325 e 2 Flashcards

1
Q

process of regulating the ECF volume, body fluid osmolality, and plasma concentrations of electrolytes

A

fluid and electrolyte balance

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2
Q

keeps the volume, osmolality, and electrolyte concentrations of fluids in body fluid areas within the normal range

A

optimal fluid and electrolyte balance

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3
Q

intake and absorption of F and E matches the output of fluid

A

optimal balance

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4
Q

major ECF cation

A

sodium

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5
Q

135 to 145

A

sodium range

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6
Q

present in most body fluids or secretions

A

sodium

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7
Q

major role in maintaining the concentration and volume of ECF

A

sodium

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8
Q

essential for maintence of acid base and fluid

A

sodium

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9
Q

maintenance of active and passive transport mechanisms, and irritability and conduction of nerve and muscle tissue

A

sodium

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10
Q

3.5 to 5

A

POTASSIUM

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11
Q

vital role in cell metabolism and transmission of nerve impulses

A

potassium

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12
Q

helps with functioning of cardiac, lung and muscle tissues and acid base balance

A

potassium

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13
Q

reciprocal action with sodium

A

potassium

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14
Q

major ICF cation and major factor in resting membrane potential of nerve and muscle cells

A

potassium

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15
Q

cause clinical problems

A

potassium changes

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16
Q

moves fluid into cell

A

hypotonic

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17
Q

moves fluid out of cell

A

hypertonic

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18
Q

equal balance…dont effect cell fluid shifts

A

isotonic

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19
Q

infants and old people

A

RF for imbalances

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20
Q

impaired ability to conserve water(less lean muscle mass); blunted thirst sensation; decreased renal reserve(less able to respond to ADH)

A

older population and imbalances

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21
Q

immature kidneys and large surface area of skin and lungs

A

infants and imbalances

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22
Q

high metabolic rate and large exchange ratio making RR increase

A

infants and imbalances

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23
Q

vomiting, diarrhea, malabsoprtion, fever, inadequate or excessive intake of F or E

A

RF for fluid and electrolyte distrubances(conditions)

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24
Q

diurtetics, laxatives, antacids, corticosteroids, IV fluid infusion, blood transfusion

A

RF for fluid and electrolyte disturbances(ADR)

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25
Q

hemorrhage, burns, crush and head injury, pancreatitis, kidney injury

A

RF for fluid and electrolyte disurbances(acute med conditions, injury or trauma)

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26
Q

heart failure, DM, cancer, oliguria renal disease, liver disease, alcoholism, eating disorders(anorexia nervosa, bulimia)

A

RF for fluid and electrolyte disturbances(chronic med conditions)

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27
Q

old(reduced kidney function and over dose of fluids (oral, enteral, IV))

A

hypervolemia RF

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28
Q

provides energy for cell metabolism, tissue maintence and repair

A

nutrition

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29
Q

give energy for organ function, growth and development, and exercise

A

nutrition

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30
Q

water is the most basic _____

A

nutrition

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31
Q

water

needed for all body fluids and cell functions

A

water

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32
Q

science of optimal cellular metabolism and impact on health and disease

A

nutrition

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33
Q

sum of processes where one takes in and uses nutrients

A

nutrition

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34
Q

SES and race

A

nutrition RF

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35
Q

very young and very old

A

nutrition RF

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36
Q

pregnancy, young, old, preemie, institutionalized old ppl

A

nutrition RF (age)

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37
Q

vit D DEFICIENT in hispanic and AA

A

nutrition RF(race)

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38
Q

type 2 DM in hispanic, NA and AA

A

nutrition RF(race)

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39
Q

IN WHITES…TYPE 1 DM, CELIAC DISEASE, MS, HUNNINGTONS DISEASE

A

NUTRITION RF(RACE)

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40
Q

low SES for malnutrition bc of food insecurity/availability)

A

nutrition RF(poor and underserved)

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41
Q

low SES bc of lack of access, insufficient funds, distance to markets, limited food prep options, high prices, and cheap prices

A

nutrition RF(poor and underserved)

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42
Q

genes, lifestyle, inconsistent eating patterns, poor food choices

A

nutrition RF

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43
Q

IMPAIRED ORAL INTAKE, IMPAIRED DIGESTION AND ABSOORPTION, AND INCREASED METABOLIC DEMAND

A

nutrition RF

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44
Q

starvation RT, chronic diseases RT, acute disease RT

A

3 causes of malnutrition

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45
Q

common sequence of illness, surgery, and hospitalization

A

malnutrition

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46
Q

NUTRITIONAL INTAKE IS PART OF

A

A HISTORY TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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47
Q

DIET RESTRICTIONS ARE PART OF

A

A HISTORY TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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48
Q

CHANGES IN APEPTITE AND INTAKE ARE PART OF

A

A HISTORY TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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49
Q

CHANGES IN WEIGHT ARE PART OF

A

A HISTORY TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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50
Q

MED HISTORY IS PART OF

A

A HISTORY TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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51
Q

CURRENT MED CONDITIONS ARE PART OF

A

A HISTORY TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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52
Q

CURRENTS MEDS AND TREATMENTS ARE PART OF

A

A HISTORY TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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53
Q

ALLERGIES ARE PART OF

A

A HISTORY TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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54
Q

FAMILY HISTORY ARE PART OF

A

A HISTORY TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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55
Q

SOCIAL HISTORY are part of

A

A HISTORY TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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56
Q

chief complaint/presenting symptoms are

A

exam findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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57
Q

general observations are

A

exam findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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58
Q

anthropomorphic measurements are

A

exam findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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59
Q

nausea, vomit, diarrhea are

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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60
Q

constipation, flaccid muscles, and mental status changes are

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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61
Q

loss of appetite, change in bowel patterns, and poor dental health are

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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62
Q

change in bowel patterns and spleen/liver enlargement are

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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63
Q

DRY, BRITTLE HAIR AND NAILS ARE

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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64
Q

LOSS OF SUBQ FAT IS

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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65
Q

DRY, SCALY SKIN AND INFLAMMATION OF GUMS ARE

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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66
Q

dry, dull eyes and enlarged thyroid are

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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67
Q

prominent protrusions in bony prominences are

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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68
Q

weakness and fatigue are

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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69
Q

changes in weight and poor posture are

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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70
Q

anorexia nervosa, binge eating disorder, and bulimia nervosa are

A

expected findings TO SEE IF SOMEONE IS HAVING INADEQUATE NUTRITION

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71
Q

LBW and fears being fat is

A

anorexia nervosa

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72
Q

sees self as fat and restricts food and not gain weight is

A

anorexia nervosa

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73
Q

repeated episodes of eating and when overweight/obese are signs of

A

binge eating disorder

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74
Q

episodes from 1-14+ times in a week are signs of

A

binge eating disorder

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75
Q

doesnt use purging and feels a loss of control when eating then has guilt, shame, or depression are signs of

A

binge eating disorder

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76
Q

cycle of binge eating then purging and lack of control during binges are

A

bulimia nervosa

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77
Q

avg of at least one cycle of binge eating and purging per week for at least 3 months

A

bulimia nervosa

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78
Q

inadequate intake and impaired nutrient absorption are

A

insufficient nutrition

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79
Q

ineffective nutrient utilization is

A

insufficient nutrition

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80
Q

insufficient nutrition leads to

A

malnutrition

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81
Q

associated with insufficient calorie intake(weight loss and not enough nutrients)

A

malnutrition

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82
Q

anorexia nervosa correlates to ___ malnutrition

A

starvation RT

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83
Q

burn injury or trauma correlates to _____

A

acute disease RT malnutrition

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84
Q

sarcopenic obesity or pancreatic cancer correlates to ______

A

chronic disease RT malnutrition

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85
Q

assist in advancing the diet as prescribed is a

A

nursing interventions for optimizing nutrition

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86
Q

instruct cts about the app diet regimen is a

A

nursing interventions for optimizing nutrition

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87
Q

provide interventions to promote appetite(good oral hygeine, fav foods, minimal odors) is a

A

nursing interventions for optimizing nutrition

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88
Q

edu cts abt meds that affect nutritional intake is a

A

nursing interventions for optimizing nutrition

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89
Q

assisting cts with feeding to promote optimal independence is a

A

nursing interventions for optimizing nutrition

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90
Q

individualize menu plans according to cts preferences is a

A

nursing interventions for optimizing nutrition

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91
Q

assisting with aspiration is a

A

nursing interventions for optimizing nutrition

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92
Q

position in fowlers position and support upper back/neck/head are

A

ways to prevent aspiration

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93
Q

avoid straw use and cts tuck chin are

A

ways to prevent aspiration

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94
Q

observing for indications of dysphagia is a

A

way to prevent aspiration

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95
Q

coughing, choking, gagging, and food drooling

A

dysphagia

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96
Q

keep the cts in semi fowlers 1 hour after a meal is

A

a way to prevent aspiration

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97
Q

providing oral hygiene after meals and snacks is

A

a way to prevent aspiration

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98
Q

provide therapeutic diets is a

A

nursing intervention to optimize nutrition

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99
Q

administering and monitoring enteral feedings by NG, gastrostomy, or jejeunostomy tubes are a

A

intervention to optimize nutrition

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100
Q

administering and monitoring parenteral nutrition to cts who are unable to use their GI tract to eat is a

A

intervention to optimize nutrition

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101
Q

lipids, electrolytes, minerals, vitamins, dextrose, and amino acids are

A

parenteral nutrients

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102
Q

maintaining a fluid balance is a

A

intervention to optimize nutrition

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103
Q

administering IV fluids and restricting oral intake and encourage oral intake of fluids are

A

way to maintain fluid balance

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104
Q

removing water pitcher from bedside is

A

restricting oral intake

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105
Q

informing the dietary staff of the amt of fluid to serve with each meal tray is

A

restricting oral intake

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106
Q

inform the staff of each shift of the amt of fluid cts may have in addition to what they get at meals is

