3. Dental disease Flashcards

1
Q

What is the WHO definition of health?

A

Health is the state of complete physical, mental and social well-being and not merely the absence of disease or infirmity.

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2
Q

What are the main micro-organisms responsible for caries? (3)

A

Streptococcus mutans
Lactobaccilli
Actinomyces

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3
Q

What is the main micro-organisms responsible for periodontitis?

A

Porphyromonas gingivalis
Many other bacteria.

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4
Q

Name 6 causes of tooth surface loss

A
  1. Dental caries
  2. Attrition
  3. Abrasion
  4. Abfraction
  5. Erosion
  6. Trauma (injuries)
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5
Q

What is dental caries?

A

A dynamic process caused by acids from carbohydrate fermentation by oral micro-organisms, involving the exchange of calcium and phosphate ions between tooth structure and saliva.

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6
Q

What 4 criteria are required for caries?

A
  1. A tooth surface
  2. Bacteria
  3. Fermentable carbohydrate (sugars)
  4. Time
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7
Q

What teeth are at highest risk for carious lesions? and why?

A

Permanent first and second molars due to length of time erupted in the oral cavity, and their complex surface anatomy to which plaque adheres.

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8
Q

What acid does bacteria produce to form caries?

A

Lactic acid

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9
Q

What sugars are mainly implicated in caries? (2)

A

Sucrose and glucose

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10
Q

What ions are removed during demineralisation from the tooth structure? (2)

A

Calcium and phosphate

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11
Q

What is the critical pH of enamel, below of which decalcification occurs?

A

pH 5.5

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12
Q

What are methods of detecting caries?

A

Visual examination
Bitewing radiograph
Fibreoptic transillumination
Lasers
Dyes
Probe (but may cause damage)

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13
Q

How are streptococci involved in the cariogenic process?

A

A group of gram-positive cocci which includes Streptococcus mutans and Streptococcus sobrinus metabolise sugars at low pH (acidogenic) and are v important in caries initiation.
They are also called viridians streptococci.

Streptococcus mutans adheres in the in the ‘biofilm’ on teeth by converting sucrose into an extremely sticky substance called dextran. The bacteria also act by converting the sugars in the diet to acids (especially lactic acid). The acids destroy (decalcify) the enamel and dentine of the teeth. The acids cause the pH to fall and when the tooth surface plaque pH drops below 5.5 tooth demineralisation proceeds faster than remineralisation. (net tooth surface loss)

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14
Q

How are lactobacillus species involved in the caries process?

A

Gram-positive bacilli which survive at low pH (aciduric). Isolated in large numbers from carious dentine.

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15
Q

What is a white spot>

A

The earliest clinical appearance of caries, caused by loss of calcium and phosphate ions from enamel prisms.

Decalcification produces opaque whitish areas on the tooth, which are painless.

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16
Q

Is decalcification reversible?

A

To a point if the person changes their diet and reduces intake of more cariogenic carbohydrates.

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17
Q

What is the critical pH of dentine?

A

pH 6.5.

The critical pH for dentine demineralisation is higher, and as dentine is softer than enamel, caries spreads more rapidly once it reaches dentine.

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18
Q

What are the consequences if caries is not treated?

A
  1. Destroys enamel causing a cavity.
  2. Reaches dentine and spreads rapidly.
  3. Pt experiences reversible pulpitis symptoms: pain sweet/sour or hot/cold, subsides within secs of removing stimulus, poorly ‘localised’, approximate area.
  4. The inflammation causes swelling of pulp but, since pulp is confined in rigid pulp chamber, the pressure builds up. Thus severe and persistent pain in tooth.
  5. Swelling also stops the blood flow into the pulp - which then dies. Pain may subside for a while.
  6. However, dead pulp is infected with bacteria from mouth (odontogenic infection). Infection spread through root apex into alveolar bone and cause apical periodontitis. Painful, when touch or bites.
  7. Tooth must be RCT or XLA otherwise dental abscess, granuloma or cyst will form.
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19
Q

What diet advice will minimise tooth damage by caries?

