4.1.1 Communicable Diseases Flashcards

1
Q

Bacteria (pathogen)

A
Prokaryotes 
Shapes:
• bacilli (rod)
• cocci (sphere)
• vibrios (comma shape)
• spirilla (spiral)
• spirochaetes (corkscrew shape)

Cell walls:
• Gram positive bacteria (purple/blue stain)
• Gram negative bacteria (red stain)

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2
Q

Viruses

A

Non-living

Invade living cells, host cell

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3
Q

Protoctista (Protista)

A

Eukaryotic
Wide variety of feeding methods
Use animals as host
May need a vector

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4
Q

Fungi

A

Eukaryotic
Multicellular
Digest food exracellularly before absorbing nutrients
Saprophytes - feed on dead/decaying matter
Produce spores which can spread

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5
Q

Pathogens; damaging host directly

A
  • viruses take over cell metabolism

* fungi digest living cells and destroy them

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6
Q

Pathogens; producing toxins to damage host tissues

A
  • most bacteria, toxins may break down cell membranes

* some fungi

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7
Q

Plant diseases; ring rot

A

Bacterial

No cure

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8
Q

Plant diseases; tobacco mosaic virus

A

Virus

No cure but resistant crop strains are available

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9
Q

Plant diseases; potato blight

A

Fungus
Host cells
No cure but resistant strains, management and chemicals used to reduce risk

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10
Q

Plant diseases; black Sigatoka

A

Fungus
No cure
Resistant strains being developed
Fungicides

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11
Q

Animal diseases; tuberculosis

A

Bacterial
Destroys lung tissue
Suppresses immune system

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12
Q

Animal diseases; bacterial meningitis

A

Bacterial
Infection of meninges of brain
Antibiotics will cure is early
Vaccines

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13
Q

Animal diseases; HIV/AIDS

A

Virus

Destroys immune system

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14
Q

Animal diseases; influenza

A

Viral
Kills ciliated epithelial cells
Flu vaccine
No cure

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15
Q

Animal diseases; malaria

A
Protoctista 
Mosquitos 
No vaccine 
Limited cures 
Control vectors
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16
Q

Animal diseases; ring worm

A

Fungal

Antifungal creams

17
Q

Animal diseases; athlete’s foot

A

Fungal

Antifungal creams

18
Q

Direct transmission in animals

A
  • direct contact
  • inoculation (break in skin)
  • ingestion
19
Q

Indirect transmission in animals

A
  • fomites (bedding,socks)
  • droplet infection / inhalation
  • vectors
20
Q

Factors affecting transmission of communicable diseases in animals

A
  • overcrowding
  • poor nutrition
  • HIV
  • climate change
  • culture and infrastructure
  • socioeconomic factors
21
Q

Direct transmission in plants

A

Contact of healthy plant w diseases plant

22
Q

Indirect transmission in plants

A
  • soil contamination

* vectors (wind,water,animals)

23
Q

Factors affecting the transmission of communicable diseases in plants

A
  • over crowding
  • poor mineral nutrition
  • damp, warm
  • climate change
24
Q

Plant defended against pathogens; recognising an attack

A

Receptors respond to molecules

Defensive chemicals give alarm to other cells before they’re attacked

25
Plant defended against pathogens; physical defences
Produce high levels of callose - act as barrier to pathogens entering
26
Plant defended against pathogens; chemical defences
* insect repellant * insecticides * antibiotics * antifungal compounds
27
Animals; non-specific defences — Keelung pathogens out
* skin * mucous membranes * lysozymes in tears and urine Blood clotting and repair • thromboplastin • serotonin Inflammatory response • mast cells activated, release histamines and cytokines • histamines make blood vessels dilate, more leaky • cytokines attract white blood cells
28
Animals; non-specific defences — Getting rid of pathogens
* fevers - high temp inhibits pathogen production | * phagocytosis - wbc engulf and destroy pathogens (neutrophils/macrophages)
29
The stages of phagocytosis
1) pathogens produce chemicals that attract phagocytes 2) recognise non-human proteins 3) phagocyte engulfs pathogen, eclipsed in vacuole called phagosome 4) phagosome combines w lysosomes forming a phagolysosome 5) enzymes from lysosome digest and destroy pathogen
30
Helpful chemicals
Cytokines - cell signalling molecules, can stimulate specific immune system Opsonins - ‘tag’ pathogens so their easily recognised
31
Animals; specific immune system — antibodies
Y shape = immunoglobulins Form antigen-antibody complex Hinge region provides flexibility to bind to two separate antigens
32
How antibodies defend the body
1) acts as oosonin, easily engulfed and digested 2) can’t invade once attached to antibody 3) a.b act as agglutinins cause complex’s to clump together - prevent spread, easily engulf 4) act as anti-toxins by binding to toxins
33
Animals; specific immune system — Lymphocytes and the immune response
B lymphocytes - Bone marrow T lymphocytes - Thymus gland •T helper - bind to surface antigens, produce interleukins that stimulate activity of B cells •T killer - produce perforin to destroy pathogen by making holes in cell membrane •T memory - part of immunological memory •T regulator - suppress immune system to control and regulate it •B Plasma cells - produce antibodies •B effector cells - divide to form the plasma cell clones •B memory cells - immunological memory, remember specific antigen
34
Animals; specific immune system — Cell mediated immunity
T lymphocytes respond to cells that have been changed in some way 1) phagocytosis 2) receptors of some T helper fit the antigens. These become activated and produce interleukins which stimulates more T cells to divide 3) clones T cells may develop into memory, produce interleukins, or stimulate development of clone T killer cells 4) cloned plasma cells produce a.b to bind to pathogen = primary immune response