A

restricting oral intake

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107
Q

RECORD ALL ORAL INTAKE AND INFORM THE FAMILY OF RESTRICTION IS

A

restricting oral intake

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108
Q

provide fresh drinking water is

A

a way to encourage intake of fluids

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109
Q

remind and encourage a consistent fluid intake is

A

a way to encourage intake of fluids

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110
Q

ask about beverage preferences is

A

a way to encourage intake of fluids

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111
Q

HEALTHY EATING AND PHYSICAL ACTIVITY ARE

A

PRIMARY PREVENTION TO OPTIMIZE NUTRITION

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112
Q

SCREENING FOR DISEASE AND NUTRITIONAL STATUS ARE

A

SECONDARY PREVENTION TO OPTIMIZE NUTRITION

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113
Q

LIPID AND BLOOD GLUCOSE SCREENING ARE

A

SECONDARY PREVENTION TO OPTIMIZE NUTRITION

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114
Q

CHECK BMI IS A

A

SECONDARY PREVENTION TO OPTIMIZE NUTRITION

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115
Q

DOING LABS OF ALBUMIN, PREALBUMIN AND C REACTIVE PROTEIN ARE

A

SECONDARY PREVENTION TO OPTIMIZE NUTRITION IF POOR STATUS EXISTS

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116
Q

DOING LABS FOR HBA1C AND BLOOD GLUCOSE AND ELECTROLUTES ARE

A

SECONDARY PREVENTION TO OPTIMIZE NUTRITION IF POOR STATUS ALREADY

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117
Q

DOING LABS FOR HB AND HEMATOCRIT AND LIPID PROFILE IS

A

SECONDARY PREVENTION TO OPTIMIZE NUTRITION IF POOR STATUS ALREADY

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118
Q

GLUCOSE LEVELS, FINDING METABOLIC DISORDERS, AND LOOKING FOR CONGENTITAL FACTORS ARE

think infants and prevention

A

SECONDARY PREVENTION TO OPTIMIZE NUTRITION IN INFANTS

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119
Q

DIETARY INTERVENTIONS, PAHARMACOLOGICAL AGENTS, AND SURGICAL INTERVENTIONS ARE ALL

A

COLLABORATIVE INTEREVENTIONS TO OPTIMIZE HEALTH

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120
Q

MEDICAL NUTRITION THERAPY ADN DIETARY SUPPLEMENTS ARE

A

DIETARY INTERVENTIONS

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121
Q

BASIC THERAPEUITIC DIETS AND TUBE FEEDING/ENTERAL NUTIRITION ARE

A

DIETARY INTERVENTIONS

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122
Q

PERENTERAL NUTRITION IS A

A

DIETARY INTERVENTION

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123
Q

LOW SALT AND LOW FAT ARE

A

BASIC THERAPEUTIC DIETS

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124
Q

CALORIES REDUCTION AND FIBER SONCUMPTION ARE

A

BASIC THERAPEUTIC DIETS

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125
Q

WEIGHT LOSS MEDS ARE

A

PAHARMACOLOGICAL AGENTS

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126
Q

ANTILIPID AGENTS ARE

A

PAHARMACOLOGICAL AGENTS

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127
Q

MICRONUTRIENT SUPPLEMENTS ARE

A

PAHARMACOLOGICAL AGENTS

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128
Q

parenteral nutrition is a

A

PAHARMACOLOGICAL AGENT

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129
Q

bariatric surgery and common complications are

A

surgical interventions

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130
Q

bariatric surgery can be

A

nonmalabsorptive or malabsoprtive procedures

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131
Q

surgical complications and nutrient deficiencies are

A

common complications

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132
Q

assessment, feeding dependent pt assistance, and seeing red flags are

A

nursing skills RT nutrition

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133
Q

refer to a ____ when you see red flags

A

RDN

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134
Q

pressure ulcers and weight loss are

A

red flag ass with nutrition

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135
Q

inadequate oral intake or appetite changes are

A

red flag ass with nutrition

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136
Q

NPO or nausea are

A

red flag ass with nutrition

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137
Q

needs to happen within 24 hours of admission

A

nutrition screening

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138
Q

body composition changes like weight loss and strength loss are signs of

A

malnutrition

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139
Q

serum protein an electrolyte levels are used in conjunction with a physcial exam when diagnosing _______

A

malnutrition

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140
Q

achieve an ideal body weight, consume a number of calories, and have no adverse consequences are

A

goals for pt with malnutrition

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141
Q

impaired nutritional status and fluid balance are

A

how to care for pt with imbalanced nutrition

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142
Q

risk for impaired tissue integrity and impaired nutritional intake are

A

how to care for pt with imbalanced nutrition

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143
Q

no added salt or 1 to 2 g sodium

A

low sodium diet

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144
Q

liquids that leave little residue(clear fruit juice, gelatin, broth)

A

clear liquid diet

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145
Q

clear liquids plus liquid dairy products, all juices. some facilities incl. purred veggies

A

full liquid diet

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146
Q

a nurse is caring for a ct who is at high risk for aspiration. what should the nurse do?

A

instruct the ct to tuck their chin when swallowing

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147
Q

what gives the most energy to the body?

A

carbs

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148
Q

a pt is on a low residue diet…they will eat what?

A

vanilla custard not veggies

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149
Q

older pts nutritional needs and considerations are what?

A

older adults are more prone to dehydration

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150
Q

older pts nutritional needs and considerations are what?

A

many need calcium supplements

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151
Q

older pts nutritional needs and considerations are what?

A

older adults need the same amount of vitamins and minerals as young people

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152
Q

directly delivered into the GI tract, bypassing the oral cavity is an

A

enteral feeding indications

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153
Q

not able to swallow but has an intact GI tract is an

A

enteral feeding indication

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154
Q

=

A

kilocalories and the roles in nutrition/mal

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155
Q

fats, vitamins and carbs are

A

kilocalories

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156
Q

proteins, minerals and water are

A

kcal

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157
Q

what is critical for cell function and replaces fluids the body loses?

A

water

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158
Q

what helps complete essential biochemical reactions in the body (K, Na, Fe, Ca)?

A

minerals

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159
Q

what is neccessary for metabolism and fat/water soluble?

A

vitamins

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160
Q

what is necessary for metabolism and fat/water soluble?

A

vitamins

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161
Q

what are the fat soluble vitamins?

A

A,D,E,K

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162
Q

contribute to growth, maintenance, and repair of body tissues.

A

proteins

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163
Q

each gram produces 4 kcal

A

proteins

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164
Q

sources of complete ____ are beef, whole milk, poultry

A

proteins

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165
Q

provide energy and vitamins

A

fats

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166
Q

no more than 35% of calories

A

fat

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167
Q

produces 9kcal per gram

A

fat

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168
Q

olive oil, salmon, egg yolk

A

fat sources

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169
Q

most of the body’s energy and fiber

A

carbs

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170
Q

produces 4kcal per gram

A

carbs

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171
Q

provide glucose

A

carbs

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172
Q

whole grain bread, baked potatoes, brown rice, plant foods

A

carb sources

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173
Q

burns completely and efficiently without prodtucts to excrete

A

glucose

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174
Q

anorexia, bulimia, and fat malabsorption syndrome

A

conditions ass with insufficient nutrition

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175
Q

protein calorie malnutrition, vit deficiencies, and zinc deficiency

A

conditions ass with insufficient nutrition

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176
Q

these are the kcal energy containing nutrients(carbs, fat, proteins)

A

macronutrients

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177
Q

cant support or maintain body functions and not a macronutrient

A

alcohol

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178
Q

in ____, an anadequate intake of protein and kcal(marasmus) impairs growth and fevelopment, stunts height and brain development

A

kids

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179
Q

primary source of fuel and energy

A

carbs

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180
Q

facilitates growth and repair of tissues; energy source

A

protein

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181
Q

source of fatty acid, needed for growth and development; energy

A

fat

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182
Q

a, d, e, k

A

fat soluble vitamins

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183
Q

c, b, thiamin, riboflavin

A

water soluble vitamins

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184
Q

niacin, pyridoxine, pantothenic, biotin

A

water soluble vitamins

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185
Q

folate and cobalamin

A

water soluble vitamins

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186
Q

ca, phosphorus, Mg, Na, K, Cl

A

major minerals

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187
Q

lack of adherance to diet and guidelines

A

bariatric surgery RF

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188
Q

limited knowledge of actual needs

A

bariatric surgery RF

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189
Q

weight gain if noncompliant to calorie intake

A

bariatric surgery RF

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190
Q

insulin lowers blood glucose and stabilizes glucose range

A

glucose regulation

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191
Q

euglycemia, hyper and hypoglycemia are

A

cat of glucose regulation

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192
Q

process of maintaining optimal blood glucose levels

A

glucose regulation

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193
Q

cell use of glucose for energy (ATP synthesis)

A

end result of glucose metabolism

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194
Q

age and pregnancy

A

glucose regulation RF

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195
Q

race and genes and lifestyle are

A

glucose regulation RF

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196
Q

fam history and certain meds

A

glucose regulation RF

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197
Q

virus and toxins are

A

glucose regulation RF for type one DM

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198
Q

islet cell autoantibodies for beta cell destruction

A

glucose regulation RF for type one DM

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199
Q

pancreas can not make enough insulin to maintain normal glucose

A

glucose regulation RF for type one DM

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200
Q

lack of exercise and overweight are

A

glucose regulation RF for type two DM

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201
Q

being older and family history are

A

glucose regulation RF for type two DM

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202
Q

insulin resistance is type __ of DM

A

2

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203
Q

more prevelant in AA, asians, hispanics, native hawaiins, NA

A

type 2 DM

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204
Q

marked decreased in the ability of the pancreas to make insulin is

A

type 2 DM

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205
Q

inappropriate glucose production by the liver is

A

type 2 DM

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206
Q

altered production of hormones and cytokines by adipose tissue relates to

A

type 2 DM

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207
Q

brain, kidneys, and gut have roles in development of

A

type 2 DM

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208
Q

state of insufficient or low blood glucose levels and below 70

A

hypoglycemia

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209
Q

state of elevated blood glucose levels and more than 100 in fasting and more than 140 in nonfasting

