28. Common shoulder conditions Flashcards

1
Q

How will a dislocated shoulder appear?

A

Visibly deformed and there may be visible swelling and/or bruising around the shoulder. Movement of the shoulder will be severely restricted

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2
Q

What is the most common type of shoulder dislocation?

A

90-95% of dislocations are anterior (i.e. the head of the humerus sits anterior to the glenoid fossa)

2-4% posterior
0.5% inferior

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3
Q

Which aspect of the shoulder joint is the weakest?

A

the joint is strengthened on its superior, anterior and posterior aspects, it is weak at its inferior aspect

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4
Q

What are the 2 types of anterior dislocations?

A

The head of the humerus usually dislocates anteroinferiorly, but then often displaces in an anterior direction (subcoracoid location = 60% of cases) due to the pull of the muscles and disruption of the anterior capsule and ligaments.
Alternatively, the head of the humerus may come to lie antero-inferior to the glenoid (subglenoid location = 30% of cases). Both are types of anterior dislocation.

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5
Q

How is the arm positioned in an anterior dislocation?

A

Arm is held in a position of external rotation and slight abduction

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6
Q

What are common mechanisms of anterior dislocation.

A

The first episode of anterior dislocation usually occurs when an individual has their arm positioned in abduction and external rotation (“hand behind head”), and an unexpected small further injury forces the arm a little further posteriorly, pushing the shoulder into an extreme position, such that the humeral head dislocates antero-inferiorly from the glenoid. An alternative mechanism is a direct blow to the posterior shoulder.

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7
Q

What are 5 complications of anterior dislocation?

A
  • Bankart lesion
  • Hill-Sachs lesion
  • Axillary nerve damage
  • humeral circumflex artery damage
  • recurrent dislocation
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8
Q

What is a Bankart lesion?

A

Force of the humeral head popping out of the socket often causes part of the glenoid labrum to be torn off.
Sometimes a small piece of bone can be torn off with the labrum

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9
Q

What is a Hill-Sachs lesion?

A

compression fracture of posterior humeral head

Tone of the infraspinatus and teres minor muscles means that the posterior aspect of the humeral head becomes jammed against the anterior lip of the glenoid fossa. This can cause a dent (indentation fracture) in the posterolateral humeral head

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10
Q

When do posterior dislocations tend to occur?

A

Occur when there are violent muscle contractions due to an epileptic seizure, electrocution or a lightning strike; when there is a blow to the anterior shoulder; or when the arm is flexed across the body and pushed posteriorly

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11
Q

How is the arm positioned in a posterior dislocation?

A

Arm internally rotated and adducted

They demonstrate flattening / squaring of the shoulder with a prominent coracoid process. The arm cannot be externally rotated into the anatomical position.

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12
Q

How can posterior shoulder dislocations be sptted on x-rays?

A
  • A posterior dislocation of the shoulder can easily be missed on an X-ray as it looks ‘in joint’.
  • However, because the arm is internally-rotated, the projection of the humeral head onto the X-ray film changes to a more rounded shape – the ‘light bulb’ sign. • The glenohumeral distance is also increased.
  • The scapular or ‘Y’ view is also very useful for detecting dislocations of the shoulder radiologically.
  • The head of the humerus should be directly in line with the glenoid fossa i.e. at the bifurcation of the Y.
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13
Q

WHat injuries are commonly associated with posterior dislocations?

A

fractures, rotator cuff tears, and Hill-Sachs lesions

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14
Q

What type of injury causes an inferior dislocation?

A

After inferior dislocation, the head of the humerus sits inferior to the glenoid. The mechanism is forceful traction on the arm when it is fully extended over the head, as may occur when grasping an object above the head to break a fall i.e. a hyperabduction injury.

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15
Q

What structures are commonly injured in inferior dislocations?

A

damage to nerves (60%), rotator cuff tears (80%), and injury to blood vessels (3%).

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16
Q

What is the most common complication of shoulder dislocation and why?

