36. Vascular/Pericardial Simulation Flashcards
MI occurs when
O2 supply < O2 demand
PMI ECG changes
Q wave (leads V1-V3)
>40 ms wide
>2 mm deep
>25% depth of QRS
ST segment abnormality
Septal ST segment
V1-2
anterior ST segment
V3-4
Lateral ST segment
1+aVL
V5-6
Inferior ST segment
2
3
aVF
right ventricular ST segment
V1
V4R
posterior ST segment
V1-3 depression
most common ECG abnormality
ST segment
Right Coronary Artery ECG lead
2
3
aVF
RCA myocardial area affected
RA
RV
SA node
inferior LV
AV node
circumflex CA ECG lead
1
aVL
Circumflex myocardial area affected
lateral LV
LAD ecg lead
V3-5
LAD myocardial area affected
anterolateral aspect of LV
high risk surgery
abdominal aortic aneurysm
peripheral vascular
thoracotomy
major abdominal
ischemic heart disease
hx of MI
hx of positive exercise stress test
angina
nitrates
Q wave
congestive heart failure
hx of CHF
hx of pulm edema
hx of paroxysmal nocturnal dyspnea
rales
S3 gallop
CXR w/pulm vascular redistribution
cerebrovascular disease
hx of VA
hx of TIA
renal function
pre-op Cr >2mg/dL
diabetes
insulin dependent
PMI management
prevent MI
avoid hyperventilation
baseline HR/BP
avoid sympathetics
events that decr O2 delivery
decr coronary BF
tachycardia
hypotension
hypocapnia
CA spasm
decr O2 content (FiO2)
anemia
arterial hypocemia
Left shift O2-Hb curve
events that incr O2 requirements
sympathetics
tachycardia
HTN
incr inotropy
incr afterload
incr preload
when do you treat PMI
1mm change in ST segment
PMI medications
NTG
esmolol
inotropics
what do you give for elevated afterload
NTG
what do you give to lower O2 demand
esmolol
what do you give for hypotension
inotropics
PMI post-op avoid:
shivering
pain
hypoxemia
hypercarbia
sepsis/infection
hemorrhage/anemia
aneurysms are asociated with
marfans
ehler danos
bicuspid aortic valve
family history
threshold for aortic aneurysm operations
> 5cm
aortic dissection is associated with
HTN
bicuspid aortic valve
TOF
atherosclerosis
trauma
bypass
pregnancy
weight lifting
abdominal aortic aneurysm rupture triad
hypotension
severe back pain
pulsatile abdominal mass
what improves survival from aortic aneurysm
short time from rupture to OR
ascending aorta dissection treatment
surgery
aortic arch dissection treatment
surgery
bypass if innominate involved
what artery requires bypass if dissected
innominate artery
descending aorta dissection treatment
medical management unless rupture is imminent
cause of anterior spinal artery syndrome
aortic cross clamping
anterior spinal artery syndrome symptoms
flaccid paralysis
motor dysfunction
acute back pain
autonomic dysfunction
ways to decrease anterior spinal artery syndrome
limit cross clamp to <30 min
incr BF
aortic cross clamp: SVR proximal
incr SVR proximal to clamp
aortic cross clamp: HR
no change
aortic cross clamp: volume
venous vasoconstriction
incr CVP
incr PAP
incr PAWP
aortic cross clamp: distal SVR
decr distal SVR
what is perfusion dependent on in aortic cross clamp?
proximal pressure
MAP above cross clamp
100mmHg
MAP below cross clamp
50 mmHg
vasodilators during aortic cross clamp
incr ICP
decr distal PP
SEPs
sensory neurons
dorsal cord
MEPs
motor neurons
ventral cord
LP drain/monitor
< 10 cmH20
promotes BF
aortic clamping
vasocnostriction
incr venous return
incr lung volume
incr intracranial blood vol
HTN
aortic un-clamping
peripheral blood pooling
central hypovolemia
decr venous return
hypotension
mannitor
incr renal BF
incr GFR
risks during unclampiong
cold
acidosis
clot formation
should you use a bair hugger during aortic cross clamping
no lower body
what should you minimize during aortic cross clamping
sympathetics
what type of aortic endovascular repair is more difficult
thoracic
why is thoracic endovascular repair difficult?