A

hyperglycemia

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210
Q

lispro(humalog) is a ____ acting insuline

A

rapid

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211
Q

regular/humulin R is a ___ acting insulin

A

short

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212
Q

NPH / Humulin N is a ___ acting insulin

A

intermediate

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213
Q

reduced cognition and tremors are signs of

A

hypoglycemia

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214
Q

diaphoresis and seizure are signs of

A

hypoglycemia

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215
Q

irritability and headache are signs of

A

hypoglycemia

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216
Q

weakness and hunger are signs of

A

hypoglycemia

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217
Q

diaphoresis is a sign of

A

hypoglycemia

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218
Q

polyuuria ad polydispia are signs of

A

hyperglycemia

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219
Q

dehydration and fatigue are signs of

A

hyperglycemia

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220
Q

fruity smelling breath and kussmal breathing are signs of

A

hyperglycemia

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221
Q

weight loss and hunger are signs of

A

hyperglycemia

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222
Q

poor wound healing is a sign of

A

hyperglycemia

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223
Q

70-140 is the range for

A

euglycemia

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224
Q

LT 70 is the range for

A

hypoglycemia

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225
Q

severe hypoglycemia is LT __

A

50

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226
Q

severe hyperglycemia is GT

A

180

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227
Q

4-6 or 6.5 to 8.5 with a target goal of LT 7%

A

A1C reference range

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228
Q

fasting plasma glucose level is

A

GT 126 mg/dL

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229
Q

casual plasma glucose levels are

A

GT 200 mg/DL

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230
Q

casual glucose levels are often ass with

A

polyuria, polydispia, and sudden weight loss

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231
Q

the range for the oral glucose tolerance test is

A

GT 200mg/dL

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232
Q

with the oral glucose tolerance test, you give ____

A

75 g of oral glucose and then check BG after 2 hr

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233
Q

A1C is diagnosed at GT ___

A

6.5%

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234
Q

the blood glucose target for preprandial plasma glucose is

A

70-130 mg/dL

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235
Q

the blood glucose target for postprandial plasma glucose is

A
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236
Q

the blood glucose target for bedtime plasma glucose is

A

100-140

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237
Q

the blood glucose target for HBA1C is

A
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238
Q

nervousness and muscle tremors are

A

consequences of hypoglycemia

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239
Q

normal hydration and irritability are

A

consequences of hypoglycemia

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240
Q

no ketones and blurred vision are

A

consequences of hypoglycemia

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241
Q

diaphoresis and hunger are

A

consequences of hypoglycemia

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242
Q

anxiety and palpitations are

A

consequences of hypoglycemia

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243
Q

weakness and dizziness are

A

consequences of hypoglycemia

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244
Q

headache and tachycardia are

A

consequences of hypoglycemia

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245
Q

no RR change and seizures are

A

consequences of hypoglycemia

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246
Q

neurological changes are

A

consequences of hypoglycemia

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247
Q

shaking and coma are

A

consequences of hypoglycemia

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248
Q

confusion and clamminess are

A

consequences of hypoglycemia

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249
Q

difficulty concentrating and unconsiousness are

A

consequences of hypoglycemia

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250
Q

death and being cool are

A

consequences of hypoglycemia

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251
Q

dehydration is a a

A

short term consequences of hypoglycemia

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252
Q

end organ disease due to microvascular damage is a y

A

long term consequences of hypoglycemia

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253
Q

macrovasucular angiopathy is a

A

long term consequences of hypoglycemia

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254
Q

peripheral neuropathy is a

A

End-organ disease due to microvascular damage

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255
Q

retinopathy is a

A

■ End-organ disease due to microvascular damage

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256
Q

nephropathy is a

A

■ End-organ disease due to microvascular damage

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257
Q

hypertension is a

A

■ Macrovascular angiopathy

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258
Q

cardiovascular and peripheral vascular disease are examples of

A

■ Macrovascular angiopathy

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259
Q

symptoms ass with dehydration or acidosis are

A

consequences of hyperglycemia

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260
Q

mental status is a

A

consequences of hyperglycemia

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261
Q

skin being warm and moist are

A

consequences of hyperglycemia

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262
Q

alert to confused and coma, esp. in untreated in ketoacidosis is

A

mental status

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263
Q

nausea and vomit are

A

■ Symptoms ass with dehydration or acidosis

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264
Q

ab cramps and fatigue are

A

■ Symptoms ass with dehydration or acidosis

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265
Q

excessive thirst(polydispia) and polyuria are

A

■ Symptoms ass with dehydration or acidosis

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266
Q

ketones and excessive hunger (polyphagia) are

A

■ Symptoms ass with dehydration or acidosis

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267
Q

wash and dry hands completely. not needed to clean the site with ETOH…will interfere and increase results

A

glucose regulation ct teaching

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268
Q

hard to get blood? warm hands and let arms dangle to get blood

A

glucose regulation ct teaching

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269
Q

lance the side of finger(fewer nerves)

A

glucose regulation ct teaching

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270
Q

record results and compare to personal blood glucose goals

A

glucose regulation ct teaching

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271
Q

brisk walking can do glucose reducing effects you need

A

glucose regulation ct teaching (exercise)

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272
Q

choose exercise that is enjoyable

A

glucose regulation ct teaching (exercise)

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273
Q

use proper fitting footwear to avoid rubbing or injury

A

glucose regulation ct teaching (exercise)

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274
Q

exercise session should have a cool down period

A

glucose regulation ct teaching (exercise)

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275
Q

start the exercise program gradually and increase slowly

A

glucose regulation ct teaching (exercise)

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276
Q

exercise is best donw after meals

A

glucose regulation ct teaching (exercise)

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277
Q

monitor blood glucose levels bf, during and after exercise to see the effect on BG level

A

glucose regulation ct teaching (exercise)

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278
Q

bf exercise, if LT/<100, eat a carb snack then check BG level after 15 min. delay exercise if <100

A

glucose regulation ct teaching (exercise)

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279
Q

bf exercise, if BG is GT/>250 in a type one person and ketones are present. delay activity until ketones are gone. drink fluid

A

glucose regulation ct teaching (exercise)

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280
Q

MAY GET EXERCISE INDUCED HYPOGLYCEMIA SEVERAL HOURS AFTER

A

glucose regulation ct teaching (exercise)

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281
Q

COMPENSATE FOR ACTIVITY BY MONITORING BLOOD GLUCOSE LEVELS AND MAKING INSULIN ADJUSTMENTS AND FOOD INTAKE ADJUSTMENTS

A

glucose regulation ct teaching (exercise)

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282
Q

for diet…use myplate ad guidelines

A

glucose regulation ct teaching

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283
Q

for exercise…150 min a week or 30 min a day 5 times a week

A

glucose regulation ct teaching

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284
Q

when preventing type 2 DM, weight loss and diet and exercise

A

glucose regulation ct teaching

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285
Q

eat carbs regularly with good timing due to drug onset and duration

A

collaborative interventions to optimize glucose regulation (Non-Pharmacological) for hypoglycemia

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286
Q

exercise and limit carb intake

A

collaborative interventions to optimize glucose regulation (Non-Pharmacological) for hyperglycemia

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287
Q

educate on self management, monitor and manage blood glucose

A

collaborative interventions to optimize glucose regulation (Non-Pharmacological) for type 2 DM

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288
Q

do nutrition therapy as a

A

collaborative interventions to optimize glucose regulation (Non-Pharmacological) for type 2 DM

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289
Q

control glucose by diet, exercise and weight control as a

A

collaborative interventions to optimize glucose regulation (Non-Pharmacological)

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290
Q

expected range of what test is 4-6/6.5-8 but must be LT 7 as a target goal?

A

Hemoglobin A1C lab

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291
Q

what lab is the best indicator of the avg BG level for the past 120 days?

A

Hemoglobin A1C lab

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292
Q

what lab assists in evaluating treatment effectiveness and compleiance?

A

Hemoglobin A1C lab

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293
Q

what lab is recommended quarterly or twice a year depending on glycemic level?

A

Hemoglobin A1C lab

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294
Q

a 2nd generation sulfonylurea

A

glipizide/Glucotrol

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295
Q

oral hypoglycemic

A

glipizide/Glucotrol

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296
Q

insulin release from the pancreas

A

glipizide/Glucotrol

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297
Q

can increase tissue sensitivity to insulin over time

A

glipizide/Glucotrol

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298
Q

notify the provider if there is a recurrant problem

A

glipizide/Glucotrol

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299
Q

lower glucose levels

A

glipizide/Glucotrol

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300
Q

high risk of hypoglycemia

A

glipizide/Glucotrol

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301
Q

pregnancy cat c risk

A

glipizide/Glucotrol

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302
Q

less effective over time

A

glipizide/Glucotrol

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303
Q

more potent with fewer drug interactions than 1st generation

A

glipizide/Glucotrol

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304
Q

for type 2 DM

A

glipizide/Glucotrol(TU)

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305
Q

blocks ATP-sensitive K channels, allowing influx of Ca

A

glipizide/Glucotrol(MOA)

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306
Q

Ca influx stimulates insulin release by pancreatic beta cells

A

glipizide/Glucotrol(MOA)

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307
Q

insulin release diminishes as glucose declines

A

glipizide/Glucotrol(MOA)

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308
Q

insulin sensitivity may increase with prolonged use

A

glipizide/Glucotrol(MOA)

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309
Q

hypoglycemia and nausea are

A

glipizide/Glucotrol(ADR)

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310
Q

epigastric fullness and heartburn are

A

glipizide/Glucotrol(ADR)

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311
Q

photosensitivity(sulfa) and diarrhea are

A

glipizide/Glucotrol(ADR)

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312
Q

possible sudden cardiac death is a

A

glipizide/Glucotrol(ADR)

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313
Q

monitor for signs of hypoglycemia is a

A

glipizide/Glucotrol(interventions)

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314
Q

diaphoresis and tachycardia are

A

signs of hypoglycemia

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315
Q

fatigue and tremors are

A

signs of hypoglycemia

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316
Q

excessive hunger is a

A

sign of hypoglycemia

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317
Q

if conscious, give glucose orally in pill form , 2-3 tsp of sugar, OJ, honey, or corn syrup dissolved in water

A

glipizide/Glucotrol(interventions)

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318
Q

if unconscious, give IV glucose; give parenteral glucagon if IV not available

A

glipizide/Glucotrol(interventions)

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319
Q

Check the client’s blood glucose every 15–20 minutes

A

glipizide/Glucotrol(interventions)

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320
Q

continue treatment until BG has returned to expected refence range and ct no longer symptomatic

A

glipizide/Glucotrol(interventions)

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321
Q

monitor for persistent nausea, vomiting, or diarrhea

A

glipizide/Glucotrol(interventions)

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322
Q

monitor CBC levels

A

glipizide/Glucotrol(interventions)

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323
Q

give orally 30 min bf selected meal

A

glipizide/Glucotrol(administration)

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324
Q

make sure cts swallow the SR form whole and dont crush or chew it

A

glipizide/Glucotrol(administration)

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325
Q

best taken 30 min bf breakfast. withhold dose if ct will not be able to eat…sometimes split dose BID

A

glipizide/Glucotrol(administration)

