3a (exchange/transport) Flashcards

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1
Q

do bigger or smaller animals have a larger surface area to volume ratio?

A

smaller organisms

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2
Q

what is fick’s law?

A

rate of diffusion = (surface area x conc diff) / thickness of membrane

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3
Q

how do you work out surface area to volume ratio?

A

surface area/volume

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4
Q

how do single celled organisms exchange substances? is this fast or slow?

A

diffusion directly into or out of cell across the cell surface membrane
fast rate due to short diffusion pathway
dont have gas exchange system

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5
Q

why is diffusion across the outer membrane in multicellular organisms too slow? how is this combated?

A

some cells are deep in the body so big distance to the outside environment
larger animals have smaller sa:v and its hard to exchange enough substances to supply a large volume animal across a small outer surface
so, instead of just diffusion, multicellular organisms have exchange organs and mass transport systems

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6
Q

what is a mass transport system? what is this in animals and plants

A

a efficient system to carry substances to and from cells
mammals- circulatory system
plants- xylem and phloem

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7
Q

factors affecting heat exchange

A

body size- rate of heat loss depends on sa:vol (larger sa;vol means heat lost more easily so smaller organisms need faster metabolic rate to generate enough heat to stay warm.)

body shape- compact shape means small sa:vol so minimised heat loss from surface. vice versa. if animal is compact or nor depends on temperature of its environ (eg arctic fox has smaller sa:vol than african bat-eared fox)

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8
Q

behavioural and physiological adaptations to aid exchange

A

animals w high sa:v lose more water so some small desert animals have kidney structure adaptations - produce less urine

to support high metabolic rates, small animals in cold places eat high energy food

some smaller mammals have thick layer of fur/ hibernate in cold

larger organisms in hot places have larger ears to increase sa to lose more heat/ spend lots of time in water

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9
Q

3 major adaptations of gas exchange surfaces

A

large surface area
thin walls- short diffusion pathway
also maintain conc grad

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10
Q

gas exchange in fish

A

counter current flow
1. water enters mouth and out gills
2. blood flows through lamellae one way and water the other- maintains large conc gradient between water and blood (always higher conc of oxygen in water so as much amount of oxygen diffuses into blood as possible

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11
Q

what is the structure of gills?

A

made of lots of thin plates called gill filaments which provide large sa
gill filaments covered in lamellae which increase sa more
lamellae have lots of blood capillaries and thin surface layer of cells to increase diffusion rate

pictures on page 60 of revision guide
check book

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12
Q

gas exchange in insects

A

microscopic air filled pipes called tracheae (supported by strengthened rings) which are used for gas exchange
air moves into them through spiracles (pores on surface)
oxygen travels down conc gradient to cells
trachea branch into tracheoles which have thin permeable walls and go to individual cells- oxygen diffuses directly into respiring cells
co2 moves down conc gradient to spiracles to be released into atmosphere
rhythmic abdominal movements to move air in and out spiracles
fluid at end of tracheoles drawn into anaerobically respiring muscles (low water pot as has lactic acid) so vol of tracheoles increase so pressure decrease so air is drawn further into tracheoles

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13
Q

why are insects small?

A

rely mostly on diffusion to exchange gases so need small pathway so size of growth is limited

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14
Q

gas exchange in plants

A

main exchange surface is the surface of mesophyll cells- large sa
air spaces- gases come into contact often with mesophyll cells- conc grad
mesophyll cells are inside leaf, gases move in and out of leaf through stomata in epidermis short diffusion pathway
stomata opened and closed by guard cells to get balance between loss of water and exchange of gases

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15
Q

structure of leaf

A

waxy cuticle
upper epidermis
palisade mesophyll
spongy mesophyll and xylem and phloem
lower epidermis with stomata and guard cells
waxy cuticle

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16
Q

how do insects prevent loss of water?

A

close spiracles
waterproof waxy cuticle and tiny hairs around spiracles to prevent evaporation

17
Q

how do plants prevent loss of water?

A

stomata open in day for gas exchange
water enters guard cells making them turgid which opens stomata
if plants get dehydrated, guard cells become flaccid which closes the pores

18
Q

what is a xerophyte? 5 examples of their adaptations?

A

plants adapted for warm, dry, windy environments where water loss is a prob
eg cacti, marram grass
1. stomata in sunken pits that trap water vapour reducing conc gradient of water in air and leaf so less water diffuses out of leaf and evaporates
2. layer of hairs on epidermis to trap water vapour around stomata
3. curled leaves with stomata inside- protect them from wind so decrease evaporation
4. fewer stomata so fewer places for water to escape
5. waxy, waterproof cuticles on leaves and stems to reduce evaporation

19
Q

what is the gas exchange system in humans? structure?