A

recurrent dislocation due to damage to the stabilising tissues surrounding the shoulder (glenoid labrum, capsule, ligaments etc.)

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17
Q

What are the chances of recurrent dislocation?

A

60% but depends on age and activity level - As we age, our tissues lose elasticity, so the risk of recurrent dislocation is approximately 90% in 20-year-olds, falling to only 10% in 40-year-olds

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18
Q

What is the effect of each dislocation on the risk of osteoarthritis?

A

Each dislocation results in further damage to the humeral head and glenoid, therefore it is unsurprising that the risk of osteoarthritis increases with the number of dislocations.

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19
Q

How might axillary artery damage be detected in shoulder dislocations?

A

Patient may have a haematoma, absent pulses and/or a cool limb

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20
Q

How common are axillary nerve injuries in dislocations?

A

10-40% of shoulder dislocations

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21
Q

How would you test for axillary nerve damage?

A

test abduction

test sensation of the regimental badge area

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22
Q

How are the symptoms of axillary nerve damage resolved?

A

Shoulder reduced (put back into place)

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23
Q

In a shoulder dislocation, would you test axillary nerve damage by getting them to abduct arm?

A

NO - too painful - test sensation and then motor after shoulder has been put back in place

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24
Q

Who are shoulder dislocation associated fractures more common in and in which bones?

A

More common when there is a traumatic mechanism of injury, first-time dislocation, or the person is aged over forty.
Commonly affected bones include the humeral head, greater tubercle, clavicle and acromion

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25
Q

Which muscles are mainly affected in shoulder dislocation?

A

Rotator cuff muscle tears can also occur in association with shoulder dislocation, most commonly in older people. they are also a common complication of inferior dislocation in all age group. The integrity of the rotator cuff should always therefore be assessed as part of the follow-up of patients after reduction of a dislocated shoulder.

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26
Q

What are the functions of the clavicle?

A
  • transmits force from the upper limb to the axial skeleton

- protection to the brachial plexus, subclavian vessels and the apex of the lung

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27
Q

Which part of the clavicle is most commonly fractured?

A

middle third - Most result from falls onto the affected

shoulder or onto the outstretched hand

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28
Q

How are most clavicle fractures treated?

A

Treated conservatively (i.e. without surgery), using a sling

29
Q

What are the indications for surgical fixation of a clavicle fracture?

A
  • Complete displacement (so the bone ends are not in apposition and cannot unite)
  • Severe displacement causing tenting of the skin, with the risk of puncture
  • Open fractures (fracture associated with a break in the integrity of skin)
  • Neurovascular compromise
  • Fractures with interposed muscle
  • Floating shoulder: clavicle fracture with ipsilateral fracture of glenoid neck
30
Q

What will happen to the position of the arm and clavicular fragments in a displaced mid-clavicular fracture?

A
  • The sternocleiodomastoid muscle elevates the medial segment
  • Because the trapezius muscle is unable to hold the lateral segment up, and also because of the weight of the upper limb, the shoulder drops
  • The arm is pulled medially by pectoralis major (adduction).
31
Q

What are some general complications associated with clavicle fracture healing?

A

nonunion (failure to unite) and malunion (uniting in a suboptimal position)

32
Q

What are complications of clavicle fracture?

A
  • pneumothorax
  • suprascapular nerve may be damaged by the elevation of the medial part of the fracture
  • supraclavicular nerves (C3,4) may also be damaged resulting in paraesthesia over the upper chest anteriorly
33
Q

Which rotator cuff muscle is most commonly torn?

A

Supraspinatus

  • where it passes beneath the coracoacromial arch, tearing at the site of its insertion into the greater tubercle of the humerus
  • The tendons of the rotator cuff are torn much more frequently than the muscles and, of these, the supraspinatus tendon is the most frequently affected
34
Q

What is the most common cause of rotator cuff tears and how does this lead to tears?

A

Age-related degeneration

- blood supply to the rotator cuff tendons decreases, impairing the body’s ability to repair minor injuries

35
Q

What does degenerative-microtrauma model hypothesise?

