3: Peds Neuro Flashcards

1
Q

What is cerebral palsy

A

Defined as brain damage that happens before, during or soon after birth
o Does not reach motor milestones
o Exhibits abnormal muscle OR
o Shows qualitative differences in movement patterns

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2
Q

CP is classified by?

A

· type of muscle tone
· topography (if spastic muscle tone)
· GMFCS level (I-V)

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3
Q

o Motor types

A

spastic, dyskinetic, ataxic, mixed

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4
Q

Spastic

A

Most common
“Stiff” or permanently contracted muscles in synergy pattern

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5
Q

Dyskinetic

A

Alternating tone with slow, uncontrolled writhing movements

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6
Q

Ataxic

A

rare form
poor coordination
difficulty with quick or precise movements

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7
Q

Mixed-

A

a child with two or more forms
most common mixed form is a blend of the spastic and dyskinetic

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8
Q

Mono

A

one limb affected

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9
Q

Hemiplegia

A

half body

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10
Q

Diplegia

A

legs more affected than arms

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11
Q

Triplegia

A

3 limbs affected, more affected on one side

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12
Q

Quad

A

both arms and legs equally affected

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13
Q

Common impairments/symptoms-CP

A

Hip Dysplasia and spasticity

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14
Q

Common PT treatments-CP

A

o Exercise and range of motion
o Braces Ex: AFOs, Hip braces
o Careful positioning and activities which decrease tone Ex: Seating, sleeping position
o Strategies to reduce/inhibit tone Ex: PNF
o Proper use of equipment

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15
Q

Hip dysplasia-

A

-Caused by forces on the joint by spastic muscles, positioning
- shallow acetabulum

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16
Q

Hip dysplasia prevention

A

· BC Hip Surveillance Program
· Assessment every 6-12 months
· early intervention= fewer salvage treatment operations

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17
Q

Hip dysplasia tx

A

· Standing programs, weightbearing
· Bracing, specialized taping
· Positioning
· Addressing muscle imbalances

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18
Q

What is Spina Bifida

A

· Neural tube fails to close in utero, due to a defect

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19
Q

Classification of SB by type/severity

A

Occulta- defect present, but structures are intact- may not know that you have it (least severe)
Meningocele- defect present, meninges protrude out of the canal

Myelomeningocele- defect present, meninges & spinal cord protrude out of the canal

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20
Q

Occulta

A
  • hairy patch at birth
  • dimple’ in back
    - Often symptom free
    -May lead to tethered cord
21
Q

Meningocele

A

· Fluid filled sac, containing meninges, present at birth
· Rare
· Needs surgery
· May have abnormal development of spinal cord
· May have minor neurological symptoms & issues with bowel & bladder

22
Q

Myelomeningocele

A

· Fluid filled sac, containing meninges & spinal cord, present at birth
· Needs surgery
· Loss of function below level of injury
· Full motor and sensory loss (like a spinal cord injury)

23
Q

Chiari II

A

· Think of a balloon stretching downwards
· characterized by beaked midbrain, downward displacement of the tonsils, and cerebellar vermis, and spinal myelomeningocele

24
Q

Hydrocephalus

A

· May require shunt
· Can cause cognitive defecits

25
Clubfoot:
Plantarflexion and inversion caused by a shortened Achilles tendon
26
Secondary Orthopedic Issues: Spina Bifida
o Caused by decreased functional movement of joints & lack of mobility o Can result in: loss of ROM, contractures, pain, weakness, injuries, etc. o Examples: Club foot, knee flexion contracture, dislocated or subluxed hips, kyphosis, scoliosis, osteoporosis with or without fractures
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SB ROM limitations
Common limitations: · Tightness in hip flexors & hip adductors · Tightness in the dorsiflexors or evertors of the ankle
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SB Common PT treatments
· Maximize ROM · Increase/maintain strength · Maintain alignment of the extremities and joints · Stabilize the spine and extremities · Maximize function · Provide comfort · Skin protection
29
What is Muscular Dystrophy | What kind is most common, who gets it more?
Inherited disorder o Most common type is Duchenne's Musclar Dystrophy (DMD)only shows in males o More common in male
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o how do you get MD?
X-Linked recessive
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o Pathophysiology of MD
· Muscle tissue is replaced by fat · Characterized by rapidly progressive muscle weakness which starts in the legs and pelvis and later affects the whole body · Pseudohypertrophy- looks like muscle but is fat
32
MD symptoms:
weakness due to progressive loss of myofibrils
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Secondary Symptoms MD | What do you see a child do
o contractures o postural malalignment, especially in antigravity postures o Scoliosis etc. From postural issues o Gowers sign: "walks" hands up thighs =due to weakness in proximal muscle groups
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Early childhood: ROM fairly normal (changes at 5 years +)
· Mild tightness in some muscles=Generally notice gastroc, soleus and TFL first · Normal lordotic curve increased with accompanying winging of the scapula · Scoliosis typically develops at or just before adolescence
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Childhood:
· Clumsiness · Falling · Inability to keep up with peers · Difficulty with stair climbing · Difficulty rising from sitting · Gait pattern changes (ex: Trendelenberg, toe walking, retracting of shoulders, decreased reciprocal arm swing)
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Adolescence MD:
· Increased disability · Progression of weakness · Contractures · Walking may be lost · Walking cessation (stopping) usually occurs by the age of 10 to 12
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Physiotherapy: MD
o Family- centered approach o Exercise can be controversial- eccentric exercise can be detrimental o Best exercise is isometric- weight bearing o ROM, stretching and positioning to prevent contracture o Supportive bracing (Ex: AFOs) o Focus on scoliosis prevention
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What is pediatric TBI & why is it different than adult TBI?
Because it is happening in a brain that has not fully developed yet
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EtiologyTBI- Infants
· 2/3+ caused by falls
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Etiology TBI Preschool Children
falls 55%, MVA 22%
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Etiology TBI: School-age children
: MVA 31%, falls 31%, sports and recreation activities 32%
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Etiology: Adolescents
sports & rec 43%
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Other reasons for TBI in children
Shaken Baby Syndrome Near drowning (under 4 and adolescents most common) Brain tumours
44
Common impairments/Symptoms TBI
· Can depend on the stage of development of the brain · Some symptoms will look much like adults and be based on part of the brain affected · Damaged areas may never get a chance to develop
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DCD
o Developmental Coordination Disorder o General clumsiness, lack of coordination, poor strength
46
Down’s Syndrome
o Trisomy 21 (extra chromosome) o Low tone, late achieving motor milestones o Ligamentous laxity
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Other:· General population physiotherapy goals:
· Children with low tone and joint hypermobility need to be stabilized · Children with increased muscle tone and limited joint range need mobility and muscle extensibility · All children must be able to move freely from one position to another
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· General Pop. PT treatment strategies (including equipment): | think techniques
Hands on * Manual stretching, PNF & other facilitation techniques, positioning, manual resistance Exercise (play based!) * Varied positions to encourage therapy goals Other techniques * Adapted activities (ex: Hippotherapy), partial WB treadmill for gait training, pool therapy, sports
48