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Internal Medicine > 3) Cardiology (Part 2) > Flashcards

3) Cardiology (Part 2) Flashcards

1
Q

Neck signs of heart failure

A
  • Elevated jugular venous distension
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2
Q

Lung signs of heart failure

A
  • Pulmonary crackles (rales)

- Wheezing (cardiac wheezes)

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3
Q

Heart signs of heart failure

A
    • S3 (s4 may be present in the case of decreased LV compliance)
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4
Q

Abdomen signs of heart failure

A
  • May have ascites

- Hepatomegaly

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5
Q

Extremity signs of heart failure

A
  • Lower extremity edema
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6
Q

CXR findings in CHF

A
  • Cardiomegaly
  • Vascular redistribution (redistribution of flow to apices)
  • Pulmonary venous congestion
  • Kerley B lines
  • Pleural effusions
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7
Q

Kerley B lines

A
  • Reflect chronic elevation of LA pressure

- Chronic thickening of intralobular septa from edema

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8
Q

Laboratory tests in heart failure

A
  • Elevated B-Type Natriuretic Peptide/Brain Natriuretic Peptide (BNP)
  • Used in combination with clinical evaluation to distinguish dyspnea from HF from other causes
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9
Q

Best diagnostic test

A
  • Echocardiogram

- 2D echocardiogram with doppler flow studies

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10
Q

What to look for in echocardiogram

A
  • Ejection Fraction
  • Structural abnormalities of the left ventricle
  • Abnormalities of the myocardium
  • Valvular heart disease
  • Abnormalities of the pericardium
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11
Q

Types of heart failure

A
  • HF with preserved Ejection Fraction (formerly known as Diastolic Dysfunction)
  • HF with diminished Ejection Fraction (formerly known as Systolic Dysfunction)
  • Mixed systolic/diastolic
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12
Q

HFpEF diagnosis includes

A
  • Clinical signs or symptoms of HF
  • Evidence of preserved or normal LVEF
  • Evidence of abnormal LV diastolic dysfunction that can be determined by Doppler echocardiography or cardiac catheterization
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13
Q

Etiologies of diastolic dysfunction

A
  • Marked LVH
  • Diabetes mellitus
  • Restrictive cardiomyopathies
  • Hypertrophic cardiomyopathy with or without obstruction
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14
Q

Marked LVH

A
  • HTN

- Advanced Aortic Stenosis (AS)

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15
Q

Restrictive cardiomyopathies

A
  • Amyloidosis
  • Sarcoidosis
  • Hemochromotosis
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16
Q

Hypertrophic cardiomyopathy with or without obstruction Tx

A
  • Beta blockers are first line
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17
Q

Diagnosis of HF w/ preserved EF

A
  • Echocardiogram is a valuable study for DX
  • Contractility is preserved and ejection fraction is usually normal
  • Concentric hypertrophy on echo Inwardly directed ventricular hypertrophy
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18
Q

Tx for (HFpEF)

A
  • Manage volume overload w/ diuretics

- Treat BP w/ guideline-directed medical therapy such as ACE inhibitors

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19
Q

New updates for Tx of class I HFpEF

A
  • Attain SBP <130 mm Hg in patients with HFpEF and persistent hypertension after management of volume overload
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20
Q

New updates for Tx of class IIa HFpEF

A
  • Use of aldosterone antagonists in appropriately selected patients with HFpEF (with EF ≥45%, elevated BNP or HF admission within 1 year, estimated glomerular filtration rate >30 and creatinine <2.5 mg/dl, potassium <5.0 mEq /L), to decrease hospitalizations
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21
Q

HFrEF definition

A
  • HF and EF ≤40%
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22
Q

Etiologie sof systolic dysfunction

A
  • Most common = ischemia: coronary Heart Disease (CAD)
  • Valvular Heart disease (AS, AR, MR)
  • HTN
  • Non-ischemic Dilated Cardiomyopathies
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23
Q