A

Age-related tendon degeneration, compounded by chronic microtrauma, results in partial tendon tears that then develop into full rotator cuff tears. Inflammatory cells are recruited and oxidative stress leads to tenocyte (tendon cell) apoptosis, leading to further degeneration, thus a ‘vicious circle’ is created

36
Q

Are rotator cuff tears usually acute or chronic?

A

Although acute tears of the rotator cuff can occur (e.g. following shoulder dislocation), most rotator cuff tears are chronic, resulting from extended use in combination with other factors such as poor biomechanics or muscular imbalance.

37
Q

Which shoulder are rotator cuff tears more common in?

A

Dominant shoulder

38
Q

What type of activities are associated with rotator cuff tears?

A

Recurrent lifting and repetitive overhead activity are also risk factors (e.g. carpenters, painters), as are sports that involve repeated overhead motion (e.g. swimming, volleyball, tennis, weightlifting)

39
Q

What is the most common clinical presentation of rotator cuff tears?

A

Anterolateral shoulder pain, often radiating down the arm

- (most are asymptomatic)

40
Q

What actions cause pain in a rotator cuff tear?

A
  • shoulder activity above the horizontal position, but itmay also be present at rest.
  • when they lean on their elbow and push downwards, as this pushes the head of the humerus superiorly and decreases the space between the humeral head and the coracoacromial arch.
  • They also experience pain in the shoulder when reaching forward (flexing the shoulder)
41
Q

What movement is restricted in rotator cuff tear?

A

weakness of shoulder abduction

42
Q

How are rotator cuff tears diagnosed?

A

As well as the history and examination, MRI and ultrasound both have a role in the diagnosis

43
Q

What is the treatment for rotator cuff tears?

A

The management may be conservative (rest, analgesia etc.) or operative

44
Q

What is impingement syndrome?

A

Supraspinatus tendon impinges (rubs or catches) on the coraco-acromial arch, leading to irritation and inflammation

45
Q

What can cause impingement syndrome?

A

Anything that narrows the space between the head of the humerus and the coracoacromial arch further e.g.

  • thickening of the coracoacromial ligament,
  • inflammation of the supraspinatus tendon,
  • subacromial osteophytes (in osteoarthritis)
  • (grinding & popping)
46
Q

What are the symptoms of impingement syndrome?

A

pain, weakness and reduced range of motion

47
Q

What causes the symptoms of impingement syndrome?

A

When the shoulder is abducted or flexed, the space becomes narrowed further

48
Q

What is the most common form of impingement?

A

Impingement of supraspinatus tendon under the acromion during abduction of the shoulder.

49
Q

What does Impingement of supraspinatus tendon lead to?

A

‘painful arc’ between 60 and 120 degrees of abduction

Patients often report pain on reaching upwards to brush their hair or to lift a food can from an overhead shelf

50
Q

Describe the onset of the pain in impingement sydrome

A

The onset of the pain may be acute if it is due to an injury, or may be insidious (gradually increasing) if it is due to a gradual process such as osteophyte formation. The pain is described as dull rather than sharp, and lingers for long periods of a time, making it hard to fall asleep at night. Other symptoms can include a grinding or popping sensation during movement of the shoulder

51
Q

How are impingement syndromes treated?

A

Treatment is directed at the underlying cause.

52
Q

What characterises Calcific supraspinatus tendinopathy?

A
  • Presence of macroscopic deposits of hydroxyapatite in the tendon of supraspinatus
  • It can occur in any tendon of the rotator cuff but is by far most commonly seen in supraspinatus.
53
Q

What are the mechanical symptoms of Calcific supraspinatus tendinopathy and what are they due to?

A

Physical presence of a large deposit, leading to stiffness, a snapping sensation, catching, or reduced range of movement of the shoulder

54
Q

What are 2 theories of calcific tendinopathy?