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326
Q

wear a medical alert bracelet

A

glipizide/Glucotrol(CI)

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327
Q

watch for and reportsymptoms of hypoglycemia

A

glipizide/Glucotrol(CI)

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328
Q

test BG to confirm

A

glipizide/Glucotrol(CI)

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329
Q

consume a snack of carbs

A

glipizide/Glucotrol(CI)

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330
Q

retest in 15 to 20 min and repeat if still low

A

glipizide/Glucotrol(CI)

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331
Q

carry a carb snack at all times

A

glipizide/Glucotrol(CI)

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332
Q

pregnancy and lactation(neonatal/infant hypoglycemia) are

A

glipizide/Glucotrol(Contraindications)

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333
Q

DKA

A

glipizide/Glucotrol(Contraindications)

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334
Q

sulfa allergy and hepatic/renal impairment(drug acculumation)

A

glipizide/Glucotrol(Contraindications)

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335
Q

insulin is _____ in pregnancy

A

recommended

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336
Q

renal and hepatic dysfunction

A

glipizide/Glucotrol(precautions)

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337
Q

adrenal or pituitary insufficiency are

A

glipizide/Glucotrol(precautions)

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338
Q

ETOH poses a risk for a disulfiram like reaction and increases hypoglycemia effects

A

glipizide/Glucotrol(interactions)

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339
Q

nausea and palpitations and flushing are

A

antabuse/disulfiram like reaction

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340
Q

Sulfonamide antibiotics, NSAIDs, oral anticoagulants, salicylates, monoamine oxidase inhibitors, and cimetidine (Tagamet) increase hypoglycemic effects are

A

glipizide/Glucotrol(interactions)

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341
Q

thiazides counteract hypoglycemic effects

A

glipizide/Glucotrol(interactions)

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342
Q

beta blockers mask manifestions of hypoglycemic

A

glipizide/Glucotrol(interactions)

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343
Q

hypoglycemia (acute ETOH ingestion, antidiabetic, NSAIDs, sulfonamide antibiotic, Tagamet/cimetidine)

A

glipizide/Glucotrol(interactions)

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344
Q

disulfiram like reaction with alcohol(flushing, palpitations, nausea)

A

glipizide/Glucotrol(interactions)

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345
Q

beta blockers suppress insulin release, inhibit breakdown of glycogen to glucose, and masks hypoglycemic symptoms

A

glipizide/Glucotrol(interactions)

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346
Q

use of alcohol can result in disulfiram like reaction(intense nausea, vomiting, flushing, palpitations)

A

glipizide/Glucotrol(interactions)

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347
Q

NSAIDs, sulfonamide antibiotics, ranitidine, and cimetidine have additive hypoglycemic effect(the nurse will monitor glucose levels when these other agent are used concurrently)

A

glipizide/Glucotrol(interactions)

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348
Q

tachycardia, tremors, palpitations, and disphoresis are

A

signs of hypoglycemia

349
Q

beta blockers decrease effectiveness by inhibiting insulin release(mon glucose levels)

A

glipizide/Glucotrol(interactions)

350
Q

a meglitinide

A

Repaglinide

351
Q

Repaglinide is an

A

oral hypoglycemic

352
Q

insulin release from the pancreas and lower glucose levels are actions of

A

Repaglinide

353
Q

Repaglinide is in pregnancy cat ___ risk

A

C

354
Q

type two DM are the TU of

A

Repaglinide

355
Q

increase pancreatic insulin production during and after meals(shorter acting than sulfonylureas) is the MOA of

A

Repaglinide

356
Q

hypoglycemia, nausea and vomiting are ADR of

A

Repaglinide

357
Q

mon for signs of hypoglycemia as in intervention for

A

Repaglinide

358
Q

diaphoresis, tachycardia, and fatigue are signs of

A

hypoglycemia

359
Q

excessive hunger and tremors are signs of

A

hypoglycemia

360
Q

if conscious, give glucose orally in either pill form, OJ, 2-3 tsp of sugar, honey or corn syrup dissolved in water are interventions for

A

Repaglinide

361
Q

if unconscious, give IV glucose; give parenteral glucagon if IV not available are interventions for

A

Repaglinide

362
Q

check the cts BG Q 15-20 min is an intervention for

A

Repaglinide

363
Q

continue treatment until the BG has returned to the expected range and the ct is no longer symptomatic is an intervention for

A

Repaglinide

364
Q

monitor for persistent nausea, vomiting, or diarrhea and CBC levels is an intervention for

A

Repaglinide

365
Q

give orally 30 min or less bf meals, usually 3 xs a day

A

Repaglinide(administration)

366
Q

tell cts to skip a dose if they skip a meal and to add a dose if they add a meal

A

Repaglinide(administration)

367
Q

dont exceed more than 4 doses a day

A

Repaglinide(administration)

368
Q

instruct cts to take the med within 30 min of meal time, 3xs per day

A

Repaglinide(administration)

369
Q

notify provider if any recurrant problems

A

Repaglinide(CI)

370
Q

wear a med alert bracelet

A

Repaglinide(CI)

371
Q

watch for and report symptoms of hypoglycemia

A

Repaglinide(CI)

372
Q

test blood glucose to confirm

A

Repaglinide(CI)

373
Q

if hypoglycemia occurs, tell pt to take OJ, or 2-3 tsp of sugar, honey, or corn syrup dissolved in water

A

Repaglinide(CI)

374
Q

retest in 15 to 20 min and repeat treatment if still low

A

Repaglinide(CI)

375
Q

carry a carb snack at all times

A

Repaglinide(CI)

376
Q

low down when nauseated

A

Repaglinide(CI)

377
Q

consume adequate carbs

A

Repaglinide(CI)

378
Q

liver or renal impairment and DKA are

A

Repaglinide(contraindications)

379
Q

liver, kidney or endocrine disorders are

A

Repaglinide(Contraindications)

380
Q

renal or hepatic dysfunction is a

A

Repaglinide(precaution)

381
Q

systemic infection and older adults are

A

Repaglinide(precaution)

382
Q

use of ETOH, NSAIDs, warfarin, loop diuretics, and anabolic steroids are

A

Repaglinide(precaution)

383
Q

gemfibrozil/Lopid inhibits metabolism and causes hypoglycemia is an

A

Repaglinide(interaction)

384
Q

Lopid , erythromycin, and chloramphenicol increase hypoglycemic effects are a

A

Repaglinide(interaction)

385
Q

alcohol, corticosteroids, and rifampin decrease hypoglycemic effects are

A

Repaglinide(interaction)

386
Q

concurrent use of Lopid results in inhibitition of repaglinide metabolism, which leads to increased hypoglycemic risk are

A

Repaglinide(interaction)

387
Q

with ____, you need to avoid concurrent use of pioglitazone and gemfibrozil

A

Repaglinide

388
Q

with _________, you want to closely mon for manifestations of hypoglycemia

A

Repaglinid

389
Q

an oral hypoglycemic

hint (M)

A

Metformin

390
Q

Metformin is a

A

biguanide

391
Q

Metformin is pregnancy cat ____

A

B

392
Q

Metformin modulates a _____ in postprandial glucose level

A

rise

393
Q

what drug reduces the production of glucose within the liver thru suppression of gluconeogenesis?

A

Metformin

394
Q

what drug increases glucose intake and use in fat and skeletal muscles?

A

Metformin

395
Q

what drug decreases glucose absorption in the GI tract?

A

Metformin

396
Q

what drug is the first choice med for most type 2 DM?

A

Metformin

397
Q

anorexia and nausea are GI effects of

A

Metformin

398
Q

diarrhea and weight loss(6.6-8.8lbs/3-4 kg) are GI effects of

A

Metformin

399
Q

with Metformin, you see vitamin ___ and folic acid deficiency bc of altered absorption

A

B12

400
Q

hyperventilation and myaglia are

A

signs of lactic acidosis

401
Q

sluggishness and somnolence are

A

sign of lactic acidosis

402
Q

there is GI effects and lactic acidosis ass with what drug?

A

Metformin

403
Q

there is a 50% mortality rate with lactic acidosis ass with what drug?

A

Metformin

404
Q

type 2 DM is the TU for

hint (M)

A

Metformin

405
Q

lower basal and postprandial blood sugar is a TU of what drug?

A

Metformin

406
Q

decrease androgen levels of PCOS is a TU of what drug?(M)

A

Metformin

407
Q

treating prediabetics in young and obese pts is a TU in what drug?

A

Metformin

408
Q

the MOA of ___ is to reduce glucose production by liver(this decreases breakdown of glycogen and decreases gluconeogensis

A

Metformin

409
Q

the MOA of ____ is to enhance insulin sensitivity and glucose transport into fat and skeletal muscle cells(M)

A

Metformin

410
Q

the MOA of ____ is to decrease intestinal glucose absorption(slightly)

A

Metformin

411
Q

lactic acidosis with Metformin can be fatal and produce a ___ effect

A

black box, 50% mortality

412
Q

lactic acidosis with Metformin can be fatal and produce a ___ effect

A

hyperventilation and myglia

413
Q

lactic acidosis with Metformin can be fatal and produce a ___ effect

A

malaise and unusual somnolence

414
Q

dehyrdation and lack of appetite are ADR of

A

Metformin

415
Q

nausea and diarrhea are ADR of

A

Metformin

416
Q

weight loss that is ind of GI distress are ADR of

A

Metformin

417
Q

decreased absorption of B12 and folic acid are ADR of

A

Metformin

418
Q

mon for signs of lactic acidosis and then stop therapy if symptoms occur are interventions of

A

Metformin

419
Q

expect that severe lactic acidosis with Metformin will require ____

A

hemodialylsis

420
Q

mon for persistent nausea, vomiting, or diarrhea are interventions for which drug?

A

Metformin

421
Q

mon for indications of vit B12 or folic acid deficiency are interventions for what drug?

A

Metformin

422
Q

recommend an appropriate supplement for which drug as an intervention?

A

Metformin

423
Q

mon renal function upon initial therapy and yearly afterward as in intervention for what drug?

A

Metformin

424
Q

instruct cts to take IR tablets 2xs a day or SR tablets once a day when administering

A

Metformin

425
Q

what is the best time to give Metformin for slower GI transit and giving better absorption?

A

daily in the evening meal

426
Q

Metformin can be given in combo with other drugs. true or false?

A

true

427
Q

what drug do you give with food to decrease GI side effects?

