18. Emergencies and Primary Measures in the ear, nose and throat. Foreign Bodies. Anatomy and physiology of the Esophagus. Corrosive Esophagitis. Treatment. Flashcards
What are the EAR / OTOLOGIC EMERGENCIES & their PRIMARY MEASURES?
1) Bleeding from the ear canal = Investigated to confirm INTEGRITY of TM / Ossicular Chain / Inner Ear
2) Damage to TM / Ossicular Chain = IMMEDIATE Evaluation via Specialist
3) Inflammation of Ear are considered EMERGENCIES IF = Process TRASCENDS Ear Boundaries (Mastoiditis / Brain Abscess / Meningitis / Sepsis)
4) Signs of INNER EAR Involvement = Labyrinthitis / HL / Vertigo
5) Auricular Inflammations CARRY RISK of Cartilage Damage
6) Acute Vestibulocochlear Dysfunction = HL / Tinnitus / Vertigo (Immediate Diagnosis / Therapy)
What are the NOSE / SINONASAL EMERGENCIES & their PRIMARY MEASURES?
1) Nose Bleed / Epistaxis = MOST COMMON!
2) Acute Sinusitis = IF Inflammation spreads to Orbit / Eye / Meninges of Frontal Lobe (Immediate!)
3) Inflammation of External Nose = Cause Cartilage Liquefaction, due to RISK of Angular Vein Thrombosis / Cerebral Venous Thrombosis
4) Septal Hematomas /Abscesses = AFTER Nasal Trauma, require IMMEDIATE attention
5) Fractures of Nasal Bone / Zygoma / Orbital Floor / Intranasal Foreign Bodies
What are the THROAT (Oropharyngeal / Laryngotracheal) EMERGENCIES & their PRIMARY MEASURES?
1) OROPHARYNGEAL
a. Inflammatory Complications of Tonsillitis = Peritonsillar / Parapharyngeal Abscess
b. Post-Op Bleeding AFTER Tonsillectomy
c. Erosion of BVs via Malignant Tumors can provoke MASSIVE Bleeding
d. Impalement Injuries = Creates NIDUS for Abscess Formation
e. Bilateral Choanal Atresia (Newborns) Require IMMEDIATE INTUBATION, as they’re nose breathers, esp during feeding
2) LARYNGOTRACHEAL
- OBSTRUCTION of Airways via Swelling / Tumor / Foreign Body / Blood, Secretions
- Require EMERGENCY Airway intervention via Intubation / Tracheotomy
What are FOREIGN BODIES of Larynx?
- Aspirated FBs are found 4x MORE in RIGHT Bronchus»_space;> Left
- Peanuts (Swell in Airway) / Watermelon Seeds / Tablets (Adults)
0 SYMPTOMS
- Immediate Coughing Fit
- Dyspnoea
- Stridor
- Pain
** Larger FBs IMPACTED in Larynx, may cause DEATH from ASPHYXIATION
** Smaller FBs cause Hoarseness / Cough
0 DIAGNOSIS = Radiographs to determine LOCATION of ASPIRATED FB
Describe the ANATOMY of the Oesophagus
- Begins at UPPER Oesophageal Sphincter (C6 / C7 Vertebrae)
- Terminates at Gastric Media in Plane of T10 Vertebrae
0 LAYERS of OESOPHAGEAL WALL
- Mucosa = Stratified / Non-keratinising Squamous
- Submucosa
- Muscularis = Inner Circular / Outer Longitudinal Muscle Fibres
- Adventitia
What is the BLOOD SUPPLY of the Oesophagus?
0 BLOOD SUPPLY
- Cervical Part = Inferior Thyroid Artery
- Thoracic Oesophagus = Aorta / Intercostal Arteries
- Abdominal Oesophagus = Left Gastric Artery / Inferior Phrenic Artery
- Venous Blood in Neck = Inferior Thyroid Vein
- Thoracic / Abdominal Drainage = Azygos / Hemiazygos / Oesophageal Veins
What is the LYMPHATIC DRAINAGE & INNERVATION of the Oesophagus?
0 LYMPH DRAINAGE
- Posterior Mediastinum LN
- Pulmonary Hilum LN
0 INNERVATION
- Upper / Cervical par = Branches from Recurrent Nerve
- Lower Part = Vagus Nerve
- BELOW Tracheal Bifurcation = Oesophageal Plexus (2 Vagus Nerves)
What is the PHYSIOLOGY of the Oesophagus?
3 Physiologic CONSTRICTIONS:
a. Upper = At Oesophageal Inlet BTW Cricoid Cartilage AND Cricopharyngeal
b. Middle = Aortic Arch CROSSES OVER Tracheal Bifurcation
c. Lower = Oesophagus PIERCES the Diaphragm
1) VOLUNTARY INITIATED Oral Phase of Swallowing FROM INVOLUNTARY Pharyngeal Phase / Oesophageal Phase
2) Controlled via Reflex Mechanisms
3) Oesophageal Phase
(i) Primary Peristaltic Wave, in response to moving bolus thru Pharynx
(ii) Secondary Peristaltic Wave = Triggered in Oesophagus via PRESSURE of Bolus AGAINST Oesophageal Wall
What is CORROSIVE OESOPHAGITIS?
- Accidental / Suicidal Ingestion of CAUSTIC Substances
- Household Cleaners / Bleach / Washing Soda
- Harmful DUE to, ALKALI Medium
- Damage DEPENDS ON Concentration of Substance
- Affects MIDDLE / LOWER 3rd of Oesophagus
What is the PATHOGENESIS of CORROSIVE OESOPHAGITIS?
{Acute, Subacute, Chronic Phases}
1) ACUTE Stage
- First 10 Days from ingestion
- Acute Necrosis w/ Mucosal Blurring
- Dilated Atonic Oesophagus
2) SUBACUTE Stage
- 10 - 20 Days AFTER Ingestion
- Oesophageal Ulceration
3) CHRONIC Stage
- AFTER 21 Days
- Oesophageal Inflammation
- HEALED via FIBROSIS
What are the 3 DEGREES OF BURN in CORROSIVE OESOPHAGITIS?
1st Degree = Mucosal Hyperemia / Oedema
2nd Degree
- Small Bleeding
- Exudates
- Ulcers
- Pseudo-membrane
3rd Degree
- Mucosal Slough
- DEEP Ulcers
- Massive Bleedings
- COMPLETE Obstruction
- Perforation
What are the SYMPTOMS and TREATMENT for CORROSIVE OESOPHAGITIS?
0 SYMPTOMS
- Oropharyngeal / Epigastric Pain
- Dysphagia
- Hypersalivation
- Vomiting
- Haematemesis
0 TREATMENT
- EMERGENCY Surgery IF = Signs of Perforation / Mediastinitis / Peritonitis
- Gastrointestinal Endoscopy WITHIN FIRST 24h
a) 1st Degree Burns
- 48h Observation
- Oral Feeding once Px swallows painlessly
- Endoscopy on Months 1,2 and 8th
b) 2nd / 3rd Degree Burns
- Fluid Therapy
- Antibiotics
- Nutrition
- PPIs
- Aerosolised Steroids
- Tracheostomy / Intubation
- Endoscopic Oesophageal Stenting