greater pressure
tortuous anatomy
higher complication risk
optimal fluid and BP state for aortic cross clamp
euvolemic
normotension
cerebral blood flow
2 internal carotid
2 vertebral arteriesw
hat do the vertebral arteries combine to form
basilar artery
where is the highest amount of plaque build up
carotid bifurcation
what has the majority of cerebral blood flow
internal carotids
hemorrhagic stroke cause
vascular deformity
trauma
coagulopathy
ischemic stroke cause
thrombotic
embolic
what type of stroke is most common with carotid disease
ischemic
when is surgery indicated for carotid artery stenosis
70-99% occlusion indicates carotid endarterectomy
carotid artery stenosis diagnosis
auscultation
angiography
CT
MRI
doppler
pts with carotid artery stenosis will present with what during auscultation?
carotid bruit
CEA pts usually have what
severe CAD
– incr intraop MI risk
HTN
CEA regional
real time neuro assessment
cervical plexus block
CEA general
mx hemodynamics
carotid sinus body cranial nerves
CN IX
CN X
stump pressure
art line in common carotid placed by surgeon
cerebral oximetry measures
tissue O2 saturation
tissue O2 saturation reflects
Hb saturation in arterial, venous, and capillaries
tissue Hb is _____% venous and ____% arterial in the cerebral cortex
tissue Hb is 70% venous and 30% arterial in the cerebral cortex
CAE cardiac complications
HTN
hypotension
MI
CAE neuro complications
stroke
thrombosis
hypoglosssal dysfunction
recurrent laryngeal dysfunction
superior laryngeal dysfunction
hypoglossal dysfunction
motor efferent to tongue
recurrent laryngeal dysfunction
vocal cord paralysis
hoarness
change in pitch
noisy breathing
superior laryngeal dysfunction
phonation changes
pericardial sac
fibrous sac that produces fluid to lubricate movement of heart
normal pericardial sac volume
15-50 mL
is the pericardial sac essential for life?
no
pericarditis
recurrent frictional rub of pericardial sac
how can you differentiate between pericarditis and MI
pericarditis does not improve with NTG
pericardial effusion
accumulation of fluid within the pericardial sac
causes of pericardial effusion
cancer
infection
MI
trauma
drugs
radiation
autoimmune
cardiac tamponade
fluid pressure = cardiac pressure
cardiac tamponade is independent of
volume of fluid
what determine physiologic manifications of tamponade
timeframe of fluid accumulation
cardiac tamponade symptoms
incr CVP
pulsus paradoxus
equalization of cardiac filling P
hypotension
blunted ECG
sympathetic activation
esophageus compression
dysphagia
hiccups
tracheal compression
dyspnea
hoarsness
deviation
lung compression
cough
cardiac tamponade diagnosis
chest XRay
CT
TTE/TEE
kussmaul’s sign
distension of jugular veins during inspiration
beck’s triad
muffled heart sounds
incr jugular venous pressure
hypotension
pulsus paradoxus
exaggerated ventricular interdependence
decr >10 mmHg SBP during respiration
cardiac tamponade echo sizes
small: <10 mm
med: 10-20 mm
lg: > 20 mm
mild/small cardiac tamponade treatment
observation
symptomatic cardiac tamponade treatment: emergent
pericardiocentesis
symptomatic cardiac tamponade treatment: chronic
pericardial window
should you do GA in emergent tamponade pt
No - needle decompression first, then surgery
cardiac tamponade goals
good CO
inotropy
chronotropy
mx SV
cardiac tamponade avoid
myocardial depression
hypovolemia
hypervolemia
arterial vasodilation
bradycardia
PPV
what induction drug is best for cardiac tamponade
ketamine
pericardial decompression syndrome
profound hypotension
acute HF
pulm edema