37. Electrophys Flashcards
intraop 5 lead
II
V5
intraop 5 lead covers
80% of ischemic changes
lead II
MEA
RV
inf LV
V5
lateral LV
polarized state is mx by
Na+/K+ atpase
what propagates AP through myocardium
Na+/Ca2+ channels
what is involved with excitiation/contraction coupling
Ca2+
what is involved with myosin power stroke
Ca2+
right heard leads
V1
V2
interventricular septum leads
V3
V4
left heart leads
V5
V6
inotropy
speed and strength of cardiac contraction
lusitropy
relaxation and ventricular filling
issue with inotropy causes
systolic heart failure
issue with lusitropy causes
diastolic heart failure
1st degree AV block: cause
athlete
digoxin
B blocker
CCB
ischemia
MD
1st degree AV block: avoid
vagal stimulation
electrolyte abnormalities
ischemia
1st degree AV block
2nd degree AV block type 1
2nd degree AV block type 2
3rd degree AV block
adams-stoke attack
sudden fainting spells due to drop in CO
(3rd degree block)
RBBB ecg
wide QRS > 120 ms in leads 1/2
deep S in leads 1/V6
RBBB
Left BBB Ecg
wide QRS > 120 ms
no Q wave in 1, V5, V6
broad/notched R wave in 1, aVL, V5, V6
LBBB indicates
ischemic serious heart disease
lab for ischemia
troponin C
LBBB
tachydysrhythmia with narrow QRS
ABOVE BOH:
sinus tach
a fib
a flutter
junctional tach
paroxysmal atrial tach
accessory pathway rachycardia
wide complex tachycardia
BELOW BOH:
V TACH
SVT w/intraventricular defect
SVT w/aberrant conduction
SVT w/preexcitation pathway
arrythmia initiation mechanism
- incr automaticity
- reentry through abnormal pathway
- trigger of potential after depolarization
what can induce automaticity
any myocyte
what sets the normal value
the inerrant baseline automaticity
what decreases timeframe for depolarization
phase 4 shift
sinus tachycardia
PAC
paroxysmal SVT
orthodromic
more common
narrow QRS
no delta