A

Metformin

428
Q

what drug causes decreased appetite, nausea or diarrhea?

A

Metformin

429
Q

avoid drinking alcohol when on what drug?(M)

A

Metformin

430
Q

report weakness and fatigue with what drug?(M)

A

Metformin

431
Q

report lethargy or hyperventilation with what drug?(M)

A

Metformin

432
Q

expect adverse effects to diminish as drug therapy continues with what drug(M)?

A

Metformin

433
Q

lie down when nauseated with what drug?(M)

A

Metformin

434
Q

maintain adequate carb and fluid intake with what drug?(M)

A

Metformin

435
Q

report weakness and fatigue with what drug?(M)

A

Metformin

436
Q

report pallor and reddened tongue with what drug?(M)

A

Metformin

437
Q

if ADR occur with Metformin…what is the action you do?

A

withhold med if these findings occur, and inform the provider immediately

438
Q

DKA and ETOHism are contraindications for what drug?

A

Metformin

439
Q

heart failure and shock are cotraindications for what drug?

A

Metformin

440
Q

cardiopulmonary, hepatic, or renal insufficiency are contraindications of what drug?

A

Metformin

441
Q

severe infection and acute MI are contraindications for what drug?(M)

A

Metformin

442
Q

hypoxemia and lactic acid are contrainidications of what drug?(M)

A

Metformin

443
Q

kidney and liver impairment are contraindications of what drug?

A

Metformin

444
Q

Metformin increases the risk for what?

A

LACTIC ACIDOSIS

445
Q

DIARRHEA AND ANEMIA ARE PRECAUTIONS FOR WHAT DRUG?

A

Metformin

446
Q

dehydration and pituitary insufficiency are precautions for what drug?

A

Metformin

447
Q

gastroparesis and obstruction are precautions of what drug?(M)

A

Metformin

448
Q

hyperthyroidism and old pts are precautions of what drug?

A

Metformin

449
Q

ETOH and Tagamet increase the risk of lactic acidosis with what drug?

A

Metformin

450
Q

any contrast dye increasing acute renal failure and lactic acidosis is ass with what drug?

A

Metformin

451
Q

Procardia, Lasix, morphine, Zantac andantifungals increase the hypoglycemic effects with what drug?

A

Metformin

452
Q

with Metformin, you want to ____ drug 1-2 days bf procedure adn mon BUN and Cr

A

stop

453
Q

with Metformin, mon BUN and Cr 48 hours ___ bf restarting drug

A

afterwards

454
Q

hypoglycemia with sulfonylureas, glitazones, Byetta, and insulin interacts with

A

Metformin

455
Q

avoid alcohol with Metformin bc _______

A

hypoglycemia and lactic acidosis

456
Q

cimetidine interacts with Metformin and increases what

A

risk of lactic acidosis

457
Q

brand name is Humalog

A

Lispro

458
Q

this is an rapid acting insulin

A

Lispro

459
Q

this is an injectable hypoglycemic drug

A

Lispro

460
Q

the onset is 15-30 min

A

Lispro

461
Q

the peak is 0.5 to 2.5 hours

A

Lispro

462
Q

the duration is 3 to 6 hours

A

Lispro

463
Q

this promotes cell uptake of glucose(it decreases glucose levels)

A

insulin

464
Q

converts glucose into glycogen and promotes energy storage

A

insulin

465
Q

moves K into cells(along with glucose)

A

insulin

466
Q

this lowers glucose levels and is for all DM types

A

insulin

467
Q

this is preg cat b

A

insulin

468
Q

for all types of DM and for glycemic control of DM to prevent complications is the TU for

A

insulin

469
Q

use insulin when type 2 pt is on….

A

oral antidiabetic med, diet, and exercise are unable to control BG levels

470
Q

use insulin when type 2 pt has…

A

severe renal or liver disease or neuropathy is present

471
Q

use insulin when type 2 pt is….

A

undergoing surgery or tests or under infection and trauma

472
Q

use insulin when type 2 pt is…

A

undergoing treatment for DKA and HHS or hyperkalemia

473
Q

hypoglycemia and anaphylaxis are ADR of

A

insulin

474
Q

hypokalemia and lipodystrophy and injection site reactions are ADR of

A

insulin

475
Q

mon for signs of hypoglycemia with

A

insulin

476
Q

tachycardia and palpitations are

A

abrupt onset of hypoglycemia

477
Q

diaphoresis and shakiness are

A

abrupt onset of hypoglycemia

478
Q

headache, tremors and weakness are

A

gradual onset of hypoglycemia

479
Q

check BG level to confirm then give OJ or oral glucose or glucose tablets for hypoglycemia with

think I

A

insulin use

480
Q

if unconsious, give glucagon parenterally when using

A

insulin

481
Q

monitor skin for subq fat accumulation when on

A

insulin

482
Q

mon K levels when on

A

insulin

483
Q

mon ECG and hypokalemia signs when on

A

insulin

484
Q

give sub Q with insulin syringe or IV (Humulin R) when giving

A

insulin

485
Q

when giving insulin, an intradermal needle is too ___

A

short

486
Q

when giving insulin, an IM needle is too___

A

long

487
Q

if a cloudy insulin, gently _____ the vial between your palms to disperse the particles

A

rotate

488
Q

when mixing short acting and long acting insulin, you want to ___

A

put short acting into syringe first

489
Q

dont mix glargine or detemir with other insulins true or false

A

true

490
Q

keep ____ premixed in syringes for 1-2 weeks in fridge and vertical with needles up

A

insulins

491
Q

if the insulin is _____, resuspend the insulin via gentle motion bf giving

A

premixed

492
Q

make sure adequate glucose is available at onset and peak insulin times when giving ____

A

insulin

493
Q

an ____ pump is SC and short acting only

A

insulin

494
Q

an ___ pump is expensive, and may have microdeposits that decrease the amt of drug delivered

A

insulin

495
Q

IV ____ is for short acting only and in emergencies

A

insulin

496
Q

IV _____ is diluted in NS(1:1) and is started in 0.1 U/kg/hour

A

insulin

497
Q

for IV ______, it is 100U in 100mL

A

insulin

498
Q

for SC ____, roll NPH insulin to disperse suspension

A

insulin

499
Q

for SC ___, draw regular insulin if mixed with NPH insulin and use the abdomen for best____ levels

A

insulin

500
Q

for SC insulin, you can use arm nd thigh but they are the _______________ ____

A

slowest absorption

501
Q

for SC insulin, you want to rotate sites within the general areato prevent _____

A

lipohypertrophy

502
Q

use the same site only once a month for the injection of SC

A

insulin

503
Q

when giving SC insulin, keep sites at least ___in apart

A

1

504
Q

increase ____, if calorie intake, infection or stress

A

insulin

505
Q

increase ____, if growth spurts, and in 2 and 3 trimester

A

insulin

506
Q

decrease ___, if exercise or 1 trimester

A

insulin

507
Q

why do you mix short acting then long acting together?

A

This prevents the possibility of accidentally injecting some of the longer-acting insulin into the shorter-acting insulin vial. (This can pose a risk for unexpected insulin effects with subsequent uses of the vial.)

508
Q

NPH and premixed insulin appear cloudy…if other are cloudy, _____

A

dont give them!

509
Q

lispro and regular insulin are given SC, and continuous and ___

A

IV

510
Q

give NPH insulin via ____

A

SC

511
Q

when giving insulin use a U-100 syringe with ____

A

U-100 insulin

512
Q

when teaching pts about enhancing diabetes med therapy, tell them to keep a __ diet and ____ activity

A

proper; consistent

513
Q

for unopened vials of ___, keep in fridge until expired

A

insulin

514
Q

vials of premixed ___, can be stored for up to 3 months in fridge

A

insulin

515
Q

syringes of remixed ___, can be kept in fridge for 1-2 weeks and get resuspended bf giving them

A

insulin

516
Q

keep the vial of ____that is in use at room temp and keep away from sunlight and intense heat

A

insulin

517
Q

inhaled human ___ is in dry powder form, packed in cartridges and then into an inhaler

A

insulin

518
Q

IV ____ can be given to pts who need rapid glucose reduction

A

insulin

519
Q

____insulin is the most common type given

A

regular

520
Q

The typical concentration is _____ of NaCl (0.9%)(1U/mL)

A

100U/100mL

521
Q

Lispro insulin is okay for ___ administration

A

IV

522
Q

When giving IV insulin , you want to allow 50 mL of solution to flow thru the IV tubing and _____ Is the safety alert

A

Waste

523
Q

The insulin will ___ to the tubing , so the ct will get the right concentration of insulin

A

Bind

524
Q

Wear a med alert bracelet if you are on _____

A

Insulin

525
Q

Watch for symptoms of hypoglycemia and test BG to confirm, then eat a carb snack and retest in 15 to 20 min and repeat treatment if still low if on

A

Insulin

526
Q

Carry a carb snack at all times if on what drug?

A

Insulin

527
Q

Report recurrring episodes of hypoglycemia to provider if on what drug?

A

Insulin

528
Q

Rotate injection sites systemically and space them one in apart if on what drug?

A

Insulin

529
Q

Don’t inject cold ____

A

Insulin

530
Q

Report weakness or nausea if on what drug?

A

Insulin

531
Q

Report palpitations or paresthesia if on what drug?

A

Insulin

532
Q

Hypersentivity to Insulin is a contraindication for what drug?

A

Insulin

533
Q

Hypoglycemia and hypokalemia are contraindications for which drug?

A

Insulin

534
Q

Anaphylaxis and lipodystrophy are contraindications for which drug?

A

Insulin

535
Q

Precautions for which drug are altered nutrition and stress?

A

Insulin

536
Q

Fever and older adults are precautions for what drug?

A

Insulin

537
Q

Sulfonylureas, meglitinides, beta blockers, salicylates, and alcohol increase hypoglycemic effects are interactions for which drug?

A

Insulin

538
Q

Thiazides and loop diuretics are interactions for which drug?

A

Insulin

539
Q

Sympathomimetics and thyroid hormones and glucocorticoids are interactions with which drug?

A

Insulin

540
Q

Thiazides, loop diuretics, sympathomimetics, thyroid hormones, and glucocorticoids increase BG levels(counteracting hypoglycemic effects) are interactions with what drug?

A

Insulin

541
Q

Beta blockers and manifestations of hypoglycemia are interactions of what drug?