A
  • Avoid consuming sugars completely.
  • Minimising non-milk sugar intake
  • Eating sugar-containing products all at once and over a short period of time.
  • Not eating sugars as the last thing at night.
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20
Q

What is arrested caries?

A

Under favourable conditions, a lesion may become inactive - black or dark brown in colour - and has a hard or leathery consistency.

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21
Q

How does caries spread into dentine?

A

Occurs when enamel caries extends to the amelodentinal junction.
Spreads laterally and, as it progresses, is cone shaped with base on amelodentinal junction.
As dentine is vital, it can respond by laying down reactionary or secondary dentine at surface of the pulp chamber.

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22
Q

What is Early Childhood Caries (ECC)?

A

also known as ‘nursing bottle caries’.

Describes extensive caries in primary incisors due to prolonged exposure to sugar-containing drinking in a feeding bottle or cup. Teeth most likely affected are maxillary anterior teeth.

Children fall asleep with sweetened liquids or feed children sweetened liquids multiple times during the day.

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23
Q

What is occult caries?

A

Describes extensive dentine caries in the presence of minimal or no clinically evident enamel breakdown. Most commonly occurs under occlusal surfaces.

An increasing problem win older children/teenagers.

May be due to increased resistance to enamel breakdown as a result of exposure to fluoride.

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24
Q

What is radiation caries?

A

Caries in people after irradiation that damages salivary glands causing hyposalivation, predisposing to caries.

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25
Q

What is rampant caries?

Causes in adults?

A

Gross caries, frequently in deciduous dentition.

‘Rampant caries’ in adults is advanced or severe decay on multiple surfaces of many teeth seen in individuals with poor oral hygiene, stimulant use (due to drug-induced dry mouth), dry mouth such as after radiotherapy in head and neck region or Sjogren syndrome, and or large sugar intake.

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26
Q

What is recurrent caries?

A

Continuation of caries after placement of restoration.

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27
Q

What is root caries?

A

Occurs following gingival recession. Varies from light yellow to dark brown in colour. Increasing problem in older patients.

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28
Q

What is secondary caries?

A

New caries occurring at restoration margins.

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29
Q

Why should you not use a sharp probe to detect caries?

A

Risk collapsing incipient lesions.

Probe should only be used to remove plaque/food debris.

30
Q

What is Blacks classification?

A

Class I cavity
= Occlusal two-thirds, pits and fissures.
- Molar and premolars (posterior teeth). Anteriors lingually.

Class II cavity
= involves two or more surfaces. Interproximal surfaces.
- Molars and premolars (posterior teeth).

Class III cavity
= interproximal surfaces
- anterior teeth (incisors and canines)

Class IV cavity
= interproximal surfaces, including incisal edge
- anterior teeth (incisors and canines)

Class V cavity
= gingival third of facial or lingual surfaces
- anterior or posterior teeth

Class VI cavity
= cusp tips
- molars, premolars, and canines

31
Q

What does DMFT index stand for?

A

Decayed, missing, filled teeth

32
Q

What is attrition?

A

Wearing away of occlusal surface caused by opposing tooth.
e.g. habits such as bruxism.
Incisal edges of anterior teeth and premolars and molar cusps wear down.

33
Q

What is abrasion?

A

Wearing away of hard tissues at the neck of the tooth by a habit such as toothbrushing with a hard brush and coarse toothpaste.
Gingivae recedes but is otherwise healthy.

34
Q

What is erosion?

A

Tooth surface loss caused by dissolution of the tooth minerals by acids (pH 5.5 or lower) other than those produced by caries.

In most patients, there is little more than a loss of normal enamel contour, but in mores severe cases, dentine or pulp may also become involved.

35
Q

What are causes of erosion?

