advances the hyoglossus muscle and increases posterior airway space.

Can cause dysphagia, infection, rupture of hyoid suspension sutures with relapse and voice changes.

61
Q

General maxillofacial exam of orthognathic patient (outline of things to mention)

A

SKELETAL
1. TRANSVERSE (midlines, chin point, occlusal plane cant, arch widths)

  1. ANTEROPOSTERIOR (facial profile, nasolabial angle, chin position/size, labiomental angle)
  2. VERTICAL (upper/middle/lower facial thirds)

TMJ
1. Range of motion, deviation

DENTAL
1. Angle’s classification
2. OB/OJ
3. Crossbite
4. Arch form (level/crowding?)

SOFT TISSUE
1. Upper lower lip thickness, length, eversion, mentalis strain.

62
Q

Normal nasion-ANS : ANS-menton ratio

A

7:8

63
Q

Blood supply to the maxilla

A

Branches of external carotid system:
Ascending palatine
Ascending pharyngeal
Palatine
Nasopalatine
Posterior superior
Infraorbital

64
Q

Maxillary artery location with respect to pterygoid plates

A

Internal maxillary artery 25mm superior to the most inferior junction of the maxilla and the pterygoid plate, leaving a 1cm margin of safety if a 15mm wide curved osteotome is used.

65
Q
A