3/10 Benign Skin Lesions Flashcards

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1
Q

What is a Nevus? What are the various types? (3)

A
  • benign, circumscribed overgrowth of cells composed of tissue elements; aka “birthmarks”
  • can be acquired (usually before 30yo) or congenital (present at birth)

3 Types
• junctional – at the DEJ (but in the epidermis)
• compound - in both epidermis and dermis
• intradermal – in the dermis

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2
Q

What is a Seborrheic keratosis?

How is it treated?

A
  • benign epidermal growth that can arise anywhere body surfaces except palms and soles; often multiple and can be extensive
  • lesions do not go away on its own
  • waxy character with a “stuck on” appearance - glob of wax smushed onto the skin (scratching the lesion reveals its waxy character)

Treatment

  • curette (picked off)
  • electrodessication
  • liquid N2
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3
Q

What are the 2 variants of Seborrheic keratosis?

A

Dermatosis papulosa nigra

  • Common with darker skin
  • Strong familial tendency

Stucco Keratoses
- small white-gray SK’s that pepper the dorsal feet and ankles of older, fair skinned individuals

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4
Q

What are Acrochordons?

What are the risk factors for these lesions?

If a patient who is obese comes in with these skin tags, what should you do?

How are these treated?

A

“skin tags”

  • fleshy papules that arise in areas where there is friction (axillae, neck, groin, and eyelids)
  • often pendunculated
  • risk factors: obesity, friction, genetics
  • Can be a marker for insulin resistance/DM (like acanthosis nigricans), so get a fasting blood glucose, esp if the patient is overweight

Treatment

  • snipping
  • Liquid N2 (may result in hypopigmentation)
  • electrodessication
  • occasionally skin tags will outgrow their blood supply or become torsed such that they necrose and fall off on their own
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5
Q

What are Cherry Angiomas?

What can cherry angiomas be confused with?

A
  • benign lesions that develop in areas of high friction, usually in the trunk (chest/back
  • common around age 30; likely to develop more with age
  • Occasionally cherry angiomas may bleed or thrombose, thereby mimicking melanoma – “When in doubt, cut (or refer) it out”
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6
Q

What are Dermatofibroma? What are some maneuvers that you can do to confirm?

A
  • spindle cell dermal proliferation; looks and feels like a wad of scar tissue
  • Peripheral rim of darkening pigment is common
  • tends to occur in the legs, possibly due to minor skin insults (esp. women who shave)

Dimple Sign = lesion tends to dimple down due to that scar-like tethering of the dermis

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7
Q

What are Solar lentigines? What should worry about if you see them? How do you treat them?

A

“sun spots”
“liver spots”
“age spots”

  • blemishes on the skin associated with aging and exposure to ultraviolet radiation from the sun
  • NOT cancerous or precancerous, BUT extensive solar lentigines indicate a history of UV exposure, and therefore can identify patients at risk for skin cancer
  • if you see an “ugly duckling”, differentiate from melanoma using ABCDEs

Treatment: - none required, but there are cosmetic treatments available (bleaching creams, liquid nitrogen, chemical peels, lasers…) - first step: sun protection

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8
Q

What is Sebaceous hyperplasia?

What should you worry about if you see these?

How do you tell the difference?

How is it treated?

A

sebaceous gland overgrowth

  • lesions are skin-colored or slightly yellow, umbilicated smooth papules on forehead and central face (umbilication is due to gland growth around a central hair follicle)
  • patients tend to accumulate more with age
  • hard to differentiate with basal cell carcinoma!
    (BCC tends to be solitary, more friable (bleeds, scabs) and often pearly translucent, often with telangiectasia)
    when in doubt, get a - not medically required and is cosmetic
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9
Q

What is a keloid?

How is it treated?

A
  • overgrowth of scar tissue beyond the original scar site
  • common on upper trunk and earlobes
  • genetic influence (most common in African-Americans)

Treatment - inject it with steroid - DO NOT excise it because it will come back bigger than it was before

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10
Q

What is an Epidermal inclusion cyst?

How is it treated?

A
  • arise from hair follicles (not sebaceous glands) to form mobile subcutaneous nodules, often with an overlying punctum
  • debris (dead skin cells, oil, etc.) collects within a sack, and may result in a foul smelling cheesy white material
  • Ruptured EIC tends to be sterile, but it may induce an inflammatory reaction that lead to abscess formation; requires drainage

Treatment: surgical excision

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11
Q

What is a miia?

How is it treated?

A
  • tiny epidermoid cyst - often forms on the face

Treatment - extraction (does not scar)

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12
Q

What is a pilar cyst?

How is it treated?

A
  • Firm, mobile subcutaneous nodules, lacking punctum that does not discharge any material (compared to an EIC, less likely to rupture or get inflamed b/c there’s no punctus)
  • Nearly always on the scalp and can cause hair follicle loss due to pressure buildup
  • Slowly enlarges over months to years

Treatment - excision

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13
Q

What is a Lipoma?

How do you treat it?

How do you differentiate it from a pilar cyst?

A
  • soft, mobile subcutaneous nodule without any overlying skin change
  • Often solitary, frequently on the trunk and proximal extremities
  • If familial (AD inheritance), lipomas tend to be multiple and begin in early adulthood

Treatment: excision

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14
Q

What is this?