Non-ischemic dilated cardiomyopathies

A
  • Peripartum
  • Various infections (mostly viral) –> myocarditis
  • Toxins: Alcohol, cocaine, Doxorubicin (Adriamycin)
  • HIV
  • Rheumatologic conditions: ie scleroderma
  • Nutritional deficiencies: ie Beriberi
  • Other causes
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24
Q

Primary goals and management of HF

A
  • Improve symptoms & quality of life
  • Avoid/reduce hospitalizations
  • Slow or Reverse disease progression
  • Decrease mortality
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25
Q

General measures of improving HF

A
  • Dietary sodium restriction
  • Daily weights
  • Physical activity to prevent deconditioning
26
Q

Dietary sodium restriction

A
  • Improves diuresis

- Prevents volume overload

27
Q

Daily weights

A
  • Monitor volume status
28
Q

Physical activity to prevent deconditioning

A
  • No heavy labor or intensive sports

- Avoid in acute exacerbations or myocarditis

29
Q

2016 additional guidelines for systolic failure with reduced EF

A
  • Loop diuretics (Lasix, furosemide)

- Relieve volume overload

30
Q

Chronic medications that reduce mortality

A
  • ACE inhibitor (preferred) OR ARB (if intolerant to ACE-I 2/2 cough or angioedema) OR angiotensin receptor–neprilysin inhibitor (ARNI)
  • Evidence based beta blockers
  • Aldosterone antagonists
31
Q

Evidence-based beta blockers

A
  • Bisoprolol
  • Carvedilol
  • Sustained release metoprolol succinate
32
Q

Aldosterone antagonists

A
  • Spironolactone

- Eplerenone

33
Q

Hydralazine + long acting nitrate used for

A
  • Patients with moderate-severe symptoms on ACE-I, bblockers, and diuretics (Especially African Americans)
  • An alternative to those who cannot take ACE Inhibitors
34
Q

Sinoatrial node modulator Ivabradine (Corlanor) can benefit patients with

A
  • Symptomatic (NYHA class II-III) stable chronic HFrEF (LVEF ≤35%) who:
  • Are receiving guideline directed evaluation and management, including a beta blocker at maximum tolerated dose
  • Who are in sinus rhythm with a heart rate of 70 bpm or greater at rest
35
Q

Rheumatic fever

A
  • Consequence of infection with group A b-hemolytic streptococci
  • Late sequelae includes valvular disease: Mitral >Aortic > Tricuspid
36
Q

Rheumatic fever Tx

A
  • Course of Penicillin for streptococcal infection
  • Aspirin for arthralgia
  • Treat HF with standard medications (ACE-I, diuretics, etc.)
  • Recurrent attacks are common - Prophylaxis with Monthly Benzathine Penicillin IM should be continued for the first 5-10 years after infection
37
Q

Infective endocarditis (IE)

A
  • Infection of the endocardium
  • Usually involves the valves and adjacent structures
  • Caused by a wide variety of bacteria and fungi
38
Q

Infective endocarditis is higher in patients with

A
  • Underlying valvular heart diseases

- Intravenous drug abuse (IVDA)

39
Q

Invasive procedures performed may cause

A
  • Bloodstream infections

- May result in endocarditis (i.e. pacemaker endocarditis, IV catheters, surgical wounds)

40
Q

IE signs and symptoms

A
  • Fever
  • Weight loss
  • Skin lesions
  • New murmur
41
Q

Best diagnostic tests for IE

A
  • Blood cultures x3 different sites (do NOT use IV)

- Echocardiogram (TEE is more sensitive than TTE)

42
Q

Medical treatment of IE

A
  • Various IV high potency antibiotic regimens are available (ie, vancomycin or Ceftriaxone plus gentamicin)
  • All involve 4-8 wks of therapy
  • Surgical Intervention of the valve for many patients
43
Q

When surgically manipulating an actual infection (abscess, cellulitis, etc.) in a patient with a “high risk” for infective endocarditis

A
  • Prosthetic cardiac valve disease
  • Previous infective endocarditis
  • Congenital heart disease (CHD)
  • Cardiac transplantation with valvular defects
44
Q

Congenital heart disease (CDH)

