2e - RFOP - Smoking Flashcards

1
Q

primary risk factors of periodontitis?

A

accumulation of dental plaque

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2
Q

common modifiable risk factors of periodontitis

A
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3
Q

smoking stats NEED TO KNOW???
- how many people smoke world wide
- how many people smoke in the uk?
- how does smoking use effect lifespan?

A
  • 1.3 billion people smoke
  • 7.6 million in the UK
    (ONS.gov.uk)
  • Tobacco kills half its lifetime
    users
  • A lifetime user loose on
    average 15 years of life (WHO
    2011)
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4
Q

how does smoking affect periodontal disease?

A
  • plaque had a greater effect on bone loss in smokers than in nonsmokers
  • Smoking contributes to formation of dysbiotic biofilm – affects bacterial acquisition, colonization, aggregation, and results in higher levels of key pathogen colonization
  • increases odds ratio for the patient to have periodontal disease (ranges 2.5-6)
  • Patients who smoke are more likely to develop Periodontitis than non-smokers (2.5-6x more likely)

EXTRA
Odds ratio = the odds that an outcome will occur given a particular exposure, compared to the odds of the outcome occurring in the absence of that exposure

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5
Q

how does tobacco effect the body? (what are smokers exposed to?)

A
  • at least 4,000 chemicals - directly in the oral cavity
  • direct cytotoxins and free radicals, carbon monoxide

*psycho- and vasoactive chemicals

*chemicals influencing cellular activity:
–tissue turnover (biological
homeostasis)
–healing and wound repair
–immunological activity
–inflammatory cell activity

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6
Q

how does smoking affect cells in periodontal tissues?

A

NEUTROPHILS
- reduce numbers in area and capacity to phagocyte and kill extracellular pathogen

VASOCONSTRICTION (nicotine causes)
- results in less bleeding than smokers

IMPAIRED HEALING
- due to effect on inflamm and vascular response, affects healing on healing after NSPT + surgical PT

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7
Q

Clinical features seen in smokers

A
  1. Increased keratinisation of the gingiva
  2. Less bleeding on probing
  3. Less gingival inflammation
  4. Increased probing depths
  5. Increased loss of attachment
  6. Increased bone loss
    - Longitudinal studies 10yr
    smoking, a predictor of
    future bone loss and at risk of
    attachment loss
    - Acute necrotizing ulcerative gingivitis (ANUG) more prevalent in smokers due to less vascularisation in tissues
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8
Q

Considerations when treating smoker

A
  • explain to patient why stopping or reducing smoking is important
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9
Q

how does quitting smoking impact periodontitis?

A
  • Quitting smoking was associated with a significant reduction in the odds ratio (OR) for periodontitis
  • Each year that passes there is a decrease on the likelihood of having periodontitis
  • It takes an average of 13 years for a former smoker to have the same odd ratio a non-smoker has
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10
Q

SUMMARY

A
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