A

lungs
air enters trachea (supported by c shaped rings of cartlige) which splits into 2 bronchi (1 bronchus going to each lung). each bronchus branches off into bronchioles which end in alveoli (small air sacs where gases are exchanged)
ribcage, intercostal muscles and diaphragm all work together to move air in and out

20
Q

what does ventilation consist of?

A

inspiration (breathing in)
expiration (breathing out)

21
Q

what happens in inspiration?
is it active or passive?

A

external intercostal and diaphragm muscles contract internal relax
so ribcage moves up and out and diaphragm flattens
this increases volume of thoracic cavity
so lung pressure decreases below atmospheric pressure
air flows from high pressure to low so flows down trachea into lungs
active process requires energy

22
Q

what happens in expiration?
isit active or passive?

A

external intercostal and diaphragm muscles relax internal contract
so ribcage moves down and in and diaphragm curves
this decreases volume of thoracic cavity
so lung pressure increases above atmospheric pressure
air is forced down pressure gradient so out of lungs
normally passive so doesnt require energy
but can be forced

23
Q

what happens in forced expiration?

A

external intercostal muscles relax and internal intercostal muscles contract- pulling ribcage further down and in
the movement of the 2 sets of intercostal muscles is antagonistic

24
Q

how do you work out pulmonary ventilation?

A

tidal volume (dm-3) x breathing rate (/min)

25
Q

what are alveoli made of?

A

single layer of thin, flat cells called alveolar epithelium

26
Q

how does gas exchange occur in alveoli?

A

lots of alveoli so large sa
alveoli surrounded by capillary network
o2 diffuses out of alveoli, across alveolar epithelium and the capillary epithelium into haemoglobin in the blood
co2 diffuses into alveoli from blood and is breathed out

27
Q

how are alveoli adapted for efficient gas exchange?

A

thin exchange surface- alveolar epithelium is 1 cell thick so short diffusion pathway
large sa- lots of alveoli
steep conc gradient of o2 and co2 between alveoli and capillaries maintained by flow of blood and ventilation

28
Q

tidal volume

A

volume of air in each breath
0.4-0.5 dm3 for adults

29
Q

ventilation rate

A

number of breaths per min
15 at rest if healthy

30
Q

forced vital capacity

A

max vol of air possible to breathe forcefully out of lungs after really deep breath in

31
Q

forced expiratory volume 1

A

max vol of air that can be breathed out in 1 second

32
Q

TB
(pulmonary tuberculosis)

A

bacteria (spread in drops etc)
immune system cells build a wall around bacteria in lungs forming tubercules (lumps)
infected tissue dies and gas exchange surface is damaged so tidal vol decreases and have to breathe faster to get more o2 (faster ventilation rate)
can cause fibrosis
symptoms- persistent cough, coughing blood, chest pain, shortness of breath, fatigue

33
Q

Fibrosis

A

formation of scar tissue in lungs (thicker and less elastic than normal) due to infection/ asbestos/dust
lungs cant expand as well so tidal vol and FVC decreases
reduction in gas exchange as thick membrane
symptoms- shortness of breath, dry cough, chest pain, fatigue, weakness
faster ventilation rate

34
Q

Asthma

A

inflamed airways often due to allergic reaction to pollen/dust
in asthma attack, smooth muscle lining bronchioles contracts and mucus produced which constricts airways. air flow reduced and FEV1 reduced
symptoms- wheezing, tight chest, shortness of breath
relieved by drugs which open airways my causing muscles to relax

35
Q

emphysema

A

caused by smoking/ air pollution when foreign particles get trapped in alveoli
causes inflammation and attracts phagocytes to area where they release enzymes that break down elastin in walls of alveoli
wo this, alveoli cant return to normal shape after exhalation so cant expel air well
also reduces sa of alveoli so gas exchange decreases
symptoms- shortness of breath, wheezing, increased ventilation rate

36
Q

lung cancer

A

see research homework
also for causes/ risk factors, symptoms and treatments of all

37
Q

why do the lung diseases cause tiredness?

A

reduced rate of gas exchange so less o2 diffuses into blood so cells receive less o2 so aerobic resp rate decreases so less
energy released

38
Q

what is a risk factor?

A

factor that increases someones chance of getting disease
correlation not cause

39
Q

practicals in this topic

A

dissecting gas exchange systems