A

Insulin

542
Q

Tachycardia and tremor are signs of ______

A

Hypoglycemia

543
Q

Beta blockers impair the body’s natural ability to breakdown glycogen stores to raise BG levels as an interaction with what drug?

A

Insulin

544
Q

Sulfonylureas are ass with _____ when looking at insulin

A

Hypoglycemia

545
Q

Alcohol is ass with _____ when looking at insulin

A

Hypoglycemia

546
Q

Thiazide diuretics are ass with _____ when looking at insulin

A

Hyperglycemia

547
Q

Beta blockers are ass with ____ when taking insulin

A

Hypoglycemia

548
Q

Lispro insulin is also called

A

Humalog

549
Q

Humalog is short duration and ______ ___

A

Rapid acting and analog

550
Q

The onset for humalog is 15 to ____ min

A

30

551
Q

The peak for humalog is 30 min to ____ hours

A

2.5

552
Q

The duration for humalog is 3 to ____ hours

A

6

553
Q

Lispro can be given ___ 5-15 min before meals

A

SC

554
Q

Lispro can be dosed after meals…true or false?

A

True

555
Q

Lispro can be given IV…true or false?

A

True

556
Q

Lispro can be external insulin pumps and sliding scale…true or false?

A

True

557
Q

Humbling R(regular) is short duration and _______

A

Slower acting

558
Q

Regular insulin’s onset is 30 to ___ min

A

60

559
Q

Regular insulin’s peak is 1 to __ hours

A

5

560
Q

Regular insulin’s duration is __ to 10 hours

A

6

561
Q

Regular insulin can be give SC __ min bf meals

A

30

562
Q

Regular insulin can be given IV….true or false?

A

True

563
Q

Regular insulin can be used for external insulin pumps and sliding scale….true or false?

A

True

564
Q

Regular insulin is ___ given IM

A

Rarely

565
Q

NPH insulin is also called ____ __

A

Humulin N

566
Q

NPH insulin is an intermediate duration and can be _____

A

Suspension with protamine

567
Q

NPH’s duration is 16 to ___ hours

A

24

568
Q

NPH’s peak is 6 to __ hours

A

14

569
Q

NPH’s onset is 1-___ hours

A

2

570
Q

NPH may mix in syringe with short duration insulins(____ and shower acting)

A

Rapid acting

571
Q

NPH is the only ____ insulin

A

Cloudy

572
Q

NPH insulin usually given SC with ____ insulin bf breakfast and dinner (2x a day)

A

Regular

573
Q

____ is also called insulin glargine

A

Lantus

574
Q

Lantus is a __ duration insulin and is an analog one

A

Long

575
Q

Lantus’ onset is ____

A

70 min

576
Q

Lantus’ peak is ____

A

No peak

577
Q

Lantus’ duration is ____

A

18-24 hours

578
Q

Lantus is given ___ 1-2 xs a day and with regular insulin adjusted for each meal

A

SC

579
Q

Lantus insulin does not ___in the same syringe with other insulins

A

Mix

580
Q

Pramlintide is an amylin mimetic and _____injectable med

A

Non insulin

581
Q

This is a preg cat c drug

A

Pramlintide

582
Q

This drug lowers glucose levels and for all types of DM

A

Pramlintide

583
Q

This drug indicates the actions of the naturally occuring peptide hormone amylin to decrease gastric emptying and inhibit secretin of glucose

A

Pramlintide

584
Q

Pramlintide satiates as well(meaning-____)

A

Helps decrease caloric intake

585
Q

Glucagon _____ postprandial glucose levels

A

Reduces

586
Q

Pramlintide is ass with nausea and the edu with that is _____

A

To report manifestations to the provider and they can decrease the dose

587
Q

The Pramlintide reaction at injection sites are _______

A

Generally self limiting

588
Q

The TU with Pramlintide is for ____

A

Type 1 and 2 DM as an insulin or hypoglycemic drug supplement

589
Q

The TU for Pramlintide is for a supplemental ____control for CT’s with TYPE 1 and 2 DM and with ineffective glucose control with insulin therapy

A

Glucose

590
Q

Pramlintide is used in conjunction with ____ ____

A

Insulin therapy

591
Q

The MOA for Pramlintide is an amylin analog which is a ____ hormone released with insulin

A

Peptide

592
Q

Pramlintide inhibits postprandial release and ______ gastric emptying

A

Slows

593
Q

Slower gastric emptying ____ appetite

A

Suppresses

594
Q

Nausea and injection site reactions are ADR of

A

Pramlintide

595
Q

Hypoglycemia that is severe when combined with insulin is an ADR of

A

Pramlintide

596
Q

With Pramlintide, you want to recommend a ____ insulin dosage when starting therapy

A

Reduced

597
Q

With Pramlintide, you want to monitor for signs of hypoglycemia , that tend to occur ____ hours after dosing

A

3

598
Q

recommend ____ titration of doses with Pramlintide

A

gradual

599
Q

mon for perisitent ___and vomiting(more common with type 1 than 2) with Pramlintide

A

nausea

600
Q

dont mix____ with insulin in the same syringe

A

Pramlintide

601
Q

give Pramlintide SC in ___ or abdomen

A

thigh

602
Q

give ____ prior to meal that have at least 30 g of carbs

A

Pramlintide

603
Q

rotate injection sites with ___

A

Pramlintide

604
Q

expect the peak action of Pramlintide ___ after dosing

A

20 min

605
Q

refridgerate ____ unopened vials until the exp date

A

Pramlintide

606
Q

Give ____ SC bf major meals

A

Pramlintide

607
Q

Ensure the injection is at least 5cm/2in from the injection site for any ___ given at that time

A

Insulin

608
Q

Give oral meds 1 hour bf or 2 hr after Pramlintide injections, to prevent ____ abosorption of the oral med

A

Delayed

609
Q

When on what drug do you wear a medical alert bracelet and carry a carb snack at all times?

A

Pramlintide

610
Q

With what drug do you watch for hypoglycemia esp. 3 hours after dosing?

A

Pramlintide

611
Q

You want to test BG to confirm then take a carb snack if low then test again in 15 to 20 min and repeat if still low with what drug?

A

Pramlintide

612
Q

You want to lie down when nauseated with what drug?

A

Pramlintide

613
Q

You want to keep unopened vials in the fridge not freezer with what drug?

A

Pramlintide

614
Q

Opened vials of __ can be kept cool or at room temp and then discarded after 28 days and you keep the vials out of sunlight

A

Pramlintide

615
Q

Don’t mix_____ med with insulin in the same syringe

A

Pramlintide

616
Q

A poor insulin regimen adherence is a contraindication to what drug?

A

Pramlintide

617
Q

Gastroparesis and drugs that affect GI motility are contraindications for which drug?

A

Pramlintide

618
Q

Kidney failure and dialysis are contraindications for which drug?

A

Pramlintide

619
Q

Thyroid disease and ETOH use are contraindications for what drug?(P)

A

Pramlintide

620
Q

Osteoporosis is a contraindication for what drug?

A

Pramlintide

621
Q

Visual or dexterity impairments are precautions with what drug?

A

Pramlintide

622
Q

Insulin increases hypoglycemia risk and is a interaction for what drug?

A

Pramlintide

623
Q

The absorption of oral drugs slows, the ct should take them one hour bf or ____ after Pramlintide

A

Two hours

624
Q

Opioids ______ gastric emptying

A

Slow

625
Q

Precose and Glyset ___ food absorption and further slow gastric emptying

A

Delay

626
Q

When on Pramlintide and insulin, hypoglycemia risk is increased, so you ________ 50% when on both

A

Decrease the ct per meal rapid or short acting insulin dose by

627
Q

Rapid acting insulin’s onset is 15- ___ min

A

30

628
Q

Lispro/Humalog are rapid acting insulin’s and have a peak of 30 mi to ____

A

2.5 hours

629
Q

The duration for lispro is 3 to_____ hours

A

6

630
Q

Short acting insulin is regular insulin and have an onset of

A

30 min to 1 h

631
Q

Humulin R has a peak of

A

1-5 hours

632
Q

The duration for Humulin R is

A

6-10 hours

633
Q

Intermediate acting insulin’s are NPH or Humulin N and has an onset of 1-2

A

Hours

634
Q

NPH insulin’s peak is

A

6-14 hours

635
Q

NPH’s duration is

A

16 to 24 hours

636
Q

Long acting insulins are insulin glargine(Lantus) and has an onset of ____ min

A

70

637
Q

There is ____ peak for glargine insulin

A

No

638
Q

The duration of lantus is

A

18-24 hours

639
Q

Why are sulfonylureas not used for type 1 DM?

A

High risk of hypoglycemia, they lower glucose levels

640
Q

Why should pts with kidney failure, alcoholism, heart failure, or COPD not take meta form in?

A

Contrast dyes increase risk of acute renal failure and lactic acidosis

641
Q

Why should pts with kidney failure, alcoholism, heart failure, or COPD not take metformin?

A

Avoid alcohol due to hypoglycemia and lactic acidosis

642
Q

Why should pts with kidney failure, alcoholism, heart failure, or COPD not take meta form in?

A

Cimetidine increase risk of lactic acidosis

643
Q

Why should pts with kidney failure, alcoholism, heart failure, or COPD not take meta form in?

A

Liver impairment, alcoholism, shock and infection inhibit breakdown of lactic acid

644
Q

Why should pts with kidney failure, alcoholism, heart failure, or COPD not take meta form in?

A

Renal impairment and dehydration elevates drug blood levels and increasing lactic acidosis risk

645
Q

regular Insulin should be administered when?

A

SubQ 30 min bf meal cuz its short acting due to onset (30m-1h)

646
Q

The nurse can give insulin in which ways?

