3.6 Organisms Respond To Changes In Internal And External Environments Flashcards

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1
Q

What is a stimulus and why is it important to respond to them?

A

•A detectable change in the environment. These changes can be detected by cells that are called receptor.
•Increases chance of survival

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2
Q

What is kinesis?

A

•if organism moves to an area with harmful stimuli= increase the rate it changes direction to return to the favourable conditions quickly
•if it is in an area with beneficial stimuli it decreases speed
•non-directional response to unfavourable conditions

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3
Q

What is taxis?

A

A directional response, where they move towards or away from a stimulus

E.g -ve chemotaxis = moving away from beneficial chemical
/ +ve phototaxis = moving towards light

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4
Q

Why do organisms respond to temperature and humidity via kinesis?

A

Less directional stimuli where there is often no clear gradient from one extreme to the other

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5
Q

What is the effect of IAA in the shoot tip?

A

•in unilateral light, IAA will diffuse towards the shaded side
•it causes the cells to elongate more and this will bend the top to the light source
=positive phototropism

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6
Q

What is the effect of IAA in the root tip?

A

•IAA will move to the lower side of the root
•this will inhibit cell elongation so that roots anchor into the soil
= positive gravitropism and negative phototropism

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7
Q

What are the events involved in a response?

A

1.stimulus= change in internal/external environment
2.receptor= detects stimulus and respond by producing action potential in neurones
3.sensory neurone= carries impulses from receptor to CNS
4.coordinator=in CNS where info is interpreted
5.motor neurone= impulse from CNS to effector (gland/muscle)

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8
Q

What are the features of the endocrine system?

A

•widespread action
•hormones transported in blood
•long lasting response
•slow effect

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9
Q

What are the features of the nervous system?

A

•localised in a specific area
•neurotransmitters (chemical coordinator)
•short lived
•rapid effect
•impulses directly to target cells

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10
Q

What is a reflex action?

A

•rapid, involuntary response to a stimulus
•innate (not learned)

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11
Q

Why are reflexes important?

A

•increase survival =escape predators
•role of homeostasis
•leave brain free to carry out complex responses
•protect body from harm
•fast as neurone pathway is short

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12
Q

What are the structures of a neurone and their functions?

A

•cell body
•dentrites: receive info and carry it towards cell body
•axon: transmit impulses away from cell body
•myelin sheath: fatty material which insulates the axon so no loss of impulses or crossing over occurs
•Schwann cells wrap around axon to form myelin sheath, gap between called nodes of ranvier
•axonites

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13
Q

How is a resting potential established?

A

•3Na+ out and 2K+ into the axon by active transport via the Na-K pump
•electrochemical gradient produced = K+ diffuse out and Na+ in by facilitated diffusion
•membrane is more permeable to K+ as more K+ channels so more K+ moves out = -70 mv inside the axon

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14
Q

What are the steps involved in an action potential?

A

Depolarisation:
•neurone stimulated opens voltage gates Na+ channels
•+ve feedback = more channels open
•Na+ flood down conc. gradient into axon = +40 mv in axon

Repolarisation:
•+40 mv reached, Na+ voltage gated channels close
•K+ voltage gated channels open and K+ flood down conc. gradient out of axon so -70 mv in axon

Hyper polarisation:
•too many K+ move out of axon so temporarily more -ve than -70 mv

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15
Q

What is the all-or nothing principle?

A

•if depolarisation < -55mv= no action potential
•all at -55 mv will trigger a depolarisation of same magnitude to +40 mv max
•bigger stimuli increases the frequency of action potentials

This is important as only large stimuli are responded to for increased survival

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16
Q

What is the refractory period and why is it important?

A

(Action potential cannot be stimulated right away after one already)=time delay between AP
•action potentials are separate form one another (distinct)
•AP will travel in one direction
•limits frequency of AP so prevents over reaction to a stimulus so senses not overwhelmed

17
Q

Why does a myelinated axon conduct impulses faster than non-myelinated axon?