A

Regional hypoxia leads to tenocytes being transformed into chondrocytes and laying down cartilage in the tendon. Calcium deposits are then formed through a process resembling endochondral ossification.

Another theory involves ectopic bone formation from metaplasia of mesenchymal stem cells normally present in tendons into osteogenic cells.

55
Q

What are the symptoms of Calcific supraspinatus tendinopathy?

A

It can present with acute or chronic pain, often aggravated by abducting or flexing the arm above the level of the shoulder, or by lying on the shoulder

56
Q

What are the 2 stages of calcific tendinopathy?

A
  • resting stage (crystalline calcific deposits)

- reabsorption stage (by phagocytes)

57
Q

When is the most pain felt in calcific tendinopathy?

A

Re absorption stage

58
Q

What is the treatment for calcific tendinopathy?

A

Treatment is initially conservative with rest and analgesia. Surgical treatment is sometimes required for persistent symptoms

59
Q

In the reabsorption stage, how do the calcific deposits appear on x-rays?

A

During the reabsorption stage they look macroscopically like ‘toothpaste’ and often appear ‘cloudy’ (i.e. less well-defined) on Xray

60
Q

What is Adhesive capsulitis (“Frozen shoulder”)?

A

a painful and disabling disorder in which the Capsule of the glenohumeral joint becomes inflamed and stiff, greatly restricting movement and causing chronic pa

61
Q

What exacerbates pain in Adhesive capsulitis?

A

The pain is usually constant, Worse at night and exacerbated by movement and cold weather

62
Q

What are the risk factors for adhesive capsulitis?

A

 Female gender
 Epilepsy with tonic seizures (i.e. sudden muscle contractions)
 Diabetes mellitus (the theory is that glucose molecules bond to the capsular collagen)
 Trauma to the shoulder
 Connective tissue disease
 Thyroid disease (hypo and hyperthyroidism)
 Cardiovascular disease
 Chronic lung disease
 Breast cancer
 Polymyalgia rheumatica (an inflammatory condition causing muscle pain and weakness)
 Parkinson’s disease
 Long periods of inactivity (from injury, stroke or illness) can precipitate frozen shoulder, and it can also occur alongside other shoulder problems e.g. calcific tendinopathy or rotator cuff tear

63
Q

What is the treatment for adhesive capculitis?

A
  • physiotherapy, analgesia and anti-inflammatory
    medication
  • Patients sometimes undergo manipulation under anaesthesia, which breaks up the adhesions and scar tissue in the joint to help restore range of motion.
  • Intense post-operative physiotherapy then helps to maintain the movement that has been gained
64
Q

What is the prognosis for adhesive capsulitis?

A

Frozen shoulder typically resolves with time and most patients ultimately regain 90% of their shoulder motion. However, once frozen shoulder has resolved, the opposite shoulder becomes affected in 6% to 17% of patients within 5 years, lending further weight to the autoimmune hypothesis

65
Q

What are the effects of frozen shoulder on the patient?

A

Patients with frozen shoulder often experience severe pain and sleep deprivation for prolonged periods, resulting in severe interference with their work and activities of daily living. Some develop depression as a result.

66
Q

Which joint is more commonly affected in osteoarthritis of the shoulder?

A

acromioclavicular joint

67
Q

What is the treatment for OA of the shoulder?

A
Activity modification (avoiding activities that
precipitate symptoms), analgesia, and anti inflammatories (NSAIDs).

Some patients report a benefit from taking nutritional supplements e.g. glucosamine and chondroitin sulfate

68
Q

What can be done to reduce swelling of the joint?

A

Steroid injections
- Hyaluronic acid injections into the joint (viscosupplementation) may increase lubrication, although the evidence for this is limited.

69
Q

What surgical treatment can be done done?

A
  • Arthroscopy (keyhole surgery): remove loose pieces of damaged cartilage from the glenohumeral joint
  • hemiarthroplasty (replacement of the humeral head)
  • or total shoulder replacement (replacement of the humeral head and the glenoid)