A
  • Unrepaired cyanotic CHD
  • Completely repaired CHD with prosthetic materials for 6mo, allowing endothelium formation
  • Incompletely repaired CHD with residual defects at prosthetic patches or devices
45
Q

American College of Foot and Ankle Surgeons’ Clinical Consensus Statement: Perioperative Prophylactic Antibiotic Use in Clean ElectiveFoot Surgery (2015)

A
  • The panel reached consensus that antibiotic prophylaxis is appropriate in patients who may be at increased risk for infection including those with diabetes, those who are immunocompromised, and those at risk for endocarditis
  • The panel noted that patient factors may more strongly drive the decision to use antibiotic prophylaxis than type of procedure performed
46
Q

Diastolic murmurs (heard between s2 and s1) “ARMPITS”

A
  • Aortic
  • Regurgitation
  • Mitral
  • Stenosis
  • Pulmonic
  • Insufficiency (regurgitation)
  • Tricuspid
  • Stenosis
47
Q

Systolic murmurs (heard between s1 and s2)

A
  • Aortic stenosis
  • Mitral regurgitation
  • Pulmonic stenosis
  • Tricuspid regurgitation
  • Atrial Septal Defect
  • Ventricular Septal Defect
  • Hypertrophic cardiomyopathy
48
Q

Continuous murmur (heard in Diastole and Systole)

A
  • Patent Ductus Arteriosis
49
Q

Aortic stenosis

A
  • Most common acquired valve disease
  • Systolic murmur that commonly radiates to the carotids
  • Narrows over several years
  • Severe stenosis may have a narrow pulse pressure, syncope, heart failure, or chest pain
50
Q

Aortic regurgitation

A
  • Diastolic murmur
  • Caused by aortic root abnormalities or aortic valve abnormalities
  • May be acute or chronic
  • Have an increased cardiac output (due to increased end diastolic volume)
51
Q

Mitral regurgitation

A
  • Systolic murmur
  • Ischemia/Infarction is an important cause (because the mitral valve has a specific blood supply)
  • May be acute or chronic
  • May cause atrial fibrillation
52
Q

Mitral valve prolapse

A
  • Mid-systolic click
  • May progress to mitral regurgitation and have an associated systolic murmur
  • Most common valve disorder in United states
53
Q

Mitral stenosis

A
  • Diastolic murmur
  • Most common cause is Rheumatic Fever as a child
  • May cause atrial fibrillation
54
Q

Pulmonic stenosis

A
  • Systolic murmur

- Rare – most cases are congenital

55
Q

Pulmonary insufficiency (regurgitation)

A
  • Diastolic murmur

- Most cases are due to pulmonary Hypertension and stress on the valve

56
Q

Tricuspid regurgitation

A
  • Systolic murmur
  • Most cases are from pulmonary hypertension and stress on the valve
  • Most common valve disorder involved in endocarditis from IV drug use
57
Q

Tricuspid stenosis

A
  • Diastolic murmur

- Very Rare

58
Q

Atrial septal defect

A
  • ASD is an opening in the atrial septum permitting free communication of blood between the atria.
  • Left – Right shunt
  • Systolic murmur with a wide, fixed-split s2
59
Q

Ventricular septal defect (VSD)

A
  • VSD is an opening in the ventricular septum permitting free communication of blood between the ventricles.
  • Left to Right shunting of blood
  • Systolic Murmur
  • Most common defect in infancy/childhood
60
Q

Patent ductus arteriosus

A
  • Ductus arteriosus that never closed – blood shunts from the aorta to the pulmonary artery
  • Continuous (heard in systole and diastole), “machinery,” “washing machine-like” murmur
61
Q

Tetralogy of Fallot

A
  • Most common cause of cyanosis in infancy/childhood

- Severity of cyanosis is related to the severity of right ventricular outflow tract obstruction

62
Q

TOF: Hypercyanotic “tet” spell

A
  • Decreased systemic vascular resistance (hot bath, fever, exercise) increases R-> L shunt, increasing cyanosis
  • Agitation increases subpulmonic obstruction
  • Life-threatening if untreated

Internal Medicine (14 decks)

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