What are the various types? (3)

A

NEVUS

  • benign, circumscribed overgrowth of cells composed of tissue elements; aka “birthmarks”
  • can be acquired (usually before 30yo) or congenital (present at birth)

3 Types
• junctional – at the DEJ (but in the epidermis)
• compound - in both epidermis and dermis
• intradermal – in the dermis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is this?

How is it treated?

A

Seborrheic keratosis

  • benign epidermal growth that can arise anywhere body surfaces except palms and soles; often multiple and can be extensive
  • lesions do not go away on its own
  • waxy character with a “stuck on” appearance - glob of wax smushed onto the skin (scratching the lesion reveals its waxy character)

Treatment

  • curette (picked off)
  • electrodessication
  • liquid N2
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What is this?

A

2 variants of Seborrheic keratosis

Dermatosis papulosa nigra

  • Common with darker skin
  • Strong familial tendency

Stucco Keratoses
- small white-gray SK’s that pepper the dorsal feet and ankles of older, fair skinned individuals

17
Q

What is this?

What are the risk factors for these lesions?

If a patient who is obese comes in with these skin tags, what should you do?

How are these treated?

A

Acrochordons “skin tags”

  • fleshy papules that arise in areas where there is friction (axillae, neck, groin, and eyelids)
  • often pendunculated
  • risk factors: obesity, friction, genetics
  • Can be a marker for insulin resistance/DM (like acanthosis nigricans), so get a fasting blood glucose, esp if the patient is overweight

Treatment

  • snipping
  • Liquid N2 (may result in hypopigmentation)
  • electrodessication
  • occasionally skin tags will outgrow their blood supply or become torsed such that they necrose and fall off on their own
18
Q

What are these things?

What can they be confused with?

A

Cherry Angiomas

  • benign lesions that develop in areas of high friction, usually in the trunk (chest/back
  • common around age 30; likely to develop more with age
  • Occasionally cherry angiomas may bleed or thrombose, thereby mimicking melanoma – “When in doubt, cut (or refer) it out”
19
Q

What is this?

What are some maneuvers that you can do to confirm?

A

Dermatofibroma

  • spindle cell dermal proliferation; looks and feels like a wad of scar tissue
  • Peripheral rim of darkening pigment is common
  • tends to occur in the legs, possibly due to minor skin insults (esp. women who shave)

Dimple Sign = lesion tends to dimple down due to that scar-like tethering of the dermis

20
Q

What are these lesions called?

What should worry about if you see them?

How do you treat them?

A

Solar lentigines

“sun spots”
“liver spots”
“age spots”

  • blemishes on the skin associated with aging and exposure to ultraviolet radiation from the sun
  • NOT cancerous or precancerous, BUT extensive solar lentigines indicate a history of UV exposure, and therefore can identify patients at risk for skin cancer
  • if you see an “ugly duckling”, differentiate from melanoma using ABCDEs

Treatment: - none required, but there are cosmetic treatments available (bleaching creams, liquid nitrogen, chemical peels, lasers…) - first step: sun protection

21
Q

What is this?

What should you worry about if you see these?

How do you tell the difference?

How is it treated?

A

Sebaceous hyperplasia

sebaceous gland overgrowth

  • lesions are skin-colored or slightly yellow, umbilicated smooth papules on forehead and central face (umbilication is due to gland growth around a central hair follicle)
  • patients tend to accumulate more with age
  • hard to differentiate with basal cell carcinoma!
    (BCC tends to be solitary, more friable (bleeds, scabs) and often pearly translucent, often with telangiectasia)
    when in doubt, get a - not medically required and is cosmetic
22
Q

What is this?

How is it treated?

A

Keloid

  • overgrowth of scar tissue beyond the original scar site
  • common on upper trunk and earlobes
  • genetic influence (most common in African-Americans)

Treatment - inject it with steroid - DO NOT excise it because it will come back bigger than it was before

23
Q

What is this?

How is it treated?

A

Epidermal Inclusion Cyst

  • arise from hair follicles (not sebaceous glands) to form mobile subcutaneous nodules, often with an overlying punctum
  • debris (dead skin cells, oil, etc.) collects within a sack, and may result in a foul smelling cheesy white material
  • Ruptured EIC tends to be sterile, but it may induce an inflammatory reaction that lead to abscess formation; requires drainage

Treatment: surgical excision

24
Q

What is this?

How is it treated?

A

miia

  • tiny epidermoid cyst - often forms on the face

Treatment - extraction (does not scar)

25
Q

What is this?

How is it treated?

What is it easily confused with? How do you tell the difference??

A

Pilar cyst

  • Firm, mobile subcutaneous nodules, lacking punctum that does not discharge any material (compared to an EIC, less likely to rupture or get inflamed b/c there’s no punctus)
  • Nearly always on the scalp and can cause hair follicle loss due to pressure buildup
  • Slowly enlarges over months to years

Treatment - excision

Often confused with a lipoma cyst, which is soft

26
Q

What is a this?

How do you treat it?

What is it easily confused with? How do you tell the difference??

A

Lipoma

  • soft, mobile subcutaneous nodule without any overlying skin change
  • Often solitary, frequently on the trunk and proximal extremities
  • If familial (AD inheritance), lipomas tend to be multiple and begin in early adulthood

Treatment: excision

Often confused with Pilar cyst, which is hard