A

•myelin sheath is an electrical insulator
•in myelinated, action potential (depolarisation) can only occur at the node
•nerve impulse jumps from node to node
•action potential does not travel along the whole length of the axon
(Saltatory conduction)

18
Q

Give the structure of a synapse and of a neurotransmitter junction

A

•synaptic cleft
•neurotransmitter (acetylcholine)
•pre and postsynaptic neurone
•neuro receptor: membrane of the post synaptic neurone has chemical gated ion channels

19
Q

What happens at the synapse?

A
  1. An action potential arrives at the synaptic knob. Depolarisation of synaptic knob leads to opening of Ca2+ channels and Ca2+ diffuses in
    2.vesicles with neurotransmitter move towards and fuse with the pre synaptic membrane and is released in the cleft
    3.neurotransmitter diffuses down a conc. gradient to post synaptic membrane (binds by complementarity of shape of receptors)
    4.Na+ ion channels on post synaptic membrane open and Na+ diffuses in (above threshold= depolarisation)
    5.neurotransmitter is degraded and released from receptor (back to pre synaptic neurone to be recycled), Na+ channels close and the post synaptic= resting potential
20
Q

What is different in a cholinergic synapse?

A

•neurotransmitter is acetylcholine and will bind to receptors on post synaptic membrane
•it is broken down to acetyl and choline, moving back to pre synaptic knob.
•ATP energy is used to recombine it and it is stored in vesicles
•more acetylcholine can be made by the smooth endoplasmic reticulum

21
Q

Compare cholinergic synapses and neuromuscular junction

A

NJ CS
•unidirectional as receptors only on post synaptic membrane
•excitatory |•excitatory or
|inhibitory
•motor neurone|•2 neurones
to muscles |
•end point for AP|•new AP
•acetylcholine |•acetylcholine binds to muscle |binds to post
fibres | synaptic
|membrane

22
Q

Why are transmissions involved in synapses unidirectional?

A

•vesicles only released in the pre synaptic neurone
•receptors only on post synaptic neurone so only bind to one side

23
Q

What is temporal summation?

A

One presynaptic neurone releases neurotransmitter repeatedly over a short period of time to add up enough Na+ diffusing to exceed threshold

24
Q

What is spatial summation?

A

Many different pre synaptic neurones collectively trigger a new action potential by combining the neurotransmitter they release to exceed the threshold

25
Q

What occurs during inhibition by inhibitory synapses?

A

•Cl- will move into and some K+ move out of the post synaptic neurone
•-ve inside and +ve outside = -80 mv (hyperpolarisation so action potential is unlikely)

26
Q

How does the diameter of the axon affect speed of conductance?

A

•wider diameter, faster speed of conductance
•larger the diameter, lower the resistance to the flow of ions so faster wave of depolarisation travels along the neurone
•there will be less leakage of ions so action potentials travel faster

27
Q

How does temperature affect speed of conductance along the axon?

A

1.ions diffuse faster as more KE energy
2.enzymes involved in respiration work faster = so more ATP for active transport in the Na/K+ pump

28
Q

How does the pacinnian corpuscle work and where?

A

1)mechanical stimulation (pressure) deforms stretch mediated Na+ channels on the membrane of the ending of the sensory neurone
2)stretch mediated Na+ channels widen and allow some Na+ to diffuse into the sensory neurone
3)generator potential is produced
4)leads to an action potential in the neurone if threshold is met

Located in joints, tendons, skin and ligaments

29
Q

Why is the pacinian corpuscle described as a transducer?

A

Transfers energy from mechanical to electrochemical energy (stimuli into a generator potential)

30
Q

What are transducers and e.g?

A

Convert light energy to electrical energy e.g rod and cone cells

31
Q

What are rod cells and function?

A

•rhodopsin pigment is broken down by low light levels to generate action potential
•sensitive to light as many rod synapses with one bipolar neurone (retinal convergence) so neurotransmitters add together to reach threshold = action potential
•low acuity as different stimuli may produce only one action potential

32
Q

What are cone cells and function?

A

•iodopsin pigment is broken down by high light levels to generate an action potential
•3 different types of cells each sensitive to different wavelengths of light
•low sensitivity as one cone synapse with bipolar cell so lots of light is needed to release enough neurotransmitter to reach threshold
•high acuity as each cone synapses with one bipolar cell so will produce one action potential for each stimulus (can distinguish between separate light sources close together)