A

SC, IV, and insulin pump(SC)

647
Q

Giving SC insulin directions

A

Roll NPH insulin to disperse suspension

648
Q

Giving SC insulin directions

A

Draw regualr insulin first if mixing with NPH

649
Q

Giving SC insulin directions

Think injection site

A

Abdomen is best for consistent levels, you can use arm and thigh(but its the slowest absorption here)

650
Q

Giving SC insulin directions

Think preventing lipohypertrophy

A

Site rotation within the same general area to prevent lipohypertrophy

651
Q

Giving SC insulin directions

Think how many times you use a site a month

A

Use the same site only once a month and they should be 1 in apart

652
Q

Giving IV insulin directions

Think short acting or long acting

A

Use short acting only

653
Q

Giving IV insulin directions

Think emergency or every day use

A

Emergency situations

654
Q

Giving IV insulin directions

Think amts and diluted or not diluted

A

Diluted in NS(1:1)

Start at 0.1 U/kg/hour

655
Q

Giving IV insulin directions

Think amts

A

100 U in 100 mL

656
Q

Isotonic solutions ____ cause a shift in cells

A

Don’t

657
Q

Giving insulin in pump form(SC) directions

Think short or rapid acting

A

Short acting insulins only

658
Q

Giving insulin in pump form(SC) directions

Think cost and microeposits

A

Expensive and there will be microdeposits that decrease the amt of drug delivered

659
Q

Fluid and electrolyte balance

A

The process of regulating the ECF volume

660
Q

Fluid and electrolyte balance

A

The process of regulating body fluid osmolality

661
Q

Fluid and electrolyte balance

A

Process of regulating the plasma electrolyte concentrations

662
Q

optimal Fluid and electrolyte balance

A

Keeps the volume, osmolality and electrolyte fluid concentrations within normal range

663
Q

To maintain an optimal Fluid and electrolyte balance, output must be ______by intake, and intake must be absorbed

A

Matched

664
Q

Sodium’s range is ______

A

135 to 145

665
Q

Sodium is the major _____ cation and maintains concentration and volume of ECF

A

ECF

666
Q

Sodium is essential for _____ of acid base and fluid balance and active/passive transport mechanisms.

A

Maintenance

667
Q

Sodium is essential for ____ and conduction of nerve and muscle tissue

A

Irritability

668
Q

K range is

A

3.5-5/5.5

669
Q

K plays a vital role in cell metabolism and _____ of nerve impulses

A

Transmission

670
Q

K is needed for cardiac, ___ and muscle tissues

A

Lung

671
Q

K is needed for _____ base balance

A

Acid

672
Q

K has a reciprocal action with ____

A

Sodium

673
Q

K is the major _____ cation and when changed in balance causes clinical problems

A

ICF

674
Q

K is needed for ______ _____potential of nerve and muscle cells

A

Resting membrane

675
Q

Hypotonic moves fluid ____cell and dilute things

A

Into

676
Q

Isotonic is an ____ ____ and dont effect the cell fluid shit

A

Equal balance

677
Q

Hypertonic solutions move fluid ___ ___ ___

A

Out of cell

678
Q

RF for impaired fluid and electrolyte balance populations at risk

A

Young kids, infants, and old people

679
Q

Hypervolemia is due to _____failure, kidney disease, and cirrhosis

A

Heart

680
Q

Hypervolemia is due to hyperaldosteronism and severe _______

A

Stress

681
Q

Hypervolemia is due to long use of corticosteroids and fluid shifts following _____

A

Burns

682
Q

Hypovolemia is due to excessive GI loss like _____, ______, ______

A

Vomit, NG suctioning, diarrhea

683
Q

Hypovolemia is due to excessive skin loss(diaphoresis without sodium and _______ _____

A

Water replacement

684
Q

Hypovolemia is due to excessive renal system losses like _____ _______, kidney disease, adrenal insufficiency

A

Diuretic therapy

685
Q

Hypovolemia is due to burns in the ___ _____

A

Third space

686
Q

Hypovolemia is due to hemorrhage or ___ loss

A

Plasma

687
Q

Hypovolemia is due to altered intake like _____ nervosa and ______swallowing

A

Anorexia; impaired

688
Q

Hypovolemia is due to nausea, confusion, and _____(decreased intake of water and Na)

A

NPO

689
Q

If fluid and electrolytes are not absorbed, they _____ in the GI tract and leave the body in feces

A

Remain

690
Q

If you have a cognitive disorder or a chronic illness you are at greater risk for ____ imbalance

A

Electrolyte

691
Q

Hyperkalemia is due to increased body ____

A

Potassium

692
Q

Hyperkalemia Is due to IV K administration and _____

A

Salt

693
Q

Hyperkalemia is due to blood ______

A

Transfusion

694
Q

Hyperkalemia is due to _____insulin and DKA

A

Insufficient

695
Q

Hyperkalemia is due to tissue catabolism which involves: sepsis and ____

A

Burns

696
Q

Hyperkalemia is due to tissue catabolism which involves: trauma and ____

A

Surgery

697
Q

Hyperkalemia is due to tissue catabolism which involves: fever and ____

A

MI

698
Q

hyperkalemia is due to uncontrolled diabetes _____

A

mellitus

699
Q

hyperkalemia is due to kidney failure and ____ dehydration

A

severe

700
Q

hyperkalemia is due to K sparing diuretics, and ___ inhibitors

A

ACE

701
Q

hyperkalemia is due to adrenal ____

A

insufficiency

702
Q

hypokalemia is due to hyperaldosteronism and ____dietary intake(rare)

A

inadequate

703
Q

hypokalemia is due to prolonged administration of non electrolyte containing IV solutions like ____

A

5% DEXTROSE IN WATER

704
Q

with age increasing, you see __ kidney function and med conditions due to salt substitutes, CE inhibitors, and L sparing diuretics

A

decreasing

705
Q

hypokalemia is due to receiving ____ parenteral nutrition

A

total

706
Q

hypokalemia is due to metabolic _____

A

alkalosis

707
Q

hypokalemia is due to excessive GI losses like vomit and ____

A

NG suctioning

708
Q

hypokalemia is due to excessive GI losses like diarrhea and ___ laxative use

A

excessive

709
Q

hypokalemia is due to renal losses like ______ use of K excreting diuretics like furosemide and corticosteroids

A

excessive

710
Q

hypokalemia is due to skin losses like diaphoresis and __ losses

A

wound

711
Q

hyponatremia is due to deficient ECF volume and excessive GI losses like(___,___,___,____)

A

vomit, NG suctioning, diarrhea, and tap water enemas

712
Q

hyponatremia is due to renal losses like(___,____,___,____)

A

diuretics, kidney disease, adrenal insufficiency, excessive sweating

713
Q

hyponatremia is due to ____ losses

A

skin

714
Q

skin losses ass with hyponatremia

think B and WD

A

burns and wound drainage

715
Q

skin losses ass with hyponatremia think GI O, and PE

A

GI obstruction and peripheral edema

716
Q

skin losses ass with hyponatremia

think a

A

ascites

717
Q

hyponatremia is due to _____ or normal ECF volume

A

increased

718
Q

hyponatremia is due to___ water oral intake and SIADH

A

excessive

719
Q

hyponatremia is due to edematous _____

A

states

720
Q

hyponatremia is due to ____ failure, cirrhosis and nephrotic syndrome

A

heart

721
Q

hyponatremia is due to ____IV administration of dextrose 5% in water

A

excessive

722
Q

hyponatremia is due to NPO status bc_____

A

inadequate sodium intake

723
Q

hyponatremia is due to ____ irrigating solutions

A

hypotonic

724
Q

hyponatremia is due to hyper or hypoglycemia

A

hyperglycemia

725
Q

old or younger adults are more at risk for hyponatremia due to chronic illnesses, diuretics, and low sodium intake?

A

older adults

726
Q

wound drainage(GI) and low aldosterone are RF of _____

A

hyponatremia

727
Q

a freshwater submersion accident and kidney disease are RF of ____

A

hyponatremia

728
Q

seizure meds, SSRIs, and desmopressin are RF of

A

hyponatremia

729
Q

NPO/water deprivation and high sodium intake can lead to

A

hypernatremia

730
Q

heat stroke and hypertonic IV fluids can lead to

A

hypernatremia

731
Q

hypertonic tube feedings and bicarb intake lead to

A

hypernatremia

732
Q

kidney failure and cushing syndrome lead to excessive sodium retention that leads to

A

hypernatremia

733
Q

aldosteronism and glucocorticoids lead excessive sodium retention that leads to

A

hypernatremia

734
Q

fever and diaphoresis can lead to

A

hypernatremia

735
Q

burns and respiratory infection can lead to

A

hypernatremia

736
Q

DI and hyperglycemia and watery diarrhea can lead to(hypo or hypernatremia?)

A

hypernatremia

737
Q

excessive intake of oral sodium can lead to

A

hypernatremia

738
Q

vomit and diarrhea are

A

causes of fluid and electrolyte balance

739
Q

organ failure and nausea are

A

causes of fluid and electrolyte balance

740
Q

fatigue and dizziness are

A

causes of fluid and electrolyte balance

741
Q

shortness of breath and muscle cramping are

A

causes of fluid and electrolyte balance

742
Q

edema and weight changes are

A

causes of fluid and electrolyte balance

743
Q

output GT intake and absorption is a

A

cause of fluid and electrolyte balance

744
Q

output LT intake and absorption are

A

causes of disrupted fluid and electrolyte balance

745
Q

altered fluid and electrolyte distribution are

A

causes of disrupted fluid and electrolyte balance

746
Q

too little or too much isotonic fluid present ARE

A

causes of volume imbalances

747
Q

body fluid becomes hypertonic or hypotonic in

A

osmolality imbalances

748
Q

hypernatremia/water deficit and hyponatremia/water excess or intoxification are examples of

A

osmolality imbalances

749
Q

decreased output not balanced by decreased intake is a

A

causes of disrupted fluid and electrolyte balance

750
Q

output LT excessive or too rapid intake

A

causes of disrupted fluid and electrolyte balance

751
Q

increased output not balanced by increased

A

causes of disrupted fluid and electrolyte balance

752
Q

normal output but deficient intake or absorption

A

causes of disrupted fluid and electrolyte balance

753
Q

normal output but deficient intake or absorption is an example of ____GT intake and absorption

A

output

754
Q

increased output not balanced by increasing intake is an example of ____GT intake and absorption

A

output

755
Q

output less than excessive or too rapid intake is an example of output ____ intake and absorption

A

LT

756
Q

decreased output not balanced by decreasing intake is an example of output LT ___ and absorption

A

intake

757
Q

shift of vascular fluid into the interstitial space causes edema is an example of altered fluid and electrolyte ____