A

Extrinsic: dietary
Fruits or fruit drinks
Carbonated drinks
Wines and other alcoholic drinks
Vinegar

Intrinsic:
- Regurgitation of stomach (gastric) acid (pH ~2), e.g. in bulimia and stomach difficulties as well as recurrent vomiting such as in anorexia or alcoholism.

36
Q

What is abfraction?

A

A notched-out area on the tooth root at the gumline. Teeth flex very slightly under improper forces in grinding and clenching and deepend notched areas.

37
Q

What are consequences of tooth surface loss?

A

Hypersensitivity and/or a cosmetic issue.

38
Q

What is apical periodontitis?

A

When the inflammation travels to the bone surrounding the tooth apex.
If the inflammation persists, it may cause an abscess which produces pain and may also result in swelling.
Pain may abate if abscess discharges.

39
Q

What is pericoronitis?

Primary treatment?

A

Infection under the operculum.

Primary treatment is by irrigation under the operculum with chlorhexidine solution (0.2%).

It may be necessary to remove the maxillary third molar to reduce occlusal trauma.

Systemic antibiotics if evidence of trismus, lymphadenopathy or spreading infection.

40
Q

What may an infant who is teething experience?

A

Irritability, disturbed sleep, flushed face, drooling, a small rise in temperature and/or a rash.

41
Q

What teeth most often get impacted?

A

Third molars, premolars and canines, because these are usually the last teeth to erupt.

42
Q

What are reasons for teeth missing from the dental arch? (3 broad)

A

Tooth may have failed to erupt.
It may not have developed
It may have been lost prematurely.

43
Q

Which teeth are most commonly genetically missing?

A

third molars, the second premolars and the maxillary lateral incisors.

44
Q

What is anodontia?

A

All teeth absent

45
Q

Supplemental teeth (extra teeth of normal shape) are uncommon. But what tooth is most frequently supplemental?

A

Maxillary lateral incisor, and in the premolar and third molar regions of either jaw.

46
Q

What are mesiodens?

A

Supernumerary, small and/or conical shaped teeth in midline of upper arch.

47
Q

Supernumerary teeth stats?
More common in x?

A

Primary teeth 0.2-0.8%.
Permanent teeth 1-3%

More common in males and maxilla.
Most common in upper incisor region.

48
Q

What is dens-in-dente?

A

Rare dental malformation that occurs when the enamel of a tooth folds into the dentin. This creates the appearance of a small tooth within a larger one.

Often requires extraction an affects lateral incisors.

49
Q

What is dilateration?

A

The abnormal angulation between the crown and root or within the root.

May be related to intrusive trauma to primary dentition.

May fail to erupt.

50
Q

Why may a first molar be impacted?

Tx?

A

Impact behind second primary molar due to:
- crowding
- abnormality in tooth eruption such as orientation of the crypt.

Tx:
- keep under observation, may self-correct if mild.
- disimpaction using separator
- extract second primary molar (but will result in space loss).

51
Q

What teeth are most commonly affected by abnormal position of tooth crypt?

A

The crypt of any tooth can be displaced or rotated.
Lower second premolar is most commonly affected.

52
Q

What is the incidence of ectopic canines?
Palatal or buccal?

A

1-2%

90% lie palatally or in line of arch.

53
Q

How can you check for an ectopic canine early?

A

At 9 should be palpable as bulge high in buccal sulcus. If not apparent by age 10, carry out clinical exam and appropriate radiographs.

54
Q

What are the treatment options for ectopic canines?

A
  • accept and review
  • extract
  • surgically expose and align orthodontically
  • transplant
55
Q

What is transposition?

Teeth most commonly affected?

A

A rare dental abnormality that occurs when two adjacent teeth switch positions.
The canine tooth is almost always involved, and the most common transpositions
- Upper jaw = canine/first premolar
- Lower jaw = canine/lateral incisor

56
Q

What is enamel hypoplasia?

