1.8 - CTG INCOMPLETE - (HR) Flashcards
What would be considered a normal foetal heart rate?
110-160
When can a CTG reliably be performed?
Third trimester => After 28 weeks
What rate of contractions would be concerning?
5 in 10 or 7 in 15 would be concerning for uterine hyperstimulation => Uterine rupture
What are all the parameters to check for in CTG. What i mean by that is include the normal ranges where applicable
Define Risk: High vs low (continuous monitoring only for high risk)
Contractions: #/10 minutes or /15 mins
Baseline foetal heart Rate: 110-160
Variability: 5-25 bpm/beat
Accelerations: 2 in 20 is reassuring
In what patients should CTG be performed?
Give 8 reasons
Any patient with high risk =>
Maternal Factors:
1) GDM
2) HTN/Pre-eclampsia/Eclampsia
3) Asthmatics
Obstetric Factors:
1) Multiple Gestation
2) Post-term
3) PROM/Pre-term delivery
4) IUGR
5) Placenta Previa
6) APH
7) Foetal anomalies
8) Induction
Environmental:
1) Smoking
2) Drug abuse
What is recorded on a CTG paper?
Maternal details
Date and time
Maternal posture (why? early Decelerations)
Speed of paper
drugs administered esp. Epidural and induction
Vaginal examinations
How does the foetal heart rate respond to Acute hypoxia
Chronic Hypoxia
Acute = increased PNS => bradycardia
Chronic = chronic increase in SNS => tachycardia
How does the foetal heart rate respond to
Hypertension
Hypotension
Hypertension: increased PNS => bradycardia
Hypotension: Increased SNS => Tachycardia
Give 4 causes of Persistent foetal tachycardia and 4 causes of persistent foetal bradycardia
Persistent Tachycardia
Chronic Hypoxia
GA<32
Infectious: Maternal pyrexia, foetal infection, chorioamnionitis
Hyperthyroidism
Catecholamines
Persistent Bradycardia:
Post-term
Cord compression
Cord prolapse
Congenital heart disease/heart block
Maternal seizure
Maternal benzos
Why would GA<32 cause persistent bradycardia?
Vagal immaturity (vagus in charge of PNS + unopposed SNS)
Same thing for post-dates causing persistent bradycardia as there is increased vagal tone => more PNS
Define reduced variability
Give 3 causes for reduced variability
Reduced variability is variability <5 for >40 minutes (>90 if pathological)
1) Foetal sleep (should not last for more than 40 minutes anyways)
2) Analgesia: Epidural and Opioids
3) Induction agents: Oxytocin and prostaglandin
4) Betamethasone (steroid given. Thats why we give dexamethasone instead)
What is a sinusoidal pattern?
Give 2 causes
Smooth, undulating sinusoidal wave-like pattern with no beat to beat variability.
Causes
Idiopathic
Anaemia due to Rhesus isoimmunization (haemolytic anaemia), or large APH
Severe hypoxia
Accelerations occur as a response to fetal movements or to uterine contractions. Define Accelerations
Abrupt increase of 15bpm for >15s
(decelerations is the opposite)
Define reactive accelerations
reduced bp followed by an abrupt increase of 15bpm for >15s
Decelerations are an abrupt decrease of 15bpm for >15 seconds. Define early deceleration.
Give 2 causes of early decelerations
Begins at the onset of a contraction and recovers by the end of it with the lowest point preceding contraction peak
Compression of the fetal head during contraction (=> increased ICP => increased vagal tone => reduced HR)
or Change in maternal posture (thats why we record it)