1.8 - CTG INCOMPLETE - (HR) Flashcards

1
Q

What would be considered a normal foetal heart rate?

A

110-160

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2
Q

When can a CTG reliably be performed?

A

Third trimester => After 28 weeks

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3
Q

What rate of contractions would be concerning?

A

5 in 10 or 7 in 15 would be concerning for uterine hyperstimulation => Uterine rupture

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4
Q

What are all the parameters to check for in CTG. What i mean by that is include the normal ranges where applicable

A

Define Risk: High vs low (continuous monitoring only for high risk)

Contractions: #/10 minutes or /15 mins
Baseline foetal heart Rate: 110-160
Variability: 5-25 bpm/beat
Accelerations: 2 in 20 is reassuring

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5
Q

In what patients should CTG be performed?
Give 8 reasons

A

Any patient with high risk =>
Maternal Factors:
1) GDM
2) HTN/Pre-eclampsia/Eclampsia
3) Asthmatics

Obstetric Factors:
1) Multiple Gestation
2) Post-term
3) PROM/Pre-term delivery
4) IUGR
5) Placenta Previa
6) APH
7) Foetal anomalies
8) Induction

Environmental:
1) Smoking
2) Drug abuse

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6
Q

What is recorded on a CTG paper?

A

Maternal details
Date and time
Maternal posture (why? early Decelerations)
Speed of paper
drugs administered esp. Epidural and induction
Vaginal examinations

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7
Q

How does the foetal heart rate respond to Acute hypoxia
Chronic Hypoxia

A

Acute = increased PNS => bradycardia
Chronic = chronic increase in SNS => tachycardia

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8
Q

How does the foetal heart rate respond to
Hypertension
Hypotension

A

Hypertension: increased PNS => bradycardia
Hypotension: Increased SNS => Tachycardia

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9
Q

Give 4 causes of Persistent foetal tachycardia and 4 causes of persistent foetal bradycardia

A

Persistent Tachycardia
Chronic Hypoxia
GA<32
Infectious: Maternal pyrexia, foetal infection, chorioamnionitis
Hyperthyroidism
Catecholamines

Persistent Bradycardia:
Post-term
Cord compression
Cord prolapse
Congenital heart disease/heart block
Maternal seizure
Maternal benzos

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10
Q

Why would GA<32 cause persistent bradycardia?

A

Vagal immaturity (vagus in charge of PNS + unopposed SNS)

Same thing for post-dates causing persistent bradycardia as there is increased vagal tone => more PNS

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11
Q

Define reduced variability
Give 3 causes for reduced variability

A

Reduced variability is variability <5 for >40 minutes (>90 if pathological)

1) Foetal sleep (should not last for more than 40 minutes anyways)
2) Analgesia: Epidural and Opioids
3) Induction agents: Oxytocin and prostaglandin
4) Betamethasone (steroid given. Thats why we give dexamethasone instead)

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12
Q

What is a sinusoidal pattern?
Give 2 causes

A

Smooth, undulating sinusoidal wave-like pattern with no beat to beat variability.

Causes
Idiopathic
Anaemia due to Rhesus isoimmunization (haemolytic anaemia), or large APH
Severe hypoxia

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13
Q

Accelerations occur as a response to fetal movements or to uterine contractions. Define Accelerations

A

Abrupt increase of 15bpm for >15s
(decelerations is the opposite)

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14
Q

Define reactive accelerations

A

reduced bp followed by an abrupt increase of 15bpm for >15s

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15
Q

Decelerations are an abrupt decrease of 15bpm for >15 seconds. Define early deceleration.

Give 2 causes of early decelerations

A

Begins at the onset of a contraction and recovers by the end of it with the lowest point preceding contraction peak

Compression of the fetal head during contraction (=> increased ICP => increased vagal tone => reduced HR)
or Change in maternal posture (thats why we record it)

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16
Q

Accelerations often proceed after variable decelerations. Define variable decelerations

Give the etiology and 2 causes of variable decelerations

A

Decelerations (abrupt decrease by 15bpm lasting >15s) that is inconsistent with contractions

It indicates a transient compression of the umbilical cord not related to contractions
=> Nuchal cord, Cord prolapse

17
Q

Define late decelerations
Give the etiology and 5 reasons for its occurance

A

Decelerations (abrupt decrease by 15bpm lasting >15s) that occur after the onset of contraction and the lowest point being 15s after peak of contraction

This is typically caused by Faetal acidosis from:
Acutely:
Maternal hypotension
Placental abruption
Antepartum hemorrhage
Rhesus isoimmunisation

Chronic:
GDM
Hypertension/PET
Renal disease
TTTS

18
Q

Prolonged decelerations are often preceded by variable decelerations. Define Prolonged deceleration
Give 3 causes

A

Drop in FHR of >30bpm for >2 minutes

Cord prolapse/total cord occlusion
Uterine hyperstimulation
After AROM or vaginal exam

19
Q

you notice this CTG. What is it demonstrating and what is your management plan?

A

Terminal bradycardia
Emergency C section or immediate operative delivery if enough descent.