A

Between birth and 6 years of age, the permanent incisors and canines are developing. If the developing tooth bud is damaged, it can produce cosmetic problem.

Enamel hypoplasia is when the tooth crown appears opaque, or yellow-brown or deformed.

Infections such as German measles (rubella), cancer treatments or jaundice may cause this type of hypoplasia.

The defects correspond to the site of tooth enamel formation at the time of the insult (‘chronological’ hypoplasia). Enamel is reduced in thickness or deficient in structure. Presentation ranges from pits and grooves to gross abnormalities.

57
Q

What is enamel hypominerasliation?

A

Enamel is of normal structure but not fully mineralised. Presents as changes in colour and translucency.

58
Q

What is the aetiology of enamel hypomineralisation?
local (4). general? Hereditary?

A

Local = infection, trauma, irradiation, idiopathic. Usually affects only one or two teeth.

General aetiology = environmental results from systemic distrubance during period of tooth formation.
- Pre-, peri-, or post-natally e.g. rubella, syphilis, childhood infections, excess exposure to fluoride.

Hereditary = amelogenesis imperfecta or ectodermal dysplasia.

59
Q

What are 2 common variants of amelogenesis imperfecta?

A
  • Hypomineralisation type. Matrix formation normal, calcification is abnormal. Mainly autosomal dominant.
  • Hypoplastic type. Matrix formation abnormal, but any tooth (enamel) matrix formed is normally calcified. Mainly X-linked.
60
Q

What is dentinogenesis imperfecta?

A

Dentine consists of reduced number of wide irregular tubules, with areas of atubular dentine.
Loss of scalloping at ADJ.
Teeth have bluish appearance.
Teeth wear rapidly as enamel is lost.

61
Q

What are 5 causes of extrinsic staining?

A
  1. Poor OH
  2. use of substances such as tobacco, betel nut, khat, tea, coffee, red wine, coloured foods, or chlorhexidine.
  3. caries
  4. trauma
  5. tooth filing materials
62
Q

How can tetracycline staining occur?

A

Brown or grey colour tooth staining caused by use of tetracyclines by pregnant or lactating mothers, or by children under the age of 8 years.

Tetracycline can cross the placenta and then enter breast milk and are taken up by developing teeth and by bone.

63
Q

How can fluorosis present?

A

white flecks or spotting or diffuse cloudiness to yellow-brown or darker patches and staining and ‘pitting’ of enamel.

64
Q

What may hypercementosis be associated with?

A

Inflammation, over-/underloading.
Paget’s disease.

65
Q

What is hypocementosis associated with?

A

Hypophosphatasia

66
Q

Individuals who have poor oral hygiene soon develop halitosis, but it is made worse by any form of oral infection, such as:

A
  • gingivitis
  • periodontitis
  • dental abscess
  • dry (infected) extraction socket
  • sinusitis
  • tonsillitis
  • ulceration
67
Q

What is sinusitis?

A

Infection of the paranasal air sinuses (maxillary most commonly, but also ethmoid, sphenoid and frontal) is usually bacteria.
It may be preceded by viral, or other factors

Diagnose from history plus tenderness over the sinus, dullness on transillumination, and radio-opacity or a fluid level on x-rays. Sinus puncture and aspiration. Computed tomography.

68
Q

What are clinical features of sinusitis?

A
  • Headache on waking is typical
  • Pain worse on tilting the head or lying down.
  • Nasal obstruction with mucopurulent nasal discharge.
69
Q

What bacteria most commonly cause acute sinusitis?

A

Streptococcus pneumoniae and Haemophilus influenzae.

Resolves spontaneously in 50%, but analgesics often indicated and antibiotics may be required if symptoms persist or there is a purulent discharge.

Tx is draininge using vasoconstrictor nasal drops such as ephedrine.

Inhalation of warm, moist air.

70
Q
A