A

distribution

758
Q

ECV deficit, too high osmolality, and plasma electrolyte deficit are examples of

A

normal output but deficient intake or absorption

759
Q

ECV deficit, too high osmolality, and plasma electrolyte deficit are examples of

A

increased output not balanced by increased intake

760
Q

ECV excess, osmolality too low, plasma electrolyte excess are examples of

A

output LT excessive or too rapid intake

761
Q

ECV excess, osmolality too low, plasma electrolyte excess are examples of

A

decreased output not balanced by decreased intake

762
Q

ECV deficit causes are

think NOBDIOSAW

A

normal output but deficient intake of sodium and water bc of lack of access

763
Q

ECV deficit causes are

think IONBBIIOSAW

A

increased output not balanced by increased intake of sodium and water

764
Q

vomit, diarrhea, and diuretics lead to

A

increased output not balanced by increased intake of sodium and water

765
Q

DRAINING GI FISTUAL , GASTRIC SUCTION , AND INTESTINAL DECOMPRESSION LEAD TO

A

increased output not balanced by increased intake of sodium and water

766
Q

addison disease and adrenal insufficiency lead to

A

increased output not balanced by increased intake of sodium and water

767
Q

hemorrhage or burns lead to

A

increased output not balanced by increased intake of sodium and water

768
Q

acute intestinal obstruction and ascites lead to

A

rapid fluid shift from ECV into a third space

769
Q

ECV deficit causes are

think RFSFECVIATS

A

rapid fluid shift from ECV into a third space

770
Q

ECV excess causes are

think OLTETRIOSAW

A

output LT excessive or too rapid intake of sodium and water

771
Q

ECV excess causes are

think DONBBDIOSAW

A

decreased out not balanced by decreased intake of sodium and water

772
Q

excessive IV infusion of Na containing isotonic solution(0.9% NaCl, ringer) are causes of

A

output LT excessive or too rapid intake of sodium and water

773
Q

isotonic solutions are

A

0.9% NaCl and ringers

774
Q

high oral intake of salty foods and water with renal retention of Na and water are causes of

A

output LT excessive or too rapid intake of sodium and water

775
Q

oliguria and aldosterone excess and high levels of glucocorticoids are causes of

A

decreased out not balanced by decreased intake of sodium and water

776
Q

normal output but deficient intake of water causes

A

hypernatremia

777
Q

body fluids are too concentrated and osmolality too high with ___natremia

A

hyper

778
Q

increased output not balanced by increased intake of water causes

A

hypernatremia

779
Q

no access to water or inability to respond to or communicate thirst like in aphasia, coma, and infancy is an example of

A

normal output but deficient intake of water

780
Q

tube feeding without additional water intake causes

A

normal output but deficient intake of water

781
Q

vomit and diarrhea with replacement of Na but not enough water can cause

A

increased output not balanced by increased intake of water causes

782
Q

DI due to lack of ADH can cause

A

increased output not balanced by increased intake of water causes

783
Q

this is when body fluids too dilute and osmolality too low is ____natremia

A

hypo

784
Q

output LT excessive or too rapid intake of water can cause

A

hyponatremia

785
Q

decreased output not balanced by decreased intake of water can cause

A

hyponatremia

786
Q

IV 5% dextrose in water(D5W) infusion with excess rate or amt is an example of

A

output LT excessive or too rapid intake of water

787
Q

rapid oral ingestion of massive amts of water (like in child abuse, club initiation, psychiatric disorder) is an example of

A

output LT excessive or too rapid intake of water

788
Q

overuse of tap water enemas or hypotonic irrigating solutions is an example of

A

output LT excessive or too rapid intake of water

789
Q

massive replacement of water without Na during vomitting or diarrhea is an example of

A

output LT excessive or too rapid intake of water

790
Q

excessive ADH is an example of

A

decreased output not balanced by decreased intake of water

791
Q

plasma K deficit is

A

hypokalemia

792
Q

normal output but deficient K intake can cause

A

hypokalemia

793
Q

increased output not balanced by increased K intake can cause

A

hypokalemia

794
Q

prolonged anorexia or lack of K-rich foods is an example of

A

normal output but deficient K intake

795
Q

no oral intake plus IV solutions not containing K can casue

A

normal output but deficient K intake

796
Q

vomit and diarrhea are an example of

A

increased output not balanced by increased K intake

797
Q

use of K wasting diuretics or drugs that increase renal K excretion is an example of

A

increased output not balanced by increased K intake

798
Q

excessive aldosterone effects like when cirrhosis, heart failure and hyperaldosteronism is an example of

A

increased output not balanced by increased K intake

799
Q

high level of glucocorticoids(cushing disease and corticosteroid therapy) is an example of

A

increased output not balanced by increased K intake

800
Q

rapid K shift from ECF into cells can cause

A

hypokalemia

801
Q

alkalosis and excessive insulin is an example of

A

rapid K shift from ECF into cells

802
Q

excessive beta adrenergic stimulation is an example of

A

rapid K shift from ECF into cells

803
Q

plasma K excess that is

A

hyperkalemia

804
Q

output LT excessive or too rapid K intake can cause

A

hyperkalemia

805
Q

decreased output not balanced by decreased K intake can cause

A

hyperkalemia

806
Q

rapid K shift from cells into ECF that can cause

A

hyperkalemia

807
Q

IV K infusion with excess rate or amt that is an example of

A

output LT excessive or too rapid K intake

808
Q

massive transfusion (GT 8 U for adults) of stored blood that is an example of

A

output LT excessive or too rapid K intake

809
Q

oliguria and use of K sparing K diuretics, ACE inhibitors and drugs that decrease renal K excretion that is an example of

A

decreased output not balanced by decreased K intake

810
Q

lack of aldosterone is an example of

A

decreased output not balanced by decreased K intake

811
Q

lack of insulin or acidosis that is an example of

A

rapid K shift from cells into ECF

812
Q

massive sudden cell death that is an example of

A

rapid K shift from cells into ECF

813
Q

check K level first then put on a pump for that is a

A

nursing and collaborative interventions to support fluid and electrolyte balance

814
Q

prevention examples are pt teaching, dietary measures, and fluid management as

A

nursing and collaborative interventions to support fluid and electrolyte balance

815
Q

do adequate intake with vomiting or diarrhea as ___ management

A

fluid

816
Q

limit intake when prone to edema as ______management

A

fluid

817
Q

check skin tugor and VS and I & O’s as primary prevention for

A

nursing and collaborative interventions to support fluid and electrolyte balance

818
Q

tenting is a sign of ECV ____

A

deficit

819
Q

rapid thredy pulse and postural hypotension are signs of ECV ___

A

deficit

820
Q

bounding pulse is a sign of ECF _____

A

overload

821
Q

oliguria is a sign of ECF ___

A

deficit

822
Q

weight loss is ass with __ deficit

A

ECF

823
Q

weight gain is ass with ECF _____

A

overload

824
Q

check weight and do a mental health exam and check muscle strength as a primary prevention for

A

nursing and collaborative interventions to support fluid and electrolyte balance

825
Q

impaired cerebral function occurs with _____ and hypernatremia

A

hypo

826
Q

flaccid muscle occur with ____ and _____

A

hyper and hypokalemia

827
Q

this is based on the principle that intake must balance output

A

nursing and collaborative interventions to support fluid and electrolyte balance

828
Q

teach pts taking K wasting diuretics to increase K consumption is

A

nursing and collaborative interventions to support fluid and electrolyte balance

829
Q

teach pts with oliguria renal disease to decrease intake of Na and K bc their output is decreased is

A

nursing and collaborative interventions to support fluid and electrolyte balance

830
Q

nurses must give ____ when intervening and at the right temp

A

fluids

831
Q

collab intervention as ordering a med true or false?

A

true

832
Q

provide safety and comfort is a

A

nursing interventions to support fluid and electrolyte balance

833
Q

facilitate oral intake if needed is a

A

nursing interventions to support fluid and electrolyte balance

834
Q

administering collab interventions

A

nursing interventions to support fluid and electrolyte balance

835
Q

adjust the fluid I and O is a

A

nursing interventions to support fluid and electrolyte balance

836
Q

treat the underlying cause is a

A

nursing interventions to support fluid and electrolyte balance

837
Q

monitor for complications of therapy is

A

nursing interventions to support fluid and electrolyte balance

838
Q

teach pts how to prevent imbalances or when to seek help is

A

nursing interventions to support fluid and electrolyte balance

839
Q

the nurse can do this for pts on fluid restrictions…

A

keep fluids out of sight, lubricate the lips, do oral hygiene, and swish fluid in mouth bf swallowing

840
Q

screening for F and EB is not routine for the general population is a

A

secondary prevention to support fluid and electrolyte balance

841
Q

mon serum blood levels as part of disease management is a

A

secondary screening process

842
Q

focus on intake, and maybe use dialysis or diuretics is

A

collaborative interventions to support fluid and electrolyte balance

843
Q

fluid and electrolyte support and med management is

A

collaborative interventions to support fluid and electrolyte balance

844
Q

treat underlying condition and pharmacotheray with diuretics, insulin, and vasopressin is

A

collaborative interventions to support fluid and electrolyte balance

845
Q

electrolyte supplements(Na, K) and water replacement therapy(oral fluids, IV fluids) are

A

collaborative interventions to support fluid and electrolyte balance

846
Q

NS 0.9% and lactated ringer solution are ____solutions

A

isotonic

847
Q

D5W and 10% dextrose in 0.225% saline are ____ solutions

A

isotonic

848
Q

give hypotonic for a ___ state

A

hypertonic

849
Q

0.45% NaCl and 0.33 NS and 0.225 NS are ___ solutions

A

hypotonic

850
Q

3% NS and 5% dextrose in .45% saline are

A

hypertonic fluids

851
Q

10% dextrose in water is a

A

hypertonic fluid

852
Q

130-145 is the range for

A

Na

853
Q

3.5 to 5 is the range for

A

K

854
Q

9-11 is the range of

A

Ca

855
Q

1.5 to 2.5 is the range for

A

Mg

856
Q

no dyspnea and no distended neck veins are

A

EF

857
Q

no visible edema and moist lips are

A

EF

858
Q

alert and attentive is an

A

EF

859
Q

dry lips are a sign of ECF _____

A

deficit

860
Q

impaired cerebral functions occurs with hypo and hyper____

A

natremia

861
Q

edema is a sign of ECF __

A

overload

862
Q

distended neck veins are a sign of ECF ____

A

overload

863
Q

dyspnea is a sign of ECF ___

A

overload

864
Q

osmotic forces are

A

the amt of pressure required to stop the osmotic flow of water

865
Q

osmosis is from high concentration to ____concentration

A

low

866
Q

osmosis is from a region of low solute to ___solute

A

high

867
Q

osmosis is

A

passive

868
Q

diffusion is the movement of ____ and water between the ECF

A

electrolytes

869
Q

diffusion is passive and __ a concentration gradient

A

down